GIT BLOCK EXAM Flashcards

1
Q

This type of stool is NOT included among the most common symptoms of a pathogenic bacterial GIT
Infection:

  • None of the above
  • fatty stool
  • prolonged diarrhea
  • bloody diarrhea
  • mucoid stool
A

fatty stool

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2
Q

Hemolytic uremic syndrome caused by a common bacteria in stool, Escherichia coli, is most frequently seen in the following population EXCEPT:

  • pregnant women
  • none of the above
  • children
  • immunocompromised
  • elderly
A

NONE

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3
Q

The following are the strict rules when requesting Stool Exam for your patients EXCEPT:

  • specimen must be sent to the lab within one hour
  • stool sample should be collected during chronic phase of diarrhea
  • contamination with urine should be avoided
  • All of the above
  • a clean, wide neck container should be used
A

stool sample should be collected during chronic phase of diarrhea

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4
Q

Though, together with other known normal flora, an increase in the population of this organism may still lead to diarrhea.

A

Clostridium difficile

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5
Q

The following are the instructions you should give your patient for stool examination, EXCEPT?

  • Stool sample should be collected in acute phase of diarrhea.
  • Specimen must be sent to the lab within 30min.
  • Stool sample may be collected from the sides of the toilet bowl
  • Contamination with urine should be avoided,
  • A clean, wide neck container should be used.
  • none of the above
A

Stool sample may be collected from the sides of the toilet bowl

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6
Q

This is NOT among the most common symptoms of a pathogenic GIT bacterial infection:

  • fat globules in stool
  • mucus in the stool,
  • none of the above
  • bloody diarrhea,
  • prolonged diarrhea,
  • abdominal pain and cramping
A

fat globules in stool

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7
Q

Complications of diarrhea EXCEPT:

  • light-headedness
  • edema
  • dehydration
  • electrolyte imbalance
  • All of the above
  • fatigue
A

edema

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8
Q

Which is NOT CORRECT regarding Pancreatic Carcinoma?

  • Tumor markers used are serum Carcinoembryonic and CA19-9antigens
  • highly early invasiveness
  • 60% are in pancreatic body
  • may manifest Migratory thrombophlebitis (Trousseau syndrome)
  • None of the above
A

60% are in pancreatic body

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9
Q

Transport Medium of Choice for Stool samples: worm infestation

A

saline

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10
Q

Transport Medium of Choice for Stool samples: suspected Salmonella and Shigella

A

tetrathionate broth

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11
Q

Transport Medium of Choice for Stool samples: Vibrio cholera

A

alkaline peptone water

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12
Q

Transport Medium of Choice for Stool samples: virus

A

phosphate buffer

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13
Q

Diagnose these STOOL COLORs: Red

A

Can indicate bleeding of the lower digestive tract or rectum

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14
Q

DIAGNOSE THESE ORAL LESION FINDINGS: red, velvety, flat or slightly depressed tongue lesion

A

Erythroplakia

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15
Q

IDENTIFY THE ASSOCIATED CAUSATIVE ORGANISM FOR EACH OF THE FOLLOWING DISORDER:
Herpes Labialis

A

Herpes simplex virus -1 (HSV -1)

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16
Q

IDENTIFY THE ASSOCIATED CAUSATIVE ORGANISM FOR EACH OF THE FOLLOWING DISORDER:
Squamous Cell Carcinoma of Oral Cavity & Esophagus

A

Human papillomavirus -16 (HPV-16)

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17
Q

IDENTIFY THE ASSOCIATED CAUSATIVE ORGANISM FOR EACH OF THE FOLLOWING DISORDER:
Sialadenitis of parotids

A

Mumps virus

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18
Q

IDENTIFY THE ASSOCIATED CAUSATIVE ORGANISM FOR EACH OF THE FOLLOWING DISORDER:
Chronic Gastritis

A

Helicobacter pylori

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19
Q

IDENTIFY THE ASSOCIATED CAUSATIVE ORGANISM FOR EACH OF THE FOLLOWING DISORDER:
Esophagitis with endothelial cell inclusions

A

Cytomegalovirus (CMV)

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20
Q

DIFFERENTIATE THE TYPES OF ODONTOGENIC CYSTS: the tooth apex resulting from long-standing
pulpitis

A

Periapical cyst

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21
Q

IFFERENTIATE THE TYPES OF ODONTOGENIC CYSTS: typically located within posterior mandible

A

Odontogenic Keratocysts

22
Q

DIFFERENTIATE THE TYPES OF ODONTOGENIC CYSTS: originates around the crown of impacted wisdom tooth

A

Dentigerous cyst

23
Q

DESCRIBE THE MECHANISM OF ESOPHAGEAL OBSTRUCTION: narrowing by fibrous thickening of
submucosa & secondary epithelial damage

24
Q

DESCRIBE THE MECHANISM OF ESOPHAGEAL OBSTRUCTION: abnormal connection between
esophageal pouches to bronchus or trachea

