HF Focused 2 Flashcards
Systolic HF causes
impaired contractile function (MI), increased afterload (HTN), cardiomyopathy, valve issues
Diastolic HF causes
LV hypertrophy (from HTN), DM, Obesity, age
Left sided HF causes
HTN CAD
Right sided HF causes
left sided, RV infarction, PE, pulmonary HTN
how does the RAAS compensate
decrease volume so it hold fluid and salt to vasoconstrict and increase BP
how does ventricular remodeling compensate
hypertrophy and dilation of ventricles
what is a complication of ventricular remodeling
Dysrhythmias and sudden cardiac death
how does the SNS compensate
releases catecolamines to increase HR, increase myocardial contractility and peripheral constrict to increase BP and afterload
what are some meds that can be used to treat to hearts compensatory mechanisms
ACE, aldosterone agonist (sprinolactone) for RAAS and b-blockers for SNS
what do you need to monitor for ACE
cough, renal insufficency, low BP, hyperkalemia, and angioedema
what do you need to monitor for ACE
low BP,hyperkalemia, and renal insufficency
how do diuretics help with compensatory mech of the heart and what do you need to watch for
decrease edema, pulmonary venous pressure, and preload watch for hypokalemia
what is the progression of acute decompensated HF
increase pulmonary venous pressure, interstitial edema, alveolar edema
what is the first ss of acute decompensated HF
pulmonary edema
what and how should you monitor for acute decompensated HF
conitous monitor of VS, O2, and UOP, hemodynamic monitoring if unstable, aterial line for BP and PAWP
