Heart Failure Flashcards
Spring 2019
S/S of Digoxin toxicity
Abdominal pain anorexia nausea vomiting bradycardia arrhythmias
Percutaneous Coronary Revascularization (PCR)
purpose if to improve blood flow through a coronary artery
LVAD and VAD (2 types of therapy)
DESTINATION THERAPY——long term tx for patients not qualified for heart transplant.
BRIDGE TO TRANSPLANT THERAPY—–used until heart becomes available
How does the “compensatory mechanism” work?
Increased volume in the vascular system that this creates only makes the struggling heart work harder
The retention of water and sodium and vasoconstriction of the arteries make it harder for the heart to pump
It increases blood pressure
It increased heart rate
It increases afterload and all of these things are bad for a heart that is already struggling
Contractility
Strength of the hearts cells to shorten and contract
Medications for Pulmonary Edema (5)
“MAD IV”
M-orphine——decrease anxiety—–cause vasodilation
A-ntihypertensives—decrease afterload (ACE inhib
beta blockers)
D-iuretics—-promotes fluid excretion (furosemide)
I-notropes—-improve cardiac output (digoxin)
V-asodilators—decrease preload & afterload (nitro)
Nursing Care of Pulmonary Edema (10)
“BIV Phobias”
B-lood gases
I-ntake & Output
V-itals
P-Potassium H-igh fowlers O-xygen w/hi-flo rebreather B-ipap I-ntubation or/ventilation A-irway S-low (or) d/C Iv's
S/S of RIGHT-SIDED heart failure (
“SWELLING”
S-Swelling W-eight gain E-dema (ankles-legs-sacrum) L-Large distended neck vein L-lethargic I-rregular heart rate N-octuria G-irth of abdomen / ascites
Loop and thiazide diuretics can cause___________, so ________ supplementation should be given
hypokalemia
potassium
Describe S/S stage 4 heart failure
1) severe
2) signs of heart failure at rest
3) any activity increases signs of failure
Implantable Cardioverter Defibrillator
provides electrical countershock when lethal arrhythmia is detected
Stroke Volume
Amount of blood pumped by a ventricle with each beat
What happens to the BNP level if the heart failure continues to worsen?
The BNP continues to increase as long as HF is worsening
What makes “heart function” successful? (2)
1) blood flow is forward
RIGHT SIDE GOES TO LUNGS
LEFT SIDE GOES TO THE BODY
2) Blood flow is efficient = adequate cardiac output
PRELOAD
volume of blood in ventricles at the end of diastole
—PALLIATIVE CARE—
END-STAGE HF SYMPTOMS (4)
1) DYSPNEA
2) PAIN
3) FATIGUE
4) DEPRESSION
S/S of LEFT-SIDED Heart Failure (10)
“DROWNING AD”
D-ifficulty breathing R-Rales/crackles O-Orthopnea W-eakness/fatigue N-Nocturnal Paroxysmal Dyspnea
I-ncreased heart rate
N-Nagging cough
G-aining 2-3 lb/day (or) 5lb/week
A-ltered mental status
D-ecreased Urine Output
_______, ________ and ________ are the 1st to show decreased function when heart failure and poor oxygenation occur.
brain
lungs
kidney
AFTERLOAD
resistance that the left ventricle must overcome to circulate the blood
PRE LOAD
Amount the ventricles stretch at the end of diastole
teaching points for LVAD & VAD
NO chest compressions
NO MRI
OK to give ACLS drugs
OK for cardiac defibrillation