Exam 1 Flashcards
risk factors for sepsis
immunocompromised, CLABSI, open wounds, malnutrition, invasive procedures, cancer, older than 80, alcoholism, diabetes mellitus, transplants, hepatitis, HIV/AIDS, GI ischemia, Hepatitis, transplant recipient, CKD, infection with resistant oraganisms, mucous membrane fissures in prolonged contact with bloody/ drainage soaked packing
stages of sepsis
SIRS, Sepsis, Severe Sepsis, Septic Shock, MODS
s/s of infection and sepsis
SIRS and confirmed infection
SIRS=2/4 criteria
body temp >100.5 or <96.8
HR>90
RR>20/PaCO2<32
WBC >12,000 or <4,000 or >10% immature bands
Infection:
fever
tachycardia
pain
swelling
warmth
redness
inc WBC
labs to assess for sepsis
sepsis labs: increased WBC, Decreased PLTs and Clotting factors, increased lactate, increased procalcitonin, BUN/creatinine, ABG, H&H, blood cultures, ALT, AST
labs to assess for infection progress
CRP(C-reactive protein), ESR (erythrocyte sedimentation rate) to track inflammation(is there a downtrend?), blood cultures, and labs listed for sepsis
nursing interventions for sepsis
hour-1 bundle
observe for organ dysfunction/worsening of s/s (decreased output, low BP, cyanosis, jaundice, etc)
qSOFA
SOFA
prioritization of nursing interventions for sepsis
hour-1 bundle
1. measure Lactate level
2. obtain blood cultures before adminstering antibiotics
3. administer broad-spectrum antibiotics
4. begin rapid adminstration of 30mL/kg crystalloid for hypotension or lactate > 4mmol/L
5. apply vasopressors if hypotensive during or after fluid resuscitation to maintain a mean arterial pressure >65 mm Hg
tx of sepsis
treat infection/fungal (broad-spectrum first, specific after blood cultures result), treat hypotension with vasopressors, start fluids 30mL/kg
prioritization of tx for sepsis
obtain blood cultures and lactate level, begin broad-spectrum abx, start 30mL/kg crystalloid fluid, adjust abx as blood culture sample gets back, monitor for worsening of s/s, monitor VS, and for rxns to abx and fluids.
adrenal function related to sepsis
decreased kidney function= toxins not released->furthering inflammation and decreasing perfusion r/t edema–> can indicate/lead to MODS
blood glucose levels related to sepsis
high glucose levels released from liver due to stress response; the more severe the response, the higher the glucose level
expected outcomes to sepsis tx
improvement in BP, return of WBC counts, increased O2 sat, decreased lactate level, increased perfusion, lack of edema, decreased procalcitonin
what has the highest risk of death over sepsis alone?
septic shock
DIC
disseminated intravascular coagulation
Disseminated intravascular coagulation; patho
microthrombi formed where not needed widespread; this consumes AVAILABLE PLTs and clotting factors and leads to hypoxia–> metabolic acidosis and organ dysfunction(inc hypoxia= inc metabolites-toxic-which amplifies inflammation and repeats poor gas exchange and perfusion)
discharge teaching for a pt at risk for sepsis
s/s of sepsis:
s/s of infection:
identify pt at risk for CV disease
male, over 40/45,smoker, alcohol, family hx, obese, sedentary lifestyle, african american, high cholesterol diet, uncontrolled HTN, uncontrolled stress
Modifiable risk factors
lifestyle, diet, exercise, smoking, drinking alcohol
non-modifiable risk factors
age, gender, ethinicity, family hx
NSR
normal sinus rhythm
describe normal sinus rhythm
impulse intiated by SA node
reg rhythm
rate 60-100bpm
normal P wave in lead
P wave before each QRS
normal PR, QRS, and QT intervals
sinus bradycardia
impulse initiated in SA node
sinus rhythm w/ rate <60
normal p wave
p wave before each QRS
causes for sinus bradycardia
vagal, drugs, ischemia, disease of nodes, ICP, hypoxemia, athletes(normal)
s/s of sinus bradycardia
can be asymptomatic
OR
confusion, SOB, CP, diaphoresis, syncope, lightheadedness, hypotension
sinus tachycardia
sinus rhythm with rate 100-150
normal P wave
p wave before every QRS
what does P wave represent
atrial depolarization
what does QRS complex represent?
ventricular depolarization
what does QT interval represent?
measures the complete ventricular cucles (depolarization AND repolarization)
what does T wave represent?
ventricular repolarization
normal values for P wave
0.12-0.2
normal values for QRS complex
0.06-0.10 seconds (longer=slower)
normal values for PR interval
0.12-0.20 seconds
normal values for QT interval
<0.44 seconds
how do you determine HR on an ECG strip?
find the tick marks, make sure strip is 6 seconds long. count the QRS complexes and multiply it by 10 (or count QRS in 30 boxes and multiply by 10)
how may seconds is one single box on an ECG strip?
