General monitoring + ECG Flashcards

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1
Q

What is a monitor?

A

A device which measures an output , processes the information and displays it in a form that can be interpreted by the user.

may also include alarms which will sound when values deviate from the normal range

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2
Q

what is the minimum AABGI for general anaestheia?

A

core monitoring = ECG, pulse oximetry, NIBP

capnography

airway pressures and volumes and respiratory rate
Volatile anaesthetic gas monitoring and inspired oxygen concentration.
Airway pressure monitoring (if ventilated).

temperature if op >30mins
NMB monitoring when NMBA used.
depth of anaesthesia - TIVA
invasive BP/ CVP - critically il/ significant operation.

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3
Q

what is minimum AABGI monitoring for sedation and regional anaesthesia?

A

NIBP
Sats
ECG

for sedation need ETCO2 if loss of response to voice.

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4
Q

what is the minimum AABGI in recovery post op?

A

core monitoring
NIBP
Sats
ECG

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5
Q

how often should BMs be monitored in diabetics in general anaesthesia?

A

once hourly

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6
Q

how often does an anaesthetist need to record data on the anaesthetic chart?

A

every 5 mins for BP, HR and sats
every 15 mins for CO2, ventilatory parameters etc
more frequently if unstable.

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7
Q

what is the purpose of monitoring?

A

monitors allow us to measure clinical parameters and warn us of deviations from the norm so we can act on them.
they should supplement clinical assessment

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8
Q

what are the problems with monitoring?

A

over reliance - treating numbers vs clinical picture. distracts anaesthetists from the patient.

fault in equipment - incorrect medications given - give example of arterial line transducer being above patient and treating hypotension incorrectly.

complications from monitoring - interference and current leak, ecg stickers irritating skin etc

constant alarming - can become less significant

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9
Q

what types of biological potentials can be measured?

A

Tissues that produce action potentials and changes to membrane potential. There tissues produce currents as their membrane potential changes and action potentials transmit.

ECG - compound potentials in myocardium
EEG (electroencephalogram)- compound potential records from brain activity
EMG *electromyogram) - muscle membrane potential changes

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10
Q

what components are needed to measure biological potentials?

A

Electrodes - detection of potential - since this is already in form of current, no transducer is required

cables - to carry current to processor

amplifier and processor - amplification of signal and filtering out noise - often by common mode rejection and high/low pass filters

display unit - displayed on a screen or printed on paper.

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11
Q

can you describe how an ECG electrode works?

A

ECG electrodes constist of silver/silver chloride electrode in contact with conductive gel and surrounded by an adhesive mount.

chloride ions in gel in direct contact with skin can allow current to pass to silver/silverchloride electrode, which again transmits current through to wire. this gel improves contact hence reduces impedance and signal loss.
hence currents present at skin surface can be transmitted through the electrode to the wire.

surrounded by adhesive mount which sticks to skin and improves contact between electrode and skin.

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12
Q

what factors can reduce and improve contact with ecg electrodes

A

anything on the skin surface can reduce contact e.g. skin or water and greese.

hence skin should be shaved and cleaned and allowed to dry before electrodes attached.

positioning on bony prominences reduces artifacts from respiratory muscles

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13
Q

other than ECG monitoring, when else are electrodes used?

A

EEG and EMG monitoring

nerve stimulators in looking at effects of NMBA and also in diagnosis of neuromuscular diseases.

defibrillators

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14
Q

what are ECG cables ?

A

cables connect electrodes to processing unit / amplifier.

cables conduct currents

the cables are colour coded / labelled such that they are positioned correctly and the processor can display correct signal for each lead for interpretation of regional abnormalities.

can be 3 lead or 12 lead or 5 lead depending on indication.

