gingival diseases Flashcards

1
Q

What are the key features of the gingiva in children compared to adults?

A

More reddish, rounded/rolled margins, less stippling, flabbier texture, and greater sulcular depth.

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2
Q

How does the cementum differ in children from adults?

A

It is thinner and less dense in children.

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3
Q

What are the differences in the periodontal membrane between children and adults?

A

Wider periodontal membrane space, less fibrous, and greater blood and lymph supply.

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4
Q

How does the alveolar bone in children differ from that in adults?

A
  • Thinner lamina dura,
  • fewer trabeculations,
  • wider marrow spaces,
  • decreased mineralization,
  • flatter alveolar crests.
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5
Q

What color is the healthy gingiva in children?

A

More reddish due to thinner epithelium and greater vascularity.

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6
Q

What is the texture of healthy gingiva in children?

A

Flabbier and with less stippling compared to adults.

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7
Q

What is the sulcular depth in children compared to adults?

A

Greater sulcular depth in children.

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8
Q

What is an eruption cyst or hematoma?

A

A type of dentigerous cyst associated with erupting teeth, containing blood-stained fluid.

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9
Q

What is the clinical appearance of an eruption cyst?

A

Bluish fluctuant swelling over an erupting tooth, with color varying from normal to dark blue.

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10
Q

What is the treatment for an eruption cyst?

A

Typically unnecessary unless there is delayed eruption or excessive parental concern.

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11
Q

What is eruption gingivitis?

A

Temporary localized inflammation associated with tooth eruption, subsiding after the tooth emerges.

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12
Q

What is the primary treatment for eruption gingivitis?

A

Oral hygiene and topical anesthesia.

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13
Q

What is pericoronitis?

A

Acute inflammation of the gingival tissue covering an erupting tooth, often due to debris and bacterial accumulation under the operculum.

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14
Q

What are the clinical features of pericoronitis?

A

Red, swollen, painful operculum with purulent exudates and possible systemic symptoms in severe cases.

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15
Q

What is the treatment for pericoronitis?

A

Gentle debridement, warm saline mouthwash, antibiotics for severe cases, and surgical removal of the operculum if necessary.

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16
Q

What causes gingival problems associated with exfoliation of primary teeth?

A

Uneven root resorption, increased tooth mobility, and accumulation of debris.

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17
Q

What is the treatment for gingival problems during exfoliation?

A

Extraction of the primary tooth and improved oral hygiene.

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18
Q

What is the etiology of acute herpetic infection?

A

Caused by the herpes simplex virus, typically occurring before the age of five.

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19
Q

What are the systemic symptoms of acute herpetic infection?

A

Fever, malaise, irritability, anorexia, and lymph node involvement.

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20
Q

What are the oral manifestations of acute herpetic infection?

A

Fiery red gingiva, vesicles that rupture to form ulcers, and severe pain.

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21
Q

How is acute herpetic infection treated?

A

Supportive care including bed rest, analgesics, topical anesthetics, and high fluid intake.

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22
Q

What is the etiology of recurrent aphthous ulcers?

A

Unknown, but associated with stress, trauma, nutritional deficiencies, and autoimmune reactions.

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23
Q

What are the clinical features of minor recurrent aphthous ulcers?

A

Well-defined round or oval shallow ulcers with a gray-yellowish center and erythematous halo.

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24
Q

What is the treatment for recurrent aphthous ulcers?

A

Topical anesthetics, CHX mouthwash, and corticosteroids for severe cases.

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25
Q

What is ANUG?

A

An infectious disease caused by Borrelia vincenti and Fusiform bacilli, involving rapid gingival tissue destruction.

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26
Q

What are the clinical features of ANUG?

A

Punched-out papillae, pseudomembranous necrotic covering, bleeding, foul odor, and systemic symptoms in severe cases.

27
Q

What is the treatment for ANUG?

A

Removal of irritants, debridement with hydrogen peroxide, and systemic antibiotics for severe cases.

28
Q

What causes acute oral candidiasis?

A

Overgrowth of Candida albicans due to factors like antibiotic use or immunosuppression.

29
Q

What are the clinical features of oral candidiasis?

A

Raised white plaques that leave raw bleeding surfaces when removed, and potential extension to circumoral areas.

30
Q

What is the treatment for oral candidiasis?

A

Cessation of antibiotics, antifungal medications like Nystatin, and improved hygiene.

31
Q

How is acute bacterial gingivitis identified?

A

Bright red, painful gingiva that bleeds easily, requiring culture tests for confirmation.

32
Q

What is the treatment for acute bacterial gingivitis?

A

Broad-spectrum antibiotics if bacterial origin is confirmed.

