Exam 2 Flashcards
The biological stress response
The effect stressors create within an organism; a by-product of poor or inadequate coping
Variety of coping strategies; Fight or Flight response activated by stress (activation of the Sympathetic Nervous System (SNS) and Hypothalamic-Pituatry-Adrenal Axis (HPA Axis)) which leads to increased heart rate and blood pressure, Release of adrenaline (epinephrine) and noradrenaline
(norepinephrine)
Characteristics of acute stress disorder and posttraumatic stress disorder
During and immediately after trauma, high levels of arousal and upset may be temporarily experienced (PTSD is long term)
PTSD Criteria: T - Trauma exposure R - Re-experiencing A - Avoidance of reminders U - Undermined cognition and mood M - Magnified arousal and reactivity A - Active symptoms for 1 month
Childhood experiences and later development of stress disorders
Growing up in poverty * Psychological disorders in the family * Assault, abuse, or catastrophe at an early age * Parental separation or divorce in early childhood
Combat exposure * Childhood physical abuse * Sexual violence * Physical assault * Being threatened with a weapon * An accident
Gender differences in stress disorders and traumatic events
Lifetime prevalence - 6.8%; Women show higher rates of PTSD and tend to have more severe symptoms.
Men tend to be more exposed to traumatic events
Male gender role stress refers to the experience of emotional distress as a result of violating or not adhering to traditional masculine gender role norms.
Traditional masculine gender roles play a part in the severity of PTSD in some men.
Complex PTSD
Frequent exposure to traumatic events can lead to more severe and chronic PTSD symptoms
Distinctions between dissociative amnesia and dissociative fugue
Dissociative amnesia: Loss of memory triggered by a specific upsetting event
Often recover without treatment * Leading treatments: psychodynamic therapy, hypnotic therapy, and drug therapy
* May have unpredictable consequences
Dissociative fugue: subtype of dissociative amnesia
person loses awareness of their identity
and engages in some form of unexpected travel
* Causes: severe stress
* Travels: * From a few hours to several months * Sudden “awakening” with confusion
* Amnesia for the fugue episode
Risk factors for dissociative identity disorder
Abuse in childhood a common factor:
Characteristics of dissociative identity disorder; subpersonalities
Dissociative identity disorder:
characterized by alternating between
multiple self states (subpersonalities
or alters)
The “host” has executive control most
of the time
“Switching” is usually sudden
Relationships among subpersonalities
* Mutually amnesic relationships
* Mutually cognizant patterns * One-way amnesic relationships
- Often display dramatically different: * Identifying features
- Abilities and preferences
- Roles
- Alters often display psychobiological differences
Theoretical explanations for dissociative identity disorder
Psychodynamic view: repression
Behavioral view: response learned through operant conditioning (dissociation is an escape behavior)
State-dependent learning: rigid state-to- memory links
* Self-hypnosis
Depersonalization and derealization
Derealization: “The world doesn’t feel real”
Depersonalization: “I don’t feel real”
- Lifetime prevalence: 1-2% * Equal numbers of males and females * Mean age of onset: 16 years old
- Comorbid conditions include mood or anxiety disorders.
Considered a disorder when:: * Depersonalization or derealization occurs on its own (that is, it
is not caused by drugs or another mental disorder), and it
persists or recurs. * The symptoms are very distressing to the person or make it
difficult for the person to function at home or at work.
Symptoms of depression
a low, sad state in which life seems dark and its
challenges overwhelming
- Emotional * Motivational * Behavioral * Cognitive symptoms
- Physical symptoms
Postpartum depression with psychosis (Andrea Yates case)
- Postpartum psychosis constitutes a medical emergency. * Case example: Andrea Yates * Developed post-partum psychosis after her 4th child
- Took Effexor and the antipsychotic Haldol off and on * Told of risk if she had more children * After the birth of her 5th child in 2001 she developed
postpartum psychosis again and drowned her 5 children - Diagnosed with peripartum-onset depression with psychotic
features
Charged with capital murder. Found not guilty by reason of
insanity in 2006. Committed to a high-security mental health
facility for an indefinite stay
The cognitive-behavioral perspective and the family-social perspective regarding the causes of depression
- Behavioral dimension
- Learned helplessness
- Negative thinking
Treatment includes
Interpersonal Therapy (IPT) * Focuses on changing
maladaptive interaction
patterns in relationships
* IPT as effective as
medications or CBT for
unipolar depression
Efficacy of the different classes of antidepressants
SSRI’s: * SSRIs inhibit the reuptake of serotonin following its release
into the synapse
* Fewer side effects
* Not fatal in overdose
* No more effective than other types of antidepressants
SNRI’s: * Serotonin and norepinephrine reuptake inhibitors (SNRIs)
(e.g., Effexor and Cymbalta) * Similar side effects to SSRIs
* Relatively safe in overdose
* Slightly more effective than SSRIs
Bupropion: Dopamine and norepinephrine reuptake inhibitor * Lacks side effects associated with SSRIs
* Side effects can include anxiety, restlessness, tremor,
insomnia, seizures
* Also marketed as a smoking cessation (Zyban)
Fail to help at least 40% of clients with depression
* Actual failure rate is likely higher (publication bias); study
designs * Most effective for severe depression
Issues with Study 329 (paroxetine)
Article used to promote paroxetine for
off-label use in teenagers
* Ghost-written by PR firm * Misrepresented negative effects
* Misrepresented efficacy
Article was never retracted
Ketamine and depression
- Ketamine: 80% efficacy for treatmentresistant depression * Esketamine: approved by FDA in 2019
(article on Sakai) * Dangerous without medical supervision
Brain stimulation treatments for depression
Electroconvulsive therapy
* Vagus nerve stimulation
* Transcranial magnetic stimulation * Deep brain stimulation