facial pain Flashcards
facial pain causes
dental cause vascular infective -sinitis sinogenic - better wth abx injury migraine cluster headache
mumps
siatholasis
paradoitis
CAD Sinus disease Dental caries/abscess/cyst Salivary gland disorder such as sialolithiasis, mumps, parotitis Temporomandibular disorders (TMDs) Neoplasia (such as nasopharyngeal, brainstem) Cranial neuralgia Primary: trigeminal or glossopharyngeal nerve Secondary: intracranial Vascular Giant cell arteritis (GCA) Migraine Cluster headache
if headache with runny eye and nose
cluster
unilateral headache with photohpohobia
migraine
bilateral frontal headache temproal
tension headache
scalp tenderness, sudden loss of vision, jaw pan
gca
stabbing pain trigger such as washing face, teeth brushing
v2 v3
trigeminal neuralgia
differentiate trigeminal neuralgia from gca
tri gem is usually unilateral
gca - eye disturbance, pain wit waking, chewing yawning
Trigeminal neuralgia causes a severe, unilateral, “shock-like” and paroxysmal facial pain, often triggered externally (for example by wind or shaving). Peak incidence is between 50-60 years age
what is paroxysmal hemicranias
sjstaad syndrome
unilateral facial pain
females
lacrmination nasal congestion rihoerrhoea
unilateral headache usually affecting the area around the eye. It normally consists of multiple severe, yet short, headache attacks affecting only one side of the cranium
CPH is a long-term disease with symptoms lasting for longer than a year, either without remission or with remissions that last less than a month.[1] In order to be diagnosed with CPH, a patient needs to have had at least 20 attacks filling the following criteria:
Attacks of severe unilateral orbital, supraorbital, or temporal pain lasting between 2 and 30 minutes.
The headache needs to take place with one of the following:
Ipsilateral conjunctival injection and/or lacrimation
Ipsilateral nasal congestion and/or rhinorrhoea
Ipsilateral eyelid oedema
Ipsilateral forehead and facial sweating
Ipsilateral miosis and/or ptosis
Attacks need to occur more than five times a day for more than half of the time, although periods of lower frequency can occur.
Attacks can be prevented completely by therapeutic doses of indomethacin.
The symptoms cannot be attributed to another disorder.[5]
condition above how diagnosed
usually post indomethacin nsaid treatment
mmenonic for bells palsy
BELLS PALSY blink reflex abnormal earache loss of sensation loss of taste and lacrimation sudden pals of 7th nerve entire side loss
what is seen In supranuclear lesion
paralaysis of contralateral lower face
upper face has dual innervation so spared
