exam 1 Flashcards

1
Q

natural passive vs natural active

A

passive: gets passed down from mother to baby
active: person is exposed nd makes the antibodies for it

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2
Q

artificial passive vs artificial active

A

passive: person get injected without other antibodies
artificial: vaccines

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3
Q

Preventing
Primordial? Primary? Secondary? Tertiary? Quaternary

A

Primordial: community
primary: lifestyle
secondary: early detection with exams
tertiary: helping with chronic illness
quartinary: medicine reconciliation

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4
Q

Hyperglycemia
what is it?

A

-the body either stops making insulin or the receptors become resistant
-this causes the liver to release glucagon which ultimately increases the sugar in the body
-sugar is more than 126 mg/dl or more athn 180 after eating

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5
Q

Hyperglycemia
manifestations?
treatment?
labs?

A

-polydipsia/phagia,low BP , tachycardia , confusion
-can lead to DKA, fruity breath,N/V,GI issues
-HbA1c:

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6
Q

what happens if it becomes too advanced?

A

DKA and kussmaul respirations which is deep rapid breathing to expel 02

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7
Q

hypoglycemia

A

-failure of glucose homeostasis which will be less than 70mg/dl
-CAN HAPPEN IF HAVE EXCESS INSULIN
-if low enough, nor/epinephrine will be released to increased sugar

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8
Q

hypoglycemia
RF? Manifestations?
labs?

A

RF: insulin dependent, DMT1, time change, abx, beta blockers
-symptoms: tachycardia, hunger, anxiety, blurry vision, chills/sweating
15-15-30 rule: eat 15 grams of complex carbs

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9
Q

what is neuroglycopenia?

A

this is when the sugar is too low and the neurons start to malfunction which causes the dizziness and confusion

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10
Q

what are the three levels of hypoglycemia?

A

level 1: 54-70; tremors, diaphoresis,confusion
level 2: less than 54 mg/dl; erratic behavior, can’t move
level 3: doesnt have level but causes seizures, coma, agitioan or DEATH

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11
Q

Type 1 DM
RF? labs?

A

-body destroys the insulin making cells
-HbA1c: more than 7%
-RF:genetics, age
-more prone to heart attacks and strokes
-non fasting: 200 or more
fasting: 126 or more
-labs: urinalysis (ketones/proteins), low peptide
-can lead to dka
-THE THREE Ps

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12
Q

what is the normal level of HbA1c?

A

its 6.5-7

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13
Q

is type 1 DM associated with hypoglycemia?

A

its associated with both

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14
Q

type 2 DM
RF? clinical presentation? labs?

A

-insulin is produced but not enough , resistance
-RF: older than 45, sedentary lifestyle, bad diet, poor sleep
-fasting higher than 126 or non fasting two hours post prandial 200 or more
HbA1c: more than 6.5
- can be treated with metformin or canaglifozin

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15
Q

metformin:

A

-its oral hypoglycemic which lowers liver production of glucose
-can cause indigestion, bloating, metallic taste

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16
Q

canagliofozin

A

-oral hypoglycemic: helps kidneys remove more glucose
-can cause fungal infections, increased urin output
-MONITOR RENAL FUNCTION

17
Q

metabolic syndrome

A

-INSULIN RESISTANT, high bp, glucose and cholesterol, central obesity
-can be reversed by eating healthy
-can lead to DMT2, cardiac, and neurological problems
-treatment: metformin, or weight loss drug

18
Q

To recognize, metabolic syndrome look out for?

A
  • high cholesterol, hyperglycemia, central obesity and HTN
19
Q

Hypothyroidism

A

-high TSH, low T3/4
-common cause: iodine deficiency , hashimotos, thyroid surgery
-can cause memory impairment, fatigue, aches, stiff muscles, weight gain, thinning hair , feel colder cold intolerance
-meds: levothyroxine

20
Q

What could happen if left untreated for too long?

A

it will lead to myxedema coma which is when the body starts to show down, low temp, extreme fatigue, lethargy

21
Q

If hypothyroidism is overtreated, it will cause?

A

-tachycardia, fever, weight loss, sweating

22
Q

hyperthyroidism

A

-excessive hormone production
-can be caused by graves disease
-RF: DMT1, pernicious anemia,
-symp: swelling of eye tissues, tachycardia, fever, weight loss, heat intolerance, a fib, insomnia, tremors, loose bowel movement, BULGING EYES
-LOW TSH, HIGH T3/4
-treatment: beta blockers, anti thyroid meds PTU, eye drops (methylcellulose)

23
Q

If it gets severe enough, it will lead to thyroid storm which is?

A
  • high temp (104-106), tachycardia, hypotension, systolic HTN, N/V
24
Q

methimazole

A

-prevents thyroid from producing T3/T4
- adverse effects: liver toxicity
-contraindications: warfarin, beta blockers

25
PTU
-prescribed if methimazole doesnt work, blocks thyroid peroxidase
26
Death: early, middle and late stage?
-early: loss of mobility, decreased vein appetite, increased sleeping -middle: brief period of wakefulness, noisy respirations "death rattle" -late: fever w/ period of dyspnea, mottling of skin -can range from 24 hrs-14 days
27
clinical vs biological death
-clinical: heart and lungs stop working but brain is viable can use CPR -biological: brain is dead, no way of reviving, heart and lungs are on machines
28
what needs to happen for a person to be considered an organ donor?
- brain dead
29
Palliative care vs hospice
-palliative: improves quality while still receiving curative care -hospice: provides comfort while having less than 6 months left to live
30
HOPE
-used for spiritual assessment H-hope O- organized religion P- personal practices/spirituality E- effects on medical care
31
justice? nonmaleficence? beneficence? fidelity? autonomy?
-justice: treated equally -nonmaleficence: do no harm -beneficence: do right by the patient -fidelity: providers/nurses provide info about disease to patient
32
what are manifestation of impending death?
-urinary retention, hypotension, tachycardia, cheyenne stokes (shallow, rapid, period of apnea)