Jesse's Part ( Exam 1) pg. 32-44 Flashcards

1
Q

what is an example of an irritating agent to smell that does not require olfaction for our bodies to sense it. and shouldn’t be used on a cranial nerve 1 exam?

A

ammonia

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2
Q

what order neurons of cranial nerve 1 are true olfactory nerves?

A

second order

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3
Q

a complete loss of smell

A

anosmia

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4
Q

perversion , hallucination, or diminution of smell is more likely associated with what type of lesion?

A

cortical

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5
Q

lesions of the gyrus of the anterior temporal lobe may cause hallucinations of smell associated with strong feelings of deja vu and are termed *** fits or seizures

A

Uncinate gyrus

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6
Q

a decreased sense of smell

A

hyposmia

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7
Q

an increased sense of smell

A

hyperosmia

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8
Q

a perversion of smell

A

parosmia

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9
Q

abnormally disagreeable smell

A

cacosmia

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10
Q

is cranial nerve 1 a true nerve

A

no it is a fiber tract of the brain

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11
Q

what order neuron of CNI
unmyelinated processes of the ciliated receptors in the upper part of the nasal mucosa gathered in about 20 branches which pass through the cribriform plate of the ethmoid bone to the olfactory bulb

A

primary neurons

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12
Q

what order neuron of cranial nerve I
myelinated processes of the bipolar cells of the bulb form the olfactory tract and terminate in the primary olfactory cortex( periamygdaloid area and prepiriform cortex)

A

Secondary neurons.. second order

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13
Q

what order neurons of cranial nerve 1
neurons extend from the primary olfactory cortex to the entorhinal cortex Area 28, lateral preoptic area, amygdaloid body, and medial forebrain bundle

A

tertiary neurons

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14
Q

is cranial nerve 2 a true nerve?

A

no it is a fiber tract of the brain just like CN I

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15
Q

what are the three central connections of the optic nerve?

A
  1. from the pretectal region the edinger-westphal via posterior commissure
  2. from the superior colliculi via the tectobulbar and tectospinal tracts to other cranial spinal nuclei
  3. from the occipital cortex to other cortical and subcortical areas
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16
Q

fibers from what region are responsible for the simple and consensual light reflexes

A

pretectal region

17
Q

this lesion involves the optic nerve or tract the most common cause is multiple sclerosis

A

retrobulbar neuritis

18
Q

this lesion of the visual apparatus includes various forms of retinitis ex. proteinuric, syphilitic, diabetic, hemorrhagic, and hereditgary,

A

optic or bulbar neuritis

19
Q

this lesion of the CN2 is aka’ed choked disc

A

papilledema

20
Q

this optic lesion is associated with decreased visial acuity and change in the color of the optic disc to light pink, white, or gray

A

optic atrophy

21
Q

is caused by processes that involve the optic nerve and do not produce papilledema

A

primary optic atrophy

22
Q

this type of optic atrophy is a sequel of papilledema

A

secondary optic atrophy

23
Q

this maybe due to tabes dorsalis multiple sclerosis or herditiary and is an atrophy of the optic nerve

A

primary ( simple) optic atrophy

24
Q

this syndrome of CN 2 may be caused by tumors at the base of the frontal lobe and is characterized by ipsilateral blindess and anosmia, ( with atrophy of the optic and olfactory nerves) and contralateral papilledema

A

foster Kennedy syndrome

25
Q

this syndrome involving the optic apparatus is AKAed Tay-Sachs disease, cerebromascular degeneration with severe mental deficiency occuring in jewish families and is associated with blindess, optic atrophy, and a dark cherry red spot in place of the macula lutea

A

Amaurotic familial idiocy

26
Q

syndrome involving the optic apparatus where the pupil reacts only to accommodation. I has neither a direct or indirect reaction to light. found to occur as a diabetic complication

A

argyll robertson pupil

27
Q

Argyll robertson pupil was once thought to be pathognomonic of tabes dorsalis but now is known to be a complication of what

A

diabetes

28
Q

a syndrome involving the optic apparatus characterized by a tonic pupillary reaction and the absence of one or more tendon reflexes. the pupil is said to be myotonic with a very slow almost imperceptible contraction to light and in near vision. a slower dilation upon removal of the stimuli.

A

Holmes-Adie syndrome

29
Q

what test is done to confirm Holmes-Adie Syndrome

A

Adler-Scheie test

30
Q

deviation of bilateral eye alignment is termed

A

heterotropia

31
Q

outward/ lateral deviation of the patients eyes when looking straight ahead

A

exotropia

32
Q

inward medial deviation of the eyes when looking straight ahead

A

esotropia

33
Q

upward deviation of the eyes when looking straight ahead

A

hypertropia

34
Q

downward deviation of a patients eyes when looking straight ahead

A

hypotropia

35
Q

what section of the brain has a primary function to coordinate eye movements also known as yoked movements by interconnecting the nuclei of cranial nerves 3 4 and 6.

A

Medial longitudinal fasciculus MLF

36
Q

a common disease to affect the MLF ( medial longitudinal Fasciculus) would be?
the patient will be unable to laterally gaz but convergence will be spared.

A

Multiple Sclerosis

37
Q

what supranuclear pathway of the eye controls mostly saccadic ( rapid or darting ) eye movements?

A

frontal lobe

38
Q

what supranuclear pathway of the eye controls mostly smooth or following eye movements

A

occipital lobe