Hef - Motor Control I&II Flashcards

1
Q

motor unit

A

one lower motor unit + all the muscle fibers it innervates

  • affects sensitivity
  • large motor units –> gross motor (inaccurate)
  • small motor units –> fine motor (accurate)
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2
Q

2 types of lower motor neurons

A
  • alpha - extrafusal fibers

- gamma - intrafusal fibers; keep sensitivity alive so muscle spindles keep responding

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3
Q

where are alpha lower motor neuron cell bodies located?

A
  • ventral horn of spinal cord for spinal nerves

- brainstem for cranial nerves

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4
Q

different types of alpha neurons

A
  • large motor neurons –> fast twitch muscle fibers, fast AP
  • small motor neurons –> slow twitch muscle fibers, slow AP
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5
Q

muscle spindles

A
  • non-contractile in center, but contractile in periphery
  • gamma fibers –> supply tips, polar region
  • alpha fibers –> supply bulk of the muscle
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6
Q

dynamic vs. static contraction

A
  • dynamic = sudden elongation/stretch of muscle (only type 1a fibers)
  • static = contracting constantly (ex. maintain posture) type 1a and II fibers
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7
Q

difference b/w nuclear chain and nuclear bag fibers

A
  1. nuclear bag - contain type 1a fibers (afferent); static and dynamic stretch
  2. nuclear chain - contain type 1a and type 2; static stretch only
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8
Q

myotatic (muscle spindle) reflex

A

stretching muscle spindle carries afferent info. on type Ia or II to ventral horn synapsing with alpha or gamma motor neurons to initiate reflex

  • type Ia = monosynaptic; fast response
  • type II = mono or polysynaptic; slower response
  • both types have collaterals to inhibitory neurons of antagonist muscle
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9
Q

upper vs. lower motor neuron lesion

A

upper motor neuron –> hyperreflexia

lower motor neuron –> hyporeflexia

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10
Q

Golgi tendon reflex (inverse myotatic reflex)

A

contraction stimulates Golgi tendons –> send info. via type 1b fibers –> inhibitory to same muscle and stimulatory to antagonist muscle

  • equalizes contractile forces
  • extreme stimulation - lengthening relaxation
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11
Q

flexor withdrawal reflex

A
  • multisynaptic
  • harmless stimulation –> limited response and localized flexion response
  • harmfull stimulation –> abrupt flexor contraction and withdrawal and cross extension (stimulate flexors inhibit extensors on same side; stimulate extensors inhibit flexors on contralateral)
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12
Q

afterdischarge

A

keeps withdrawal response longer

-needed for cross extension (want to last longer than flexion)

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13
Q

central pattern generator

A

in spinal cord - responsible for locomotion independent of cerebral cortex
-sensory feedback needed for fine tuning

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14
Q

reflexes of spinal cord

A
  • positive supportive rxn = extending leg upon stimulus of foot
  • rhythmic stepping = alteration b/w flexion/extension
  • stumble reflex = ipsilateral flexion, contralateral extension
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15
Q

primary motor cortex

A

brodmann area 4

  • conscious and voluntary movements (association motor is more complex movement)
  • low level electrical stimulation (highly sensitive)
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16
Q

M1

A

where corticospinal tract fibers originate and synapse monosynaptically in ventral horn

17
Q

supplementary motor area (SMA)

A

connected to M1 and is involved in complex bilateral movements (ex. hands)
-work with premotor for complicated sequential movement as well as planning

18
Q

premotor cortex

A

complex and planning movements with SMA

-mirror neurons –> transfer sensory to motor movement

19
Q

motor apraxia

A

damage to premotor, association, or SMA - not fibers

  • cannot plan/organize actions
  • ex. forgot how to put jacket on
20
Q

corticospinal tract

A

originate from M1 = primary motor cortex

  • get info. from parietal somatosensory (fine movements)
  • most fibers cross –> lateral (fine motor)
  • some fibers ipsilateral –> anterior (gross motor)
  • lower motor neurons in spinal cord
21
Q

corticobulbar tract

A

impulses from primary motor or parietal somatosensory to brainstem to synapse with lower motor neurons of cranial nerves

  • bilateral in most CNs
  • unilateral for CN7 in lower face –> right side lesion –> left side paralysis
  • info. from limbic system along with primary motor
22
Q

corticospinal tract

A

monosynaptic with LMNs in spinal cord

  • more monosynaptic connections –> more accurate and specificity
  • damage –> lose motor functions (especially fine motor)
  • pyramidal tract fibers regulate reflex arc –> increase or decrease receptor firing with interneurons
  • controls flexors more than extensors
23
Q

corticorubrospinal tract

A

info. from cortex –> red nucleus –> spinal cord
- supplementary role (antigravity) and lacks fine motor control
- may help corticospinal tract regain some function

24
Q

reticulospinal tract

A

posture, moving toward stimulus, crude movements of extremities, locomotion

  • medial tract –> pons –> excitatory info. –> exaggerate response
  • lateral tract –> medulla –> inhibitory info. by interneurons to dampen pons
25
Q

tectospinal tract

A

turning head or body in response to visual or auditory stimuli

  • info. coming from descending fibers of superior colliculus to spinal cord –> auditory or visual reflex
  • fibers decussate after superior colliculus to terminate on interneurons and motor neurons
26
Q

damage to descending corticospinal tract

A

muscle rigidity and paresis, exaggerated stretch reflex, + babinski, lose voluntary movements, hard to recover fine motor

  • UMN lesion
  • have descending inhibitory and stimulatory neurons