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Practice Quiz > Inflammation/Immunity > Flashcards

Inflammation/Immunity Flashcards

1
Q

type of WBC, most plentiful, phagocytosis of microorganism. First responder

A

neutrophil

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2
Q

type of disease-fighting WBC (parasitic infection, allergic reaction, or cancer)

A

eosinophil

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3
Q

type of WBC, produced in bone marrow, if low could be due to severe allergic reactions, release of histamine leading to inflmmations

A

basophil

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4
Q

type of WBC, produced in bone marrow, phagocytosis-eat everything

A

monocytes

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5
Q

WBC, produced in bone marrow, found in blood and lymph tissue

A

lymphocytes

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6
Q

active in cell mediated immune; fights against infection

A

T lymphocytes

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7
Q

produces antibodies; book of knowledge

A

B lymphocytes

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8
Q

locate and eat bacteria, virus, fungi, parasite, born from monocytes

A

macrophage

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9
Q

immune cells of myeloid linage and present in connective tissues immediate inflammatory response such as hypersensitivity and allergic reaction

A

mast cells

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10
Q

promotes stimulation of nerve receptors at site and increase pain

A

bradykinin inflammatory mediator

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11
Q

promotes stimulation of nerve receptors at site and increase pain; initiate fevers

A

prostaglandin inflammatory mediator

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12
Q

increase capillary permeability-vasodilation, histamine, serotine, prostaglandins, leukotrienes

A

chemical mediators

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13
Q

store multiple inflammatory molecules interact with leukocytes and support their interaction. they do not remove debris and dead cells

A

platelets

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14
Q

Injury
Vasoconstriction
Mast cells degranulation releasing chemical mediators
Vasodilation
Bottleneck of blood
Increased capillary permeability-allowing blood components to move into the interstitial space (including fluid and WBC’s to start the cleanup process and continues the inflammatory response)
Edema-due to excess fluid in the interstitial compartment

A

Steps of inflammation

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15
Q

most common, fight infection, passive immunity for fetus

A

IgG

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16
Q

first responder, incompatibility reaction to blood

A

IgM

17
Q

tears and saliva, protection for newborns via colostrum

A

IgA

18
Q

response to allergens, release histamine and trigger inflammtion

A

IgE

19
Q

activate B-cells

A

IgD

20
Q

Defend against bacteria

Activated with IgG or IgM antibodies

A

complement systems

21
Q

process of acquiring an immunity-primary response

A

i. person is first exposed to antigent
ii. antigen recognized and processed
iii. development of antibodies is initiatiate (1-2 weeks)

22
Q

process of acquiring an immunity-secondary response

A

i. second or repeated exposure to antigent

ii. rapid and results in increased level of antibody

23
Q

i. pathogens enter body and cause illness
ii. antibodies form in host
iii. memory
iv. person has chicken pox once

A

active natural immunity

24
Q

i. vaccine is injected into person
ii. no illness
iii. antibodies form
iv. memory
v. person had measles vaccine and gains immunity

A

active artificial immunity

25
Q

i. antibodies pass directly from mother to child to provide temporary protection
ii. No memory
iii. Pacental passage during pregnancy or ingestion of breast milk

A

natural passive immunity

26
Q

i. antibodies injected into person (antiserum ) to provide temporary protection or minimize severity of infection
ii. No memory
iii. Gammaglobulin if recent exposure to microbe

A

articial passive immunity

27
Q

a. begins with exposure to allergen and develops IgE antibodies from B lymphocytes
b. antibodies attach to mast cells creating sensitized mast cells
ex. hay fever, anaphylaxis
effect-immediate inflammation and pruritus

A

Type 1 allergic reaction

28
Q

a. example of ABO blood incompatibility
b. mechanism- IgG or IgM reacts with antigen on cell. complement activated
c. effects: cell lysis and phagocytosis

A

Type II cytotoxic hypersensitivity

29
Q 
examples:
a. autoimmune disorders: SLE  and glomerulonephritis
b. mechanism
i.antigen-antibody complex deposits in tissue
ii. complement activated
effects
i. inflammation
ii. vasculitis
A

Type III immune Complex

30
Q 
examples
a. contact dermatitis
b. transplant rejection
mechanism
a. antigen bind to t-lymphocyte
b. sensitized lymphocyte release lymphokines
effects
a. delayed inflammation usually more than 24 hours
A

Type IV cell-mediated or delayed

31
Q 
I.Compromised or lack of immune response
II. Causes
a. primary- basic developmental failure in system 
b. secondary-or acquired, loss of immune response resulting from a specific cause
Risk
a. increased risk for cancer
b. opportunistic infections
c. AIDS
A

Immunodeficieny

32
Q

spread to distant sites by blood or lymphatic channels

A

metastasis

33
Q

classification process; describe the degree of metastasis and involvement of lymph nodes

A

staging

34
Q

degree of differentiation of malignant cells

A

grading

Practice Quiz (13 decks)

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