Extraction Complications Flashcards

1
Q

Types of peri-operative complications

A
  • difficult access
  • abnormal resistance
  • fracture of tooth/root fracture of alveolar bone
  • jaw fracture
  • involvement of maxillary antrum
  • fracture of tuberosity
  • loss of tooth
  • soft tissue damage
  • damage to nerves/vessels
  • haemorrhage
  • dislocation of TMJ
  • damage to adjacent teeth/restorations
  • extraction of permanent tooth germ
  • broken instruments
  • wrong tooth
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2
Q

Reasons for difficult extraction access

A

Trismus
Reduced aperture of mouth - congenital, syndrome
Crowded/malpositioned teeth

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3
Q

Reasons for abnormal resistance in extraction

A
  • thick cortical bone - less flexibility
  • shape/form of roots eg. divergent roots/hooked roots - upper 6 and lower premolars
  • number of roots e.g. 3 rooted lower molars
  • hypercementosis -build up of cementum on roots
  • ankylosis - bonded to surrounding bone no PDL
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4
Q

Reasons for tooth/root fracture during extraction

A
  • caries
  • alignment - position its lying in can make it more susceptible to fracture
  • size
  • root morphology
    • fused
    • convergent or divergent
    • extra roots
    • hypercementosis
    • ankylosis

CONSENT patient for this

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5
Q

Reasons for alveolar bone fracture

A

Tooth taken out too quickly
Usually buccal plate
Common in canine and molar region

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6
Q

What needs to be assessed when alveolar bone is broken

A
  • is bone still attached to periosteum
  • how big is chunk of bone
  • if its still attached can push back in and reattach and suture to hold in soft tissue
  • if doesn’t reattach will work its way out - can cause problems when sequestreum
  • if its still attached to periosteum and you remove you may need to dissect it free with scalpel
  • try and retain especially in canine region as it can lead to thin ridge which affects denture design and retention
  • if remove bone need to smooth edges as they can be jaggy - can pierce through and cause wound to break down
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7
Q

How to treat alveolar bone fracture (molars and canines)

A

Molars
- periosteal attachment
-suture
-dissect free

Canines
-stabilise
-free mucoperiosteum
-smooth edges

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8
Q

Where and why does jaw fracture occur

A
  • mandible
  • impacted wisdom tooth, cyst, atrophic mandible
  • usually caused by application of force
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9
Q

Tx for jaw fracture

A
  • inform patient
  • OPT
    -refer to max fax - DONT EAT
  • analgesia
    -stabilise
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10
Q

Signs of jaw fracture

A

-crack during extraction
-moving in two different parts
-tear in gingivae
- teeth no longer meeting in occlusion

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11
Q

Reasons for involvement of maxillary antrum in extractions

A

Maxillary sinus just above molars, roots can be very close to

  • can fracture tuberosity
    -can lose root into antrum
  • can cause OAF/OAC
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12
Q

How to diagnose OAC/OAF

A

-size of tooth
-position of roots in relation to antrum
-bone at trifucation of roots
-bubbling of blood
-nose holding test
- juice through nose
-direct vision
-suction
-blunt probe

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13
Q

Risk factors of OAC

A
  • extraction of upper molars and premolars
  • close relationship of roots to sinus on radiograph
  • last standing molars
  • large bulbous roots
  • older patient - sinus bigger and drops down slightly
  • previous OAC - on other side
  • recurrent sinusitis - tooth often close to sinus
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14
Q

Management of OAC/OAF

A

Small
- encourage clot
- suture margins
-AB

Large
- buccal advancement flap

Give post op instructions

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15
Q

Aetiology of tuberosity fracture

A
  • single standing molar
  • unknown un-erupted molar wisdom tooth
  • pathological gemination
  • extracting in wrong order
  • inadequate alveolar support
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16
Q

Diagnosis of tuberosity fracture

A
  • noise
  • movement noted both visually or with supporting fingers
  • more than one tooth movement
  • tear on palate
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17
Q

Management of tuberosity fracture

A
  1. Dissect out and suture
  2. Reduce and stabilise
    - reduction then splint
    - need to make sure it doesn’t affect occlusion
    - AB
    -remove tooth 8 weeks after
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18
Q

What type of nerve injuries can occur during extraction

A

-crush injuries
-cutting/shredding injuries
-transaction
-damage from surgery or LA

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19
Q

Definitions of neurapraxia, axonotmesis, neurotmesis (anatomical descriptions of nerve damage)

A
  • neurapraxia - confusion of nerve/continuity of epineural sheath and axons maintained
  • axonotmesis - continuity of axons but not epineural sheath disrupted
  • neurotmesis -complete loss of nerve continuity/nerve transected
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20
Q

