HEENT Flashcards
False sense of motion (or exaggerated sense of motion)
Vertigo
2 types of vertigo
Peripheral and central
Horizontal nystagmus indicates
Peripheral vertigo - due to labyrinth or vestibular issues
Episodic vertigo, no hearing loss
BPPV or
Vestibular neuritis
Episodic vertigo, hearing loss
Meniere or
Labyrinthitis
Management of N/V in pts with vertigo
- Antihistamines (Meclizine, cyclizine, dimenhydramine, diphenhydramine)
- Metoclopramide, prochlorperazine
- Scopolamine
- Lorazepam, diazepam
Inflammation of both eyelids. Common in pts with _________ and ________
Blepharitis
Down syndrome
Eczema
Two types of blepharitis
- Infectious (staph aureus or staph epidermidis)
2. Seborrheic
Signs/symptoms of blepharitis
- Eye irritation/itching
2. Eyelid burning, erythema, crusting, scaling, red-rimming and eyelash flaking
Management of blepharitis
Warm compresses, eyelid scrubbing/washing with baby shampoo
May give azithromycin ointment/solution
Most common etiology of conjunctivitis
Adenovirus
Most common cause of viral conjunctivitis
Swimming pools
Signs/symptoms of viral conjunctivitis
Foreign body sensation
Erythema
Itching
Normal vision
Preauricular lymphadenopathy, copious watery discharge from eyes, scanty mucoid discharge.
Often bilateral
Viral conjunctivitis
Management of viral conjunctivitis
Supportive - cool compresses, artificial tears
Antihistamines for itching/redness
Signs/symptoms of allergic conjunctivitis
Conjunctival erythema paired with other allergic symptoms
Cobblestone mucosa appearance to the inner/upper eyelid, itching, tearing, redness, stringy discharge. Usually bilateral, +/- conjunctival swelling
Allergic conjunctivitis
Treatment for allergic conjunctivitis
Topical antihistamine: olopatadine
Topical NSAID: ketorolac
Most common causes of bacterial conjunctivitis
S. aureus
Strep pneumoniae
H. influenzae
Purulent discharge from eye, lid crusting, usually no vision changes
Bacterial conjunctivitis
Management of bacterial conjunctivitis
Topical abx - erythromycin, fluoroquinolones (moxi), sulfonamides, aminoglycosides
Management of bacterial conjunctivitis if contact lens wearer
Cover pseudomonas
Fluoroquinolones or aminoglycoside
Blowout fracture:
Fracture to the orbital floor as result of trauma. May lead to trapping of eye structures
Signs/symptoms of blowout fracture
- Decreased visual acuity (trapped orbital tissue)
- Diplopia especially with upward gaze (if inferior rectus muscle entrapment)
- Orbital emphysema (eyelid swelling after blowing nose - air from maxillary sinus)
- Epistaxis, anesthesia to the anteromedial cheek
Diagnosis of blowout fracture
CT - may show teardrop sign
Management of blowout fracture
- Initial: nasal decongestants, avoid blowing nose, corticosteroids, antibiotics
- Surgical repair - severe cases, patients with enophthalmos
Foreign body sensation in the eye, tearing, red and pain that is relieved with instillation of ophthalmic analgesic drops
Ocular Foreign body
Corneal abrasion
Diagnosis of ocular foreign body /corneal abrasion
Pain relieved with instillation of ophthalmic analgesic drops
Fluorescein staining- abrasions
Management of ocular foreign body
Check visual acuity first
Remove foreign bodies with sterile irrigation
Avoid sending pts home with topical anesthetics
Antibiotic drops - erythro, polymyxin/trimethoprim
Management of corneal abrasion
Check visual acuity first
Patching not indicated for small abrasions and no longer than 24 hrs
Ciprofloxacin, erythromycin
Infection of the lacrimal sac
Dacryocystitis
Tearing, tenderness, edema and redness to the nasal side of lower eyelid
Dacryocystitis
Management of dacryocystitis
Antibiotics - clindamycin
Dacryocystorhinostomy
Signs/symptoms of foreign body in the ear
Ear pain, drainage, conductive hearing loss. May be asymptomatic
Management of foreign body in ear
- Lidocaine drops if insect (to paralyze)
- Foreign body removal
- Assess for tympanic membrane rupture or complications
Signs/symptoms of foreign body in nose
Mucopurulent discharge Foul odor Epistaxis Nasal obstruction (mouth breathing)
