Feeding of goats Flashcards

1
Q

Breeds of goats

A

Dairy: Alpine, Anglo-Nubian, Saanen
Meat: Boer, Hungarian imperial

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2
Q

Digestive physiology of goats

A
  • better adapted to environmental conditions than sheep and cattle
  • more selective eaters; browsers, prefer shrubs, tree leaves (80%)
  • have high tolerance towards bitter taste as well as tannins (in tree leaves)
  • DMI capacity is higher - 2-5% BW
  • larger volume of rumen
  • dense papillae in rumen —> huge surface area —> efficient absorption of VFAs
  • higher number of cellulolytic bacteria
  • have higher level of ammonia (due to higher DMI)
  • higher level of microbial protein synthesis
  • longer retention time —> netter digestibility
  • high amount of saliva secreted —> increased fibre fermentation
  • intensive urea recycling
  • water turnover is unique: water intake is low, goats can tolerate absolute water deprivation, concentrated urine
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3
Q

Breeding cycle of goats

A
  • BW: 60-100 kg
  • pregnancy: ~150 days
  • lactation: 8,5 - 10 months (dairy goats: 900 days!)
  • dry period: 2 - 3,5 months
  • chemical composition of milk is close to cow’s but better (more essential AAs)
  • daily milk yields is ~4 kg/day
  • ~1000kg of milk per lactation
  • mating: autumn (Aug-Sep)
  • calving: Jan-Feb
  • seasonal breeders
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4
Q

Expression of energy and protein requirements for goats

A
  • NE l
  • MP
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5
Q

BCS, flushing

A
  • desirable BCS 3
  • flushing is not as common as in ewes, done if BCS «< 3
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6
Q

Feeding of dairy goats. Gestation without lactation

A
  • first 15 weeks in first pregnancy and mid-pregnancy (6-15 weeks) in later pregnancies
  • maintenance level
  • pasture, forages/roughages
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7
Q

Feeding of dairy goats. Late gestation and lactation

A
  • last 4-6 weeks of gestation
  • high energy and nutrients demand
  • forages/roughages + concentrates (2,7-3 kg/day, twice at milking)
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8
Q

Feeding of breeding bucks

A
  1. In breeding season: pasture or forages + concentrates (oats) (1-1,5 kg)+ Zn, Se supplementation
  2. Outside of breeding season: pastures/forages + concentrates (0,5 kg)
    - **NE m, MP
    - BCS: 3
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9
Q

Feeding of kids (goatlings)

A
  • birth BW: 2-4 kg
  • colostrum! (In dairy: bootle feeding); 10% of BW
  • after colostrum period, on the 2nd day of life kids are separated from life, milk replacer is fed
  • milk replacer (25% CP, 28% EE)
  • from 7 days creep feeds are introduced: starter diet/ oats, alfalfa hay, straw ad lib
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10
Q

Feeding of young goats

A
  • weaning at 6 weeks with BW of 12 kg
  • mating: 7-12 months, BW ≥ 35 kg
  • NO maize because rapid fermentation may lead to problems
  • goat diet, concentrates (barley/oats/sorghum)
  • alfalfa ad lib
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11
Q

Nutritional disorders in sheep and goats

A
  • pregnancy toxaemia
  • hypocalcemia
  • enterotoxaemia
  • poliencephalomalacia
  • white muscle disease
  • urolithiasis
  • cobalt deficiency
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12
Q

Pregnancy toxaemia

A
  • common disorder of ewes and does
  • condition when drastically low level of blood glucose damages the brain, causes hydration, renal failure and results in death
  • occurs in late gestation (last 4-6 weeks)
  • caused by inadequate energy intake (energy demand is increased a lot because of tense foetal growth) —> fat is broken down —> ketone bodies are produced
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13
Q

Which dams are most at risk of pregnancy toxaemia?

A
  • late pregnancy with multiple foetus
  • fat animals!!
  • feed intake is decreased ( foetal pressure, decreased rumen volume)
  • grazed on pastures low in nutrients
  • diseases (foot rot, worm infestation)
  • extreme weather condition
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14
Q

Signs of pregnancy toxaemia

A
  • decreased feed intake
  • decreased rumen motility
  • nervous system signs: depression, tremors, etc
  • laying down for days and eventually will die (3-4 days)
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15
Q

Chow can pregnancy toxaemia be treated?

A
  • glucose administration
  • good quality hays and oats
  • inducing parturition / c-section
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16
Q

How can pregnancy toxaemia be prevented?

