dysrhythmias Flashcards

1
Q

dysrhythmias

A

abnormal cardiac rhythm

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2
Q

ECG

A
  • graphic tracing of electrical impulses produced by the heart
  • waveforms represent activity of charged ions across membranes of myocardial cells
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3
Q

phases of cardiac action potential

A
  • phase 0
  • phase 1,2,3
  • phase 4
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4
Q

ECG leads

A
  • six leads (I, II, III, aVR, aVL and aVF) measure electoral forces in frontal plane
  • six leads (V1-V6) measure forces in horizontal plane
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5
Q

counting HR in ECG

A
  • number of QRS complexes in 1 min
  • R-R intervals in 6 seconds, and multiply by 10
  • number of small squares between one R-R interval and divide the number by 1500
  • number of large squares between one R-R interval and divide the number by 300
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6
Q

client prep for ECG

A
  • clip excessive hair on chest wall
  • gently rub skin with dry gauze until skin is slightly pink
  • may need to use alcohol for oily skin
  • apply electrical conductive gel
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7
Q

sinus bradycardia

A
  • sinus node fires <60 bpm
  • normal rhythm is aerobically trained
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8
Q

sinus tachycardia

A

discharge rate from sinus node is increased

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9
Q

premature atrial contraction (PAC)

A
  • travel across atria by abnormal pathway
  • distorted P wave
  • may be stopped, delayed, or conducted normally at the AV node
  • can result from: emotional stress, physical fatigue, use of caffeine and tobacco, hypoxia
  • significance: isolated PACs are not significant in healthy hearts but in those with heart disease it can be a warning of more serious dysrhythmias
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10
Q

paroxysmal supraventricular tachycardia (PSVT)

A
  • originates in ectopic focus anywhere above bifurcation bundle of HIS
  • run of repeated premature beats
  • abrupt onset and termination
  • clinical associations: overexertion, emotional stress, stimulates, CAD
  • clinical significance: prolonged episode and HR > 180 may precipitate decreased CO, hypotension, dyspnea, angina
  • tx: vagal manoeuvres (Valsalva stimulation, coughing, holding breath), IV adenosine
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11
Q

atrial flutter

A
  • recurring, regular, sawtooth-shaped flutter waves
  • originates form single ectopic focus
  • clinical associations: CAD, hypertension, mitral valve disorders, pulmonary embolus, chronic lung disease, cardiomyopathy, hyperthyroidism, medications (digoxin, quinidine, epinephrine)
  • clinical significance: high ventricular rates (>100) and loss of atrial “kick” can decrease CO and precipitate HF
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12
Q

atrial fibrillation

A
  • total disorganization of atrial electrical activity due to multiple ectopic foci, resulting in loss of effective atrial contraction
  • significance: decrease in CO, thrombi may form in atria, embolus may develop
  • pts usually on blood thinners
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13
Q

first degree AV block

A
  • every impulse is conducted to the ventricles but duration of AV conduction is prolonged
  • PR interval is long
  • usually asymptomatic
  • client should not be on beta blockers
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14
Q

premature ventricular contractions (PVC)

A
  • contraction originating in ectopic focus of the ventricles
  • premature occurrence of a wide and distorted QRS complex
  • associated with: stimulants, electrolyte imbalance, hypoxia, fever, exercise, MI, HF, CAD
  • significance: benign in healthy heart, in heart disease can decrease CO and precipitate angina and HF if >10/minute
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15
Q

ventricular tachycardia

A
  • run of 3+ PVCs
  • considered life threatening because of decreased CO and the possibility of deterioration to ventricular fibrillation
  • no P or T waves, widened and more frequent QRS complex
  • HR is >180
  • sustained VT can cause hypotension, pulmonary edema, decreased cerebral flow and MI
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16
Q

ventricular fibrillation

A
  • irregular undulations of varying contour and amplitude
  • no effective contraction or CO
  • do CPR or shock
  • associations: acute MI, CAD, occur in cardiac procedures, may occur with coronary reperfusion, accidental electrical shock
  • pt unresponsive, pulseless, apneic state
17
Q

asystole

A
  • flatline
  • no CO
  • cannot shock, continue CPR until activity becomes present
18
Q

sudden cardiac death (SCD)

A

result from ventricular dysrhythmias (v. tach, v. fib)

19
Q

defibrillation

A
  • most effective method of terminating VF and pulseless VT
  • depolarizes cells in myocardium to allow the SA node to resume pacemaker role
  • after initial shock, initiate CPR
20
Q

ischemia

A
  • ST segment depression and/or T wave inversion
  • only significant if it is at least 1 mm (one small box) below the isoelectric line
21
Q

cardiac injury

A

ST segment elevation is significant if >1mm above the isoelectric line

22
Q

cardiac infarction

A
  • pathological Q wave is deep and >0.03 seconds in duration (indicates that at least half of the thickness of the heart wall is involved)
  • T wave inversion