B10: Clinical Dietetics Of Beef Cattle Flashcards
1
Q
Clinical dietetics of beef cattle. Key points
A
- focus on prevention because treatment is difficult as these animals are kept in pastures
- antiparasitic treatment! - anemia, malnutrition, reduced BW gain
2
Q
Most important nutritional disorders of beef cattle
A
- “Washy pastures”
- Nutritional deficiencies
- Urea toxicity
- Nitrate/nitrite toxicity
- Heat stress
- Urolithiasis
- Sand impaction
- Bloat
- Acidosis, rumenitis, liver abscesses
- Hardwarre disease
- Hydrocyanic acid toxicity
- Others (phytoestrogens, poisonous plants)
3
Q
“Washy pasture”
A
- early spring
- grasses: high moisture and protein content —> diarrhea
- prevention: transition period for 2 weeks, increasing DMI by feeding hays and straw
4
Q
Nutritional deficiencies. Vitamin A deficiency
A
- antioxidant —> in their deficiency protection of skin and MMs decreases —> higher chance of infections, decreased absorption of nutrients
- reproduction is negatively affected: decreased quality of sperm and eggs, decreased progesterone production
- deceased calf’s growth and bone development (should be supplemented in late pregnancy)
- vitamin A deficiency is really rare in pastures! but excess of sun can destroy vit A
- to prevent deficiency: carrots as supplementation
5
Q
Nutritional deficiencies. Magnesium deficiency = grass tetany
A
- lactating cattle !!
- early spring: lush perennial grasses are low in magnesium
- clinical signs: hyper-excitability, muscle twitching, muscle spasms, collapse, death
- grass-legume mixed pasture should be grazed first!
- ## salt blocks !, as salt can support magnesiumtransport into the cells and its uptake
6
Q
Nutritional deficiencies. Vitamin E and Selenium deficiency = white muscle disease
A
- in calves: muscular dystrophy. If cardiac muscle affected -> sudden death. If skeletal -> slow onset, difficulty swallowing (tongue)
- in adults: impaired immune responses
- muscles are pale
7
Q
Nutritional deficiencies. Calcium and vitamin D
A
- quite rare
- happens in intensive fattening animals because excess grains are fed with low calcium content
- breaking down the heel bone (os calcaneus) together with Achilles tendon
- lameness
8
Q
Nutritional deficiencies. Thiamine (vit B1) deficiency
A
- CCN: corticocerebral necrosis (=polyencephalomalacia, PEM)
- in intensive fattening
- more grains are fed -> higher demand of B1 (Krebs cycle)
- also more grains in diet decrease rumen pH -> proliferation of thiaminase producing bacteria
- molasses, corn gluten -> increase of phosphate content -> decrease thiamine utilisation
- clinical signs: circling head, convulsion
9
Q
Nutritional deficiencies. Copper deficiency
A
- rare: either soil is low in copper or other minerals inhibit copper utilisation (Mo, Fe, S)
- hair coat will be pale
10
Q
Urea toxicity
A
- urea supplementation (often in beef cattle) (protein balance!)
- urea supplementation can be done when protein balance is NEGATIVE
- recently is rare because usually urea is supplied together with molasses for good utilisation
- in case of urea toxicity -> ammonia toxicity (salivation, convulsion, in coordination, death)
- treatment: adding vinegar
11
Q
Nitrate/nitrite toxicity
A
- after fertilisation of soil -> water contamination
- in rumen nitrate is converted to nitrite (more toxic)
- in case of drought fertilisers are not utilised by plants, they stay on plants -> consumption
- clinical symptoms: DMI decreases, abortion, methaemoglobinaemia in calves
12
Q
Heat stress
A
- clinical signs: DMI significantly lower