EATING BEHAVIOUR-neural mechanism in control eating b

Question 1

what is metabolism?

Answer 1

regulated by physiological mechanism & aims to keep body in homoestastics

Question 2

what is homeostatics?

Answer 2

automatic functioning within body e.g temp,heart rate,hunger & satiety

Question 3

what is satiety?

Answer 3

keeps us full

Question 4

what is the dual control theory?

Answer 4

idea of hunger is regulated by 2 parts of hypothalamus

Question 5

what is dual control theory based on?

Answer 5

hypothalamus

Question 6

what is process with hypothalamus?

Answer 6

eat meal and releases certain chemicals- brains see levels of g increase and stop eating. glucose burnt then low so danger of losing energy. low= start eating. if we ignore other parts of brain stimulate and recog levels of other chemicals

Question 7

what is the chemical it releases?

Answer 7

glucose

Question 8

what is it also known as (process)?

Answer 8

negative feedback loop

Question 9

process of what happens when we’re hungry?

Answer 9

lateral hypothalamus= activated & ghrelin= weak w o no glucose. lepton goes into free fat cells in blood. 2 types ceplin goes in and when poefed = full when out get thin.

Question 10

not just 1 mechanism we go through then how many?

Answer 10

3 but we gonna do 2.

Question 11

what 2 theories/mechanism do we go?

Answer 11

dual centre theory

set point theory

Question 12

what is dual centre theory also linked to?

Answer 12

boundary model theory in dieting

Question 13

what is set point theory?

Answer 13

our body weight is regulated by a biologically determined standard

Question 14

who suggested our body weight is regulated by a biologically determined standard ?

Answer 14

keeley

Question 15

point 2 for set point theory?

Answer 15

our homoestatic mechanism alters our metabolism and appetite if we eat too much/little which returns us to our normal weight

Question 16

point 3 for set point theory?

Answer 16

if we have boundary of weight how people get obese/anorexic - we screw with our own biology

Question 17

what did passer et al say?

Answer 17

but if we are persistent over or under eaters then the mechanisms struggle results = new set weight

Question 18

what is dual centre also?

Answer 18

automatic function

Question 19

what is the two important components for the dual centre?

Answer 19

lateral & vetrimenal hypothalamus

Question 20

when does hypothalamus mean?

Answer 20

main area of the brain implicate in appetite regulation (deepest part of head to damage)

Question 21

what is 2 areas of the hypothalamus?

Answer 21

lateral & ventromedial hypothalamus

Question 22

what is lateral hypothalamus?

Answer 22

switches hunger on. damage to this area= food intake automatically reduced

Question 23

what is lateral hypothalamus also known as?

Answer 23

hunger centre

Question 24

what is vmh?

Answer 24

switches eating off, tells us we are full (satiety) and appetite reduces - damage 2 area = increased appetite

Question 25

vmh also known as?

Answer 25

satiety centre

Question 26

what does the set point theory suggest? (in relation to dual centre)

Answer 26

suggests that food consumption & weight control are a result of balance between these 2 areas & other metabolic processes

Question 27

diagram intro?

Answer 27

as glucose levels decrease hunger levels increase increase in g lev means satiation released

Question 28

what happens in lh with glucose levels?

Answer 28

low in g level activates area and feel hungry. individ look for food and eats

Question 29

what happens in vmh with glucose levels?

Answer 29

increase in g levels activates this area and feel satiation -inhibits further eating

Question 30

what is the function of the dual centre area?

Answer 30

homoestatis- dont need to think bout this stuff

Question 31

what about type 2 diabetes and this diag?

Answer 31

we develop later in life.increase in type 2 because glucose lev high

Question 32

A02 for pathway cases?

Answer 32

berthound & morrison wood leibowtz

Question 33

what did bethard & morrison?

Answer 33

not just g that affects b/hunger

Question 34

in depth bethard & morrison?

Answer 34

look at circuits within hypothalamus - found many of the pathways involve the para ventricular nucleus (receptors) for transmitters which stimulate or reduce appetite are found in this cluster of neurons.

Question 35

what is influenced by signals from pvn?

Answer 35

metabolic and digestive processes

Question 36

what did woods say?

Answer 36

neuropeptide is one signal. increase levels of npy in pvn = food intake & weight increase when leptin levels low ,npy lev increase

Question 37

what did leibowtz?

Answer 37

rats injected with npy consume large amounts of food even when full. preference of food shown in carbs

Question 38

what is galanin?

Answer 38

another peptide found in pvn (leibowtz) when thi is injected into rats, again food intake increased food preference shown fats

Question 39

what do cravings work on?

Answer 39

individ not enough of something bod & stimulas vmh to find food (lh)