HF 2 Flashcards
Heart Failure Classifications
RIght or left side
Left Sided HF
Blood backs up in pulmonary circulation (left atrium first)
Right Sided HF
Blood backs up in systemic circulation (right atrium first)
Left Sided HF: Symptoms
- Congestion in LEFT chambers
- LV becomes larger (LVH)
- Backflow into pulmonary veins
- Congestion in lungs
Left Sided HF: Findings
- Cough, crackles, wheezes
- Frothy sputum, may be blood tinged (PE)
- Paroxysmal nocturnal dyspnea (smothered and coughing at night suddenly)
- Orthopnea (tripod position to breath) (sleep sitting up)
Right Sided HF: Symptoms
- Often due to COPD
- Congestion in RIGHT chambers
- RV becomes larger (RVH)
- Backflow into Vena Cava, decreases perfusion to lungs
- Congestion in jugular veins, Liver, Lower extremities
Right Sided HF: Findings
JVD
Dependent Edema (swelling in lower extremities)
Weight gain
Hepatosplenomegaly
Left-Sided HF most common cause
Poorly controlled HTN
LUNGS
Right-Sided HF most common cause
COPD
-Pulmonary hypertension
(BODY)
Ejection Fractions
The amount of blood pumped out by LEFT VENTRICAL with each squeeze (55-65%)
HF: Reduced Ejection Fraction (HFrEF) [Systolic HF]
Determined by EF < 40%
Caused by:
- Impaired contractile function
- Inc afterload
- Cardiomyopathy
- Mechanical problems
- Left ventricle loses ability to generate pressure to eject blood
- Weakened muscle cannot generate stroke volume and then lowers cardiac output
- LV fails, blood backs up, causes fluid backup and accumulation
HF: Preserved Fraction (HFpEF) [Diastolic HF]
Inability of ventricles to relax and fill during diastole
HTN is the primary cause
Being female, older, diabetes, and obesity are risk factors
(FOOD)
LV is stiff and noncompliant leading to high filling pressures, leads to decreased stroke volume and decreased cardiac output
Reduced CO leads to fluid congestion
EF is normal or moderately reduced
Chronic HF
Episodes of “decompensated” HF
- New or worsening signs/symptoms
- Frequent visits to the ER
- Hospitalization
- Less common- new onset of HF (20%)
Ventricular Remodeling in HF
A weakened heart muscle
-Secretion of molecular substances
-Angiotensin II, aldosterone, endothelin
TNF-alpha, catecholamines, insulin-like
growth factor, and growth hormone.
-Provoke genetic changes, apoptosis and hypertrophy of cardiac myocytes, as well as collagen deposits and myocardial fibrosis
These molecules cause changes that lead to ENLARGEMENT AND DILATION OF THE LEFT VENTRICLE
-Worsens HF
S3 gallop in HF
- Low pitched sound heard after s2
- During rapid filling of the ventricle in the early part of diastole
- High ventricular end-diastolic volume
- Increased pressure within ventricles