exam 2 reverse cards Flashcards

1
Q
  1. insomnia/hypersomnia
  2. psychomotor agitation or retardation
  3. feelings of worthlessness or guilt
  4. hard to think or concentrate
A

Major Depressive Disorder Symptoms

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2
Q

two ppl can have depression and have different symptoms

A

heterogenous presentation

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3
Q

loss of pleasure, despair, lack of mood reactivity
highly heritable and associated w childhood trauma

A

MDD with melancholic features

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4
Q

mood reactivity, weight gain, hypersomnia, leaden paralysis

A

MDD with atypical features

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5
Q

delusions or hallucinations

A

MDD with psychotic features

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6
Q

immobility to extensive psychomotor activity
mutism & rigidity

A

MDD with catatonic features

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7
Q

episodes occur and resolve at the same times, no other nonseasonal episodes

A

MDD with seasonal pattern

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8
Q

less intense, more chronic
worse outcomes, equal impairment

A

persistent depressive disorder

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9
Q

Manic episode
1. elevated, expansive, irritable mood
2. psychotic symptoms
3. lack of insight
4. circumstantiality (over-explaining)
5. tangentiality (not getting back to question)
6. clanging (alliteration & rhyming)

A

Bipolar I

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10
Q

Hypomanic episodes
shorter duration, no psychotic symptoms or grandiosity

A

Bipolar II

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11
Q

numerous period of hypomanic and depressive symptoms without meeting criteria for episodes

A

Cyclothymic Disorder

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12
Q
  1. helps slow down
  2. learn to avoid painful relationships
  3. make corrections for next time
A

how are low-level depressive symptoms adaptive

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13
Q

Nature
1. Genes (serotonin transporter gene)
2. Neurotransmitters (imbalance theory incorrect)
3. Hormones (cortisol, hyperthyroidism)
4. Brain influences
5. Biological rhythms (sleep, seasons)
Nurture
1. Stress
2. Psychological factors

A

Causes of MDD

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14
Q

Excluded in DSM-5 so someone depressed following bereavement can get diagnosed
Pathologizing grief

A

Issue of Bereavement

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15
Q
  1. CBT! Negative cognitive triad–about self, world, and future–maintained by errors in logic and thinking (excessive responsibility, fortune-telling)
  2. Behavioral activation: reduce withdrawal & avoidance, increase positive reinforcement (schedule activities)
A

Therapeutic Treatments of MDD

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16
Q

Tricyclics
SSRIs
Esketamine nasal spray
Psilocybin-assisted treatment

A

Pharmacological Treatments of MDD

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17
Q

Individualistic = higher manic
Collectivistic = higher depressive

A

Culture in Bipolar

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18
Q
  1. Neurotransmitters
  2. Brain structure (frontal-limbic)
  3. Hormones (HPA axis, thyroid)
  4. Genetics
  5. Biological rhythms
A

Causes of Bipolar

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19
Q
  1. Mood stabilizers (Lithium)!
  2. Family-focused treatment
  3. Psychoeducation
  4. CBT
A

Treating Bipolar

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20
Q

Add to typical functioning
1. delusions and hallucinations
2. disorganized speech and behavior

A

Schizo Positive Symptoms

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21
Q

Deficit in typical functioning
1. Flat affect (no facial express)
2. Anhedonia (cant experience positive emotions)
3. Asociality
4. Avolition (apathy)
5. Alogia (poverty of speech)

A

Schizo Negative Symptoms

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22
Q

Major mood episode (depressive or manic) with delusions/hallucinations.
Delusions/hallucinations in the absence of a major mood episode.

A

Schizoaffective Disorder

23
Q

Schizophrenia-like psychosis but shorter

A

Schizophreniform Disorder

24
Q

Delusions & no other psychotic symptoms
Erotomanic, Grandiose, Jealous, Persecutory, Somatic

A

Delusional Disorder

25
Mostly positive symptoms of schizophrenia lasting at least 1 day to less than 1 month
Brief Psychotic Disorder
26
Highly heritable Prenatal factors (exposure to infection, complications, malnutrition) Communication deviance Expressed emotion
Genetic Influence on Schizo
27
Loss of brain volume--frontal & temporal lobes (amygdala, hippocampus, thalamus)
Brain Abnormalities in Schizo
28
Urban living Immigration Socioeconomic status Better prognosis in less industrialized nations (less expressed emotion & less long-term maintenance pharmacology)
Social Factors on Schizo
29
Antipsychotics (GOLD STANDARD) 1. Thorazine 2. Haldol 3. Zyprexa 4. Seroquel
Pharmacological Treatments of Schizo
30
Family therapy Psychoeducation ACT
Psychosocial Treatments of Schizo
31
Support network, intensive treatment at home Demedicalize the illness
Finland's Open Dialogue for Schizo
32
Relate body sensations to catastrophic illness High negative affect, alexithymia (trouble naming emotions), history of childhood sickness/absence
Somatic Symptom Disorder
33
CBT: induce symptoms to show they're harmless; prevent body scans and reassurance-seeking
Treating Somatic Symptom Disorder
34
No somatic symptoms High level of anxiety about health
Illness Anxiety Disorder
35
Begins after stressor, psych symptoms become physical Not explained by known medical condition
Conversion Disorder
36
Eclectic: Successive exercises with reinforcement for progress. Reinforcements for symptoms are eliminated
Treatment of Conversion Disorder
37
Commonalities: 1. Prevalence 2. Demographics 3. Motor symptoms, loss of consciousness, pseudo-seizures Differences: 1. Symptoms in asia and africa include unresponsiveness, peppery sensations 2. Overlap with culture-bound syndromes
Culture and Conversion Disorder
38
loss of awareness for important personal info, a way of managing stress and anxiety
Dissociative Episodes
39
Sense of self and of one's own reality temporarily lost
Depersonalization
40
Sense of reality of the outside world temporarily lost
Derealization
41
Inability to recall previously stored info: Name, Address, Occupation, Family. Retrograde--impaired episodic and autobiographical memory
Dissociative Amnesia
42
Individual leaves home. When home, amnesia for fugue period sets in
Dissociative Amnesia with Fugue
43
Remove patient from threatening situation, recover and work through memories
Treatment for Dissociative Amnesia
44
2+ identities rotate in controlling behavior. "Switching" is sudden, often involuntary
Dissociative Identity Disorder
45
1. Movies and media 2. Increased attention to history of child abuse 3. Diagnostic criteria 4. Therapists subtly suggesting multiple personalities
Reasons DID is Increasing
46
Posttraumatic theory: reaction to severe childhood trauma (diathesis-stress) Sociocognitive theory: highly suggestible person adopts different identities as clinicians suggest, legitimize, and reinforce them (biopsychosocial)
How DID Develops
47
Eclectic: relying most heavily on insight-oriented techniques (psychodynamic)! Goal is to merge sub-personalities. After merging, therapy needed to maintain complete personality and prevent future dissociations
Treating DID
48
Binge eating and purging to prevent weight gain
Bulimia Nervosa
49
Strict dieting Tension and cravings Binge eating Purging Shame and disgust Repeat
Binge-Purge Cycle
50
Antidepressants and CBT
Treating Bulimia
51
Recurrent episodes of binge eating with no purging
Binge-Eating Disorder
52
Interpersonal Therapy! Antidepressants + CBT
Binge Eating Disorder Treatment
53
Denial Pride Ambivalence (baggy clothes) Preoccupation with food
Anorexia Nervosa Features
54
First restore weight Medications: Olanzapine (antipsychotic that helps weight gain) For teens: Family therapy For adults: CBT, IPT, general supportive therapy
Anorexia Treatment