exam 3: abnormal muscle tone Flashcards

1
Q

resistance of muscles to passive stretch or elongation
amount of tension a muscle as at rest

A

muscle tone

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2
Q

normal muscle tone is high enough to ______ yet low enough to _____

A

counter the effects of gravity
allow freedom of movement

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3
Q

hypotonia is:
- ____ or _____ muscle tone
- stretch reflexes _____
- _____ resistance to passive movement
- results in ___
- a finding with ______ syndrome
- what are 2 other synonymous terms?

A
  • decreased or absent
  • diminished or absent
  • little to no
  • neurogenic muscle atrophy
  • lower motor neuron
  • flaccidity, low tone
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4
Q

true or false. acute UMN lesions can initially produce temporary hypotonia due to spinal or cerebral shock.

A

true

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5
Q

hypertonia is:
- ______ muscle tone
- resistance to passive movement that (is/is not) dependent on velocity
- can be with or without _____

A
  • increased
  • is NOT
  • spasticity
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6
Q

what are the 4 types of hypertonia?

A
  • spasticity
  • rigidity
  • dystonia
  • decorticate and decerebrate rigidity
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7
Q

increased, involuntary, velocity-dependent muscle tone
the faster the passive movement, the stronger the resistance

A

spasticity

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8
Q

spasticity originates from injury to ____
and as a result, what happens?

A

descending motor pathways or brainstem
lack of inhibition of spinal reflexes causing them to be hyperexcitable

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9
Q

can a spastic muscle be manual muscle tested?

A

no - you need isolated, graded movement to perform MMTs

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10
Q

what is the most common flexor synergy pattern? extensor?

A

flexor: UE
extensor: LE

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11
Q
  • involuntary and sustained muscle contractions (can be repetitive movements)
  • increased muscular tone
  • can affect one or more body parts
  • commonly seen from lesion to the basal ganglia
A

dystonia

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12
Q

what are 3 diseases that result in dystonia?

A
  • primary idiopathic dystonia
  • wilson’s disease
  • parkinson’s disease
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13
Q

dystonia is a type of _____ _which is?

A

dyskinesia
–> abnormal involuntary writhing movements of a body part (face, UEs, LEs)
–> can be smooth, rapid jerking, or tics

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14
Q

identify these common types of dyskinesia:
1. most commonly seen in CP, involuntary writhing movement that is slow and continuous, more twisting movements, less jerky
–> ability to let go of hand

  1. involuntary and sustained muscle contractions, twisting writhing repetitive movements, involving co-contraction of agonist and antagonist
    –> unable to let go of hand
  2. involuntary, rapid, abrupt twisting, writhing movements that may appear to jump from one extremity to another
    –> dance like movements
A
  1. athetosis
  2. dystonia
  3. chorea
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15
Q

stiffness
resistance to movement that is independent of velocity of movement
associated with lesions of basal ganglia
not related to spinal reflex mechanisms

A

rigidity

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16
Q

two types of rigidity:
1. constant increase in muscular tone and stiffness of affected muscles

  1. rigidity w/ tremor resulting in ratchet-like jerkiness when moved, seen in UE at elbow and wrist
A
  1. leadpipe rigidity
  2. cogwheel rigidity
17
Q

posturing with elbows, wrists and fingers flexed and legs extended and rotated inward

A

decorticate rigidity

18
Q

decorticate rigidity:
- severe injury to ___
- long term impact is ___

A
  • cortex
  • severe ROM contractures
19
Q

posturing with lots of extension –> shoulder IR, full ext at elbow, flexion at wrist and fisted hand.
LEs in extension with severe PF contracture

A

decerebrate rigidity (lots of e’s)

20
Q

what is opisthotonos?

A

strong, sustained, muscle contraction of the extensors of the neck and trunk
rigid, hyperextended posture

21
Q

your patient has a severe neurologic disease including neurodegeneration and trauma to the CNS rostral to the spinal cord. what should you check for?

A

primitive reflexes

22
Q

what is the primitive reflex ATNR?

A

asymmetric tonic neck reflex
ext. of UE and LE side head rotates to
flx. of UE and LE occiput side

23
Q

how do you assess for ATNR and what is your intervention?

A

palpate for change in mm tone with head rotation
head position matters and is critical to reduce muscle tone that results from this reflex

24
Q

what is the primitive reflex STNR?

A

symmetric tonic neck reflex
neck extension = UE ext and LE flx
neck flx = UE flx and LE ext
** legs opposite arms

25
Q

how do you assess for STNR and what is your intervention?

A

move head into flx and then ext while palpate for change in muscle tone at elbow
head position to neutral which may require good postural control and positioning of trunk and pelvis

26
Q

tonic labyrinthine is linked to _______ development and important precursor to development of _____

A

vestibular system
postural reflexes

27
Q

forward TLR:
backward TLR:

A
  • neck flexes –> arms + legs flex
  • neck ext –> arms + legs ext
28
Q

what makes up the bermuda triangle of spasticity and what do they have in common?

A

posturing/rigidity
TLR
STNR
–> all deal w extension

29
Q

how do you examine muscle tone?

A

passive motion testing –> repeat specific motions with increased velocity
clonus –> quick stretch of mm + when spasmodic contraction of antagonist mm

30
Q

what scale is used to measure spasticity? what is a high and low score?

A

Modified Ashworth Scale (MAS)
0 = no increase in muscle tone
4 = affected part(s) rigid in flexion or extension

31
Q

what are results of unmanaged spasticity?

A

joint contractures
spinal deviation/scoliosis
wounds
inability to access active movement

32
Q

what are some PT interventions for managing spasticity?

A

WB
prolonged stretch
static splinting (prolonged stretch & maintain ROM)
dynamic splinting (low load prolonged stretch)
serial casting
modalities (goal is to change motor neuron excitability)
positioning (in treatment, bed and wheelchair)

33
Q

where do you start to manage spasticity?

A

manage primitive reflexes (address head position in bed and sitting)
manage postural deviations
manage LE extensor tone
reduce destructive hip position