HEART AND VASCULAR Flashcards

1
Q

HEART SIZE IN WOMEN AND MEN

A

9OZ AND 10.9 OZ

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2
Q

The large veins and arteries leading directly to and away from
the heart are referred to as the

A

great vessels.

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3
Q

Collagen
—— anchor the AV valve flaps to
papillary muscles within the ventricles.

A

Collagen
fibers,chordae tendineae,

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4
Q

tough, inextensible, loose-fitting, fibroserous
sac that attaches to the great vessels and surrounds the heart.

A

pericardium

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5
Q

thickest layer of the heart, made up of
contractile cardiac muscle cells.

A

myocardium

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6
Q

thin layer of
endothelial tissue that forms the innermost layer of the heart

A

endocardium

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7
Q

in the
myocardium of both ventricles, causing them to contract almost
simultaneously.

A

Purkinje fibers

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8
Q

filling and emptying of the heart’s
chambers.

A

cardiac cycle

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9
Q

endures for approximately
two thirds of the cardiac cycle and systole is the remaining one
third

A

Diastole

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10
Q

the amount of blood pumped from the heart with each
contraction (SV from the left ventricle is usually 70 mL).

A

Stroke Volume

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11
Q

veins lie deep and medial to the
sternocleidomastoid muscle.

A

internal jugular veins

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12
Q

lie lateral to the sternocleidomastoid muscle and
above the clavicle

A

external jugular veins

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13
Q

determining the hemodynamics of the right side of the heart.

A

jugular venous pulse

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14
Q

reflects rise in atrial pressure that occurs with atrial
contraction

A

a wave

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15
Q

reflects right atrial relaxation and descent of the
atrial floor during ventricular systole

A

x descent

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16
Q

reflects right atrial filling, increased volume, and
increased atrial pressure

A

v wave

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17
Q

reflects right atrial emptying into the right ventricle
and decreased atrial pressure

A

y descent

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18
Q

May be seen with
weak heart muscles, an attempt
by the heart to increase CO.

A

Tachycardia

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19
Q

may occur with an
abnormality of the heart’s
conduction system
(arrhythmias) or during the
heart’s attempt to increase CO
by increasing the HR.

A

Palpitations

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20
Q

may result from
compromised CO.

A

Fatigue

21
Q

is usually
described as a sensation of
squeezing around the heart; a
steady, severe pain; and a
sense of pressure.

A

Angina

22
Q

may result from
congestive heart failure,
pulmonary disorders, coronary
artery disease, myocardial
ischemia, and myocardial
infarction (MI).

A

Dyspnea

23
Q

is the need
to sit more upright to breathe
easily due to fluid accumulation
in the lungs

A

Orthopnea

24
Q

A sudden onset of
edema in one leg with pain may
be due to a blood clot in that leg

A

deep vein thrombosis

25
Q

the most
common cause of peripheral
edema.

A

Venous insufficiency

26
Q

affect the heart’s ability to pump, decreasing the oxygen
supply to the tissues.

A

Congenital or acquired defects

27
Q

develops after exposure
to group A beta hemolytic
streptococci and results in
inflammation of all layers of the
heart, impairing contraction and
valvular function.

A

Rheumatic
carditis

28
Q

inflammation of
the parietal pleura
next to the
pericardium)

A

Pericarditis

29
Q

tear in
the wall lining the
aorta)

A

Aortic dissecting
aneurysm

30
Q

irreversible heart
damage due to
myocardial
ischemia)

A

Myocardial infarction
(irreversible heart

31
Q

Distention of jugular
vein on one side
may be caused by a

A

kink or aneurysm.

32
Q

may
occur in clients with
severe constrictive
pericarditis.

A

Kussmaul sign

33
Q

a blowing or
swishing sound
caused by turbulent
blood flow through
a narrowed vessel,
is indicative of
occlusive arterial
disease. However, if
the artery is more
than two-thirds
occluded, a bruit
may not be heard.

A

bruit

34
Q

Always
auscultate the
carotid arteries
before palpating
because

A

palpation
may increase or
slow the HR

35
Q

Loss of elasticity
may indicate—–

A

arteriosclerosis

36
Q

Loss of elasticity
may indicate

A

Fully distended
jugular veins

37
Q
A
38
Q

delayed upstroke

A

aortic
stenosis.

39
Q

Pulsations, which
may also be called

A

heaves or lifts

40
Q

The apical impulse
may be impossible
to palpate in clients
with

A

pulmonary
emphysema

41
Q

If you detect an
irregular rhythm,
auscultate for a

A

pulse rate deficit.

42
Q

The radial and apical
pulse rates should
be

A

Identical

43
Q

A thrill is palpated over the second and third ICS; a thrill may indicate

A

aortic stenosis and systemic hypertension.

44
Q

A thrill palpated over
the second and third left ICSs may indicate

A

pulmonic stenosis and
pulmonic hypertension.

45
Q

A sign of pressure overload

A

accentuated apical impulse

46
Q

A sign of volume overload

A

apical impulse displaced

47
Q

These beats occur earlier than the next expected beat and are followed
by a pause. The rhythm resumes with the next beat.

A

PREMATURE ATRIAL OR JUNCTIONAL CONTRACTIONS

48
Q

These beats occur earlier than the next expected beat and are followed
by a pulse. The rhythm resumes with the next beat.

A

PREMATURE VENTRICULAR CONTRACTIONS

49
Q

faster with inhalation and slower with expiration

A

SINUS ARRHYTHMIA