Dysrhythmias Flashcards

You may prefer our related Brainscape-certified flashcards:
1
Q

stable or unstable

A
  • Interpretation based on a spectrum
  • ALOC, ↓BP, CP, pulmonary edema

-This is not a dichotomous decision. If someone is able to talk to you, they are perfusing enough to get o2 to the brain – they are STABLE even if their BP is 80

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

regular or irregular rhythm?

A
  • Irregular
    • Above the ventricles
    • NOT VT
    • Block AV Node
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

are P-waves present?

A

If there are P waves present, it is unlikely that it is a tachydysrythmia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Narrow or wide complex?

A

Narrow

  • Above the ventricles
  • Block the AV- Node
  • Not VT
  • Duration (<0.12 (adults), < 0.08 (children))
  • If it is narrow, its from the atrium. If its wide its from the ventricles
  • If you have narrow complex you need to block the av node
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Group A: narrow, regular, no p-waves

A
  • PSVT
  • Atrial Flutter
  • WPW (orthodromic)
  • Narrow complex VT (very rare)

-Cause: electrical circuit movement

  • Narrow complex VT resolves by shock etc. its not picked up very often
  • Orthodromic – narrow – starts in atrium
  • Antidromic – wide – starts in ventricle
  • WPW – accessory pathway
  • The AV node cannot go faster than 220! If it goes higher than that, there is an accessory pathway
  • A flutter = microcircuit issue
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Treatment for group A dysrhythmias

A

STABLE

  • Vagal maneuvers
  • AV Nodal blockers: Adenosine, CCB, ẞ-Blocker
  • Cardioversion

UNSTABLE

  • Can consider Adenosine but…
  • Cardioversion start at 50J and 2x every time
  • You need to block the AV node
  • If you use medication and it doesn’t work, you use synchronized cardioversion
  • Unstable: you CAN push adenosine
  • What does adenosine feel like? FEELS LIKE DYING!! You need to prime the patients so theyre ready for this
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Group B dysrhythmias: narrow, irregular, no Ps

A
  • A-fib
  • A-Flutter with variable block

-Cause: increased automaticity

  • Anyone in group A (SVT, Aflutter) – AV NODE ISSUE
  • Adenosine is FAST! Lasts for a few seconds and goes away – if you give adenosine to someone with aflutter, it will NOT CURE Aflutter
  • For Aflutter, you need a longer acting agent

-Increased automaticity – fireworks going off in the atrium all the time

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Group B treatment

A
  • it depends:
    • chronic: rate control
    • new onset: rhythm control

You will see a chronic pt if they have palpitations

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Group B treatment: Chronic - stable vs unstable

A

Stable patient

  • Stabilize Atrium by blocking AV node
    • CCB
    • ẞ-blocker
    • Digoxin…old drug
    • Diltiazem 5-10mg IVP slow. If tolerated can 2x it. (Up to 60mg in 30min)

Unstable

  • Rapid Cardioversion: Start high! Sync!
  • Support Blood Pressure: Pressers – you need to ensure perfusion of brain and heart – success rate of conversion with low blood pressure is LOW
    • If the diastolic pressure is low, you will NOT convert these patients because their heart is irritable! You need to get the BP up!
  • Slow down heart: drips not IV Push
    • If you push, you may bottom out the BP and cause cardiac arrest
    • But, if the person is bigger, you may need to push since the drip wont do anything
  • Magnesium – vasodilator
  • Repeat Cardioversion
  • Other considerations: Is it really A-Fib?

