HEENT Flashcards
False sense of motion (or exaggerated sense of motion)
Vertigo
2 types of vertigo
Peripheral and central
Horizontal nystagmus indicates
Peripheral vertigo - due to labyrinth or vestibular issues
Episodic vertigo, no hearing loss
BPPV or
Vestibular neuritis
Episodic vertigo, hearing loss
Meniere or
Labyrinthitis
Management of N/V in pts with vertigo
- Antihistamines (Meclizine, cyclizine, dimenhydramine, diphenhydramine)
- Metoclopramide, prochlorperazine
- Scopolamine
- Lorazepam, diazepam
Inflammation of both eyelids. Common in pts with _________ and ________
Blepharitis
Down syndrome
Eczema
Two types of blepharitis
- Infectious (staph aureus or staph epidermidis)
2. Seborrheic
Signs/symptoms of blepharitis
- Eye irritation/itching
2. Eyelid burning, erythema, crusting, scaling, red-rimming and eyelash flaking
Management of blepharitis
Warm compresses, eyelid scrubbing/washing with baby shampoo
May give azithromycin ointment/solution
Most common etiology of conjunctivitis
Adenovirus
Most common cause of viral conjunctivitis
Swimming pools
Signs/symptoms of viral conjunctivitis
Foreign body sensation
Erythema
Itching
Normal vision
Preauricular lymphadenopathy, copious watery discharge from eyes, scanty mucoid discharge.
Often bilateral
Viral conjunctivitis
Management of viral conjunctivitis
Supportive - cool compresses, artificial tears
Antihistamines for itching/redness
Signs/symptoms of allergic conjunctivitis
Conjunctival erythema paired with other allergic symptoms
Cobblestone mucosa appearance to the inner/upper eyelid, itching, tearing, redness, stringy discharge. Usually bilateral, +/- conjunctival swelling
Allergic conjunctivitis
Treatment for allergic conjunctivitis
Topical antihistamine: olopatadine
Topical NSAID: ketorolac
Most common causes of bacterial conjunctivitis
S. aureus
Strep pneumoniae
H. influenzae
Purulent discharge from eye, lid crusting, usually no vision changes
Bacterial conjunctivitis
Management of bacterial conjunctivitis
Topical abx - erythromycin, fluoroquinolones (moxi), sulfonamides, aminoglycosides
Management of bacterial conjunctivitis if contact lens wearer
Cover pseudomonas
Fluoroquinolones or aminoglycoside
Blowout fracture:
Fracture to the orbital floor as result of trauma. May lead to trapping of eye structures
Signs/symptoms of blowout fracture
- Decreased visual acuity (trapped orbital tissue)
- Diplopia especially with upward gaze (if inferior rectus muscle entrapment)
- Orbital emphysema (eyelid swelling after blowing nose - air from maxillary sinus)
- Epistaxis, anesthesia to the anteromedial cheek
Diagnosis of blowout fracture
CT - may show teardrop sign
Management of blowout fracture
- Initial: nasal decongestants, avoid blowing nose, corticosteroids, antibiotics
- Surgical repair - severe cases, patients with enophthalmos
Foreign body sensation in the eye, tearing, red and pain that is relieved with instillation of ophthalmic analgesic drops
Ocular Foreign body
Corneal abrasion
Diagnosis of ocular foreign body /corneal abrasion
Pain relieved with instillation of ophthalmic analgesic drops
Fluorescein staining- abrasions
Management of ocular foreign body
Check visual acuity first
Remove foreign bodies with sterile irrigation
Avoid sending pts home with topical anesthetics
Antibiotic drops - erythro, polymyxin/trimethoprim
Management of corneal abrasion
Check visual acuity first
Patching not indicated for small abrasions and no longer than 24 hrs
Ciprofloxacin, erythromycin
Infection of the lacrimal sac
Dacryocystitis
Tearing, tenderness, edema and redness to the nasal side of lower eyelid
Dacryocystitis
Management of dacryocystitis
Antibiotics - clindamycin
Dacryocystorhinostomy
Signs/symptoms of foreign body in the ear
Ear pain, drainage, conductive hearing loss. May be asymptomatic
Management of foreign body in ear
- Lidocaine drops if insect (to paralyze)
- Foreign body removal
- Assess for tympanic membrane rupture or complications
Signs/symptoms of foreign body in nose
Mucopurulent discharge Foul odor Epistaxis Nasal obstruction (mouth breathing)
Management of foreign body in nose
