Exertional Heat Illness Flashcards

1
Q

Exertional Heat Illness

A

Collapse on exertion

Body core temperature >39 degrees

Recover very quickly

Usually caused by drop in blood pressure
- struggle between pumping blood to surface to decrease heat and pumping blood to working muscles

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2
Q

Exertional Heat Stroke

A

More severe

Collapses on exertion

Body core temperature >40 degrees

Neurological disturbance - more sustained in severe cases

Multiple organ failure in very severe cases

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3
Q

EHI Incidence - Sport

A

Varies between sports and events but increases with rising ambient temperature and humidity

Observed more frequently in road races and other activities that involve continuous high intensity metabolic activity

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4
Q

EHI Incidence - Military

A

Training - on average ~100 personnel are admitted to hospital each year during training

Operations

  • average maximum air temp. in excess of 40 degrees in Iraq and Afghanistan
  • 849 cases of heat illness during first 6 months of Op Telic
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5
Q

Proposed new framework classification for ‘heat-illness’

A

Conditions in which there is a pathological increase in body heat storage

  • Exercise and environmentally induced heat stroke:
    • –> due to inadequate heat loss
    • –> due to excessive endogenous heat production
    • –> combination

Conditions in which there is no pathological increase in body heat storage
- Exercise - associated postural hypotension

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6
Q

Risk factors

A

Sleep deprivation

Lack of fitness

High solar radiation

Training at hottest hours

Absence of medical support

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7
Q

Heat stressors

A

Clothing thermal properties

Intensity and duration of exercise

Thermal environment

  • Temperature
  • Humidity
  • Air velocity

Individual heat tolerance

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8
Q

Wet Bulb Globe Temperature

A

Environmental stress measured

Threshold Limit Values

Takes into account

  • temperature
  • solar radiation
  • humidity
  • air velocity

Gives WBGT index

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9
Q

Physical Fitness

A

Rectal temperature increases as exercise intensity increases (Saltrin & Hermansen, 1994)

Mortality of sedentary rats occur at lower core temperature than endurance trained rats (Fruth & Gisolfi, 1983)

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10
Q

Menstrual Cycle Phase

A

Temperature higher in luteal phase of cycle

Endurance time reduced

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11
Q

Age

A

Increased cases of heat stroke in older individuals - probably related to physical fitness (Armstrong, 2003)

Children have a smaller surface to body mass ratio

  • lower sweat rates
  • slower rate of acclimatisation
  • greater core temperature before sweating
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12
Q

Treatment

A

Every minute counts

Rectal temperature is best gauge

Cool first, transport second

Ice-water or cold-water is best (Casa et al., 2005)

Monitor closely - remove athlete from tub when rectal temperature = 38.9 degrees

Patient can be cooled from ~42 degrees to 38.9 degree in 15-30 mins (Roberts, 1989)

Send patient to hospital after cooling

Survival rates approach 100% with fast cooling

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13
Q

Recovery

A

90% of heat stroke patients have normal heat tolerance within 2 months (Armstrong et al., 1990)

Only 1% of Marine recruits who suffered heat illness had subsequent occurrence per year (Phiney et al., 2001)

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14
Q

Role of Arteriovenous Anastomoses (AVAs) in Heat Loss

A

Location

  • Palmar side of fingers
  • Plantar side of toes, palm, sole, ear and nose

Will not constrict - allow continued blood flow to periphery even when skin temperature is low

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15
Q

Proposed Mechanisms - Incidence & Pathophysiology of EHI in Parachute Regiment Trainees - source

A

Bilzon, Walsh and Jackson, 2008

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16
Q

Proposed Mechanisms - Incidence & Pathophysiology of EHI in Parachute Regiment Trainees - Bilzon, Walsh & Jackson, 2008

A

Measured:

  • Thermal and cardiovascular strain
    • -> full blood count
    • -> inflammation
    • -> muscle damage - creatine kinase, myoglobin
    • -> liver damage
  • Lipopolysaccharide (LPS) concentration
    • -> anti-LPS antibodies
    • -> heat stroke proteins

30% of casualties from loaded march were positive for LPS only in prolonged exercise