Exam 3 Flashcards

1
Q

Anorexia Nervosa (characteristics; medical consequences)

A

Restriction of energy intake relative to requirements, leading to a significantly low body weight in the context of age, sex, developmental trajectory, and physical health (less than minimally normal/expected) (based on WHO BMI)

Intense fear of gaining weight or becoming fat or persistent behavior that interferes with weight gain

Disturbed by one’s body weight or shape, self-worth influenced by body weight or shape, or persistent lack of recognition of seriousness of low bodyweight

Two Types:
Restricting type: During the last 3 months has not regularly engaged in binge-eating or purging.
Binge-eating/purging type: During the last 3 months has regularly engaged in binge-eating or purging

Potential psychological problems: Depression, anxiety, low self-esteem, sleep disturbances, Substance abuse, Obsessive-compulsive patterns and perfectionism (similar to OCD, but different reasonings)

Potential Medical Complications (Partial List): Amenorrhea, Death from heart arrhythmias, kidney damage, gray matter brain shrinkage, brittle hair and nails, dry skin, yellowish skin from liver damage, etc.

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2
Q

Bulimia nervosa (characteristics; medical consequences)

A

Recurrent episodes of binge eating characterized by both:
1. Eating in a discrete period of time (e.g., within any 2-hour period), an amount of food definitely larger than what most individuals would eat in a similar period of time under similar circumstances
2. A sense of lack of control during the episode

Recurrent inappropriate compensatory behaviors to prevent weight gain, such as self-induced vomiting; misuse of laxatives, diuretics, or other medications; fasting; or excessive exercise.
The binge eating and inappropriate compensatory behaviors both occur, on average, at least once a week for 3 months.
Self-evaluation is unduly influenced by body shape and weight.

Bulimia can lead to damage to hands, throat, and teeth

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3
Q

Binge Eating Disorder

A

Binge eating episodes (at least 1/week) characterized by both:
1. Eating in a discrete period of time an amount of food definitely larger than what most individuals would eat in a similar period of time;
2.A sense of lack of control during the episodes

Binge eating episodes are associated with 3≥ of the following:
Eating much more rapidly than normal.
Eating until feeling uncomfortably full.
Eating large amounts of food when not physically hungry.
Eating alone because of feeling embarrassed.
Feeling disgusted with oneself, depressed, or very guilty afterwards.

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4
Q

Atypical anorexia nervosa

A

all the criteria for anorexia nervosa are met, except that despite significant weight loss, the individual’s weight is within or above the normal range

Since speed of weight loss is related to medical complications, individuals with atypical anorexia nervosa who lose a lot of weight rapidly by engaging in extreme weight control behaviors can be at high risk of medical complications

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5
Q

Biological factors in eating disorders (e.g., parts of the hypothalamus, set-point theory)

A

The set-point theory is related to homeostasis. The theory posits that the human body has a predetermined weight or fat mass set-point range. Various compensatory physiological mechanisms maintain that set point and resist deviation from it.

The hypothalamus controls eating and other body maintenance functions; * Damaged ventromedial hypothalamus * Signals of satiety no longer received

Starvation causes a release of endorphins
* Decision making about food switches to a less logical part of
the brain. * Hyperactivity in AN due in attempt to stay warm further
decreases weight

Serotonin: Low in anorexia, Normal in bulimia, Both show higher levels than control women during recovery

Dopamine: Imbalance may explain lack of pleasure from food and other typical comforts.

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6
Q

Minuchin’s views on family systems in eating disorders

A

Enmeshed family patterns (overinvolvement; overconcern)

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7
Q

Treatments for anorexia nervosa; challenges

A
  • Emergency procedures to restore weight * Antidepressants or other medications
  • Family therapy
  • Cognitive-behavioral therapy

Emergency procedures: Severe AN may require emergency
hospitalization and: * intravenous feeding. * Nasogastric tube feeding may be used temporarily when medical
stability and/or body weight continue to decline despite refeeding
efforts. Major disadvantage of using forced feeding for patients with EDs is they may become distrustful of the medical establishment and uncooperative with further treatment.
Medications: Antidepressants sometimes used; no evidence effective.
Treatment with antipsychotic medications may be beneficial.
Family therapy: treatment of choice for adolescents with anorexia. In
randomized controlled trials where patients are treated with family therapy for one year, after five years have 75%–90% of patients show full recovery

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8
Q

Medications to treat bulimia

A

Antidepressants
Cognitive-behavioral therapy

For BN antidepressants can binge frequency; improve mood and
preoccupation with shape and weight
Most effective treatment is cognitive-behavioral therapy.
* Behavioral elements:
* focus on meal planning, nutritional education, and ending binge/ purge cycles by teaching to eat small amounts more frequently. * Exposure and response prevention. In this situation a client
with bulimia would be exposed him to situations that usually
cause binge episodes and then prevented from binge eating.
* The cognitive element is aimed at changing the cognitions and
behaviors that initiate or perpetuate a binge cycle.

