Exam V -- Skin Flashcards
Describe risk factors for pressure injury
-Prolonged immobilization
-moisture
-neurologic disorders
-fractures
-impaired perfusion/ischemia
-malnutrition/dehydration
Differentiate light skin vs dark skin assessment of pressure ulcer injury
Lighter skin: erythema/redness
Darker skin: purple/blue/violet skin, non-blanchable, induration, taut/shiny skin, pain assessment
Describe altered scarring by name and description
Keloids: ‘clawing’ scars outside of original border
Hypertrophic: fibrous scar within original border
Compare allergic to irritant dermatitis
Allergic/atopic: type 1/type 4 hypersensitivity response, genetic mutation, common in younger children in crevices
Irritant/contact: older adults, response to chemical exposure
Describe Steven’s Johnson Syndrome
Form of erythema multiforme: T cell immunologic reaction to drugs
-Bulls eye lesions on skin, mucous membranes
Differentiate between virus and bacterial skin pathophysiology
Bacterial: impetigo (staph/strep_, furuncles/carbuncles, folliculitis/cellulitis,
Viral: HSV, shingles, chickenpox, warts (HPV)
List benign vs cancerous/malignant alterations of skin
Benign:
-Nevi
-Actinic keratosis
-Seborrheic keratosis
Malignant:
-Basil cell carcinoma
-Melanoma
-Squamous cell carcinoma
Identify tinea fungal infections by location: scalp, hand, foot, corporis, cruris, unguium
Capitis (scalp), manus (hand), pedis (foot), corporis (ringworm) cruris (Crotch), unguium
Explain the pathophysiology of shingles, relationship to chicken pox, and the clinical manifestations
OG chicken pox (varicella-zoster) infection –> virus latent in trigeminal/dorsal root ganglia –> present as shingles (herpes zoster) along thoracic dermatome
What is the” Rule of Nines”?
The body is split into segments of 9%: head/neck, chest/back, abdomen/lower back, each leg.
Used for estimating size of burns.
Describe the clinical presentation/ pattern of scabies
Infestation –> 3-5 weeks later: burrows/papules/vesicles
-in crooks/crannies
-intense pruritis–worse at night
-secondary infections (crusting) common
Describe the etiology/pathophysiology and clinical presentation of impetigo
PATHO:
-skin lesion r/t opportunistic staph or strep on skin
CM:
-honey crusted lesions around face/nose/mouth, common in children
Explain the pathophysiology of acne vulgaris vs acne rosacea
Vulgaris: Inflammation of pilosebaceous follicles, can be inflammatory (cystic) or non-inflammatory
Rosacea: chronic vasodilation results in lesions–adults (triggered by sun, alcohol, hormones)
Explain the pathophysiology of atopic dermatitis and its’ clinical manifestations
AKA eczema
PATHO: IgE mediated reaction–associated with filaggrin gene mutation/deficiency
MANIFESTATIONS: redness, edema, scaling, pruritis
Differentiate between virus and bacterial pediatric skin pathophysiology (name bacterial vs viral infections)
Bacterial: impetigo, SSSS—breach in skin barrier –> infection
Viral: Molluscum, rubella, rubeola, roseola, small pox, chicken pox viral infection of cells –> more systemic