Positive pressure technique (have pt close other nostril and blow)
Oral positive pressure (parent blows into mouth while occluding other nostril - small children)
Instrument removal
Increased intraocular pressure leads to optic nerve damage, leading to decreased visual acuity
Acute narrow angle-closure glaucoma
Decreased drainage of aqueous humor via trabecular meshwork and canal of schlemm in pts with preexisting narrow angle or large lens
Acute narrow angle-closure glaucoma
Leading cause of preventable blindness in US
Acute narrow angle-closure glaucoma
Precipitating factors for acute narrow angle-closure glaucoma
Mydriasis - pupillary dilation further closes the angle
Dim lights, sympathomimetics and anticholinergics
Severe, sudden onset of unilateral ocular pain +/- nausea, vomiting, headache. Vision changes, blurring, halos around lights, peripheral vision loss (tunnel)
Acute narrow angle-closure glaucoma
Conjunctival erythema, steamy cornea, mid-dilated, fixed, nonreactive pupil, eye may feel hard to palpation
Acute narrow angle-closure glaucoma
Diagnosis of acute narrow angle-closure glaucoma
Increased IOP by tonometry (> 21 mmHg)
Cupping of optic nerve on fundoscopy
Management of acute angle glaucoma
Ophthalmic emergency
Step 1: lower IOP (acetazolamide, BB, mannitol)
Step 2: open the angle (cholinergics -pilocarpine, carbachol)
Peripheral iridotomy definitive treatment
Medications to avoid with acute angle glaucoma
Anticholinergics
Sympathomimetics
Visible blood in the anterior chamber of the ey
Hyphema
Complication of hyphema
Can lead to blindness if not properly attended to - leads to ocular hypertension
Diagnostic testing for hyphema
- Screen for sickle cell disease
2. If serious injury, CT scan for further evaluation
Treatment of hyphema
Eye shield, elevated head to 30 degrees
Give adequate analgesia (topical cycloplegics) and antiemetics to prevent increased ocular pressure
Topical steroids
Topical BB if increased pressure
Surgery indications of hyphema
Early corneal blood staining > 1/2 of anterior chamber involved
Uncontrolled intraocular pressure
Risk factors for macular degeneration
- Age > 50
- Caucasian
- Females
- Smokers
Most common cause of permanent legal blindness and visual loss in the elderly
Macular degeneration
Small, round, yellow-white spots on the outer retina (scattered, diffuse). Accumulation of waste products
Drusen - seen in macular degeneration
New, abnormal vessels grow under the central retina which leak and bleed, leading to retinal scarring - rarer than dry
Wet (neovascular or exudative) macular degeneration
Bilateral blurred or loss of central vision (including detailed and colored vision), scotomas (blind spots, shadows)
Macular degeneration
Straight lines appear bent
Metamorphopsia
Macular degeneration
Object seen by the affected eye looks smaller than in the unaffected eye
Micropsia
Macular degeneration
Diagnosis of macular degeneration
Amsler grid
Wet: fluorescein angiography
Management of wet macular degeneration
- Bevacizumab - VEGF
- Laser photocoagulation
- Optical tomography done to monitor treatment response
Acute inflammatory demyelination of the optic nerve
Optic Neuritis (Optic Nerve/CN II Inflammation)
Etiologies of optic neuritis
- Multiple Sclerosis (MC)
2. Medications (ethambutol, chloramphenicol, autoimmune)
Signs/symptoms of otpic neuritis
- Loss of color vision, visual field defects, loss of vision over a few days
- Usually unilateral
- Associated with ocular pain that is worse with eye movement
During swinging-flashlight test from the unaffected eye into the affected eye, the pupils appear to dilate (delayed response from affected optic nerve)
Marcus-Gunn Pupil
Optic Neuritis
Diagnosis of optic neuritis
- Marcus-Gunn pupil
- Fundoscopy - 2/3 normal disc/cup ratio OR 1/3 optic disc swelling/blurring
- May use MRI in some cases
Management of optic neuritis
IV methylprednisolone followed by oral corticosteroids
Vision usually returns with tx
Usually secondary to sinus infections (ethmoid 90%)
Orbital cellulitis
Orbital cellulitis most commonly occurs in ___________
children