Many GI abnormalities originate when?
During fetal life; they may or may not be detected during the neonatal period.
When obtaining a history for most GI conditions, it is important to obtain information based on:
food habits (what is eaten, how it is prepared, how much is eaten, when does eating occur and with whom), vitamins and nutritional supplements that may be used, and normal bowel habits, including frequency, appearance, and the use of aids, such as laxatives or enemas.
How much does the stomach hold?
The newborn stomach expands from 20 mL at birth as much as 90 mL by the end of the first week of life, the stomach holds up to 3,000 mL by late adolescence.
How is the infant’s gut different during the first year of life?
It is immature; some products cannot be digested and are excreted in the stool, whereas other products may be absorbed more readily, possibly leading to the development of food allergy.
Vomiting
Introduction
Vomiting
Assessment
Vomiting
Interventions
Gastroesophageal Reflux (GER)
Introduction
1. GER is the return of gastric contents into the esophagus from an incompetent or poorly developed esophageal sphincter.
2. The condition occurs almost immediately after eating and typically affects infants; it is also seen in infants and young children with spastic cerebral palsy and other conditions with decreased muscle tone.
3. Aspiriation puts them at risk for pneumonia.
Assessment
1. Assess the relationship of vomiting to feedings, positioning, and the activity level immediately after feedings.
2. Assess for failure to thrive/poor weight gain.
3. Assess for aspiration of feedings; note any relationship between apnea and GER (many infants may be placed on apnea monitors).
4. Measure the pH of gastric contents.
Interventions
1. Admin meds 30 minutes before meals to maximize the benefits of the medications.
a. H2 blockers and proton pump inhibitors are drugs used to reduce the amount of gastric acid present in gastric contents, thereby reducing esophagitis.
2. Provide thickened formula (1 tbsp cereal/1 oz formula); will need to enarge hole in nipple or use cross-cut nipple.
3. Feed child in an upright position and keep upright for 30 minutes after feed; avoid car seat sitting position.
4. Give small frequent feedings.
5. Research supports placement of infant with GER on their backs to sleep, as there is no increased risk for aspiration.
6. Prepare for surgery (Nissen fundoplication for hiatal hernia) if necessary.
Diarrhea
Introduction
What is the most common cause of diarrhea in infants?
Rotavirus
Difficile infection symptoms
fever, abdominal pain, and often bloody diarrhea
Diarrhea
Assessment
Diarrhea
Interventions
Constipation
Introduction
1. Constipation is a decreased amount and increased consistency of stool compared to the patient’s normal pattern (children do not need to have a daily bowel movement).
2. Constipation is not necessarily synonymous with straining.
3. It may be caused by diet low in liquids or high in fat and protein.
Assessment
1. Note a hard, dry, infrequent stool.
2. Test stool for blood (guaiac test). Melena is dark, tarry stool usually associated with bleeding from the upper GI tract; it may be Hematest-positive from trauma to the rectal tissue from passing a hard mass; assess skin integrity around anus.
3. Ask child about abdominal pain during stooling or intermittent pain throughout the day.
4. Assess the child’s diet for liquids, fiber, carbohydrates, and constipating foods.
5. Measure abdominal girth.
6. Note whether child is reluctant to use the toilet in school or is consciously retaining stool.
7. May present as encopresis/fecal incontinence with involuntary soiling of clothing as impacted stool collects in colon and rectum; may appear as diarrhea as only liquid leaks around the retained stool.
a. Usually occurs after age 4
b. May be due to emotional issues or fear of using toilet and pain
Interventions
1. Lubricate around the anus to ease the passage of hard stool (may have to remove stool digitally).
2. Administer stool softeners: suppositories, mineral oil, and docusate sodium (Colace).
3. Add fiber or prune juice to the diet; increase fluid intake.
4. Administer enemas, if ordered; use isotonic solutions only.
Cleft Lip and Palate
Introduction
Cleft Lip and Palate
Assessment
Cleft Lip and Palate
Interventions for cleft lip (preoperative)
Cheiloplasty
Interventions for postoperative cleft lip repair
CLEFT LIP REPAIR
1. Cheiloplasty unites the lip and gum edges in anticipation of teeth eruption, provides a route for nuturition and sucking, and improves the infant’s appearance, which promotes parent-child bonding.