Reasons for haemorrhage during extraction

A
  • local - mucoperiosteal tears , fractures
  • clotting abnormalities - haemophilia
    -liver disease
    -medications - warfarin , antiplatlet
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21
Q

How to handle haemorrhage of soft tissue (extraction)

A
  • mechanical pressure
    -sutures
    -LA with vasoconstrictor
    -diathermy
  • haemostatic forceps - larger vessels
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22
Q

How to handle haemorrhage if bone

A
  • pressure (via swab)
  • LA on a swab or injected into socket
  • haemostatic agents
  • blunt instrument
  • bone wax
  • pack
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23
Q

How to manage TMJ dislocation

A
  • relocate immediately (analgesia and advice on supported yawning)
  • if unable to relocate try local anaesthetic into masseter intra-orally
  • if still unable to relocate - immediate referral
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24
Q

Management of damage to adjacent teeth during extraction

A
  • temp dressing/restoration
  • arrange definitve restoration
  • if large restoration next to extraction site warn patient of risk
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25
Q

Common post extraction complications

A
  • pain/swelling/ecchymosis
  • trismus/limited mouth opening
  • haemorrrhage/post-op bleeding
  • prolonged effects of nerve damage
  • dry socket
  • sequestrum
  • infected socket
  • chronic OAF/ root in antrum
  • osteomyelitis
  • osteoradionecrosis (ORN)
  • medication induced osteonecrosis
  • actinomycosis
  • bacteraemia/infective endocarditis - note current guidance
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26
Q

What increases risk of oedema

A

-poor surgical technique

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27
Q

Causes of trismus after extraction

A

Surgery - Odema/IDB
LA - medial pterygoid muscle spasm
Haematoma
Damage to TMJ

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28
Q

How to manage trismus

A
  • monitor
    -mouth opening exercises
    -wooden spatula
    -trismus screw
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29
Q

What guidance is used for prolonged bleeding

A

SDCEP
Management of patients taking anti platelet or anticoagulant

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30
Q

What is warfarin

A

Vit K agonist

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31
Q

What is management of warfarin for extraction

A
  • check INR - 24 hours before
  • INR below 4 for treatment
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32
Q

What are types of antiplatelet

A

-aspirin
-clopidogrel

33
Q

How to manage antiplatelet in extraction

A

Without interrupting
- can stage treatment

34
Q

Which dental procedures are considered risk of post op bleeding complications for antiplatelet/anticoag

A

Complex extractions
Extractions
Incision and draining of swelling
6ppc
RSD
Direct or indirect with subgingival margins

35
Q

How to manage DOAC in extraction

A

Low risk procedures - treat without interrupting

High risk - miss or delay morning dose before TX

36
Q

What are types of DOAC

A

Apixiban
Dabigatran
Rivaroxaban
Edoxaban

37
Q

Specifically which dose is missed for each DOAC in extraction

A

Apixaban or dabigatran - miss morning dose - normal dose in evening
Rivaroxaban or edoxaban - delay dose to evening

38
Q

Reason for immediate post op bleeding

A

Vessels open
Vasoconstriction effects of LA wear off
Sutures loose
Patient traumatises area

39
Q

Reasons for secondary bleeding in extraction

A
  • due to infection
  • common 3-7 days
  • usually mild ooze but occasionally big bleed
  • medication related
40
Q

What are haemostatic agents

A

Adrenaline containing LA - vasoconstrictor
Oxidised cellulose - surgicel
Haemocollagen sponge
Thrombin liquid and powder
Floseal

41
Q

What are systemic haemostatic aids

A

Vitamin K
Anti-fibrinolytic - tranexamic acid
Plasma
Desmopressin

42
Q

Post op extraction instructions

A
  • don’t rinse till next day
    -avoid trauma
    -avoid hot food
    -avoid exercise
  • no alcohol or smoking
    -advise on gauze
43
Q

3 types of sensory change in nerve damage

A

Anaesthesia
Parasthesia
Dysaesthesia

45
Q

What is alveolar osteitis

A

Dry socket
2-3% of extractions
Normal clot dissapears
Intense pain
Takes up to 2 weeks to resolve

46
Q

Symptoms of alveolar osteitis

A
  • dull aching pain - moderate to severe
  • usually throbs , can radiate to ears, continuous and can keep awake at night
  • the exposed bone is sensitive and is source of pain
  • characteristic smell/bad odour and patient frequently complains of bad taste
47
Q

Predisposing factors of alveolar osteitis

A
  • molars
    -mandible
    -smoking - reduced blood supply
    -female
    -oral contraceptive
    -excessive mouth rinsing post extraction
48
Q

Management of alveolar osteitis

A

-supportive - analgesia, reassurance
-LA
-irrigate with saline
-curretage/debridement
Pack - alvogel

