Hemodynamic Monitoring Flashcards

1
Q

Systole

A

Contraction
* Ventricles eject blood into the aorta and pulmonary artery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Diastole

A

Ventricular relaxation and filling
* When blood return blood to the heart in preparation for the next ventricular contraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Four properties of the myocardial cell

A
  1. Automaticity (Chronotropic)
  2. Conductivity (Dromotropic)
  3. Contractility (Inotropic)
  4. Excitability (Bathmotropic)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Chronotropy

A
  • Chrono=time
  • The rate of contractions
  • Positive chronotropic drugs increase HR
  • Negative chronotropic drugs decrease HR
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Chronotropic effects of Beta blockers

A

Negative chronotropic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

effects of Calcium channel blockers

A

Negative chronotropic
Negative inotropic
* Calcium increases the strength of contractions, so by blocking calcium you decrease the strength of those contractions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

All beta blockers end in what

A

lol

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

All calcium channel blockers end in

A

Pine/ zem

(Amlodipine/ Cardizem)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Amiodarone chronotropic effects

A

Negative chronotropic effects
(Also turns you blue)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Inotropes

A

Affect the contractility of the heart
* Positive increase the contractility, used to pump more blood with fewer heart. beats
* Negative decrease the contractility: and to decrease the HR

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Why do we give positive inotropes

A
  • CHF
  • Cardiomyopathy
  • Recent MI
  • Cardiogenic shock
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Why do we give negative inotropes

A
  • Weaken the contractions of the heart and slows the HR
  • HTN
  • Chronic heart failure
  • Arrhythmias
  • Chest pain

Also used in MI patients to decrease the burden on the heart

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Dromotropy

A

Affects the conduction speed at the AV node, increasing/decreasing rate which electrical impulses move through the heart
* Positive increases conduction velocity
* Negative decreases velocity (Vagal stimulation)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Contractility

A

Force which the heart is able to contract
* Essential property of all muscles this is what allows the heart to act as a pump

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Frank starling laws

A

Essentially the heart is a rubber band, the more you’re able to stretch the muscle (More full), the more powerful the contraction
* Increasing preload increases contractility

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Digoxin, effects on the heart

A
  • Lowers HR (Negative chronotropic effects)
  • Increases contractility (Positive inotropic effects)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Cardiac Output (CO)

A

Amount of blood pumped by the heart in one minute (L/min)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Preload

A
  • Stretch of the ventricle, due to be filled with blood at the end of diastole
  • LVEDP
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

diseases with increased preload

A
  • HF
  • Valve diseases
  • Increasing O2 demand
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Meds that decrease preload

A
  • Ace inhibitor
  • Arbs
  • Diuretics
  • Nitrates
  • calcium channel blockers (CCB)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

LVEDP

A

Left ventricle end diastolic pressure
Stretch of the ventricle at the end of diastole, known as preload

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Afterload

A
  • The pressure which the heart must work against to be able to eject its blood during systole
  • Increased afterload makes the heart work harder decreasing CO
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Conditions that increase afterload