Management of foreign body in nose
Positive pressure technique (have pt close other nostril and blow)
Oral positive pressure (parent blows into mouth while occluding other nostril - small children)
Instrument removal
Increased intraocular pressure leads to optic nerve damage, leading to decreased visual acuity
Acute narrow angle-closure glaucoma
Decreased drainage of aqueous humor via trabecular meshwork and canal of schlemm in pts with preexisting narrow angle or large lens
Acute narrow angle-closure glaucoma
Leading cause of preventable blindness in US
Acute narrow angle-closure glaucoma
Precipitating factors for acute narrow angle-closure glaucoma
Mydriasis - pupillary dilation further closes the angle
Dim lights, sympathomimetics and anticholinergics
Severe, sudden onset of unilateral ocular pain +/- nausea, vomiting, headache. Vision changes, blurring, halos around lights, peripheral vision loss (tunnel)
Acute narrow angle-closure glaucoma
Conjunctival erythema, steamy cornea, mid-dilated, fixed, nonreactive pupil, eye may feel hard to palpation
Acute narrow angle-closure glaucoma
Diagnosis of acute narrow angle-closure glaucoma
Increased IOP by tonometry (> 21 mmHg)
Cupping of optic nerve on fundoscopy
Management of acute angle glaucoma
Ophthalmic emergency
Step 1: lower IOP (acetazolamide, BB, mannitol)
Step 2: open the angle (cholinergics -pilocarpine, carbachol)
Peripheral iridotomy definitive treatment
Medications to avoid with acute angle glaucoma
Anticholinergics
Sympathomimetics
Visible blood in the anterior chamber of the ey
Hyphema
Complication of hyphema
Can lead to blindness if not properly attended to - leads to ocular hypertension
Diagnostic testing for hyphema
- Screen for sickle cell disease
2. If serious injury, CT scan for further evaluation
Treatment of hyphema
Eye shield, elevated head to 30 degrees
Give adequate analgesia (topical cycloplegics) and antiemetics to prevent increased ocular pressure
Topical steroids
Topical BB if increased pressure
Surgery indications of hyphema
Early corneal blood staining > 1/2 of anterior chamber involved
Uncontrolled intraocular pressure
Risk factors for macular degeneration
- Age > 50
- Caucasian
- Females
- Smokers
Most common cause of permanent legal blindness and visual loss in the elderly
Macular degeneration
Small, round, yellow-white spots on the outer retina (scattered, diffuse). Accumulation of waste products
Drusen - seen in macular degeneration
New, abnormal vessels grow under the central retina which leak and bleed, leading to retinal scarring - rarer than dry
Wet (neovascular or exudative) macular degeneration
Bilateral blurred or loss of central vision (including detailed and colored vision), scotomas (blind spots, shadows)
Macular degeneration
Straight lines appear bent
Metamorphopsia
Macular degeneration
Object seen by the affected eye looks smaller than in the unaffected eye
Micropsia
Macular degeneration
Diagnosis of macular degeneration
Amsler grid
Wet: fluorescein angiography
Management of wet macular degeneration
- Bevacizumab - VEGF
- Laser photocoagulation
- Optical tomography done to monitor treatment response
Acute inflammatory demyelination of the optic nerve
Optic Neuritis (Optic Nerve/CN II Inflammation)
Etiologies of optic neuritis
- Multiple Sclerosis (MC)
2. Medications (ethambutol, chloramphenicol, autoimmune)
Signs/symptoms of otpic neuritis
- Loss of color vision, visual field defects, loss of vision over a few days
- Usually unilateral
- Associated with ocular pain that is worse with eye movement
During swinging-flashlight test from the unaffected eye into the affected eye, the pupils appear to dilate (delayed response from affected optic nerve)
Marcus-Gunn Pupil
Optic Neuritis
Diagnosis of optic neuritis
- Marcus-Gunn pupil
- Fundoscopy - 2/3 normal disc/cup ratio OR 1/3 optic disc swelling/blurring
- May use MRI in some cases
Management of optic neuritis
IV methylprednisolone followed by oral corticosteroids
Vision usually returns with tx
Usually secondary to sinus infections (ethmoid 90%)
