haemorrhagic stroke Flashcards

1
Q

pathophysiology of haemorrhagic stroke (6)

A

The expanding blood clot dissects and destroys brain
tissue
 It acts like space-occupying lesion causing high
intracranial pressure
 High ICP may affect cerebral perfusion
 High ICP in one compartment may displace structures and
cause herniation
 Brain stem compression cause death
 Mortality is high in hemorrhage

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2
Q

signs and symptoms of increased ICP 5

A

headache
seizure
n/v
AMS
photophobia and diplobia/ blurring of vision

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3
Q

what is ICH, where does it usually occur?

A

Bleeding occurs in the brain substance
 It usually from small arteries deep in the white
matter
 Common sites: basal ganglia,thalamus, pons and
cerebellum

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4
Q

clinical features of haemorrhagic stroke (3)

A

 Stroke usually occurs while the patient is awake or exerting
 Sudden onset within minutes
 Often associated with severe headache and vomiting. Seizures
are common

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5
Q

what are the complications of haemorrhagic stroke?

A

 Vasospasm: Presence of blood may irritate one or more arteries
causing vasospasm and ischemia within 1 or 2 weeks
 Hydrocephalus: Blockage of reabsorption of CSF can result in
hydrocephalus and increase in ICP
 Rerupture: chances are higher

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6
Q

symptoms of ganglionic haemorrhage

A

Contralateral hemiplegia which worsens to drowsiness and
coma

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7
Q

symptoms of thalamic haemorrhage

A

Contralateral hemiplegia with involvement of third nerve

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8
Q

symptoms of pontine haemorrhage

A

Quadriplegia, pin-point pupils, death

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9
Q

symptoms of cerebellar haemorrhage

A

Ataxia, altered sensorium, death

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10
Q

cause of SAH

A

Subarchnoid hemorhhage
 Commonly due to saccular aneurysms of cerebral arteries

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11
Q

what is saccular aneurysm

A

Saccular aneurysms (Berry aneurysm)
 Congenital pouch-like dilatations of the arterial wall.
 Rupture of the dome occurs in adolescents

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12
Q

Clinical Features of SAH

A

 Sudden onset of worst
headache ever
experienced
 Vomiting
 May or may not lose
consciousness
 commonly no focal
neurological deficits

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13
Q
A
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