high risk pregnancy Flashcards

1
Q

apgar score

A

done immediatley after birth at

  • 1 minute,
  • 5 mins =
    • 7+ pts is good
    • <3 pts suggests cerebal palsy and perm neuro damage
  • and as needed intervals

score of 7-10 means good health

4 and 6- ventilation assistance

4 means that prompt, life-saving measures may be called for.

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2
Q

what characterises a high risk pregnancy

maternal rf

fetal rf

A

Maternal

    • maternal age over 35 under 16
  • chronic disease(HTN,DM thyroid)
  • preeclampsia
  • IUGR
  • previous still birth
  • post term ( 2 weeks past DD)
  • mx preg
  • previous cervical incomp
  • rh iso immuization
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3
Q

explain placental insufficiency

A

disorder of fetomaternal circ causing insuff BF to placenta via umbillical cored leading to impaired 02 exchange impared fetal growth and fetal metabolic acidosis

the earlier this occurs in preg the worse the prognosis

can be acute or chronic

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4
Q

what is acute placental insufficiency

A

acute consequences of fetal hypoxia and intra uterine death occur in w/in mins to hours

d/2

  1. supine hypotensive IVC syndrome
  2. placental abruption/ previa
  3. prolonged uterine contraction

treatment

  • left lateral posish for shivcs
  • IMMEDIATE C SECTION
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5
Q

what is chronic placental inssuficiency

A

long term placental insufficiency causing intrauterine death or intra uterine growth restriction which may lead to preterm labour

causes

  1. maternal conditions (smoking, maternal disease-DM, HTM, ANEMIA)
  2. preg assoc conditions= PREECLAMSIA, rh isoincompatibility
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6
Q

complications of placental insufficiency

A

maternal

  • preeeclampsia
  • preterm labour

fetal

  • cerebral palsy( d/2 fetal hypoxia)
  • intrauterine death
  • learning disability
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7
Q

diagnostic procedures for placental insuff in 2st trimester

A
  • early ID of preg in 1st trimester using US
  • detects fetal anomalies (US)
  • observe fhr and breathing (NST,ST,BPP)
  • amniotic fluid index
  • locate and grade placenta (lower number is better as less calcifications)
  • amniocentesis/ CVS
  • Determine fetal position
  • doppler
    *
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8
Q

use of doppler in placental insuff

A

determines the blood flow

asseses placental function

used to manage:IUGR; preterm labour; prolonged preg; mx preg

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9
Q

NST

A

determines health of baby in the uterus by measuring FHR in relation to movement and uterine contraction

REACTIVE

  • 2+ fhr accel of 15bpm for 15sec in 20mins

NON REACTIVE

  • no reaction in 30 mins
  • other tests req
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10
Q

biophysical profile

A

preanatal US when NST is unreactive

scoring

2=normal finding

0=abnormal finding

overall score dictates management

  • 4= unduce labour at 30-32 wks
  • persistently below 4 for 120mins= imm labour induction
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11
Q

what is the oxytocin contraction stress test

A

evaluates the respiratory function of the placenta by monitoring FHR in response to uterine contraction stim by oxytocin or nipple stimulations

-ve test= good response

  • 3 good contractions that last 40 secs in 10 min period
  • no late deccelerations during this period

+ve test = bad response

  • persistent late deccelerations in 50% of contractions
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12
Q

amniocentesis = 15-17 weeks gestation

A

needle pens abdom and uterine wall to collect amniotic fluid

measurements

  1. abnormal AFP(reduced in downs increased in spina bifida)
  2. hCG lvls
  3. ue3??
  4. lecithin/ sphingomyelin rattio= for fetal lung maturation

possible complications

infection => abortion

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13
Q

chorionic villi sampling

A

10-12 weeks from placenta

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14
Q

what is the normal fetal heart rate

A

110-160 bpm = 140bpm

fetal heart sounds can be monitred by fetoscope

cardiac abnormalities measured by fetal echo in 2-3rd trimester

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15
Q

what are deccelerations

A

decrease in FHR by 15bpm caused by reduced bf/02 to fetus

Early = reduced FHR before midpoint of uterine contration

  • inconsequenction
  • d/2 head compression during contraction causing vagal nerve stim

