GI Part 1 Flashcards

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1
Q

NCLEX: If one part of the GI is sick, then what?

A

Any other parts of the GI can too!

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2
Q

Exocrine vs. Endocrine functions

A

Exocrine: Digestive enzymes
Endocrine: Insulin

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3
Q

Acute pancreatitis vs Chronic pancreatitis CAUSES

A

Acute: Alcohol #1, gallbladder disease #2

Chronic: Alcohol

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4
Q

How is pancreatitis formed?

A

USUALLY enzymes moving from the pancreas move through the pancreatic duct into the small intestine

BUT HERE, something (gallstone, alcohol scar tissue) blocks the pancreatic duct so the enzymes activate in the pancreas and auto digest itself

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5
Q

S/S Pancreatitis

Pain in regards to eating?
Where is the fluid going? What's building up?
What does the abdomen look like?
Skin color?
GI?
Temperature?
BP?
A
Pain - increases with eating
Ascites (protein and blood build-up)
Rigid/board like or a mass d/t a swollen pancreas
Bruising or jaundice if liver involved
N/V
Fever (d/t inflammation)
Hypotension (d/t bleeding or ascites)

This shit hurts!

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6
Q

What kind of bruising so we see in pancreatitis?

A

Cullen’s sign - umbilicus

Gray Turners - flank (roll over to look for the blood pooling)

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7
Q

Why might a doctor do to assess if a pancreatitis patient is bleeding?

A

Gastric lavage - Pink fluid

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8
Q

What labs are we going to do to test for peritonitis?

*Most specific?
What does the pancreas make?
Inflammation?
Enzymes?
Liver problems?
Bleeding or dehydrated will show what?
A

Lipase*
Amylase
WBC ^
Blood sugar ^ (d/t low insulin from damaged pancreas)
AST, ALT ^ (liver hurt)
PT, aPTT longer d/t liver changes and decreased clotting
Serum bilirubin^
H&H (UP if dehydrates d/t low intake d/t pain or LOW if bleeding)

*If we are losing fluid (dehydrated), this causes our urine to be dilute - and our blood to be concentrated, thus higher H&H (think RBC per unit of volume - less volume=more RBC’s)

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9
Q

Normal Amylase

Normal Lipase

A

Amylase: 30-220
Lipase: 0-110

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10
Q

Key NCLEX strategy about treating the pancreatitis patient

A

Keep the stomach EMPTY and DRY

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11
Q

What medications do we use to treat pancreatitis?

Let's think about it......
Do they hurt?
What is their pancreas not making?
Isn't there inflammation?
Remember what NCLEX wants us to be?
Their stomach is getting it's acid neutralized, now what??
A

**Pain meds - opioids, PCA, patch

Steroids - decrease inflammation

Insulin - d/t altered pancreas, taking steroids, and TPN is packed with sugar

Anti-cholinergic’s - dry you up (atropine, Benztropine)

GI - PPI, H2 antagonists (‘dine’), Antacids

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12
Q

What to monitor in pancreatitis patient? - what are they at risk for?????

What can they NOT have?

How are we going to decrease stomach secretions?

What about their diet?

A

F/E balance
Daily weights
NO Alcohol (May need AA)

NPO, suction, bed rest, meds

Diet: NPO FIRST!!!!!!!!! THEN Ease into it, may need TPN

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13
Q

What are the 4 major functions of the liver?

A
  1. Detoxify the body
  2. Helps clot
  3. Metabolize drugs
  4. Synthesize (make) albumin
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14
Q

NCLEX TESTING STRATEGY

Sick liver #1 concern?
Sick liver regarding meds?
Sick sick never give what? What is it’s antidote?

A

BLEEDING

Decrease the dose

Acetaminophen - attacks liver
Acetylcysteine

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15
Q

AGAIN, what does toxin build up cause?

A

HA and malaise - that’s why we want this patient to rest because the toxins make them so tired

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16
Q

What is happening in the body when a patient has cirrhosis?

A

Liver cells are destroyed and replaced with connective and scar tissue which alters circulation within the liver, leading to portal HTN

Portal HTN and all it’s pressure can lead to the formation of varicies or hepatic coma

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17
Q

What are the S/S of cirrhosis?

