Exam V -- Skin Flashcards

1
Q

Describe risk factors for pressure injury

A

-Prolonged immobilization
-moisture
-neurologic disorders
-fractures
-impaired perfusion/ischemia
-malnutrition/dehydration

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2
Q

Differentiate light skin vs dark skin assessment of pressure ulcer injury

A

Lighter skin: erythema/redness
Darker skin: purple/blue/violet skin, non-blanchable, induration, taut/shiny skin, pain assessment

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3
Q

Describe altered scarring by name and description

A

Keloids: ‘clawing’ scars outside of original border
Hypertrophic: fibrous scar within original border

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4
Q

Compare allergic to irritant dermatitis

A

Allergic/atopic: type 1/type 4 hypersensitivity response, genetic mutation, common in younger children in crevices
Irritant/contact: older adults, response to chemical exposure

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5
Q

Describe Steven’s Johnson Syndrome

A

Form of erythema multiforme: T cell immunologic reaction to drugs
-Bulls eye lesions on skin, mucous membranes

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6
Q

Differentiate between virus and bacterial skin pathophysiology

A

Bacterial: impetigo (staph/strep_, furuncles/carbuncles, folliculitis/cellulitis,
Viral: HSV, shingles, chickenpox, warts (HPV)

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7
Q

List benign vs cancerous/malignant alterations of skin

A

Benign:
-Nevi
-Actinic keratosis
-Seborrheic keratosis

Malignant:
-Basil cell carcinoma
-Melanoma
-Squamous cell carcinoma

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8
Q

Identify tinea fungal infections by location: scalp, hand, foot, corporis, cruris, unguium

A

Capitis (scalp), manus (hand), pedis (foot), corporis (ringworm) cruris (Crotch), unguium

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9
Q

Explain the pathophysiology of shingles, relationship to chicken pox, and the clinical manifestations

A

OG chicken pox (varicella-zoster) infection –> virus latent in trigeminal/dorsal root ganglia –> present as shingles (herpes zoster) along thoracic dermatome

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10
Q

What is the” Rule of Nines”?

A

The body is split into segments of 9%: head/neck, chest/back, abdomen/lower back, each leg.
Used for estimating size of burns.

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11
Q

Describe the clinical presentation/ pattern of scabies

A

Infestation –> 3-5 weeks later: burrows/papules/vesicles
-in crooks/crannies
-intense pruritis–worse at night
-secondary infections (crusting) common

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11
Q

Describe the etiology/pathophysiology and clinical presentation of impetigo

A

PATHO:
-skin lesion r/t opportunistic staph or strep on skin

CM:
-honey crusted lesions around face/nose/mouth, common in children

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11
Q

Explain the pathophysiology of acne vulgaris vs acne rosacea

A

Vulgaris: Inflammation of pilosebaceous follicles, can be inflammatory (cystic) or non-inflammatory
Rosacea: chronic vasodilation results in lesions–adults (triggered by sun, alcohol, hormones)

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12
Q

Explain the pathophysiology of atopic dermatitis and its’ clinical manifestations

A

AKA eczema
PATHO: IgE mediated reaction–associated with filaggrin gene mutation/deficiency
MANIFESTATIONS: redness, edema, scaling, pruritis

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13
Q

Differentiate between virus and bacterial pediatric skin pathophysiology (name bacterial vs viral infections)

A

Bacterial: impetigo, SSSS—breach in skin barrier –> infection
Viral: Molluscum, rubella, rubeola, roseola, small pox, chicken pox viral infection of cells –> more systemic

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14
Q

Rubella patho, S/S

A

PATHO: RNA respiratory virus
s/s: maculopapular rash that moves from trunk –> extremities “3 day measles”

15
Q

What infection leads to ‘Kopliks’ spots? What are they?

A

-Rubeola–classic measles
-white spots on buccal mucosa