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9
Q

Gender and eating disorders (e.g., different prevalence of)

A

Female internalized ideal, * 94% of teen girls and 64% of boys have experienced body
shaming
Boys and men account for 10-25% of all cases of Eds
Check one note for prevalence

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10
Q

Orthorexia (general characteristics)

A

“Fixation on righteous eating” * Not in DSM * Food choices severely restricted * Obsession with purity of diet (not weight)

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11
Q

Gender differences in alcohol tolerance

A

Women have less dehydrogenase (less mass to dilute alc. as well) in the stomach, become more intoxicated than men on equal doses of alcohol (even if both male and female are same size, women will get drunk faster with the same amount)

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12
Q

Physiological effects of alcohol

A

Irreversible liver damage—cirrhosis (liver is overworked and could mean liver replacement)
Damage to the heart and immune system (could lead to strokes and heart attacks)
Major nutritional problems
Korsakoff’s syndrome (Person can be delusional, also known as wet brain syndrome, damage isn’t always reversible)

Biological Processes: * Increases GABA activity at key sites in the brain * Decreases glucose levels (low energy) * Motor impairment * Cerebellum

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13
Q

Delirium tremens (DTs)

A

When a heavy drinker suddenly stops or significantly reduces their alcohol intake may experience delirium tremens (DT)
DTs usually starts two to five days after the last drink * Lasts 3-6 days * 5-25% with DTs die from seizures, heart failure, and other complications

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14
Q

Opioids (risks and treatments)

A

Most immediate danger is overdose * Impure drugs * Dirty needles and other equipment can spread infection

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15
Q

Medical uses of opioids

A

Suppresses the sensation of pain * Euphoria * CNS depressant

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16
Q

Opioid overdose and naloxone (Narcan)

A

Drug for opiod overdose

17
Q

Stimulants (effects and risks)

A

Alertness and confidence * Decrease feelings of fatigue
Mechanism of action: block reuptake of dopamine, norepinephrine, and serotonin.

Amphetamine: severe body destruction * Amphetamine psychosis ( * Paranoia * Hallucinations * Anxiety * Difficulty concentrating * Aggressiveness * Increased motor activity)

Methamphetamine: Highly addictive * Substantial increase in last 10 years * Structural changes in brain * Resistant to treatment * Relapse common

18
Q

Hallucinogens (effects and risks)

A

Disassociation, Delusions

Hallucinogen and Use Disorder: Withdrawal effects in 59-98% of users.
Other risks of hallucinogens: * Water intoxication * Serotonin syndrome * Psychosis in susceptible individuals

19
Q

Risks of polysubstance use (what happens when drugs are combined)

A

Different kind of effect results when drugs have opposite (antagonistic) effects

Polysubstance abuse refers to the concurrent use of multiple substances, which significantly increases the risk of adverse effects, including overdose, addiction, and mental health issues. Mixing substances can amplify their individual effects, leading to unpredictable outcomes and heightened health risks. It can also complicate treatment and recovery processes. Therefore, polysubstance abuse poses a serious threat to both physical and mental well-being.

20
Q

Learning explanations for substance abuse

A

Operant conditioning: Temporary reduction of drug-produced tension has rewarding effect thus likelihood that the user will seek this reaction again increases
Self-medication hypothesis

21
Q

The brain’s reward circuit (pleasure pathway)

A

Certain drugs stimulate the reward center directly * Other drugs stimulate the reward center indirectly

22
Q

Treatment modalities (medical, cognitive-behavioral, etc.) for drug use

A

Detoxification: * Systematic and medically supervised withdrawal from a drug * Outpatient or inpatient; gradual withdrawal; induced withdrawal

Common drugs for Medication Assistant Therapy (MAT): * Naltrexone (for AUD and OUD) * Methadone or buprenorphine (for OUDs) * Suboxone (buprenorphine combined with naloxone) - most commonly prescribed MAT for OUDs.

