Exam 2 Flashcards
ADHD diagnosing
symptoms last more than 6 months
symptoms begin before 7 years old
To diagnose ADHD before 17, pt must have
6 or more of ADHD characteristics
Stimulant drugs for ADHD
Dextroamphetamine - stimulant, loss of appetite, sleep problems, headaches
Atomoxetine - decreased appetite, N/V, sleep disturbance
Nonstimulant ADHD meds
Norepinephrine reuptake inhibitors
ADHD meds should be given to kids _ food
AFTER
so they eat
Monitor ADHD kids for…
Give meds early so they can _
weight loss
sleep
Therapeutic play for ADHD
Promotes communication
Increases understanding of thoughts and feelings
ADHD drugs should be stored safely to prevent
accidents or abuse
Behavioral therapy for ADHD
Positive reinforcement
Age appropriate consequences
Refer ADHD fam to
local support groups
national ADHD support groups
3 types of hyperactivity
Impulsive
Inattentive
combined
When is autism IDd
between 12 months and 36 months
Autism characteristics
little eye contact few facial expressions difficulty building relationships low emotional affect lack spontaneous enjoyment
Warning signs of autism
no babbling by 12m
no pointing or gesturing by 12m
no words by 16m
no 2-word utterances by 24m
LOSS or Regression of skills
Therapies for autism
behavioral and communication
Autism patients respond well to _ education
structured
Autism stereotyped motor behavior
Hand flapping
Body twisting
Head banging
Autism screenings
MchatR
SCQ Social communication questionnaire
PDDST pDevelopmental disorder screening test
Interventions for autism
emotional support
guidance and edu
Routine
Controversial treatment for menopause
HRT
Hormone replacement therapy
If a menopause woman still has her uterus her HRT will have
Both estrogen AND progestin
Dosing for HRT
Lowest effective
Shortest duration
HRT contraindications
Hist of breast cancer Uterine cancer Vascular thrombosis Impaired liver function Undiagnosed uterine bleeding
HRT patients need to understand they are taking
HORMONES
S/S of DVT
HRT risk
leg redness and pain/tenderness
S/S of PE
HRT risk
chest pain and SOB
Patient maintenance for HRT therapy
Weight bearing exercises
v in smoking/alcohol
Annual mammogram
Take calcium and vit D
Does intellect decline with age
NO
just get a little slower
Is depression normal in older adults
NO
Neither is loneliness
Aging and heart
HF
Atherosclerosis
Aging had lungs
Loss of elasticity
Difficulty breathing
Aging and GU
Incomplete emptying, creates need to pee
Leads to fall risk and infection
Aging and GI
v metabolism
constipation
+fiber
Aging and senses
Vision Hearing Taste and Smell
ALL DECREASE
PT will add salt
TB spread is
Airborn
N95
Tuberculosis 101
airborne
multiply in alveoli and get transported via blood and lymph
as infected cells die they accumulate in lung causing bronchopneumonia
Can recur
TB infection occurs to weeks after exposure
2 to 10
S/S of EARLY TB
insidious
Low fever
cough
sweats
fatigue
weight loss
As TB progresses S/S includes
Mucopurulent sputum expectorate
Hemoptysis - blood in cough
Elder TB patients have _ pronounced symptoms
Less
If pt presents with positive skin, blood or sputum, for TB
Do history, physical exam, Chest xray.
Drug susceptibility testing.