Abdomen?
Pain?
GI?
Bowels?
Spleen?
Albumin?
AST/ALT?
RBC?
Ammonia? Ammonia acts like a what?
A

Firm, nodular liver - can feel it on R and!! -HUGE!)

Abd. pain (liver capsule stretched)
Chronic indigestion (dyspepsia)
Change in bowel habits
Ascites d/t LOW albumin
Splenomegaly - immune!!
AST/ALT ^
Anemia
Ammonia build-up can lead to hepatic encephalopathy/coma

Ammonia = sedative!!

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18
Q

How do we diagnose cirrhosis?

A

US, CT/MRI

Confirmation with a Liver biopsy

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19
Q

Liver Biopsy

Worry about what?
Labs pre-op?
Positioning during?
Breathing?

Positioning after?
*key thing to worry about

A

Needle puncture into sick liver
Worry about the puncture and bleeding!

PT, aPTT, INR, VS pre-op

Pre-op: supine with R arm behind head
Post-op: like on R side to hold pressure (may use a towel)

Exhale and hold to get the diaphragm out of the way

** Hemorrhage

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20
Q

Treatment for cirrhosis:

Meds
What do they need?
What to monitor?
What's sick liver skin like?
What's their abdomen doing?
Procedure

*What do we worry about when giving pain meds

A

Antacids
Vitamins
Diuretics
Pain meds - Like double the dose when the liver is sick!!!! Avoid narcotics!!

I/O, daily weight
REST!
*Bleeding precautions
Abdominal girth to Mx ascites 
Jaundice - itchy skin (cut nails, good skin care)

Paracentesis

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21
Q

Paracentesis –

What is it?
What to do before it?
Positioning?
*** Risk for what??

A

Removing peritoneal cavity fluid

Void - tiny bladder decreases risk of puncture

Sitting up - Fluid will settle in 1 spot

Watch for SHOCK!!!! Any time we are taking fluid from vascular system, you can throw them into shock
*Fluid is in the wrong space

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22
Q

Diet of a cirrhosis patient?

A

LOW Protein (Protein is broken down into ammonia and ammonia is already high!)

Low Na

NO alcohol

23
Q

How is protein handled in the liver?

A

Protein is broken down into ammonia

Liver convert ammonia to Urea

Kidneys excrete the Urea

24
Q

What is happening to the body during Hepatic Coma?

A

Too much ammonia is building up when the liver becomes so impaired and LOC is decreased

When the liver is sick, it can’t convert ammonia to urea

25
Q

What are the S/S of Hepatic Coma?

What could be the first sign?

A

LOC changes
Hard to waken

Asterixis - Liver flap - hand tremor = handwriting changes - might be the first sign

Decreased reflexes
Bradycardia
Ammonia breath (fetor)

26
Q

How do we treat Hepatic Coma? - Remember that they have a lot of ammonia in there blood!

Meds?
Way to get the blood out?
Diet?
Monitor what everyday?

A

*We want blood out of GI tract

Lactulose - decrease ammonia

Cleansing enema - get blood out of GI tract

Diet: LOW protein - increased protein will cause more ammonia because the liver can’t break down the ammonia bi-product of protein metabolism

Monitor Serum ammonia every day!

27
Q

Liver people tend to be what?

A

GI bleeders! Blood is protein! Increased ammonia! Blood causes increased ammonia!

28
Q

What causes Bleeding Esophageal Varices?

Who frequently has these when GI bleeding?

Are these problematic?

A

Portal HTN forces collateral circulation to form but it forms in the stomach, esophagus, and rectum. Portal HTN causes increase pressure in all of the GI tract

*Think of a bulging vessel like a hemorrhoid in the esophagus with pressure building behind it

Alcoholics

Become a problem when they rupture - sudden onset - bright red blood

29
Q

How do we treat Esophageal Varices?
THEY ARE BLEEDING!

They need what put on?
What do we need to replace?

Meds?

Fluids? How are we going to get rid of the blood?

Surgery?

A

Replace blood
Monitor CVP, VS
Give O2 - anemic

Octreotide
Lactulose

Cleansing enema - get rid of blood
Saline lavage - get blood out of stomach

Balloon Tamponade

30
Q

What does Octerotide do?