-For elderly, TREAT UNDERLYING CAUSE FIRST!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Group B tx: new onset

A
  • Rhythm Conversion: Ottawa Aggressive Protocol for A-fib
    • Clear Hx with in 48 hours
    • No structural heart dz
    • Age not a contraindication
  • Caveat in elderly don’t rush often p/w vague sx’s
  • New onset – you want to get them back into sinus rhythm
  • AGE IS NOT A DETERMINING FACTOR!
  • If you have a patient with longstanding Afib, the risk of converting them is STROKE!
-Chemical cardioversion: Procainamide 1g over 1 hour
↓
-Electrical cardioversion: Biphasic 200J
↓
-D/C home without meds
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Group C: wide, regular, no Ps

A
  • VT
  • SVT with aberrancy (BBB)
  • Antidromic WPW
  • Cause: circuit re-entry in the ventricles
  • WIDE and REGULAR – VTACH!!!!!!
  • WPW – shortened PR interval with delta wave
  • You CARDIOVERT for unstable people!!
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Group C tx: stable vs unstable

A

STABLE

  • Consider Adenosine
  • Cardioversion: 100J
  • Antiarrythmic Infusion: Procainamide, Amiodarone, Magnesium

UNSTABLE

  • CARDIOVERT
  • ACLS

-Why consider adenosine? SVT with abberancy - adenosine in this patient that is stable is like pressing a restart button on the heart

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

AV node

A
  • AV node cant go faster than 200-220! This is 300 so its WPW
  • CALL CARDIOLOGY!! In the ED, think about in this group whether they are stable or unstable. If you convert, do they go home or get admitted? ADMITTED
  • Vtach can turn to Vfib!!
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Group D: wide, irregular, no Ps

A
  • A-fib with BBB
  • A-fib with WPW
  • A-flutter with variable block and BBB
  • Polymophic VT = Torsades
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

treatment of group D dysrhythmias

A

STABLE

  • A-Fib = Rate control: Be carful If…
  • WPW
    • Regular: Consider *Adenosine (some people don’t like this because it can send them into overdrive), electricity
    • Irregular: No AV nodal blocker, Electricity, Procainamide
  • Torsades = Magnesium

UNSTABLE

  • Defibrillate
  • ACLS
  • Orthrodromic- Narrow
  • Antidromic – Wide treat as VT Cardioversion
  • A-Fib with WPW- very hard, if in doubt reg or irreg okay to cardiovert 120-200J Biphasic and 200J, meds likely started in the Unit
  • WHEN ITS IRREGULAR YOU DEFIBRILLATE
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Supraventricular tachycardia tx

A
  • Stable
    • Vagal maneuvers
    • Block AV-node
Adenosine 6mg IVP can mix with 15ml of NS rapid push 
↓
Double dose 12mg Rapid IVP 
↓
Can Go Up to 18mg Rapid IVP
↓
Synchronized Cardioversion
17
Q

treatment of chronic A-fib

A

Unstable

  • Rapid cardioversion: Pads anterior and posterior
    • Start high 360J, Sync
    • Sedation (Etomidate + Ketamine)
  • Elevate BP
    • Push dose pressers (PDP) Phenylephrine 50-200mcg IVP temporizing measure
    • Target diastolic > 60mm Hg

Slow down the heart

  • Diltiazem:
    • NO IVP!
    • Loading Drip 50 mg mix into 50mL NS (Gtts at 2.5mg per minute)
  • Amiodarone
    • 150mg over 5-10 min
  • Okay to use PDP to maintain BP PRN

Other meds to use:
-Magnesium: 1-2gm IV drip

Repeat Cardioversion

  • PUT ON PADS!!
  • What does sedation do to BP? Drops it! Ketamine is one of the only sedatives that does not drop BP
  • Etomidate has a better safety profile but can still drop BP
  • If you elevate BP, youre more likely to convert them

All antiarrhytmics can be proarrythmics!!
Don’t use CCBs in heart failure!!!