Orbital cellulitis
Orbital cellulitis most commonly occurs in ___________
children
Signs/Symptoms of orbital cellulitis
Decreased vision Pain with EOM Proptosis (bulging eye) Eyelid erythema Edema
Diagnosis of orbital cellulitis
High resolution CT scan
Infection of the fat and ocular muscles
MRI
Management of orbital cellulitis
IV antibiotics - vancomycin, clindamycin, cefotaxime, ampicillin/sulbactam
Management of preseptal cellulitis
Amoxicillin (no admit needed)
Infection of the eyelid and periocular tissue - may have ocular pain and swelling but no visual changes and no pain with ocular movement
Preseptal cellulitis
Optic nerve (disc) swelling secondary to increased intracranial pressure (classically bilateral)
Papilledema
Etiologies of papilledema (4)
- Idiopathic intracranial HTN (pseudotumor cerebri)
- Space-occupying lesion (cerebral tumor, abscess)
- Increased CSF production
- Cerebral edema, severe HTN (malignant)
Signs/symptoms of papilledema
- Headache
- Nausea/vomiting
- Vision usually well preserved, but may have changes
Diagnosis of papilledema
- Fundoscopy
- MRI or CT scan to r/o mass
- LP for increased CSF pressure
Management of papilledema
Diuretics (acetazolamide)
Most common type of retinal detachment
Rhegmatogenous
Retinal inner sensory layer detaches from choroid plexus
Most common predisposing factors for retinal detachment
Myopia (nearsightedness)
Cataracts
Photopsia (flashing lights), floaters, progressive unilateral vision loss
Retinal detachment
Shadow “curtain coming down” in peripheral initially, leading to loss of central visual field. No pain or redness of eye
Retinal detachment
Diagnosis of retinal detachment
Fundoscopy: detached tissue flapping in vitreous humor
Clumping of brown-colored pigment cells in anterior vitreous humor resembling tobacco dust
+ Shafer’s Sign
Retinal detachment
Management of retinal detachment
Ortho emergency
Keep patient supine
Don’t use miotic drops
Laser, cryotherapy
Central retinal thrombus, fluid backup in retina, acute sudden monocular vision loss
Central retinal vein occlusion (CRVO)
Risk factors for CRVO
- HTN
- DM
- Glaucoma
- Hypercoagulable states
Extensive retinal hemorrhages (blood and thunder appearance), retinal vein dilation, macular edema, optic disc swelling
Central retinal vein occlusion
Management of CRVO
No known effective tx
+/- anti-inflammatories, steroids, laser photocoagulation
May resolve spontaneously or progress to permanent vision loss
Excess H2O or local trauma changes the normal acidic pH of the ear, causing bacterial overgrowth
otitis externa
Most common eteiology of otitis externa
Pseudomonas (MC)
Proteus, S. aureus
1–2 days of ear pain, pruritus in the ear canal
May have had recent activity of swimming
Auricular discharge, pressure/fullness. hearing usually preserved
Otitis externa
Management of otitis externa
Protect ear against moisture
Ciprofloxacin/dexamethasone
Ofloxacin safe
Aminoglycoside combination
Management of malignant otitis externa
Seen in DM and immunocompromised
IV Ceftazidime or Piperacillin + fluoroquinolones
Infection of middle ear, temporal bone, and mastoid air cells. Most commonly preceded by viral URI
Acute otitis media
4 most common organisms of acute otitis media
S. pneumo, H. influenzae, M. catarrhalis, strep pyogenes
Risk factors for otitis media
Eustachian tube dysfunction Young (ET is wider, shorter and more horizontal) Daycare Pacifier/bottle use Parental smoking Not being breastfed
Fever, otalgia, ear tugging in infants, conductive hearing loss, stuffiness
Otitis media
Rapid relief of ear pain + otorrhea
Tympanic membrane perforation
Management of otitis media
- Amoxicillin 10-14 days
- Augmentin or Cefixime
- If PCN allergic, erythromycin, azithromycin, Bactrim
Management for severe, recurrent cases of otitis media
Myringotomy (surgical drainage)
Tympanostomy
Treatment for chronic otitis media - perforated TM + persistent or recurrent purulent otorrhea +/- pain
Topical ofloxacin or ciprofloxacin
Avoid water/moisture/topical aminoglycosides in ear with TM rupture
