Final Exam - new stuff Flashcards
orthosis positioning
Safe position / antideformity position PIP/DIP extension 0 MCP joint flexion at least 70 Wrist extension 20-30 Thumb in palmar abduction
Functional positioning / resting Wrist 10-15 extension MCP joints 15-20 flexion PIPs flexion 20-25 DIPs flexion 10
Consider bony, nerve, and venous anatomy
Incorporate arches
⅔ of the way down the forearm
What is the difference between dynamic, static progressive and serial casting for mobilization orthosis fabrication?
Static: Primarily to support, stabilize, protect, immobilize, prevent contractures, modify tone
Serial static: Splint is remolded or replaced to lengthen tissues, restores mobility
Static progressive: Lengthens tissues via a non-dynamic component (Velcro, turnbuckle), restores mobility. Uses a low load force
Dynamic: Has moving parts (elastic component) to permit, control, or restore movement, can act as a substitute for weak muscles
high risk groups for cumulative trauma disorders (CTD’s)
New on the job w/ no major previous exposure to repetitive tasks
After a break from this type of activity (vacation or sick leave)
Older workers (35-55 yo)
Gender, females are 10 x more common than men
Type of work, ex: manufacturing could lead to shoulder pain
commonly used CTD terms
Repetitive strain injury (RSI), occupational cervico-brachial disorder, overuse syndrome, work related disorders, repetitive trauma disorders, regional musculo-skeletal disorders
characteristics of CTD and be able to determine the presence of CTD’s
Condition in soft tissue structures (tendons, tendon sheaths (tenosynovitis), nerves, muscles or blood vessels, trigger points, myofascial pain, bursitis)
Cause or accelerated by repeated stresses and/or awkward motions
Not caused by a single incident (sprain is a single incident)
Each work activity has the potential to cause micro tears in soft tissue structures
Criteria to determine CTD in the workplace can be tendon or nerve related or both*
Symptoms: pain, numbness, tingling; symptoms lasting more than 1 week and/or occuring more than 20 x in the last year; no evidence of acute traumatic onset; no evidence of systemic disease; onset of symptoms occurring with present job
levels of CTD
Level 1 - most severe
Level 2
Level 3 - least severe
appropriate treatment for CTD conditions
Management of osis or itis: anti-inflammatory medications; NSAID, corticosteroid
Patient education, activity modification, stretches, splint, icing, proximal exercises to strengthen surrounding muscles, regenerative injections, adhering to restrictions
Rotator cuff tendonitis: NO overhead reaching
Short term treatment strategies:
Goal central sensitization
Acute/sub-acute denervation or peripheral
Goal for muscular or ligament strains
Work on posture and activity modification
What is BTE? How is it used?
Used for both assessment (strength and endurance) and treatment (work simulation)
ADLs and IADLs
Can be used in ortho, industrial, and neuro settings
Has various settings for: isotonic (eccentric and concentric), isometric, isokinetic (speed remain the same, but resistance can change), PROM, active assisted ROM
How is work defined when using BTE
Work = Force x Distance
Power = work/time
In relation to BTE = resistance, repetitions, time
What patient populations would the BTE be appropriate for
Evals: work capacity eval
Treatment: acute or work conditioning
Ergonomic analysis
How: torque control, ratchet switch, height adjustment, work head adjustment, work head position, dynamic and static
Can assess: manual dexterity, handling/grasping, ROM, strength, endurance, effects of vibration, effects of repetitions, work tolerance
Do NOT use for pts with degenerative neurological conditions
Identify CTDs
Condition in soft tissue structures (tendons, tendon sheaths (tenosynovitis), nerves, muscles or blood vessels, trigger points, myofascial pain, bursitis)
Cause or accelerated by repeated stresses and/or awkward motions
Not caused by a single incident (sprain is a single incident)
Each work activity has the potential to cause micro tears in soft tissue structures
Symptoms of carpal tunnel
Pain, tingling, & numbness in the thumb, index, middle, & radial side of ring finger
Tingling in median nerve distribution
Symptoms are worse at night
Hands feel weak in the morning
Pts drops objects more than usual
Difficulty grasping or pinching objects
Difficulty with tasks requiring dexterity, coordination & strength, such as buttoning a shirt, writing with a pen, or opening a jar lid
The muscles at the base of your thumb are smaller and weaker than they used to be.
conservative treatment for carpal tunnel
Splinting; for positioning when sleeping & in the daytime splint if the symptoms persist during ADL activities. Splinting reduces swelling. Can be off the shelf (less effective, but less restrictive) or custom made
Medications, injections of corticosteroids
Ergonomics: rest wrist and hands from repetitive activity intermittently, alternate tasks to reduce pressure on the wrist, delegate tasks that aggravate to co workers or family members, modify and change daily activities that aggravate
Patient education
surgical management carpal tunnel
open procedure
endoscopic
causes of carpal tunnel
- Entrapment of median nerve
- Injuries
- Arthritis
- Work activities and hobbies
- Associated conditions: pregnancy, birth control, diabetes, thyroid disease, amyloidosis
What is occupational therapy’s role with the management of patients with Lymphedema?
Maintenance: compression sleeve, compression bandaging, self massage, exercise, skin care, prevention and risk guidelines, psychological health promotion
What is the patient’s responsibility when managing Lymphedema?
Pt needs to buy in and know it is not a quick fix; it will require a permanent lifestyle change to manage/reduce the lymphedema
Maintenance and self-management: compression sleeve, compression bandaging, self-massage, exercise, skin care, follow prevention and risk guidelines, watch for cellulitis/ scratches
What are the different types of Lymphedema?
Primary Lymphedema
Lymphedema Congenitum: clinically present at birth (10-15% of primary lymp.)
Lymphedema Praecox: present w/ onset of puberty (75-80%)
Lymphedema Tardum: appears after 35 yrs (15-20%)
Secondary Lymphedema: cancer treatment #1 cause of lymphedema in the US; trauma/burn, infection, scar tissue formation, lymphatic system blockage, chronic venous insufficiency, filariasis (#1 cause worldwide) , radiation therapy