18
Q

Treatment of Vtach

A

SHOCK

  • PT yells and is awake
  • what do you want to know
19
Q

Solution to tachyarythmias

A

-WHEN IN DOUBT, ELECTRICITY

20
Q

how to approach bradycardia

A
  • ABC’s, IV, O2, Monitor
  • Stable vs. Unstable
  • Wide or Narrow
  • How slow?
21
Q

Wide or narrow complex brady

A

Wide

  • Block is below AV node
  • Slower
  • More likely to stop
  • Not atropine sensitive

Narrow

  • More stable
  • Faster
  • Atropine sensitive
22
Q

brady big 3

A
  • electrolytes
  • ischemia
  • drugs
23
Q

1st degree AVB

A
  • PR > 0.20 sec (1 box)
  • Usually benign
  • Normal in 5% of population
  • Usually narrow

TREATMENT
-Refer back to primary doctor - you dont really do much for this patient

24
Q

2nd degree type 1 AV block - Wenckebach

A

PR gets progressively longer then drops a QRS complex

Narrow complex

At level of AV node

Stable and Transient

25
Q

2nd degree type 2 AV block

A

PR interval consistent, Drops QRS complex

Below AV Node

Likely go into 3rd degree or asystole

26
Q

3rd degree AVB

A
  • narrow complex - AV node
  • wide complex - Associated more with asystole
  • PR interval is randomly changing
  • No associating QRS to a P-Wave
  • Great imitator of other AVB
27
Q

treatment of 3rd degree AV blocK

A
  • Atropine: start 0.25-0.5mg bolus
  • Transcutaneous Pacing: Sedation
  • Dopamine: 2-10 mcg/kg per min
  • Epinephrine: 2-10 mcg per min
    • 1ml of crash cart epi (1:10,000) = 100mcg
    • Put 1ml in 100ml of NS = 1mcg per ml
  • BRADY Are Too Darn Easy
  • You know the transcutaneous pacing is capturing by just feeling pulse and if its regular, youre good

-Pacemaker for all 3rd degree AVB and symptomatic Mobitz Type II

28
Q

Other abnormal EKG rhythm

A

Prolong QT syndrome
-Lead to Ventricular dysrhythmia = Torsades

Brugada syndrome/HOCM
-Associated with sudden death

Wellen’s Syndrome
-High Risk for extensive anterior MI and death

29
Q

ddx of prolong QT

A
  • Hypokalemia
  • Hypocalcemia
  • Hypomagnesmia
  • Na Channel Blockers
  • Miscellaneous: Elevated ICP, ACS, Hypothermia, hereditary
30
Q

Consideration of QT intervals

A
  • If QT lengthen due to stretching of ST segment = Hypocalcemia and Hypothermia
  • If QT lengthen due to to stretching of T-wave = Ischemia
  • If you have prolonged QT because T wave is getting really big, THAT IS ISCHEMIA!!
31
Q

Brugada Syndrome

A
  • RBBB or incomplete RBBB with ST elevation in V1-V2
  • Two morphology
    • Saddle type
    • Cove Type (more specific)
  • Healthy with structurally normal hearts
  • Syncope or Near Syncope
  • Mortality 10% per year
  • Sudden VT or polymorph VT, spontaneously convert = syncope and if not they die!
  • 50% genetic mutation in Na channel
32
Q

HOCM

A
  • Deep Q-waves in Lateral Leads
  • Structural heart dz = Left ventricle
  • Treatment: Surgery or medical management
  • Not ischemic Q-wave these are 1 box wide.
  • HOCM narrow!
33
Q

Wellen’s

A
  • Proximal LAD lesion
  • ST Changes are absent
  • Two Morphologies
    • Type 1: Deep symmetric T-Wave in precordial leads
    • Type 2: Biphasic T-Waves in precordial leads
  • WELLENS type 2 (for having 2 waves – biphasic) – if you do a trop on this person it will NOT be positive but if you stress them, they will die! – this person has a proximal LAD lesion
  • WELLENS type 1 (deep t wave)
34
Q

treatment of wellen’s

A

-PCI/Cath is the best management

  • Medical management = not effective
    • 75% develop AMI within weeks

-Stress test = precipitate MI