What are it’s SE?

A

Lowers BP in the liver

SE: Causes vasoconstriction in other parts of the body, but we don’t know where (May be coronary arteries)

31
Q

What is a Balloon Tamponade?

Keep what at bedside?

How long can we use it?

A

Tube is put in like an NG but blown up to put pressure on the bleed - 1 balloon in stomach ad 1 balloon in esophagus: Need to mark the tube!!!!!!

Keep scissors at the bedside because if they pull on the tube, it will obstruct the airway
- Getting a syringe would take too long!

Use no longer than 12 hours - usually an ED procedure

Blakemore tube

32
Q

Path of Peptic Ulcers

Common cause?
Where is it?
M or F more common?
What is present?

A

GI bleeding common cause

Esophagus, stomach, duodenum

Males

Erosion!

33
Q

S/S Peptic Ulcers

A

Burning pain usually in the mid-epigastric region

Heartburn (dyspepsia)

34
Q

What is a Gastroscopy? (EGD)

How are the patients pre-op?

Important thing to watch for?

A

Endoscopy through the oropharynx to view the Upper GI (esophagus, stomach, part of small intestine)

NPO (after until gag reflex)
Sedated

**Watch for perforation: pain, or trouble swallowing

35
Q

What is an Upper GI series?

Pre op?
NPO? avoid what?

A

Looks at the esophagus and stomach with dye

NPO past midnight

Avoid anything that increases stomach motility
NO gum/mints/smoking/patch

Smoking ^ motility AND increases stomach secretions putting the patient at risk for aspiration

36
Q

How to treat Peptic Ulcers?

Meds? (6)

A
Antacids
PPIs
H2 antagonists
Antibiotics if H. Pylori
GI cocktail
Sucralfate to form a barrier on the wound so acid can't get on it
37
Q

What kind and when should Peptic Ulcer patients take antacids?

A

Liquid form to coat the stomach

Take on an empty stomach at bedtime because having an empty stomach causes more acid to harm it

38
Q

What is the #1 cause of Peptic ulcers?

What do we need with this?

A

H. Pylori

Antibiotics

39
Q

What do we teach the patient with a Peptic Ulcer?

Decrease what?
Diet?
What to avoid?

How long to follow-up?

A

Decrease stress
Stop smoking

Eat what they can tolerate, but will want to avoid spicy foods and caffeine because they will irritate the stomach
Avoid temperature extremes

Follow-up for a year - takes a long time for an ulcer to heal

40
Q

Differences between Gastric ulcers and Duodenal ulcers?

Location?
Nourished?
Pain timing?
What helps the pain?
what doesn't help the pain?
Where is blood?
A
GASTRIC ULCERS
Higher up in GI system
Malnourished
Pain 30 min - 1hr after meals
Vomiting helps (malnourished)
Eating hurts
Blood in the vomit
DUODENAL ULCERS
Lower in GI system
Well-nourished
Pain 2-3hr after meals, nighttime
Vomiting doesn't help
Eating helps
Blood in stool
41
Q

When do we monitor CVP?

A

Esophageal Varices

42
Q

when do we have chronic indigestion?

A

Cirrhosis

43
Q

when do we have grey turners and cullens?

A

Pancreatitis

44
Q

Who do we give diuretics, vitamins, and antacids to?

A

Cirrhosis

45
Q

Where do we have burning pain and heart burn?

A

Peptic ulcers

46
Q

What does H.Pylori cause?

A

Peptic ulcers

47
Q

When are we going to give octreotide?

A

Esophageal Varcies

48
Q

When are we going to worry about bleeding and getting rid of the blood?

A

Hepatic coma

Esophageal Varicies

49
Q

Liver people tend to be what?

A

GI bleeders

50
Q

When are you going to avoid anything that causes motility?

A

GI series

51
Q

When do we use Sucralfate?

A

Peptic Ulcer

52
Q

When do we give GI cocktail?

A

Peptic Ulcer

53
Q

What does H Pylori cause?

A

Peptic Ulcer

54
Q

When are patients sedated?

A

Gastroscopy