Down sheep Flashcards

1
Q

Ddx for a down sheep

A
  • hypocalcaemia
  • pregnancy toxaemia (twin lamb dz)
  • scrapie
  • lameness
  • respiratory dz
  • listeria
  • hypomagnasaemia
  • carb overload causing acidosis
  • copper poisoning (most often found dead)
  • rhododendron poisoning (particularly in smallholdings)
  • clostridia (botulism) (sheep less susceptible but still can get infected)
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2
Q

Ddx that can happen anytime, and will likely be relatively slow onset to point of recumbency

A
  • scrapie
  • resp dz
  • listeria
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3
Q

Ddx that can happen anytime, but always worth checking the feet and limbs of a recumbent animal

A
  • lameness
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4
Q

Ddx that can happen anytime, and will likely be relatively slow onset to point of recumbency, initial signs of pruritic and nervous signs

A
  • scrapie
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5
Q

Ddx that can happen anytime, and will likely be relatively slow onset to point of recumbency, initial signs of pyrexia and respiratory stridor expected

A
  • resp dz
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6
Q

Ddx that can happen anytime, and will likely be relatively slow onset to point of recumbency, neurological and some unilateral facial paralysis expected

A
  • listeria
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7
Q

Hypocalcaemia

A
  • occurs in late pregnancy
  • older and thinner ewes more prone
  • carrying multiple foetuses increases risk
  • any stressor can trigger the condition
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8
Q

Hypocalcaemia management

A
  • 25-40ml 40% calcium + borogluconate over 30-60s IV
  • typically eructation seen after 1-2mins, stand after 5mins then urinate/defecate and re-join flock
  • normal serum levels are slightly lower than cattle:
    – Ca 2.6mmol/l
    – P 1mmol/l
    – Mg 1.3mmol/l
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9
Q

Pregnancy toxaemia

A
  • occurs in late pregnancy
  • older or thinner ewes are more prone
  • carrying multiple foetuses increases risk
  • any stressor can trigger the condition
  • ewe separated from flock
  • ewe drowsy or comatose
  • ewe not eating: rumen compressed by foetuses in abdomen just can’t get food in properly
  • nervous system signs due to ketone bodies in the brain: tremors, blindness
  • lying on their side: long duration (3-4d)
  • die few days later
  • normal blood glucose = 50-80mg/dl
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10
Q

Nutrition during late pregnancy needed for

A
  • developing foetus
  • udder development and colostrum production
  • prevent milk fever
  • compensation for reduced feed intake
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11
Q

Why shouldn’t you feed ewes loads during late pregnancy?

A
  • perversely increases pregnancy toxaemia risk as deregulates insulin response
  • increases milk fever risk as deregulated PTH response
  • vaginal prolapses more likely
  • dystocia more likely: fat in pelvic canal with oversized foetus
  • feed = money
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12
Q

Pregnancy toxaemia management

A
  • IV glucose
  • oral glucose
  • induce parturition (steroids, PGF2a)
  • encourage eating: minerals in molasses, yeasts (promote rumen function) and propylene glycol drench, vitB12, consider minerals
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13
Q

Hypomagnesaemia

A
  • staggers
  • is more common post-partum
  • seen when moved onto fast growing spring pasture
    – sometimes pasture with high potassium fertiliser which inhibits uptakes
    – just as with cattle
    – is an emergency, as with cattle
    – volumes of IV magnesium to kill a sheep are obviously less, so even more care with administration
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14
Q

Managing staggers

A
  1. increasing gut transit times, by feeding higher fibre diet can help to increase absorption of available Mg, and reduce the incidence of dz
  2. routine soil analysis shows the Mg level of soil, use of Mg limestone rather than Ca limestone maintains the levels
  3. modify bagged fertiliser applications so K applications are carried out later in the season
  4. a regular supply of Mg in the diet e.g. offer free access to minerals
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15
Q

How do steroids induce/help parturition?

A
  • matures lambs lungs
  • increases surfactant
  • encourages them to breathe when born
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