ECGs Flashcards

1
Q

presentation of heart block

A

syncope
heart failure
regular bradycardia
wide pulse pressure
JVP - cannon waves in neck
variable intensity of S1

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2
Q

1st degree heart block

A

PR >0.2s

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3
Q

2nd degree heart block, mobitz type 1

A

progressive prolongation of PR interval until dropped beat occurs

(Wenckebach)

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4
Q

2nd degree heart block, mobitz type 2

A

PR interval is CONSTANT but P wave is often not followed by QRS

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5
Q

3rd degree heart block

A

no assoc between Pwaves + QRS

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6
Q

management of torsades de pointes

A

haemodynamically unstable = DC cardioversion

stable = IV magnesium sulphate 2g over 1-2mins

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7
Q

ST elevation in II, III, aVF

A

inferior -> right coronary artery

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8
Q

ST elevation in V1-4

A

anterior -> LAD

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9
Q

ST elevation in I, aVL +/- V5-6

A

left circumflex

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10
Q

signs of haemodynamically instability

A

shock - suggests organ hypoperfusion
syncope - brain hypoperfusion
chest pain - myocardial ischaemia
pulmonary oedema - evidence of heart failure

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11
Q

when can sinus bradycardia be normal

A

adults aged over65
young athletes

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12
Q

how can heart rate in patient with regular rhythm on ECG be calculated?

A

dividing 300 by the numbers of squares in between the QRS compleses

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13
Q

PE ECG findings

A

sinus tachycardia = commonest + sometimes only feature
RBBB
right axis deviation

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14
Q

atrial fibrillation ECG features

A

absent P waves
narrow QRS complex
irregularly irregular rhythm

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15
Q

when should rate control not be given in AF

A

a reversible cause
new onset (in last 48hrs)
heart failure
symptoms despite being effectively rate controlled

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16
Q

rate control options in AF

A

beta-blocker
CCB
digoxin - only in sedentary with persistent AF, require monitoring

(give rhythm control)

17
Q

rhythm control

A

cardioversion
- pharma -> felcainide, amiodarone
- electrical

long term rhythm control
- betablockers =1st line
- dronedarone
- amiodarone

18
Q

general treatment of atrial fibrillation

A

betablocker - rate control
DOAC - for anticoag

19
Q

ECG features in Wolff-Parkinson white

A

short PR interval <0.12s
wide QRS >0.12s
delta wave

definitive treatment = ablation of accessory pathway

20
Q

acute management of supraventricular tachycardias

A
  1. vagal manoeuvres - valsalva, carotid sinus massage
  2. adenosine (rapid bolus)
  3. verapamil or beta blocker
  4. synchronised DC cardioversion
21
Q

when is adenosine avoided

A

asthma, COPD
heart failure
heart block
severe hypotension

22
Q

causes of bradycardia

A

medications - betablockers
heart block
sick sinus syndrome

23
Q

management of bradycardia

A

IV atropine
inotropes - adrenaline
temp. cardiac pacing
pacemaker

24
Q

P wave

A

atrial depolarization

0.08-0.10 secs

25
Q

QRS complex

A

ventricular depolarisation <0.10s

26
Q

T wave

A

ventricular repolarisation

27
Q

PR interval

A

largely AV node delay 0.12-0.20sec

28
Q

ST segment

A

ventricular contraction (systole) occurs in the ST segement

29
Q

TP interval

A

ventricular relaxation (diastole) occurs in the TP interval

30
Q

what does a large box vs a small box represent on an ECG?

A

large = 0.2 secs

small = 0.04secs

31
Q

left axis deviation signs on ECG

A

lead I = positive (left thumbs up)

lead II/aVF = negation

32
Q

right axis deviation signs on ECG

A

lead I = negative

lead II/aVF = positive (right thumbs up)