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15
Q

Can you describe the set up of a 12 lead ECG

A

limb leads = 4
chest leads = 6

By different combinations of limb leads the electrical potential in different vectors of the myocardium can be recorded. e.g. chest leads record changes in the horizontal plane, directly beneath electrode. 1 recorded per electrode

limb lead combinations produce 6 vectors in the vertical plane through comparisons between the different leads with one another or a 0 point.

in total this makes 12 different leads
3 bipolar = compare signals between each of the leads e.g. Lead 1 compares signal between LA and RA
3 unipolar/ augmented = comparing limb leads to a neutral point e.g. aVF compares signal in LL to the 0 point of RA and LA (lead 1)

6 chest leads = record and display currents directly benefit hence in the horizontal plane

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16
Q

Describe the location of each of the 12 electrodes in a 12 lead ECG…

A

V1 = right side of sternum, 4th intercostal space
V2 = left side of sternum, 4th intercostal space
V4 = apex= 5th intercostal, mid clav line
V6 = 5th intercostal, mid axillary line

V3 and V4 inbetween the others

RA = red
LA = yellow
LL = green
RL = black

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17
Q

draw the einthovens triangle

A
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18
Q

what direction does depolarisation have to travel to create positive/ negative deflection?

A

positive = towards
negative = away from
right angle = biphasic

19
Q

what leads can help locate MI location?

A

aVL, lead 1, V5, V6 = lateral = circumflex artery

aVF, II and III = inferior, RCA

V1 - V2 = septal
V3- V4 = anterior

20
Q

what is the role of the black electrode - right leg?

A

acts as a differential filter - to reduce noise

21
Q

how is aVR calculated?

A

average of left arm and left leg electrode is measured

right arm electrode is then compared against this mid point.

22
Q

which ECG set up is used in anaesthesia? describe positions

A

3 bipolar leads
lead I, II and III

usually placed right arm(red), left arm(yellow) and left costal margin / left leg(green)

lead 2 is best at picking up arrythmias
not so good for ischaemic territories.

23
Q

other than 3 lead and 12 lead ECG , do you know any other ecg configurations?

A

CB5 - uses 5 electrodes, and better at viewing posterior heart. includes a central back electrode plus the other standard limb leads.

CM5 - placed closer towards manubrium. Red on manubrium (superior sternum), yellow 5th intercostal space mid clavicular line (this is known as V5 position). Green/ black right clavicle (C in the name refers to clavicle).
improves signal quality and better at diagnosing arrhythmias and MI. Switch to lead 1

posterior ECG - same but also V7,8,9 on back

24
Q

do you know any forms of invasive ECG monitoring?

A

oesophageal ECG - oesophageal electrodes used, good for atrial arrhythmias and posterior wall ischaemia

intracardiac - via pulmonary artery catheter. may be used in pacing

tracheal ECG - electrodes placed within tracheal tube - good for atrial arrhythmias

25
Q

what is the normal cardiac axis?

A

-30 to + 90mV

26
Q

describe the process of myocardial AP and currents within ecg leads

A

myocardial resting membrane potential is -90mV and rises to 30mV during an action potential. currents produced locally are large and result in AP trasmission throughout the myocardium and contraction of muscle cells.

these currents also dissipitate in other direction including towards the skin surface

however there is thoracic impedance along the way which will resist these currents and result in loss

by the time they reach the surface the record potential difference is only 1-2mV

ecg electrodes can record these changes in potential differences that occur at the surface of skin which correspond to larger changes within the myocardium

hence the ecg measures signals at skin that correspond to syndronised depolarisations of the myocardium

27
Q

how is a typical ECG calibrated?

A

25mm/second
10mm/ 1mV

each small square = 0.04seconds
5 small squares = 0.2seconds

28
Q

why is the p wave much smaller in amplitude than the QRS?

A

atrial tissue is less in mass than ventricles hence less current flows, less of a potential difference
p wave = atrial systole
QRS = ventricular

29
Q

how are ecg signals processed ?

A

amplification - electrical potentials at skin are only 1-2mV so need to be amplified for interpretation. ecg machine amplifies this - needs to have high GAIN

filtering out noise = high and low pass filters to remove frequencies of ranges that are not needed. in monitoring mode 0.5-40Hz included, in diagnostics 0.05 - 150Hz (more risk of noise)

Differential amplifier = common mode rejection - during amplification process, any signals common to all leads are removed and considered to be background noise.

analogue to digital conversion - Fouriers analysis.

30
Q

how does the frequency in diagnostic vs monitoring ecg vary?

A

0.5Hz -40Hz = monitoring
0.05Hz -150Hz = diagnostic

31
Q

how is the patient kept separate from the amplifier circuit?

