Dr. Wang says you should know... Flashcards

1
Q

How do you define active disease? Why?

A

2mm of CAL progression over 12months

Probing error is 0.75mm (0.8mm) - 2.5x the error = Progression

Goodson

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2
Q

How many milimeters deep should an implant be placed in posterior and anterior?

A

1.5-3mm below CEJ in posterior

3-4mm below CEJ in anterior

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3
Q

What rule for M-D space for implants?

A

5-8-8-9 - Space from root - center of implant, center of implant to center of implant

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4
Q

How can you create papilla

A

Surgical means: Difficult because no bone to support - unpredictable

Orthodontic means: Most predictable way to grow true papilla

Restorative means: Larger crown - longer contact point

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5
Q

What are characteristics of Gargiulo et al 1961 that we should consider?

A

Cadaver study on ERUPTION PHASE teeth

Range of Junctional Epithelium from 1 - 10mm!

Connective tissue is CONSTANT (~1mm)

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6
Q

How do you treat Excessive Gingival Display?

A

Silberberg 2009

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7
Q

Where should you create your periosteal incision in your flap to increase flap elevation?

A

3-5mm above the MGJ

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8
Q

What happens to transseptal fibers after extraction/implant placement

A

The fibers will span the space between the two teeth after extraction. When an implant is placed, it will wrap around the implant to attach to the next tooth

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9
Q

Edwards 1971

A

Supra-crestal fibrotomy every 2 weeks - 4x - reduce relapse to 30%

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10
Q

What kind of bone formation do you get from BMP - why is this significant?

A

Endochondral ossification

Oral cavity is intramembranous - resists resorption

When you take bone from extra-oral source, get a large amount of resorption/shrinkage

BMP bone shrinks

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11
Q

4 things you must know about ONJ

A

No osteoclast activity

No blood supply

Impaired epithelial cell migration

Bacterial infection

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12
Q

When predicting papilla fill. What percentage probability drop do you have from 5mm to 6mm?

A

Tarnow 1992

42%

4mm - 100%
5mm - 98%
6mm - 56%
7mm - ~25%

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13
Q

What is the bone level to papilla tip that can be expected between 2 implants? Why?

A

3mm

Tooth is 5mm - Tooth has a CEJ - Biologic width is 2mm of CT - No CT on implant so lose 2mm

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14
Q

How much of periimplantitis is caused by excess cement?

A

81% (Wilson 2015)

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15
Q

What is the primary AND SECONDARY etiology of periimplantitis

A

Primary - bacterial dysbiosis and its biproducts in a suseptable host with exaggerated host response

Secondary - Calculus and its biproducts

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16
Q

What is a predisposing factor?

A

Something that puts you at risk of developing a disease/condition (ie. overhang which traps plaque)

17
Q

What is a precipitating factor?

A

Something that causes the disease/condition (ie. Plaque causes the inflammation)

18
Q

Mechanisms of Diabetes and Smoking

A
  • Both have:
    • Vasoconstriction
      • Smoking
        • Nicotine - doesn’t tell you how much
        • Cotinine - TELLS YOU HOW MUCH - use this in research, not nicotine
      • Diabetes
        • AGE
          • RAGE is on the capilary walls
          • AGE deposite on the walls and thicken the walls, narrowing the vessels
    • Effects
      • Reduced chemotaxis of immune cells
        • impaired healing
19
Q

What are the links between Rheumatoid Arthritis and Periodontitis?

A
20
Q

How do restorations relate to periodontal disease

A

Position of the margin - must be accessible for patient to clean

21
Q

How does subgingival restoration effect gingiva?

A

Pontoreiero - recession in thin, inflammation in thick

22
Q

What is the prevalence of PIM and PI?

A

Derks & Tomasi 2015

PIM: 43%
PI: 22%

23
Q

KAN 2003

A

Interproximal bone level of a single implant is determined by the bone level of the adjacent teeth

24
Q

Dr. Wang’s graft formula for lateral window

A

4:1

Cortical : Cancellous

25
Q

How long does it take to scale posterior teeth?

A

39min Maxillary posterior

25min Mandibular posterior

Stambaugh 1981

26
Q

How is calculus adhered to the root?

How is this related to why we used to think we had to root plane?

A

RISP (Zander 1953)

Resorption

Irregularities

Secondary cuticle

Penetration (refuted by Canus 1979)

Theorized that planing would remove penetrated calculus

27
Q

How much calculus can you remove in non-surgical vs surgical? (3 citations to remember)

A

Waerhaug:

Shallow: 83%

(Flip 8 and 3)

Mod: 38%

(Add 8 + 3)

Deep: 11%

Caffesse (Non-surgical):

Shallow: 86%

(Divided by 2)

Mod: 43%

(Minus 11 from Waerhaug)

Deep: 32%

Caffesse (Surgical):

Shallow: 85%

(Minus 10)

Mod: 75%

Deep: 50%

28
Q

Furcation entrance according to Gher & Dunlap and Bower

A

G&D

Max M: 3.6

Max B: 4.2

Max D: 4.8

(Add 0.6 to each)

Bower

Mand B: 3.14

Mand L: 4.17

29
Q

Why do they only achieve 50% clean of the root surface with surgical in the Caffesse study?

A

They were using MWF - which does not expose the crest!

30
Q

What is the difference between gingivitis and periodontitis?

A

Page and Schroeder

Apical migration of JE

Bone Loss

Clinical attachment loss

31
Q

What is the most consistant fiber in the gingival fibers?

A

Trans-septal fibers

32
Q

What are the 4 risk determinants

A

Age
Gender
Ethnicity
Gentics

33
Q

What are risk predictors?

A

Factors that are created by the disease, are not part of the causal chain of disease, but can aid in future disease progression.

ie. Contributing factors
Furcation - not the cause of disease, but risk for future
Calculus…

34
Q

Mattila et al. 1995

A

poor dental health is
a predictor of the incidence of fatal and
non-fatal coronary events for persons with
preexisting coronary artery diseases.
However, it does not prove causality.

35
Q
A