EXAM 2 Flashcards
Cognitive Impairments of Aphasia
Attention: left neglect in RH stroke
Memory: WM in language processing
Executive Functioning: difficulty in discourse
Fluent Aphasia
Damage to the posterior portion of language center in the dominant hemisphere.
Talk with easy flow
Normal/fast rate, good intonation and stress.
WFD
Nonfluent Aphasia
Damage to the anterior portion of language center in DH.
Omit grammatical morphemes or function words.
Slow, effortful, frequent pauses, disrupted prosody.
Fewer than normal words.
WFD
Types of Fluent
Wernicke’s (poor repetition)
Conduction (poor repetition)
Transcortical Sensory (Good repetition)
Anomic (good repetition)
Types of Non-Fluent
Global (poor repetition)
Broca’s (poor repetition)
Transcortical Motor (good repetition)
Mixed Non-Fluent (good repetition)
Non-fluent Global
Alert
Language Characteristics: All language functions severely affected. impaired reading, writing, word retrieval, auditory comp, speech production.
May have preserved ability to read simple words and recognize pictures and gestures.
Unable to produce even a single word verbally.
Most have cognitive and motor deficits
Worst prognosis for recovery
Non-Fluent Broca’s
Comprehension: relatively preserved. Comp of syntactically complex sentences is often impaired
Oral Expression: Anomia, shorts phrases, halting, effortful, confluent speech, agrammatic output, may have some over-learned phrases.
Poor reading and writing.
Top-Down Processing
Use context and experience to function.
Does not involve language.
I.E. drama in a foreign language
preserved intelligence and cognitive functions compared to CDD
Non-Fluent Transcortical
Non-fluent and lack of spontaneous speech.
Agrammatism, short and telegraphic phrases
Short incomplete utterances that may contain some grammatical words.
Unusual delays in initiation
Fairly good auditory comp
repetition better than other nonfluents
Mixed Non-Fluent/transcortical
Between global and Broca’s (better than global worse than Brocas)
Recover from Global
Verbal output is limited to stereotypic utterances.
Poor auditory comp
Severe anomia
Better repetition
Fluent Wernickes
Comprehension: Severly impaired, great difficulty understanding speech of both others and themselves.
Oral Expression: Poor repetition, anomia (semantic and phonemic paraphasias), fluent with good prosody, well articulated, rapid rate, use jargon, Paragrammatism, paraphasia.
Alexia and agraphia
Anosognosia: inability to recognize their own deficits
Fluent Transcortical Sensory
Like Wernicke’s but good ability to repeat.
Language: fluent spontaneous speech with ease of articulation, good prosody, poor auditory comp, good repetition function.
poor word retrieval with more semantic paraphasias
Fluent Conduction
Damage to arcuate fasciculus.
Fair to good aud. comp
Fluent spontaneous speech
Fair articulation and prosody.
Anomia: may disrupt speech flow and use circumlocution
Significantly poor repetition
Show awareness and self correct
Fluent Anomic
The least severe aphasia syndrome.
Marked difficulty with naming.
No other significant receptive and expressive deficits
Fluent speech but pauses due to WFD.
May have difficulty with categories (grammatical or semantic)
Aware of deficits
Which Aphasias can repeat?
Mixed transcortical
Transcortical Motor and Sensory
Anomic
Subcortical Aphasia
Results from damage to the subcortical regions.
Diaschisis
Symptoms depend on site and severity
Crossed Aphasia
Occurs when a persons language center is not in the expected hemisphere
Unclassified aphasia
Possible Reasons:
Unusual combination of lesion sites within LH
Atypical cerebral dominance and individual variability of brain organization
Other medical conditions
Assessment of Naming
Anomia: common LH stroke symptom
Word retrieval depends on lexical semantic, lexical network, and lexical phonologic word forms
May show category specific deficits
Diagnosis of Aphasia: Anomia
WFD
Tasks: Confrontation Naming
Automatic-closure naming
Responsive naming
Generative Naming
Discourse