EMQs Flashcards

1
Q

An 18-year-old known asthmatic with a respiratory rate of 50, mx?

A

Endotracheal intubation

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2
Q

Features of sarcoidosis?

A

Exocrine- swollen parotid glands
Lupus pernio- indurated plaques + discoloration
Non- caseating granulomas- transbronchial biopsy is essential for diagnosis in most cases

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3
Q

Can measles cause bronchiectasis?

A

Yes

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4
Q

Most common identifiable cause of bronchiectasis?

A

Cystic fibrosis

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5
Q

Baker’s cyst

A

A popliteal cyst is an accumulation of synovial fluid which in this case has resulted from this woman’s arthritis. This is an accumulation of synovial fluid behind the knee, usually in response to injury or inflammation. It will self-resolve but the underlying cause should be addressed i.e. arthritis. First line treatment for grade 1 or 2 injuries is with RICE: rest, ice, compression and elevation followed by gentle mobilisation. Adjunctive analgesia can be offered with paracetamol. Treatment is conservative, particularly if asymptomatic. Surgery is only indicated in those with extensive symptoms where conservative and percutaneous treatments have failed. Corticosteroid injections (intra-articular) can also be considered.

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6
Q

Non small cell carcinoma

A

Clubbing

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7
Q

PE on CXR

A

Band atelectasis, hemidiaphragm elevation, Fleischner’s sign, Westermark’s sign and Hampton hump.

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8
Q

What can precipitate immune thrombocytopenic purpura?

A

HIV

Varicella zoster

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9
Q

Hep A

A

shellfish which is harvested from sewage contaminated water

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10
Q

Symptoms of Hep A

A

pre-icteric phase, lasting 5-7 days, consisting characteristically of N&V, abdominal pain, fever, malaise and headache. Rarer symptoms may be present such as arthralgias and even severe thrombocytopenia and signs that may be found include splenomegaly, RUQ tenderness and tender hepatomegaly as well as bradycardia. The icteric phase is characterised by dark urine, pale stools, jaundice and pruritis. When jaundice comes on, the pre-icteric phase symptoms usually diminish, and jaundice typically peaks at 2 weeks. However, a fulminant course runs in <1% of patients with worsenining jaundice and encephalopathy.

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11
Q

Gallstones ix

A

USS is the definitive initial investigation. HIDA scanning and MRI may help if the diagnosis remains unclear.

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12
Q

Long term treatment after MI

A

aspirin, ace, beta blockers, statin

Clopidogrel for twelve months

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13
Q

Risk factors for SVT

A

Infiltrative disease- eg sarcoidosis, amyloidosis- can cause scar tissue in AV node

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14
Q

angina mx

A

beta blockers first line
ccb second line

+
GTN

+ aspirin- prevention

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15
Q

What is EBV associated with?

A

Mononucleosis (associated with
lymphomas, nasopharyngeal
carcinoma)

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16
Q

How is VZV spread?

A

Respiratory

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17
Q

Shingles treatment?

A

First line: Valacyclovir

Second line:

18
Q

Hepatitis Ix?

A

FBC,U+E, LFT, Clotting, CRP
Viral serology + PCR
Liver USS
In extreme cases- Liver biopsy

19
Q

Shingles treatment?

A

First line: Valacyclovir or famacyclovir
Second line: Acyclovir
If within 72 hours of onset
Give for 7 days

20
Q

What autonomic symptoms can GBS have?

A

Urinary retention, ileus

21
Q

Risk factors for GBS

A

Cancer - esp lymphoma

Immunisation

22
Q

Ix for GBS?

A

Nerve conduction studies- decreased nere conduction

Lumbar puncture

  • Albuminocytological dissociation - high protein [due to inflammation], normal glucose, normal cell count

Bloods- Anti ganglioside antibodies in 25% of GBS and all Miller Fischer variant

Spirometry- [bedside, every 6 hours]

23
Q

Hydropcephalus first line?

A

CT or MRI

24
Q

Types of herpes simplex I- different presentations

A

j

25
Q

Coeliac gold standard?

A

Endoscopy and biopsy

26
Q

Coeliac Mx?

A

Avoid gluten

Pneumococcal vaccine every 5 years

27
Q

Types of uveitis

A

Anterior
Posterior
Complete
Intermediate

28
Q

Difference between posterior and anterior uveitis

A

Anterior= autoimmune condition, painful, red, increased lacrimation and photophobia

Posterior uveitis = infective disease, painless, floaters and scotoma

Both present with blurry vision

29
Q

ix for uveitis

A

Fundoscopy

Slit lamp

30
Q

Pain in types of conjunctivitis

A

Viral conjunctivitis is painless

Bacterial conjunctivitis is painful

31
Q

fOURTH NERVE palsy causes

A

Most common= head trauma

tumours
vascular- diabetes, atherosclerosis, hypertension

32
Q

Causes of sixth nerve palsy

A

Stroke, trauma, viral illness, brain tumour, inflammation, infection, migraine headache and elevated pressure inside thebrain

33
Q

Other symptoms of sixth nerve palsy

A

Hearing loss, facial weakness, decreased facial sensation, droopy eyelid and/or abnormal eye movement

34
Q

Causes of sixth nerve palsy

A

Stroke, trauma, brain tumour

viral illness,inflammation, infection

migraine headache and elevated pressure inside thebrain

35
Q

Bulbar palsy signs

A
Gag reflex – absent
Tongue – wasted, fasciculations
Palatal movement – absent
Jaw jerk – absent or normal
Speech – nasal
Emotions – normal
Other – signs of the underlying cause, e.g. limb fasciculations.

X, XI and XII

Motor neuron disease
Guillain-Barre

36
Q

Pseudobulbar palsy

A
UMN - limbs bilateral
Gag reflex= increased/normal
Tongue= spastic
Palatal movement= absent
Jaw jerk= increased/normal
Speech= donald duck- monotonous, slurred, high pitched
Emotions- labile

V, VII, VIII, IX, X

Stroke of internal capsule
MS
Motor neuron disease

37
Q

Causes of cranial diabetes insipidus?

A

Pituitary tumour
Meningitis/infection
Sarcoidosis

38
Q

Causes of nephrogenic diabetes insipidus?

A
Lithium
Hypercalcaemia
Hypokalaemia
AVPV2
Idiopathic
39
Q

Causes of nephrogenic diabetes insipidus?

A
Lithium
Hypercalcaemia
Hypokalaemia
AVPV2 gene- inherited
Idiopathic
40
Q

What happens to urea in acute addisons?

A

Urea

41
Q

What happens to urea in acute addisons?

What happens to glucose?

A

High urea

Low glucose