5mm or greater on TB test
When is test read
Will it be elevated
Positive
48-72h
Yes
Blood tests for TB
QuantiFERON Gold
T-Spot
rules out Active and Latent infections
Good for BCG vaccinated people
TB meds
Anti-TB agents
INH, Rifampin, Pyrazinamide, Ethambutol
Given for 6 to 12 months
Prolong treatment to ensure eradication
TB drug resistance
Primary - resistance to ONE drug in people who have not had previous treatment
Secondary - resistance to ONE OR MORE in people undergoing therapy
Multi - resistance to TWO agents
TB treatment phases
phase 1: all drugs + Vit B6 for 8 weeks
phase 2: INH and rifampin for 4 to 7 months
Nursing priorities with TB
Airway clearance
Adherence to meds
^ Activity and nutrition
Prevent transmission
Airway clearance with TB interventions
^ fluid intake
Postural drainage
Adherence to meds with TB
Take meds on empty stomach or 1h before meal
Food that messes with TB meds
Tuna
Aged cheese
Red wine
Soy sauce
Yeast extract
Rifampin makes what meds less effective
Warfarin
digoxin
corticosteroids
Contact lenses and rifampin
Will be discolored
switch to glasses
Side effects of antiTB meds
Liver/Kidney problems (BUN, creatinine, enzymes)
Hearing loss
Rash
Prevent transmission with TB
Cover mouth
dispose of tissues
Hand hygiene
Do you report TB to the health department
YES
RA patho
Immune system attacks joints causing effusion, pain and edema
After triggering event, pain subsides pannus (extra growth) occurs
Destruction of joint cartilage and bone
Pannus
Proliferation of new synovial joint tissue WITH inflammatory cells already formed
occurs due to RA
RA is autoimmune
body attack self
S/S of RA
PAIN
joint swelling
limited movement
stiffness
weakness
Diagnosing RA
RF rheumatoid factor ACPA ESR erythrocyte sedimentation rate C reactive protein CRP Radiology
Rx management of RA
salicylates (aspirin)
NSAIDs
DMARDs (work on autoimmune response)
None Rx management of RA
Heat/Paraffin baths 20 min max
Therapeutic exercises
Braces, splints, assistive devices (canes)
Treatment goals for RA
v inflammation
control pain
^ mobility
^ PT knowledge
RA exercise and activity
Physical/occupation therapy
TENS
relaxation techniques
Sleep with RA can be aided by
Low dose antidepressants
Amitriptyline
Good sleep hygiene (cold room, no tv, no eating in bed, etc.)
What joints are affected first by RA
fingers
wrists
toes
small joints first
clues for RA problems in elderly
Gait pattern change
Guarding
Joint flexion
3 Distinct characteristics of RA
Inflammation
Autoimmunity
Degeneration of cartilage
RA
Swan neck
Boutonniere
finger appearance ^v-
finger appearance ^-
Caution with biologic DMARDS
Disease modifying antirheumatic drugs
Suppress autoimmune response
RA joint deformities can result in
loss of use
Contractures
muscles bones joints get stuck due to joint destruction RA
When in they day is the pain worse
MORNING
ON TEST
RA foot deformities
Major bunion
Toes going in different directions
Why do joint deformities happen
destruction and recalcification of tissue
Biggest difference between lupus and RA
Degeneration and destruction of tissue with RA
ON TEST
NSAIDS CORTICOSTEROIDS DMARDS
with RA
STAY AWAY FROM PEOPLE
HIGH INFECTION RISK
DMARDs
stop progression but can not fix damaged joints
does not work indefinitely
Nonbiologic - take longer to work then biologic
Infliximab DMARD
Works to improve the immune system
Anaphylaxis
Type I hypersensitivity
Rapid release of IgE
severe life-threatening reaction
Common causes of anaphylaxis
Antibiotics and radiocontrast agents
Penicillin most common culprit
The faster the onset of anaphylaxis
The more severe the reaction
Mild anaphylaxis S/S
tingling and warmth
fullness in mouth and throat
nasal and periorbital swelling
sneezing and tearing of eyes
onset is first 2h of exposure
Moderate anaphylaxis S/S
flushing/warmth/itching
Anxiety
Bronchospasms
edema or airways
cough/wheezing
onset within first 2h
Severe anaphylaxis S/S
Abrupt onset
severe dyspnea
cyanosis
hypotension
V/D
seizures
Treatment for anaphylaxis
Strict avoidance
Epinephrine (EpiPen Auvi-Q)
Med management of anaphylaxis
O2
Epi
Antihistamines
Corticosteroids
IV fluids
After treatment for anaphylaxis watch for rebound reaction which happens…
4 to 8 h after
Nursing management of anaphylaxis
Check airway breathing and vitals
Notify providers
Instructions after recovery, like what to avoid and getting an epipen
Emergency nursing measures for anaphylaxis
Intubating
Admin emergency meds
IV lines + fluids
O2 admin
Anaphylactoid reaction
Not same as anaphylaxis
NON IgE
No sensitization
Fluids with Anaphylaxis
Increased permeability of capillaries
Fill up with fluid in lungs
N/V/D etc.
