EKG and the Cardiac Cycle Flashcards

1
Q

3 steps of Cardiac Action Potential (1st step)

A

Depolarization- opens fast sodium (Na+) channels, extracellular Na+ enters

  • rising phase of action potential (-90mV to +30mV)
  • influx of Na+ will stop quickly
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2
Q

3 steps of Cardiac Action Potential (2nd step)

A

Voltage change open calcium Ca2+ channels, influx of extracellular Ca2+

-Ca2+ influx prolongs depolarization - the plateau
- cells will contract as long as Ca2+ is entering

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3
Q

3 steps of Cardiac Action Potential (3rd step)

A

repolarization- results from inactivated Ca2+ channels, the opening of potassium K+ channels - an efflux of K+

  • resting potential (-70mV) is restored
  • Ca 2+ is either pumped out of the cell or into the sarcoplasmic reticulum
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4
Q

Function of Plateau

A
  • Action potential and contraction phase are longer in cardiac muscle
  • sustained contraction ensures efficient ejection of blood from the ventricles
  • longer absolute refractory period avoids tetany
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5
Q

P Wave

A
  • Last .08s
  • results from movement of the depolarization wave from SA to AV node
  • the atria contracts .1s after the P wave begins
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6
Q

QRS complex

A
  • lasts .08s
  • results from ventricular depolarization, precedes ventricular contraction
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7
Q

T wave

A
  • lasts .16s
  • results from ventricular repolarization
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8
Q

Why is atrial repolarization not seen on a typical EKG?

A

Atrial repolarization occurred during ventricular depolarization – the resultant wave was obscured by the QRS Complex

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9
Q

Interval

A

duration of the time that includes 1 segment and 1+ wave

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10
Q

Segment

A

a region between two waves

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11
Q

What is the possible clinical significance of an elevated or depressed ST segment?

A

Cardiac ischemia

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12
Q

Atrial Depolarization

A

Completed by SA Node, causes P wave

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13
Q

Atrial depolarization complete

A

Impulse delayed at AV Node

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14
Q

Ventricular Depolarization

A
  • Begins at apex, causes QRS complex
  • Atrial repolarization occurs, but is obscured
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15
Q

Ventricular repolarization

A

Begins at apex, causes T wave

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16
Q

Systole

A
  • Contraction
  • Blood is forced out the heart’s chambers
17
Q

Diastole

A
  • Relaxation
  • Blood refills the heart’s chambers
18
Q

Cardiac Cycle (1st step)

A

Ventricular Filling
- pressure is low, blood flows from atria to ventricles
- AV valves open, SL valves close
- 80% of ventricular filling
- following atrial depolarization (P Wave), atrial systole occurs, and blood is compressed into the ventricles
- EDV
- atrial diastole and the start of ventricular depolarization

19
Q

Cardiac Cycle (2nd step)

A

Isovolumetric Contraction
- atria relax, ventricles start contracting, pressure in ventricles rises
- AV valves close
- for a moment, the ventricles are closed chambers with a constant blood volume
- pressure continues to rise until it exceeds the pressure in the great vessels
- SL valves open

20
Q

Cardiac Cycle (3rd step)

A

Ventricular Ejection
- blood moves from the ventricles to the great vessels (pulmonary trunk vs aorta)
- pressure in the aorta is typically - 120mmHg

21
Q

Cardiac Cycle (4th step)

A

Isovolumetric Relaxation
- following the T-wave, the ventricles relax
- ESV
- ventricular pressure drops, blood slides from the great vessels back towards the ventricles - SL valves close
- ventricles are again closed chambers

22
Q

End Diastolic Volume (EDV)

A

the maximum volume of blood that the ventricles will contain in the cardiac cycle

23
Q

End Systolic Volume (ESV)

A

the un-ejected blood remaining in the ventricular chambers

24
Q

Dicrotic Notch

A

a brief rise in aortic pressure caused by blood rebounding off the newly closed aortic valve (isovolumetric Relaxation)

25
Q

Pulmonary Circulation

A

low-pressure circuit - pressure in the pulmonary arteries = 24/10 mmHg

26
Q

Systemic Circulation

A

higher-pressure circuit - pressure in the aorta = 120/80 mmHg

27
Q

Closure of the ____ Valves is the 1st Sound (“lub”)

A

AV

28
Q

Closure of the ____ Valves is the 2nd Sound (“dup”)

A

SL

29
Q

Murmur

A

an abnormal heart sound secondary to turbulent blood flow