Treatment for tympanic membrane perforation without infection
Most heal spontaneously. Follow up to ensure resolution
Auricular hematoma occurs after ____________ to the ear, typically during spots (wrestling, rugby, boxing, etc)
Direct trauma
If an auricular hematoma is not drained, disruption of blood supply to the auricular cartilage causes necrosis and usually results in:
Cauliflower ear
Management of auricular hematoma
All should be drained ASAP after injury
If > 7 days old, refer to otolaryngologist or plastic surgeon for debridement
Regional auricular block using local anesthetic usually provides adequate anesthesias, then either needle aspiration or I&D is performed
Inflammation of the vestibular portion of CN 8 - most commonly after viral infection
Vestibular Neuritis
Vestibular neuritis plus hearing loss/tinnitus
Labyrinthitis
Signs/symptoms of vestibular neuritis/labyrinthitis
Peripheral vertigo (usually continuous), dizziness, N/V, gait disturbances
Management of vestibular neuritis/labyrinthitis
Corticosteroids first line
Meclizine, benzos for sx
Inflammation of the mastoid air cells of the temporal bone
Mastoiditis
Etiology of mastoiditis
Usually a complication of prolonged or inadequately treated otitis media
Signs/symptoms fo mastoiditis
- Deep ear pain, fever
2. Mastoid tenderness, may develop cutaneous abscess
Complications of mastoiditis
Hearing loss, labyrinthitis, vertigo, CN VII paralysis, brain abscess
Diagnosis of mastoiditis
CT scan
Management of mastoiditis
- IV antibiotics + middle ear/mastoid drainage hallmark
2. Mastoidectomy if refractory or complicated
Tonsillitis –> cellulitis –> _____________
Abscess formation
Peritonsillar abscess
Most common causes of peritonsillar abscess
Strep pyogenes (GABHS) Staph aureus
Signs/symptoms of peritonsillar abscess
Dysphagia, pharyngitis
Muffled “hot potato voice”
Difficulty handling oral secretions, trismus
Uvula deviation to contralateral side
Tonsillitis, anterior cervical lymphadenopathy
Diagnosis of peritonsillar abscess
CT scan first line to differentiate cellulitis vs abscess
Management of peritonsillar abscess
Antibiotics + aspiration or I&D
Unasyn, clindamycin, penicillin G + metronidazole
Tonsillectomy if recurrent
Treatment for dental abscess
Augmentin and abscess drainage
Inflammation of the larynx
Laryngitis
Most common etiology of laryngitis
Viral - adenovirus, rhinovirus, etc.
Trauma (vocal abuse)
Hallmark of laryngitis
Hoarseness
Management of laryngitis
Vocal rest, warm saline gargles, anesthetics, lozenges, increased fluid intake
Inflammation of the epiglottis that may interfere with breathing (medical emergency)
Epiglottitis
Most common cause of epiglottitis
Haemophilus influenzae type B - reduced incidence due to Hib vaccination
Signs/symptoms of epiglottitis
3 D’s - dysphagia, drooling, distress
Fever, odynophagia, inspiratory stridor, dyspnea, hoarseness, muffled voice, tripoding
Suspect in pt with rapidly developing pharyngitis, muffled voice and odynophagia out of proportion to physical finddings
Epiglottitis
Diagnosis of epiglottitis
- Laryngoscopy - direct visualization - cherry red epiglottis
- Lateral cervical radiograph - thumb sign
- If high suspicion, do not attempt to visualize the epiglottis with tongue depressor
Management of mild epiglottitis - no stridor at rest, no respiratory distress
Cool humidified air mist, hydration
Dexamethasone provides significant relief as early as 6 hours after dose (oral or IM)
Supplemental O2 in pts with sats < 92%
Patients can be discharged home
Management of moderate epiglottitis - stridor at rest with mild to moderate retractions
Dexamethasone PO or IM + supportive treatment
+/- nebulized epinephrine
Should be observed for 3-4 hours after clinical intervnetion
May be discharged home if improvement is seen
Management of severe epiglottitis - stridor at rest with marked retractions
Dexamethasone + nebulized epinephrine and hospitalization
Most common etiology of corneal ulcer? In contact lens wearers? With ocular trauma? Chronic topical steroid use?