If you have Mild anaphylaxis now
DOT NOT mean will have Mild anaphylaxis next time
Solution to push IV for anaphylaxis
Isotonic
LR
NS
Nursing management basics for anaphylaxis
Stop allergen
Give epi FIRST, IV, O2
If you have allergy
awareness
wear bracelet
Inform school nurse/teachers
ask for return demonstration when showing how to use
Hold epipen in injection place for
FULL 10 SECONDS
Crohn’s disease 101
Chronic inflammation of GI
Most common in distal ileum and ascending colon
Crohn patho
small lesions that expand and thicken becoming fibrotic
intestinal lumen narrows
S/S of crohn
prominent right lower quadrant pain
unrelieved diarrhea
pain occurs after meals
Secondary complications of crohns
weight loss
malnutrition
anemia
stretorrhea and chron
fat in feces
Assessment for crohns
CT and MRI
Crohn bowl complications
obstruction
structural problems
perianal disease
Enterocutaneous fistula - opening between small bowels and skin
Enterocutaneous fistula
opening between small bowels and skin
Ulcerative colitis 101
ulcerative inflammatory disease of mucosal and submucosal layers of colon and rectum
Characteristics of Ulcerative colitis
Abdominal cramps
Bloody/purulent diarrhea
LEFT lower quadrant pain
Weight loss
six or more liquid stools a day
Patho of ulcerative colitis
mucosa become edematous and inflamed
colonic epithelium sheds
Eventually the bowel narrows, shortens and thickens
Assessment for ulcerative colitis
abdominal x ray
colonoscopy
Ulcerative colitis may lead to toxic mega colon this is treated with
NG suction
IV fluids
Lytes
Corticosteroids
Antibiotics
SURGERY
Diet for ulcerative colitis
Oral fluids
Low residue
High protein, high calorie diet
Supplemental vitamins, iron
Food to avoid with ulcerative colitis
cold food
milk
Rx or ulcerative colitis
sedatives antidiarrheals antiperistalitics for diarrhea
Aminosalicilates to reduce inflammation
Corticosteroids to reduce swelling
Immunomodulators to treat underlying cause
Partial or complete obstruction or bowels =
surgery
Colectomy and ileostomy
removal of colon and stoma for drainage
used in IBD problems
Normal elimination nursing interventions
food diary
give meds
increase fluid intake
record frequency and consistency of stool
Pain nursing interventions for IBD
Analgesics
Position changes
Heat
When doing parenteral nutrition for IBD patients monitor
glucose q6h
Mental health and ulcerative colitis
Promote rest
Reduce anxiety
Enhance coping measures
With bedridden IBD patients monitor for
skin breakdown
2 types of TB
Active- showing symptoms, neutrophils and macrophages fight infection
Latent- NOT active but carrier of encapsulated bacteria
Most obvious TB S/S
Cough lasting more than 3 weeks
low grade fever
blood in sputum
if PPD test is greater than 10mm
POSITIVE
Induration (raised) MUST be present
PPD test is considered
SCREENING not diagnostic
TB GOLD, Chest xray and Acid fast are diagnostic
ACID FAST CULTURE IS THE GOLD STANDARD OF TB TESTING
Standard number of meds to take for TB
4
isoniazide
Rifampin
Pyrazinamide
Ethambutol
All TB meds are toxic to
LIVER
do enzyme tests
For tb take B6
take meds 1 h _ meals
when to take
to prevent neuropathy
before
SAME TIME QDAY
When is a TB patient not contagious
after 2-3 weeks
AND
3 negative sputum cultures
AND
Med compliant
when are sputum samples done for TB
Q2-4W
Helps track progress
S/S or hepatotoxicity
S/S nerutoxicity
Jaundice, Right upper quadrant pain, Fever
Numbness, tingling
for alergic reactions do you shut the med off first or give epi
SHUT THE MED OFF
Ulcerative colitis only affects
Starts at
Mucosal and Submucosal layers
Low part of rectum and works its way up
Crohn’s disease affects
All layers of intestine from mouth to butt but not continuously
here and there
movement number
Ulcerative colitis UC pain will start at
15 to 20 bowel movements
Left lower side
Bleeding
movement number
CD pain
5 stool movements
pain in right lower quadrant
UC complications
Loss of Haustra - smooths out intestine, these parts can absorb nutrients
Toxic megacolon - colon dilates, inflammation. Unable to contract, distends, becomes paralyzed
Cobblestone intestine is related to
CD
IBS labs
Albumin will be LOW malnutrition
ESR, C reactive protein - inflammation
Stool sample
CBC
Chem panel
IBS diagnostics
MRE - magnetic resonance enterography
MRI/CT
Sigmoidoscopy
Colonoscope
Endoscopy