A

Aortic stenosis and elevated BP

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Stroke volume

A
  • Volume of blood pumped by LV during one contraction
  • Normal is 50-100
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Venous return
* Blood needs to come back to the heart in order to pump it * Volume of blood from the veins that returns to the atria each min * Venous has a huge impact on cardiac
26
Systemic vascular resistance
Total peripheral resistance, and the force which is exerted onto the blood by the vasculature of the body
27
Pulmonary vascular resistance
Resistance against blood flow from the pulmonary artery to left atrium
28
Cardiac output
SV * HR
29
Goal of hemodynamic monitoring
*** Maintain adequate tissue perfusion** * identify the presence and nature of shock * Guide response to resuscitation * Eval volume state * measure cardiac contractility * and systemic vascular resistance You need sufficient CO to keep BP and supply O2 rich blood to the brain and organs
30
Goal of hemodynamic monitoring
*** Maintain adequate tissue perfusion** * identify the presence and nature of shock * Guide response to resuscitation * Eval volume state * measure cardiac contractility * and systemic vascular resistance You need sufficient CO to keep BP and supply O2 rich blood to the brain and organs
31
Hemodynamic monitoring: Swan-ganz
* Indwelling line which gives info about 1. Blood volume 2. Perfusion 3. Fluid status 4. How well the heart is pumping
32
Swan-ganz/ pulmonary artery cath: Proximal lumen
Measures * right atrial pressure, (CVP) * IVF's * And can collect venous blood samples
33
Swan-ganz/ pulmonary artery cath: Distal lumen
Measures * Pulmonary artery pressure (PAP) * Pulmonary artery systolic/diastolic * Mean Pulmonary artery pressure * Pulmonary artery wedge pressure
34
Swan-ganz/ pulmonary artery cath: Balloon inflation port
* Used intermittently for pawp measurements * When not in use it must be deflated and locked
35
Swan-ganz/ pulmonary artery cath: Thermistor
* Measures temp difference between right atrium and pulmonary artery to determine CO
36
An increase in the Pulmonary artery pressure can be an early indicator of
Can show us before the patient is symptomatic * Left ventricular failure * Pulmonary edema * Fluid overload Early indication means it needs to be treated early
37
Indications for a pulmonary art cath
* Critical illness * Shock * Hemodynamic instability * Heart failure * Unstable post op patient * ARDS * Acute kidney injury * Severe burn injury * Trauma
38
Normal Central Venous pressure (CVP)
* 2-6 mm HG * (Located by the right atrium) * Can be used to measure preload on the right side of the heart
39
Normal Pulmonary Artery Systolic (PAS)
15-28 mmHg
40
Normal pulmonary artery diastolic (PAD)
5-16 mmHg
41
Normal pulmonary artery wedge pressure
Measured in the pulmonary artery * 6-15 mmHg * Can be used to measure the preload for the left side of the heart
42
Normal Cardiac output
3-6 L/min
43
Normal mixed venous (SVO2)
60-80%
44
Pre procedure nursing actions: Cardiac cath insertion ## Footnote Probably dont need to know
* Informed consent * Assemble the pressure monitoring system, purge air and maintain sterility * Place pt trendelenburg or supin * Administer sedation or analgesia as ordered * Level transducer with phlebostatic axis * Can see arrhythmias during insertion
45
Phlebostatic Axis
Anatomical point that corresponds to the RA and most accurately reflects a patient's hemodynamic status Pt lies supine with an art line entering their left forearm which is measured at the 4th intercostal space, mid axillary line where the RA exist
46
Post op Cardiac cath
* Get a chest X ray to confirm cath placement * Monitor vitals, Resp and cardio status * Maintain line placement and integrity (Secure it) * **Secure it** Observe and doc waveforms, report changes as this can indicate cath migration or displacement Doc cath placements every shift and after movements Obtain readings from the cath
47
Post op cardiac cath: Taking a reading
1. Place pt supin position prior to getting readings of hemodynamic values (HOB can be elevated 15 degrees) 2. Level the transducer at the phlebostatic axis before reading and with all position changes 3. Zero system to atmospheric pressure 4. Compare hemodynamic findings to physical assessment 5. Monitor trends in values obtained over time
48
Complications of a cardiac cath: infection/sepsis
* Change dressings per protocol and doc * Use surgical aseptic technique (Masks **Sterile gloves, sterile field)** * Monitor for S+S of infection (Fever, increased WBC) * Collect specimens or cultures as ordered * Admin antibiotics as orders * Admin IV fluids for vascular support (Sepsis) * Admin vasopressors secondary to sepsis as needed