A

plugged in at the wall
AC –> DC via transformer
the amplifier is in a closed circuit with this power source
Isolating transformer

patient is isolated and not part of this circuit

amplifier is earthed to allow stray currents to go via earth.

32
Q

what is an oscilloscope?

A

Device that measures and displays voltages overtime
continous display of ECG monitor

(Not the same as an oscilonometer which measures amplitude/freq of oscillations/vibrations)

33
Q

what might cause artifacts on an ECG trace?

A

electrodes
- dry gel, poor contact, incorrect placement

capacitance coupling
- 2 pieces of equiptment with charge come into contact and create a capacitor between them which results in interference e.g. cables/ monitors. reduced by copper screens around cables and common mode rejection

electromagnetic induction - current flowing through cable makes small magnetic field around it. if ecg cables pass through this can result in interferent. again can be reduced by shielding and common mode rejection

diathermy - can be reduced by high frequency filters but usually still effects ECG signal

patient - shivering and respiration (resp muscle potentials)

34
Q

give an overview of how the EEG works?

A

16 to 25 scalp electrodes
measure potentials at different points across the brain.

much smaller potentials 50uV due to impedance from skull, meninges and CSF

patterns of electrical activity can be recorded over time and analysed.

specific patterns correlate to certain situations e.g. B waves in awake patient frequency of 12Hz -25 hz

35
Q

give an overview of how the EMG works?

A

electrical potentials during skeletal muscle contraction recorded.
higher potential if more motor units stimulated.
can be up to 30mV
frequency 5-100Hz

36
Q

why is a chloride / silver chloride electrode chosen?

A

least likely to experience a change in electrical potential due to chloride ions in sweat

37
Q

what are the normal values for PR, QRS and QT intervals

A

PR -0.12 to 0.2s
QRS <0.12
QT - 0.35 to 0.45s

38
Q

how can the signal to noise ratio of an ECG be improved?

A

the electrical potential recorded is very small and electrodes will also pick up unwanted signals (noise)

a number of methods to reduce noise/ increase signal. we can think of these via the route the signals take during the monitoring process

electrodes: improved contact with the skin - increases signal. reduced shivering/movement reduces noise from respiratory muscles. or using bony prominence

cables - during the passage through cables they are at risk of capacitance coupling or electromagnetic interference. shielding cables can help reduce this

processor:
removing noise - common mode rejection and filters e.g. 50Hz will be common interference from mains and will be removed. high pass allows high frequency signals to pass.
amplification of signal - high gain. amplification can be either narrowband or wideband i.e. over narrow range of frequencies or wide range.

39
Q

how is capacitance coupling reduced?

A

shielding
increasing distance between electrical componetns

40
Q

compare the amplitude and frequencies measured by ECG, EEG and EMG..

A

ECG - 1-2mV, 0.05-150hz
EMG - 0.1 -1mV, 40-3000Hz
EEG - 10-50microV, 0.1-30Hz

41
Q

how can MRI affect ECG signal?

A

magnetic field can induce currents.
lead to noise / interference

can also induce currents in the wires and result in burns via the electrode

hence electrodes and wires must be carbon fibre (traditionally copper coating was used) also place the electrodes closer on the chest to maximise the amplitude of signals.

42
Q

what are the indications of EEG monitoring ?

A

in anaesthesia a modified version = BIS - indicated in TIVA to monitor level of anaesthesia

in medicine otherwise - diagnosis of seizures, status epilepticus in intubated patients

e.g. burst supression in barbiturate coma in traumatic brain injury

Prognostication in ITU

43
Q

tell me about the eeg waveform?

A

complex waveform
formed by different vectors from 16 scalp electrodes
picks up very small potential differences 10-50uV which must be amplified.

delta waves = 0 to 4 Hz deep sleep
theta waves = 4 to 8 Hz light sleep
Alpha waves = 8 to 12 Hz eyes closed
BEta waves = 12 to 25 Hz awake

donalds four theives ate 12 burgers

44
Q

what is the difference between active and passive transducer?

A

active = generate current in response to stimulation e.g. piezoelectric and thermocouples

passive = change in property e.g. resistance will alter already existing current from an external source e.g. thermistor and strain gauge.