25
DESCRIBE THE MECHANISM OF ESOPHAGEAL OBSTRUCTION: thin, non canalized cord at the tracheal bifurcation
Atresia
26
DESCRIBE THE MECHANISM OF ESOPHAGEAL OBSTRUCTION: Esophageal dysmotility in failure of neural innervation
Achalasia
27
IDENTIFY THE TYPE & INCIDENCE OF ACUTE PEPTIC ULCERATION: critically ill patients with shock, sepsis, or severe trauma
Stress ulcers
28
IDENTIFY THE TYPE & INCIDENCE OF ACUTE PEPTIC ULCERATION: in proximal duodenum associated with severe burns or trauma
Curling ulcers
29
IDENTIFY THE TYPE & INCIDENCE OF ACUTE PEPTIC ULCERATION: in stomach, duodenum, or esophagus of persons with intracranial disease
Cushing ulcers
30
IDENTIFY THE TYPE & INCIDENCE OF ACUTE PEPTIC ULCERATION: at anastomotic site following partial gastrectomy
Marginal ulcers
31
SITES OF LYMPH NODE METASTASES: upper third of esophagus
cervical lymph nodes
32
SITES OF LYMPH NODE METASTASES: middle third
mediastinal, paratracheal, & tracheobronchial nodes
33
Causes of Mechanical Bowel Obstruction: Intussusception
Intramural
34
State the Pathological Findings of the following conditions: Lynch syndrome * multiple gastrointestinal hamartomatous polyps and mucocutaneous hyperpigmentation * familial clustering of cancers at several sites including colorectum, endometrium, ureters, brain, and skin. * All of the above * teenage 100 polyps to several thousand polyps * rectal bleeding, mucus discharge, and reactive inflammatory polyp of the anterior rectal wall
ALL
35
State the Pathological Findings of the following conditions: Peutz-Jeghers Syndrome * none of the above * familial clustering of cancers at several sites including colorectum, endometrium, ureters, brain, and skin. * teenage 100 polyps to several thousand polyps * multiple gastrointestinal hamartomatous polyps and mucocutaneous hyperpigmentation * rectal bleeding, mucus discharge, and reactive inflammatory polyp of the anterior rectal wall.
multiple gastrointestinal hamartomatous polyps and mucocutaneous hyperpigmentation
36
State the Pathological Findings of the following conditions: Familial Adenomatous Polyposis * teenage 100 polyps to several thousand polyps * familial clustering of cancers at several sites including colorectum, endometrium, ureters, brain, and skin. * rectal bleeding, mucus discharge, and reactive inflammatory polyp of the anterior rectal wall. * none of the above * multiple gastrointestinal hamartomatous polyps and mucocutaneous hyperpigmentation
teenage 100 polyps to several thousand polyps
37
Colonic Polyps: Inflammatory Polyps
reactive Polyp
38
Colonic Polyps: Juvenile Polyps
solitary, pedunculated, dilated glands filled with mucin
39
Colonic Polyps: Peutz-Jeghers Syndrome Polyp
multiple large, polyp w/ arborizing connective tissue
40
Describe how should a Physician illicit the following signs of appendicitis from the patient: Dunphy sign * RLQ pain with internal and external rotation of the flexed right hip * deep tenderness at 2/3 of imaginary line from umbilicus to right anterior superior iliac spine * RLQ pain on voluntary cough * RLQ pain with flexion & extension of the right hip * RLQ pain on jarring motion while standing landing
LQ pain on voluntary cough
41
Describe how should a Physician illicit the following signs of appendicitis from the patient: Markle sign * All of the above * RLQ pain on voluntary cough * RLQ pain with flexion & extension of the right hip * deep tenderness at 2/3 of imaginary line from umbilicus to right anterior superior iliac spine * RLQ pain with internal and external rotation of the flexed right hip * RLQ pain on jarring motion while standing landing
RLQ pain on jarring motion while standing landing
42
Describe how should a Physician illicit the following signs of appendicitis from the patient: McBurney’s sign
deep tenderness at 2/3 of imaginary line from umbilicus to right anterior superior iliac spine
43
Describe how should a Physician illicit the following signs of appendicitis from the patient: Psoas sign
RLQ pain with internal and external rotation of the flexed right hip
44
Congenital Anomalies of Pancreas: Agenesis
pancreas totally absent
45
Congenital Anomalies of Pancreas: Pancreas Divisum * unilocular space containing clear serous fluid * a ring of pancreatic tissue completely encircles the duodenum * normal pancreatic tissue in Meckel’s diverticulum * pancreas totally absent * fetal pancreatic primordial ducts fail to fuse * none of the above
fetal pancreatic primordial ducts fail to fuse
46
Congenital Anomalies of Pancreas: Annular Pancreas
a ring of pancreatic tissue completely encircles the duodenum
47
Congenital Anomalies of Pancreas: Ectopic Pancreas
normal pancreatic tissue in Meckel’s diverticulum
48
Congenital Anomalies of Pancreas: Congenital Cysts
unilocular space containing clear serous fluid
49
Identify the Type of Hernia as to Pathogenesis: Indirect Inguinal Hernia
failure of deep inguinal ring to close during embryogenesis
50
Identify the Type of Hernia as to Pathogenesis: Umbilical hernia
can affect adults, due to repeated abdominal strain