Self-help organizations * Alcoholics Anonymous (AA); Al-Anon; Alateen * Narcotics Anonymous; Cocaine Anonymous * Residential treatment c

23
Q

Multidimensional (step-down) treatment approach

A

A multidimensional treatment program most effective
Step-down process is common
In-patient medical detox (biological)
Recovery center/rehab for 30-90 days (with CBT; groups; drug screening)
Sober living home – can be 1 month to several years (groups; structured programs; social and work skills)

24
Q

Harm reduction approach

A

A. Harm reduction focuses on minimizing the negative consequences of substance use without requiring abstinence.
B. Includes strategies like needle exchange programs, supervised consumption sites, and education on safer substance use practices.
C. Commonly provide linkages to MAT programs; rehabilitation programs
D. Controversial issue. Opponents argue it brings danger to communities and enables continued drug use
E. Proponents argue it addresses reality of addiction; reduces OD deaths; reduces disease transmission

25
Q

Gender differences in schizophrenia

A

More common and severe in men

26
Q

Symptoms of schizophrenia

A

Positive symptoms are “pathological excesses” - bizarre
additions to a person’s behavior: * Delusions (Types of delusions: * Persecutory
* Referential * Grandiose
* Religious
* Somatic
* Control * Disordered thinking and speech
* Hallucinations

  • Loose associations (derailment) * Word salad (failure to make sense) * Echolalia
  • Clang speech
  • Neologisms
  • Perseveration
  • Echopraxia * Dressing oddly
  • Doing things in public that are usually done only in private

Negative symptoms: * Alogia (poverty of speech) * Flat affect * Avolition
* Anhedonia
* Social withdrawal

Psychomotor symptoms: * Awkward movements
* Repeated grimaces
* Odd gestures
* Catatonia

27
Q

Concordance rates for schizophrenia in twins

A

More closer genetically, more likely to develop schizophrenia if schizophrenia is present, 50% chance in twins

28
Q

Dopamine hypothesis

A

Supported by research on L-dopa * New antipsychotics cast some doubt (glutamate appears to play complex role)

hyperactive dopamine transmission results in schizophrenic symptoms.

29
Q

Brain abnormalities in schizophrenia

A

Biochemical abnormalities

Abnormal brain structure: Decreased brain volume * Enlarged ventricles * Frontal lobe dysfunction * Reduced volume of the thalamus * Abnormalities in temporal lobe areas

Dysfunctional brain circuits

30
Q

Family influences; expressed emotion relating to schizophrenia

A

Family factors - parents of people with the disorder often: Display more conflict * Have greater difficulty communicating
Are more critical of and overinvolved with their children than other parents
Typically dysfunctional families

31
Q

Diathesis-stress model

A

The diathesis-stress model, also known as the vulnerability–stress model, is a psychological theory that attempts to explain a disorder, or its trajectory, as the result of an interaction between a predispositional vulnerability, the diathesis, and stress caused by life experiences

Dysfunctional living situations in families

32
Q

Antipsychotic medications

A

Antipsychotic drugs (aka “neuroleptics”): Alleviate or reduce the intensity of delusions and hallucinations

First-generation (“typical”) antipsychotics: * Thorazine and Haldol * Block the action of dopamine * Some clinical change within 24 hours * Extrapyramidal side effects (affect motor movement, blurred vision, dry mouth)
Do little for negative symptoms of SZ

Second-generation “atypical” antipsychotics: * E.g., Clozaril, Risperdal, Zyprexa, Seroquel, Abilify
More effective than typical antipsychotics, esp. for negative symptoms
Fewer extrapyramidal side effects, Blocks both dopamine and serotonin receptors.

Tardive Dyskinesia: Developed after a long time use of antipsychotics, repetitive movements similar to Parkinson’s

Drowsiness; dizziness * Restlessness * Weight gain (sometimes dramatic) and diabetes * Dry mouth; constipation * Nausea; vomiting * Blurred vision * Low blood pressure * Uncontrollable movements; spasms * Seizures * Low number of white blood cells

Reduce symptoms in majority of patients * Maximum improvement seen within the first 6 months
Maintenance therapy may not reduce relapse for some
Patients able to participate in psychotherapy
Duration: for those who have recovered from an acute first psychotic episode, recommended to continue antipsychotics for 2 to 3 years to prevent relapse
Longer duration for those who have experienced multiple severe episode

Non-compliance can be due to
i. Certain symptoms of SZ including disordered thinking (forget to take medication); paranoid delusions, and anosognosia (don’t believe they have a disorder, truly believe that delusions are reality), distrust of doctors
ii. side effects of the medications

33
Q

Cognitive-behavioral therapy for Schizophrenia

A

Includes cognitive restructuring, behavioral experiments / reality testing, self-monitoring and coping skills training.
Example of a behavioral experiment: The individual is asked to discriminate between a real sensation (such as a tap on the shoulder) and an imagined sensation (such as feeling a tap on the shoulder when nobody is there). This can help the individual distinguish between reality and hallucinations

34
Q

Milieu therapy

A

Milieu therapy is a safe, structured, group treatment method for mental health issues. It involves using everyday activities and a conditioned environment to help people with interaction in community settings. Milieu therapy is a flexible treatment intervention that may work together with other treatment methods

35
Q

Assertive Community Treatment (ACT)/ community-based mental health care

A

In a perfect world all services are coordinated: * Short-term hospitalization * Partial hospitalization * Supervised residences * Occupational training and support