Exam 1 Flashcards

1
Q

The mind body problem

A

How can a physical substance give rise to our feelings, thoughts and emotions?

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2
Q

Story of Thomas Willis and his most lasting contribution to neurology

A

Father of clinical neurology -linked brain damage to specific behavioral deficits -documented anatomy and disease Linked brain morphology with cognitive dysfunction by folioing patients for many years and then doing postmortem dissections

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3
Q

Phrenology

A

Franz Gall Localizationist view -attributed individual cognitive functions to specific brain regions -using a mental function cause corresponding brain region to grow bigger, creating bumps on skull -put mind in brain

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4
Q

Holism vs. Localism (role of Broca and Wernicke in debate)

A

Holism -whole brain participated in each behavior Both partially right -specific process can be localized to single brain region -complex functions are carried out by many brain regions acting in concert Broca’s aphasia -inability to generate speech Wernicke’s aphasia -comprehension loss

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5
Q

What is cognitive neuroscience?

A

Understanding the critical link between brain and behavior Understanding early misapplications of this principle

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6
Q

Four lobes

A

Dorsal (superior) HEAD Rostral (anterior) Caudal (posterior) Ventral (inferior) Frontal lobe Parietal lobe Temporal lobe Occipital lobe

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7
Q

Gyri and Sulci (e.g., central sulcus, calcarine sulcus, intreparietal suclus)

A

Gyri (protrusion) Sulci (invagination) Precentral sulcus Central sulcus Postcentral sulcus Interparietal sulcus

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8
Q

Orientation references (e.g., coronal)

A

Medial- toward the inside Lateral- toward the outside Coronal- skull to jaw cut from ear to ear Axial- cut from eye to bump Sagittal- skull to jaw, eye to back

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9
Q

Brodmann’s areas (how they are defined)

A

Based on different cell types (cytarchitectonics) in postmortem brain V1- Visual processing, primary visual cortex (far back of head- occipital lobe)

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10
Q

General differences between our brains and other animals

A

?

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11
Q

Methods of stimulation

A

Selective damage through natural accidents -which necessary for specific cog. function Invasive stimulation -strengths: precise temporal and spacial stim, see direct and downstream effects -weaknesses: invasive Non-invasive stimulation (TMS & tDCS) -strengths: good temporal resolution, spacial precision, non-invasive -weaknesses: limited to brain surface, stimulation vs. disruption not fully understood (must be combined with other techniques)

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12
Q

Methods of measuring natural signals

A

Electrophysiological -single cells recording Imaging: MEG/EEG -strengths: high temp resolution, non-invasive -weaknesses: localization hard, need many events fMRI -strengths: spacial, functional networks and props -weaknesses: indirect, expensive

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13
Q

Single vs double dissociation

A

Single

  • Two groups, one task minimum; differ for one task, but not other
  • weak evidence
  • ex. temporal (good spacial, bad object) parietal (good spacial, bad object)

Double

  • two groups, two task minimum; one impared on one task, other impared on other task
  • strong argument
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14
Q

Principles of choosing research methods

A

If interested in temporal info -> ERPs/EEG If interested in localizing a cog function -> fMRI/MEG Correlational or causal rel?

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15
Q

Pathway of visual info up to primary visual cortex

A
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16
Q

Organization of V1

17
Q

Functional specialties of V5/MT and V4

A

MT (middle temporal)

  • motion (lesion creates deficit)
  • dorsal pathway

V4

  • color (lesion causes no color perception)
  • ventral pathway
18
Q

Cortical magnification and functional overrepresentation

A

Mapping and perception is not fixed

19
Q

Visual hierarchy

20
Q

Illusory filling-in (examples)

A

Reconstructive

Example: lines

21
Q

Case study: how can emotion affect perceived brightness?

A

Positive -> brighter Moral behavior increases brightness -good and evil, metaphoric illusion

22
Q

Cortical plasticity/reorganization

A

Mapping in perception is not fixed

23
Q

What vs. where/how pathways of infor and supporting evidence

A

Where: Dorsal stream- position task (how)

  • Size of receptive feild increases along dorsal; spacial selectivity preserved
  • Interhemispheric comm: more unilateral representation
  • Lateral function deficits: lesion IPS -> gross and incorrect misreaching for target in leteral peripheral vision

What: Ventral stream- object task

  • RF size increases along stages (integrates over previous stages); central vision; more select to stimulus properties
  • Interhemispheric comm: bilateral representation
  • Lateral function deficits: lesion LOC -> cannot recognize objects, but know how to act on them

Evidence: temp area removed-> spatial irrelevant; parietal -> object irrelevant

24
Q

Grandmother cell vs. Ensemble or population coding for object recognition

A

Grandmother

Highest level representation

Each neuron responds to one specific object (knows everything about X)

Ensemble

Many neurons code parts of object together

individual cells have continuos coding for a certain feature

Need flexibility and specificity

25
Optic ataxia vs. object agnosia
Object agnosia -cannot ID objects Optic ataxia - lack coordination
26
Signatures of face processing in cog psych, cog neurosci and neuropsyc their role is debate about whether faces are special or not
Composite face effect and face inversion effect suggest that face regonition is critically dependant on a hollistic pros of facial components
27
Hollistic vs. expertise
Hollistic (images of faces or objects) Expertise (expert at objects, equiv to face recognition by long term expertis- trade off)
28
Case study: Jennifer Aniston same as grandmother cell?
Same idea