2. Maintain a patent airway; edema or narrowing of a previously large airway may make the infant appear to be in distress.
3. Observe for cyanosis, as the infant begins to breathe through the nose.
4. Maintain an intact suture line; keep the infant’s hands away from the mouth by using elbow restraints or swaddling the infant with a blanket; Steri-Strips will be used to hold the suture line in place.
5. Prevent tension on the suture line by anticipating the infant’s needs and preventing crying; do not place in prone position to avoid pressure on face.
6. Give extra care and support, because the infant cannot meet emotional needs by sucking.
7. When feeding resumes, use a special cleft lip feeder (e.g., a Haberman feeder) so they may drink from a bottle or a syringe with tubing to administer foods at the side of the mouth; this will prevent trauma to the suture line.
8. After feedings, place infant on right side to prevent aspiration.
9. To prevent crusts and scarring, clean suture line after each feeding with warm water using a Q-Tip and apply antibiotic ointment (Neosporin).
10. Monitor and treat for pain.
Staphylorrhaphy
Interventions for cleft palate (preoperative)
CLEFT PALATE REPAIR
1. Be aware that the infant must be weaned from the bottle or breast before cleft palate surgery; the infant must be able to drink from a cup.
2. Feed infant with a cleft palate nipple or a Teflon implant to enhance nutritional intake.
3. Surgery is usually scheduled before age 1 year to allow for the growth of the palate and before the infant develops speech patterns to prevent speech delays; the infant must be free from ear and respiratory infections.
4. Teach the parents that the infant is susceptible to pathogens and otitis media from the altered position of the Eustachian tube.
Staphylorrhaphy
Interventions for cleft palate (postoperative)
CLEFT PALATE REPAIR
1. Maintain a patent airway; position infant on the abdomen or side.
2. Anticipate edema and a decreased airway from palate closure; this may make the infant appear temporarily dyspneic.
3. Prevent trauma to the suture line by keeping hard or pointed objects (utensils, straws, frozen dessert sticks) away from the mouth; do not use suction catheters, except in emergency (use bulb syringe if necessary).
4. Use a cup to feed; do not use a nipple or pacifier.
5. Use elbow restraints to keep the child’s hands out of the mouth or have parent hold child in shuch a way as to pregent hands going in mouth.
6. Provide soft toys.
7. Start the child on clear liquids and progress to a soft diet; rinse the suture line by giving child a sip of water after each feeding.
8. Distract or hold child to try to keep the tongue away from the roof of the mouth.
Esophageal Atresia (EA) and Tracheoesophageal Fistula (TEF)
Introduction
1. An atresia is the termination of a passageway; it usually refers to a pathologic closure or the absence of a normal anatomic opening.
2. A fistula is a tubelike connection between two structures.
3. These conditions occur alone or in many combinations; they may be associated with other congenital defects, especially cardiac defects; TEF may be part of a syndrome called VACTERL (Vertebral defects, Anorectal malformations, Cardiac anomalies, TEF, EA, Renal defects, and Limb defects).
4. EA occurs when the proximal end of the esophagus ends in a blind pouch; food cannot enter the stomach via the esophagus.
5. TEF occurs when there is a connection between the esophagus and the trachea.
6. TEF may result in the reflux of gastric juice after feeding that can allow acidic stomach contents to cross the fistula, irritating the trachea.
7. EA with TEF occurs when either the distal end of the esophagus ends in a blind pouch and the proximal end of the esophagus is linked to the trachea via a fistula or when the proximal end of the esophagus ends in a blind pouch and the distal portion of the esophagus is connected to the trachea via a fistula; EA with TEF is the most common of these defects with EA alone being second most common.
Assessment of TEF
1. Maternal history of polyhydraminos (excess amniotic fluid, since fetus is unable to swallow it).
2. Newborn may have excessive oral secretions.
3. Assess for choking, coughing, and intermittent cyanosis during feeding from formula that goes through the fistula into the trachea; stop feeding if this occurs.