33
Q

What are the main features of chronic gingivitis?

A

Bleeding on touch, redness, swelling, false pockets, and exudate.

34
Q

How is chronic gingivitis managed?

A

By removing local irritants, maintaining good oral hygiene, and educating patients.

35
Q

What is puberty gingival enlargement?

A

Gingival enlargement associated with hormonal changes during puberty, characterized by exaggerated tissue response to plaque.

36
Q

What is the clinical picture of puberty gingival enlargement?

A

Enlargement of interdental papillae, redness, bleeding on touch, and often confined to the anterior segment.

37
Q

What is the treatment for puberty gingival enlargement?

A

Removal of irritants, oral hygiene improvement, dietary recommendations, and surgical correction if severe.

38
Q

What is hereditary gingival fibromatosis?

A

A rare, slow-progressive benign enlargement of the gingiva, often of genetic origin.

39
Q

What is the clinical picture of hereditary gingival fibromatosis?

A

Firm, pale, and dense gingiva affecting free and attached portions, possibly causing malocclusion and delayed tooth eruption.

40
Q

What is the treatment for hereditary gingival fibromatosis?

A

Gingivectomy or complete removal of teeth in severe cases to prevent recurrence.

41
Q

What causes Dilantin-induced gingival enlargement?

A

Phenytoin therapy for epilepsy, affecting about 50% of users.

42
Q

What is the clinical picture of Dilantin-induced gingival enlargement?

A

Enlargement begins at the interdental papillae, spreading to the marginal gingiva and sometimes covering tooth surfaces.

43
Q

How is Dilantin-induced gingival enlargement managed?

A

Oral hygiene, chlorhexidine mouthwash, reducing plaque, and surgical removal in severe cases.

44
Q

What is ascorbic acid deficiency gingivitis?

A

Gingival inflammation caused by vitamin C deficiency, impairing collagen formation and increasing capillary fragility.

45
Q

What is the clinical picture of ascorbic acid deficiency gingivitis?

A

Swollen, bleeding gingiva with secondary infections and impaired wound healing.

46
Q

What is the treatment for ascorbic acid deficiency gingivitis?

A

Administration of vitamin C, dietary supplementation, and oral hygiene improvement.

47
Q

What are the primary features of aggressive periodontitis?

A

Rapid attachment and bone loss with familial aggregation, localized or generalized.

48
Q

What is localized aggressive periodontitis?

A

Rapid alveolar bone loss around first molars and incisors, often in healthy children.

49
Q

What is the treatment for aggressive periodontitis?

A

Oral hygiene instructions, mechanical removal of plaque, antimicrobial therapy, and frequent maintenance visits.

50
Q

What systemic diseases are associated with periodontitis in children?

A

Conditions like neutropenia, leukemias, Down syndrome, and Papillon-Lefèvre syndrome.

51
Q

What is Papillon-Lefèvre syndrome?

A

A genetic disorder causing severe gingival inflammation, hyperkeratosis of palms and soles, and early loss of both primary and permanent teeth.

52
Q

What is the periodontal presentation in children with Down syndrome?

A

Generalized early periodontitis, often affecting incisors and molars, with rapid progression and premature tooth loss.

53
Q

What is self-mutilation in children?

A

Intentional trauma to oral structures, often due to emotional instability or family tension.

54
Q

How is self-mutilation managed?

A

Identifying and addressing the underlying psychological causes.

55
Q

What factors contribute to gingival recession in children?

A

Toothbrush trauma, tooth prominence, orthodontic movement, oral habits, and periodontitis.

56
Q

How is gingival recession treated?

A

By eliminating predisposing factors and maintaining excellent oral hygiene.

57
Q

What causes green stains on children’s teeth?

A

Chromogenic bacteria acting on the enamel cuticle, commonly on maxillary anterior teeth.

58
Q

What causes orange stains on children’s teeth?

A

Unknown, but associated with poor oral hygiene and localized to the gingival third.

59
Q

What causes black stains on children’s teeth?

A

Chromogenic bacteria like Actinomyces, often forming a thin line along the gingival contour.

60
Q

How are extrinsic stains removed?

A

Polishing with rubber cup and pumice, and improving oral hygiene.

61
Q

What factors contribute to calculus formation in children?

A

Poor oral hygiene, soft diets, and abnormal salivary function.

62
Q

How does calculus in children differ from adults?

A

Less common in children and often associated with mental retardation or high saliva stagnation.

63
Q

What is the relationship between calculus and caries in children?

A

High calculus incidence is linked to a low caries rate.

64
Q

How is calculus managed in children?

A

Removal through scaling and polishing, coupled with improved oral hygiene practices.