Variable = abrupt changes in FHR w/ no connection to uterine contractions

  • can nbe d/2 refluex tachy cardia/ cord compression
  • mild cord compression up to 40 secs ,mod up tp 60 secs= inconsequntial
  • cord compression over 60 secs leads to fetal acidosis
  • Leads to NADIR in under 30 secs
    • if under 50% (INTERMITTENT) are variable deccelrations then no intervention
    • rx: if over 50% (RECCURENT) are variable= intrauterine resuscitations
      • failure?=> emergency C-section

late deccelerations = persists after midpoint of contraction( max contraction curve)

  • suggests uteroplacental insufficiency is present as contraction worsens fetal bf that’s already compromised - hypoxia
  • onset to NADIR is gradual and over 30 secs
    • RX: intrauterine ressuc in all cases
      • failure?+ emergency c- section
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16
Q

early decelerations

A

Early = reduced FHR before midpoint of uterine contration

inconsequenction

d/2 head compression during contraction causing vagal nerve stim

17
Q

variable deccelerations

A

Variable = abrupt changes in FHR w/ no connection to uterine contractions

can nbe d/2 refluex tachy cardia/ cord compression

mild cord compression up to 40 secs ,mod up tp 60 secs= inconsequntial

cord compression over 60 secs leads to fetal acidosis

Leads to NADIR in under 30 secs

if under 50% (INTERMITTENT) are variable deccelrations then no intervention

rx: if over 50% (RECCURENT) are variable= intrauterine resuscitations

failure?=> emergency C-section

18
Q

late decelerations

A

ate deccelerations = persists after midpoint of contraction( max contraction curve)

suggests uteroplacental insufficiency is present as contraction worsens fetal bf that’s already compromised - hypoxia

onset to NADIR is gradual and over 30 secsRX: intrauterine ressuc in all cases

failure?+ emergency c- section

19
Q

what is nadir

A

the lowest or most unsuccessful point in a situation.

“asking that question was the nadir of my career”

synonyms:the lowest point, the all-time low, the lowest level, low-water mark, the bottom, as low as one can get, rock-bottom, the depths; More

20
Q

what is intrauterine growth restriction

A

lower than normal fetal growth characterized by an estimated fetal weight below the 10th percentile for a given gestational age.

  • Symmetrical IUGR is caused by direct fetal factors in the early stages of gestation
    • early intrauterine infections,
    • genetic abrnorm
    • teratogenic drugs( ACE inhib)
    • uterine malform
  • Asymmetrical caused by factors outside the fetus that affect the fetus in the later stages of gestation
    • placental insufficiency= abruption/preavia, smoking, mx preg, db, HTN
21
Q

sx of IUGR

A

fetal

  • Asymmetrical IUGR: disproportionate growth restriction

The dimensions of the head are normal while the body and limbs are thin and small.

  • Symmetrical IUGR: global growth restriction
  • Decreased or absent fetal movements

maternal

  • Mostly asymptomatic
  • Small uterus (e.g., a smaller abdomen than in previous pregnancies)
  • Possible vaginal bleeding (e.g., placental abruption); preterm labor
22
Q

dg lf IUGR

A

US:

  • BELOW 10th percentile
  • olgiohydromnious
  • placental grading shows calcifications

doppler

  • reduced diastolic flow

NST= LATE DECCEL

BPP= below 4

23
Q

rx of IUGR

A

Treatment of the underlying condition (e.g., treatment of hypertension in pregnancy, gestational diabetes mellitus)

Close monitoring; of fetal status and placentaldevelopment (NST, CST, BPP)

If the infant is close to term, administer steroids and induce labor after 48 hours.

If there are signs of nonreassuring fetal status;or deterioration of maternal vital signs (e.g., pre-eclampsia), induce labor or perform immediate cesarean section.