4. Observe for abdominal distention from air that goes through the fistula into the stomach.
5. Observe for tracheal irritation from gastric acids that reflux across the fistula.
Assessment of EA with TEF
1. Assess for all signs and symptoms of EA.
2. Observe for signs of respiratory distress: coughing, choking, and intermittent cyanosis.
Interventions
1. If EA or TEF is suspected, do not feed orally; a percutaneous endoscopic gastrostomy (PEG) tube may be inserted; keep the tube open and suspended above the child for release of gas.
2. Maintain a patent airway; have suction equipment available.
3. If feeding the child via gastrostomy tube after surgery, anticipate abdominal distention from air; keep the child upright during feedings to reduce the chance to regurgitated stomach contents and aspiration pneumonia, and keep the tub open and elevated before and after feedings.
4. Gastrostomy feeds should be administered either by gravity or by feeding pump.
5. Explain to parents how the surgery ligates the TEF and reanastomoses the esophageal ends; expect a staged repair in many instances.
Pyloric Stenosis
Introduction
1. Pyloric stenosis is an increasing hyperplasia and hypertrophy of the circular muscle at the pylorus, which narrows the pyloric canal as it exits the stomach.
2. The defect is most commonly seen in boys around 4 weeks of life.
3. Emesis may be Hematest positive, but it will not contain bile; it will increase in amount and force as the obstruction increases and will become projectile.
Assessment
1. Be aware that symptoms seldom appear during the first two weeks of life.
2. May palpate for an olive-size bulge below the right costal margin.
3. May observe for projectile emesis during or shortly after feedings (preceded by reverse peristaltic waves [going left to right] but not by nausea).
4. Note that the child will resume eating after vomiting.
5. Evaluate for poor weight gain and symptoms of malnutrition despite the child’s apparent hunger.
6. Assess for metabolic alkalosis and dehydration from frequent emesis.
Interventions
1. Provide small, frequent feedings with the head of the bed elevated; burp the child frequently.
2. Position the child to prevent the aspiration of vomitus, preferably on the right side for children and on their backs for infants with the head of the bed raised 30 degrees.
3. Correct any electrolyte imbalance.
4. An NG tube may be inserted and kept open and elevated for gastric decompression.
5. Prepare for pyloromyotomy, where the overdeveloped muscle around the pyloric valve is split, thereby spreading open the muscle and enlarging the pylorus to relieve the obstruction.
Intussusception
Introduction
1. **Is the telescoping or invagination of a bowel segment into itself, the most common site being the ileocecal valve; it usually occurs between 3 and 24 months. **
2. The condition may result from polyps, hyperactive peristalsis, an abnormal bowel lining, or for no known cause.
3. Initially, it causes inflammation and swelling at the affected site; edema eventually causes obstruction and necrosis from occlusion of the blood supply to the bowel.
4. Intussusception can be acute or chronic. Untreated, it may lead to peritonitis.
Assessment
1. Note a sudden attack of acute abdominal pain in a previously well child; the child shrieks and draws the knees to the chest.
2. Observe for an increase in bile-stained vomitus.
3. Note the passage of a red currant jelly-like stool (red blood and mucus).
4. Assess for a distended and tender abdomen.
5. Note pallor and agitation.
Interventions
1. Prepare for a barium enema or water-soluble contrast with air pressure to confirm the condition and reduce the invagination by hydrostatic pressure.
2. Be aware that, if enemas fail to reduce the invagination, surgery may be required to resect the gangrenous portion; a temporary colostomy may be necessary.
Hirschsprung Disease
Congenital Aganlionic Megacolon
Introduction
1. Parasympathetic ganglionic cells are absent in a segment of the colon (usually at the distal end of the large intestines); the lack of nerve innervation causes a lack of or alteration in peristalsis in the affected part.
2. As stool enters the affected part, it remains there until additional stool pushes it through; the affected part of the colon dialates; a mechanical obstruction may result.
Assessment
1. Observe for liquid or ribbon-like stools; only fluid can pass the obstruction caused by stool.
2. Assess for a distended abdomen from stool impaction.
3. Assess for nausea, vomitus, anorexia, lethargy, weight loss, and failure to thrive.
4. Prepare for barium studies and a biopsy of the large intestines to confirm condition.
5. Be alert for signs and symptoms of enterocolitis, volvulus, and shock, which can occur from this condition.
Interventions
1. Surgery to bypass the affected area or a resection of the aganglionic portion may be done with or without a colostomy