A
  • maintaining BCS
  • pregnancy scanning (for twins, triplets), should be fed accordingly
  • supplementary grains in last weeks of pregnancy
17
Q

Hypocalcemia in sheep and goats

A
  • very similar to dairy cows
  • calcium is low, muscular contractility is bad
  • either inadequate Ca intake (grazing cereal regrowth; consuming feed with high oxalate level) or inability to metabolise/absorb calcium
18
Q

What sheep and goat are most at risk of hypocalcemia?

A
  • big difference with cattle: hypocalcemia can develop before lactation, in late pregnancy
  • older dams!
  • multiple foetuses
19
Q

Signs of hypocalcemia in sheep and goats

A
  • rapid onset
  • weakness
  • decreased rumen motility
  • stiff, uncoordinated movement movements
  • muscle trembling (goats!)
  • inability to stand up
  • vaginal prolapse (fatty animals)
  • death (24 hours)
20
Q

How to treat and prevent hypocalcemia

A

Treatment:
- Calcium injections (rapid recovery; repeated treatment)
- rapid recovery is important because that’s what is gonna distinguish hypocalcemia and pregnancy toxaemia)

Prevention:
- adequate dietary Ca throughout gestation
- Ca : P = 2 : 1

Diseases that look like pregnancy toxaemia and hypocalcemia: acidosis, cerebral abscesses, scrapie, rabies

21
Q

Most important about pregnancy toxaemia and hypocalcemia

A
  • similar signs: decreased rumen motility, depression, in necropsy: yellow liver in toxaemia, no such sign in hypocalcemia
  • causes are different: low levels of glucose vs low levels of calcium; therefore treatments are different
  • prevention: feeding managements
22
Q

Enterotoxaemia

A

= overeating disease = pulpy kidney disease
- more frequent in sheep
- any age but most common in lambs
- causative agent is part of normal intestinal microflora
- background: diet rich in carbohydrates —> overgrowth of Clostridium perfrigens type D —> toxin —> increasing vascular permeability (in brain as well)

23
Q

Clinical signs of Tupi D enterotoxaemia

A
  • sudden death of animals with good BCS
  • pushing head against fixed objectives
  • lesions: edema and malacia in cerebellum; autolysis of kidneys
24
Q

How to diagnose and control type D enterotoxaemia?

A

Diagnose: sudden death and special toxin in small intestinal fluid

Control:
- immunization
- feeding highly fermentable CHs in small meals with additional feeds

25
Q

Polyencephalomalacia. Causes

A

= thiamine (B1) deficiency = CCN (corticocerebral necrosis)
- fattening lambs and kids
- etiology: similar to enterotoxaemia:
- high CH diet —> higher demand for vit B1
- excess of CH —> decrease of rumen pH —> more thiaminase-producing bacteria
- sulphate (molasses, CGF) will inhibit thiamine utilisation in rumen
- some weeds contain thiaminase

26
Q

Polyencephalomalacia. Clinical signs. Treatment

A
  • decreased appetite
  • recumbency, stargazing, spasms
  • death

Treatment: thiamine therapy, increase of forage intake

27
Q

White muscle disease (Se and vit E def)

A
  • sheep > goat, young animals are most affected by
  • acute degeneration of cardiac and skeletal muscles
  • soils low in Se!!!
28
Q

Clinical signs of white muscle disease. Treatment

A
  • stiffness
  • arched back
  • cardiac arrhythmia
  • respiratory distress
  • AST and LDH increase
  • death or animal can respond favourably
  • white striations in necropsy

Selenium and vitamin E supplementation on salt lick

29
Q

Urolithiasis

A
  • round sediments of different diameter of protein and minerals
  • castrated and intact rams/bucks
  • excess protein, P and Ca are excreted
  • feeding excess amount of cereal (P)
  • affected animals have alkaline urine
  • struvite calculi (NH4MgPO4)

Why is more common in males?
Narrow urethra

30
Q

Urolithiasis. Clinical signs. Treatment. Prevention

A
  • painful urination
  • kicking the area of the penis
  • lethargy
  • decreased appetite
  • bladder rupture
  • death

Treatment: pain management, NH4Cl to acidify urine, water ad lib to dilute urine

Prevention: optimum Ca : P ratio at least 2 : 1; hays instead of cereals (alfalfa)

31
Q

Cobalt deficiency

A

= white liver disease = pine
- cobalt is a component of vitamin B12 —> important for propionate metabolism
- weaned lambs
- late summer/autumn
- cause of cobalt deficiency: soil low in cobalt
- signs: poor growth rate, lethargy, poor appetite, immunosuppression, anaemia
- necropsy: liver is pale, swollen, fatty

  • cobalt supplemented salt lick. Legumes > cereals —> mixed pastures!!