0.04 seconds per small box
how many seconds is 5 boxes on an ECG strip
0.2 seconds for 5 boxes
what does amplitude measure on an ECG?
force of contraction
how is Time measured on an ECG strip?
horizontally
nursing interventions for sinus bradycardia
-monitor VS, monitor for confusion, SOB, CP, diaphoresis, syncope, lightheadedness, hypotension
-give fluids for hypotension
-anticipate orders for atropine or external pacing
nursing interventions for sinus tachycardia
assess for s/s: confusion, hypotension, lightheadedness, delay cap-refill, anxiety
monitor VS
anticipate medications
impact on beta-blockers on the heart
lower HR and BP
Risk factors for DVT/PE
endothelial injury, stasis of blood flow, hypercoagulable state (virchow’s triad)
Virchow’s Triad
endothelial injury, stasis of blood flow, hypercoagulable state
what falls under “endothelial injury”
part of Virchow’s triad for VTE risk factores:
surgery- Hip, knee, prostate
trauma
athersclerosis
smoking
catheter
what falls under the “hypercoagulable state”?
part of Virchow’s triad for VTE risk factors:
-malignancy
-congenital coagulation defect
-thrombophilia
-oral contraceptives
-inflammatory bowel disease
what falls under the “stasis of blood flow”?
part of virchow’s triad for VTE risk factors:
-immobility
-atiral fibrillation
-venous insufficiency
-venous obstruction
-Heart failure
diagnostics for DVT/PE
D-Dimer lab- tells if there is a clot that is breaking and floating
Venous Duplex Ultrasound- preferred method to dx DVT
non-surgical interventions for DVT/PE
early ambulation is the most important!
-SCDs
-ROM exercises
-TEDs
-elevation
-adequate hydration
-IS use
-coughing
-deep breathing
-turning while in bed
pt education about tx and prevention of DVT/PE
-prevent by walking frequently after procedures, perform gas pedal pumps and ROM in bed, wear compression stockings, maintain adequate hydration.coughing and deep breathing
-Rx for clot busters and anticoagulants may be given- enoxaparin/lovenox injection instructions or NOAC (novel oral anticoagulants)
-surgical:thrombectomy-remove clot by catheter aspiration suction
mechanical thrombectomy- blood clot is broken up into small pieces and removed
warfarin pt education
-vitamin K is the antidote- keep diet consistent, do not increase or decrease vitamin K consumption once therapeutic level of warfarin prescribed (leafy greens)
-s/s bleeding and prevention
-takes 3-4 days to be effective
-must have INR monitored as ordered (1.5-2)
-avoid prolonged sitting and crossing legs
-avoid smoking
-report abd pain
-pregnancy category X
-s/s hepatitis: jaundice, abd pain, nausea,etc.
ABG interpretation
pH level: 7.35-7.45 (acidosis or alkalosis)
PaCO2(respiratory):45-35
HCO3(bicarb;metabolic):22-26
pt prep for Chest X-RAY
education, no prep
Pt prep for thoracentesis
explain procedure and informed consent
supplies
pt position upright leaning on table
educate to remain absolutely still
VS
pt prep for bronchoscopy
-consent
-complete labs and diagostics needed
-NPO 4-8 hrs before
-remove dentures and oral prosthetics
-admin meds and topical anesthetics
pt prep for pulmonary function test
explain why tests are given: assess lung capacity and volumes, screen for respiratory complications and compare baseline overtime to others
-no smoking 6-8 hrs before
-no bronchodilator 4-6 hrs before
pt education for chest x-ray
used to monitor chest tubes insertion/placement daily
assess pulmonary issues, tubes,lines, evaluate condition of pneumothorax, pneumonia, abcess