49
Complications of cardiac cath: Embolism
* Use 0.9% NS for flushing system. Flush can include heparin or whatever your facilities protocol is * Avoid introduction of air into flushing system to prevent air embolism * Recognize the risk of pneumothorax with line insertion * Recognize the risk of arrhythmias with insertion or movement of the line | Plaque or a clot can be dislodged from the procedure
50
Hemodynamic instability
* **Systolic BP equal to or less than 90** * Sustained low BP and dropping * Not orthostatic hypotension * Need a baseline for the patient,, what is their usual BP
51
Goal Mean arterial pressure (MAP)
>60 mmHg
52
Mean Arterial pressure (MAP)
(SBP + (DBP*2))/3
53
Normal MAP
70-105 mmHg Greater than 60 is the goal
54
Manifestations of altered hemodynamics: Elevated preload
* Crackles in the lungs * Jugular vein distention * Hepatomegaly * Peripheral edema * Taut skin turgor Wedge measures the Left heart CVP measures the right heart
55
Manifestations of altered hemodynamics: Decreased preload
Poor skin turgor Dry mucous membranes Basically dehydrated or in shock Wedge measures the Left heart CVP measures the right heart
56
Manifestations of altered hemodynamics: Increased Afterload
More resistance means less blood flow out * Cool extremities * Weak pulses Left heart is measured by the systemic vascular resistance Right heart is measured by the pulmonary vascular resistance
57
Manifestations of altered hemodynamics: Decreased Afterload
Less resistance means more blood flow out * Warm extremities * Bounding pulses Left heart is measured by the systemic vascular resistance Right heart is measured by the pulmonary vascular resistance
58
Caution with vasopressors
Can cause too much vasoconstriction, which can lead to infarction/ blood clots in the bowel
59
What do vasopressors do
Increase SVR and increase BP * Class of drugs that increase MAP via vasoconstriction
60
Is a vasopressor an inotrope
No they differ from inotropes, however many drugs both have inotropic and vasopressor effects
61
Five vasopressor drugs
* Phenylephrine * Norepinephrine * Epinephrine * Dopamine * Vasopressin
62
Positive Inotrope examples
Epi NE DA Dobutamine Milrinone Digoxin
63
Negative inotropes examples
Flecainide (Antiarrhythmic agent) Verapamil (Calcium channel blocker) Cardizem (Calcium channel blocker) Clonidine (Antihypertensive given to psych) Atenolol (Beta blocker)
64
Drugs that affect hemodynamics: Vasodilators
* Decrease Afterload * Decrease BP
65
Drugs that affect hemodynamics: Antihypertensives
* Decrease afterload * Decrease BP
66
Drugs that affect hemodynamics: Loop diuretics
* Decrease preload * Decrease BP * Taken long term it decreases afterload
67
Drugs that affect hemodynamics: Morphine
* Decreases vascular resistance and preload
68
The three P's of perfusion
1. Pump 2. Pipes 3. Plasma
69
Reduced CO
* Reduce blood flow * Reduce perfusion * If not corrected quickly, shock
70
Three main factors affecting blood flow
* Blood volume (Plasma) * Cardiac pump function (EF) (Pumps) * Vaso Muscular tone (Pipes)
71
An RN is assessing a patient with a PA catheter in place. The CVP is 4 and PCWP is 19 mmHg. Which of the following findings would the nurse expect? 1. Poor skin turgor 2. Bilateral crackles in the lungs 3. Jugular venous distension 4. Dry mucous membranes 5. Hepatomegaly What if the CVP is 12 and PCWP is 9 mmHg?
Normal CVP ranges from 2-6, 4 is normal Normal PCWP ranges from 6-15 mmHg, 19 is elevated The PCWP measures the pressure of the left heart, higher pressure indicates congestion and a back up into the peripheral system, and due to the cvp being normal that means the lungs 2. Bilateral crackles in the lungs If the CVP is 12 it indicates an issue of increased preload on the right side which can indicate excess fluids and/or right sided heart failure
72
Arterial Line
* Placement of a cath into the lumen of an artery for continuous BP, and frequent Blood sampling
73
Nursing actions ART line
* Continually assess for the bleeding, dislodgement, hematoma formation and infection * Assess the patient for the 5 p's * If art line becomes dislodged patient can hemorrhage and die fast * Zero the system with atmospheric pressure, hemodynamic pressure lines must be calibrated to read zero atmospheric pressure * Obtain initial readings as needed, compare art line reading to noninvasive BP * Document the patient's response * Secure the art line!
74
What do you do if a art line becomes dislodged
Put pressure on it or else they will die fast