49
Q

What is sequestrum

A

-common
-bits of dead bone
-delays healing

50
Q

What is infected socket

A

-rare
-pus discharge
-look for remaining tooth or fragments

51
Q

Different types of flap for OAF

A

Buccal advancement flap
Buccal fat pad with buccal advancement flap
Palatial rotational flap
Tongue flap
Bone graft

52
Q

If root in antrum TXp

A

Flap design
Suction
Curretes
Then close as OAC

Or could to Caldwell luc approach or ENT approach

53
Q

What is osteomyelitis

A

Inflammation of bone marrow , infection of bone

54
Q

How may osteomyelitis present

A

Systemic unwellness
Mandible usuallly
If infection is deep then possibly some altered sensation
Oedema
Early presentation similar to dry socket or localised infection

55
Q

How does osteomyelitis spread

A

Usually in medullary cavity with cancellous bone
Spread to cortical bone
The periosteum

Oedema in cancellous bone (enclosed space)
This leads to increased hydrostatic pressure
Higher than the blood pressure leading to compromised blood supply and necrosis
Normal blood borne defences don’t reach tissue

56
Q

What is primary blood supply in mandible

A

Inferior alveolar artery

57
Q

Why is mandible more likely to become ischaemic in osteomyelitis

A

Poorer blood supply due to dense overlying cortical bone

58
Q

What is predisposing factors to osteomyelitis

A

Odontogenic infections
Fractures of mandible
Immunocompromised host - diabetic , leukaemia , chemo

59
Q

How would chronic osteomyelitis present

A

Pus
Bony destruction
Radiographically - after 10 days moth eaten - increased radiolucency

60
Q

Bacteria involved in osteomyelitis

A

Steptococci
Anaerobic cocci
Fusobacterium and prevotella

61
Q

Treatment of osteomyelitis

A
  • AB - Pen V - long course - can be up to 6 months
  • drain pus , remove non vital teeth, remove loose bone
    -corticotomy and excision of necrotic bone
62
Q

what is ORN

A
  • patients who received radiotherapy of head and neck
    -bone within radiation beam becomes non vital
  • reduced bloody supply and turnover of viable bone is slow
    -mandible more commonly affected
63
Q

Prevention of ORN

A
  • scaling, chlorhexidine in lead up to extraction
    -atraumatic extraction technique
    -AB
    -hyperbaric oxygen before and after extraction
64
Q

Treatment of ORN

A
  • irrigation of necrotic debris
  • don’t give AB unless secondary infection
  • remove sequestra
    -if severe - resection of exposed bone and soft tissue closure
65
Q

What is MRONJ

A

Medication related osteoradionecrosis of jaw
Side effect of anti-resorptive and antiangiogenic drugs

66
Q

Which drugs to watch out for for MRONJ

A

Bisphosphonates - inhibit bone resorption and renewal
Anti-angiogenic -
Rank L inhibitors - stop osteoclast production

67
Q

Incidence of MRONJ

68
Q

When does MRONJ occur

A

Spontaneous
Following extraction
Denture trauma

Small asymptomatic areas of exposed bone to large exposure with pain

69
Q

When was MRONJ guidance released

A

2017 SDCEP

70
Q

What classifies a patient as high MRONJ risk

A
  • Bisphosphonates for more than 5 years
  • Bisphosphonates/denosumab with a systemic glucocorticoid
  • patients being treated with anti-resorption or anti-angiogenic drugs as part of cancer treatment
    -previous MRONJ
71
Q

Treatment of MRONJ

A
  • manage symptoms
    -remove sharp bone
    -chlorhexidine
  • debridement
  • prevent invasive retreatment
72
Q

What is actinomycosis

A

Rare bacterial infection
Actinomyces israeli
Chronic
Multiple skin sinuses and swelling
Thick lumpy pus

73
Q

Treatment of actinomycosis

A
  • Incision and drainage of pus
    -excision of sinus tract
    -excision of necrotic bone and foreign bodies
    -high does AB
    Then long term AB to prevent recurrence
    Pen V, doxycycline
74
Q

Guidance for IE

75
Q

Which groups are in special consideration for IE

A
  • prosthetic valve
    -previous IE
    -cyanotic CHD
    -CHD repaired with prosthetic material
76
Q

Example of invasive procedures I.E

A
  • matrix bands
  • clamps
    -subgingival restorations
    -Endo
    -PMC
    -extractions
77
Q

Risks and benefits of IE prophylaxis

A

Risk
-hypersensitivity
-CDIFF
- antimicrobial resistance
- antibiotic colitis

78
Q

What is given for AB prophylaxis

A

Amoxicillin - 3G - 60 mins before
Clindamycin 300mx2 - 60 mins before
Azinthromycin -500mg — 60 mins before