Entrance test 6 Flashcards
Transient paralysis form of Mareks disease can be present in hens
f
Lymphoproliferative form of Mareks disease is an acute disease
t
Neurological form of Mareks disease can be present in 4 week old chicken.
f
There is in ovo vaccine against Mareks disease
t
Intake of GaHV2 happens most frequently per os.
f
Germinative infection does not occur in Mareks disease
t
Germinative infection does occur in Mareks disease
f
In case of viraemia the GaHV2 spreads to fowl’s organs in red blood cells
f
The acute form of Mareks is primarily a tumour formation.
T
Marek disease is usually seen in chickens below 2 weeks of age.
F
Marek disease virus is shed with faeces in high titres
F
Signs of Marek disease may be very similar to reticuloendotheliosis signs
T
The Marek disease virus survives for long in the environment
T
Marek disease is caused by turkey herpesvirus
F
Transient Marek paralysis may lead to visceral form
T
Transient paralysis by Marek’s disease usually ends in full recovery
F
The chronic form of Marek disease is characterized by neurological disorders
T
The chronic form of Marek disease is due to circuses of higher virulence
F
Transient Marek paralysis may lead to visceral form
T
) Marek disease is the result of air born infection.
T
Transient paralysis by Marek disease usually ends in full recovery.
F
Marek disease usually starts as an airborne infection
T
Free Marek virus particles are shed from the feather follicles.
T
The Marek’s disease virus may survive in the environment for several months
T
Marek’s disease is most frequently seen in geese and ducks.
F
Free virions of the Marek’s disease virus are formed in the feather follicle epithelial cells.
T
Humoral immunity plays the central role in the host’s defence against the Marek’s disease virus.
F
The neurological form of the Marek’s disease is seen only in day-old chicks
F
Chicken shed the Marek’s disease virus via feather follicular epithelial cells
T
The pathological lesions of acute Marek’s disease and reticuloendotheliosis can be similar.
T
Conjunctivitis is a frequent sign of acute Marek’s disease.
F
The Marek ǵs disease virus is transmitted by inhalation
T
Marek ǵs disease virus can ǵt be prevented by vaccination, because it ǵs immunosuppressive.
F
The pathology lesions of acute Marek ǵs disease and avian leucosis can be similar
T
The Marek ǵs disease virus causes immunosuppression.
T
The acute form of Marek ǵs disease is characterized by lymphoid cell proliferation.
T
The neurological form of Marek ǵs disease has a mass appearance
F
The highly virulent strains of Marek ǵs disease may cause tumours in turkey.
T
The incubation time of acute Marek ǵs disease is 1-2 day
F
The neurological form of Marek ǵs disease leads to significant liver degeneration.
F
Both serotypes of the Marek ǵs virus cause disease in geese.
F
North America is free of Marek ǵs disease.
F
Marek’s disease only occurs in domestic fowl.
T
Marek’s disease spreads primarily via the aerogenous route
T
Marek’s disease virus is shed mainly via the faeces
F
Marek ǵs disease in the blood multiplies in the endothelial cells.
F
Marek ǵs disease is a beta herpes virus.
F
Marek’s disease primarily targets day old chickens.
F
The neoplastic form of Marek disease is caused by high virulence strains.
T
Vaccination is used for the prevention of Marek ǵs disease.
T
The neoplastic form of Marek’s is caused by low virulence strains
F
We can differentiate Marek’s disease from reticuloendotheliosis only by lab diagnostics methods.
T
Prevention against Marek ǵs: no measures needed as the disease remains mostly symptomless.
F
General preventative measures and vaccination of day-old chickens must be used for the prevention of Marek’s disease.
T
Live attenuated strains are used for vaccination against Marek ǵs disease.
T
No vaccination is needed against Marek’s disease
F
The highly virulent strains of Marek’s disease may cause tumors in turkeys.
T
Marek’s disease viruses are uniform in their virulence.
F
Highly virulent Marek’s disease viruses may break through vaccine induced protection
T
Conjunctivitis is a frequent sign of duck viral enteritis
T
Only sporadic clinical cases of duck viral enteritis are seen in an affected flock
F
Mallards may transmit the duck enteritis virus to domestic ducks kept on lakes
T
Liver dystrophy is a frequent lesion of duck viral enteritis (duck plaque
T
Wild ducks may be persistently infected with duck plaque (duck enteritis) virus
T
Duck plaque virus may be shed life long by animals recovered from the disease
T
Duck plaque (viral enteritis) can’t cause high mortality without secondary bacterial infection
F
Duck plaque and duck viral enteritis are two names of the same disease
T
Duck viral enteritis is seldom fatal
F
Treatment is the most effective control method for duck viral enteritis.
F
Duck plague is more sever in wild birds than in domestic ducks
F
Duck plague virus damages blood vessel endothelium
T
Duck plague only affects young ducklings
F
Duck plague is only seen in day old ducklings
F
Duck viral enteritis is usually mild, osmotic diarrhoea.
F
Duck plague infects exclusively domestic and wild ducks
F
Antibiotic treatment is the most effective control method for Duck Viral Enteritis.
F
Duck plague infects exclusively domestic and wild ducks.
F
Beak deformity is a typical sign of Duck Plague.
F
Muscovy duck are resistant to the Duck Viral Enteritis.
F
Duck Plague/Duck viral Enteritis cause high mortality in all ages.
F
Duck Plague/Duck viral Enteritis clinical signs in young ducks are only seen in birds up to 4 weeks of age
F
Vaccines containing a live attenuated strain can be used for prevention against Duck Plague.
T
Duck Plague/Duck viral Enteritis vaccination is not needed as clinical signs are mild.
F
Duck Plague virus can “settle” in wild ducks.
T