HSV overall
pseudomonas
Bacterial
fungal
Ulceration usually has regular borders and will have accompanying purulent exudate
Corneal ulcer
Blue/green discharge with corneal ulcer
pseudomonas
Dendrites on fluorescein staining with corneal ulcer
HSV
Satellite lesions around ulceration with corneal ulcer
Fungus
3 main types of rhinitis
Allergic
Infectious
Vasomotor
Nonallergic/noninfectious dilation of the blood vessels (ex temperature change)
Vasomotor rhinitis
MC infectious cause of rhinitis
Rhinovirus (common cold)
Sneezing, nasal congestion/itching, clear rhinorrhea. Eyes, ears, nose and throat may be involved. Allergic associated with nasal polyps and tends to be worse in the morning
Rhinitis
Pale/violaceous, boggy turbinates, nasal polyps with cobblestone mucosa of the conjunctiva
Allergic rhinitis
Erythematous turbinates indicates
Viral rhinitis
Management of viral rhinitis
- Intranasal corticosteroids
Management of rhinitis
- Oral antihistamines
2. Decongestants - oral, intranasal
Acute sinusitis is defined as:
1-4 weeks
Etiologies of sinusitis
S. pneumo
H. flu
GABHS
M. catarrhalis (same as otitis media)
Signs/symptoms of sinusitis
Sinus pain/pressure - worse with bending down and leaning forward Headache, malaise Purulent sputum or nasal discharge Fever Nasal congestion
Physical exam of sinusitis
Sinus tenderness on palpation
Opacification with transillumination
Diagnosis of sinusitis
Clinical diagnosis
CT scan diagnostic test of choice
Sinus radiographs- waters view
Symptomatic management of sinusitis
- Decongestants, antihistamines, mucolytics, intranasal corticosteroids, analgesics, nasal lavage
Indicated if sx < 7 days
Antibiotic treatment for sinusitis
Sx should be present for > 10-14 days or earlier if: febrile, facial swelling, etc
Amoxicillin drug of choice x 10-14 days
Doxycycline, Bactrim
Chronic sinusitis is defined as
> 12 consecutive weeks
Most common bacterial cause of chronic sinusitis
S. aureus
Most common epistaxis form
Anterior
Most common site of bleeding in anterior epistaxis
Kiesselbach’s Plexus
Most common risk factors for posterior epistaxis
Hypertension and atherosclerosis
Most common site of bleeding in posterior epistaxis
Palatine artery
Management of epistaxis
- Pressure while seated and leaning forward
- Topical decongestants/vasoconstrictors
- Cauterization if bleeding can be seen
- Nasal packing
Most common causes of pharyngitis
Adenovirus, rhinovirus, enterovirus
GABHS for streptococcal pharyngitis
Signs/symptoms of pharyngitis
- Sore throat
2. Pain with swallowing
Management of viral pharyngitis
Fluids, warm saline gargles, topical anesthetics, lozenges, NSAIDs
Centor criteria for strep throat
- Fever > 100.4
- Pharyngotonsillar exudates
- Tender anterior cervical lymphadenopathy
- Absence of cough
Centor criteria interpretation
Score 0-4
0-1 no abx or culture needed
2-3 throat culture
4-5 give abx
Modified centor criteria
< 15 y/o add 1 point
> 44 y/o subtract 1 point
Diagnosis of strep throat
Rapid antigen detection test
Throat culture - definitive diagnosis (gold standard)
Management of strep throat
Penicillin G or VK first line, amoxicillin, augmentin
Macrolides if PCN allergic (azithromycin, clarithromycin, erythromycin)
Complications of strep throat
- Rheumatic fever
- Glomerulonephritis
- Peritonsillar abscess, cellulitis