Down cow Flashcards

1
Q

Causes of downer cow syndrome (broad categories)

A
  • metabolic
  • traumatic
  • neurological
  • toxic
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2
Q

What is down cow syndrome?

A
  • inability of cattle to rise and stand for a period of at least 12-24h for undetermined reason
  • also caused bovine secondary recumbency
  • a cow becomes a downer cow when the initial cause resolves but the cow still doesn’t rise
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3
Q

Metabolic causes of down cow syndrome

A
  • unresolved hypocalcaemia (check phosphorous levels too - creeper cows)
  • hypomagnesaemia (generally a cause of becoming a down cow, rather than a chronic situation)
  • ketosis (beware angry nervous ketosis cow, is the ketosis primary or secondary?)
  • hypokalaemia (often give potassium as part of tx, look for muscular fasciculations)
  • fatty liver dz (if fatty liver alone has result in a down cow, the prognosis is poor)
  • fog fever
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4
Q

Fog fever

A
  • autumn time most likely (foggy mornings)
  • movement from sparse to lush pasture
  • L-Tryptophan in the pasture converted by the rumen to 3-Methylindole
  • this is toxic to lungs
  • causes Acute Bovine Pulmonary (O)edema and Emphysema (ABPEE)
  • cows can get used to L-Tryptophan with a slow introduction
  • if suspicious of fog fever but currently only 1 cow down its likely many others are about to go
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5
Q

Hypomagnasaemia

A

= grass staggers
- seen in the spring with a new flush of grass
- seen in the autumn with stale grass
– situation made worse by high potassium and high ammonium fertiliser
– situation improved by more broad leaf, slower growing plants in the sward (clover, chicory)
- cows and sheep don’t store Mg
– require daily dose to top them up
- lots of magnesium goes into milk

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6
Q

Grass staggers tx

A
  • true emergency as will die very fast
  • IV magnesium will cause cardiac arrest
  • can get away with a small amount given really slowly and warmed- in ‘red top’ calcium
  • should take 15 mins to administer bottle IV
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7
Q

Grass staggers prevention

A
  • by ensuring adequate is in diet
  • can supplement water, but beware this reduces palatability of the water
  • take other cows off pasture
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8
Q

Traumatic causes of downer cows

A
  • sacroiliac luxation
  • coxo-femoral luxation
  • gastrocnemius rupture
  • ^ 3 often a sequel to struggling to stand
  • pelvic fracture
  • profound anaemia as a result of haemorrhage
  • profound foot lameness
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9
Q

Neurological causes of downer cows

A
  • obturator paralysis
  • bilateral peroneal paralysis
  • rarely - lymphosarcoma infiltration into spine (cows rarely live long enough for these to develop)
  • BSE
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10
Q

Toxic causes of downer cows

A
  • septic metritis
  • acute toxic mastitis, usually with a coliform
  • tetanus
  • botulism
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11
Q

Causes of primary recumbency

A
  • 46% dystocia (obturator nerve paralysis, pelvic fractures, tears, haemorrhage, etc)
  • 38% milk fever
  • 16% other
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12
Q

When does it occur?

A
  • 50% occur within 24h of parturition
  • can see seasonal variation -> diet when housed and room to move around when calving, so see higher incidence in winter
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13
Q

What happens when a cow becomes recumbent?

A
  • weight on muscles
  • compartment syndrome (esp in hindlimbs) (occluded blood vessels causing necrosis of the muscles/tissues - as bp not good enough to keep blood flowing to these areas when down)
  • may remain bright and alert (can be a good sign in prognosis, if eating nursing becomes easier to achieve)
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14
Q

History qs

A
  • when did she calve?
  • was it assisted?
  • what degree of traction was used?
  • calf alive or dead?
  • how long has she been down?
  • did she stand after calving?
  • is she moving around?
  • did she go down here or was she moved here?
  • how did you move her here?
  • has she eaten/drunk/been drenched?
  • has she been treated already?
  • have you had any others like this?
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15
Q

Treat any underlying conditions

A
  • oral fluids +/- additives
  • IV treatments - Ca, P, K, Mg? fluids?
  • NSAIDs
  • injectable vitamin supplements - B12 (appetite stimulant)
  • antibiotics
  • propylene glycol instead of glucose: rumen bugs will just use glucose and so none will get to the cow
  • hobbling
  • splints
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16
Q

What impact does good nursing care have on downer cows

A
  • increases recovery % massively
17
Q

Likely dx for S-shaped neck flexion

A
  • hypocalcaemia
18
Q

Likely dx for dog sitting

A
  • damaged sciatic nerve
19
Q

Likely dx for uterine prolapse

A
  • often associated with hypocalcaemia
20
Q

Likely dx for legs abducted

A
  • obturator nerve paralysis, hopeless prognosis
21
Q

Factors that affect prognosis

A
  • season: greatest risk = spring > winter > autumn > summer (relatively small changes in risk)
  • days recumbent: straight line graph over time
  • HR: brady and tachycardia = bad
  • RR: tachypnoea (>36bpm) = bad
  • temperature: hypothermic and hyperthermic equally bad
  • PCV: anaemic = bad
  • leukocytes: leukopenia and neutrophilic = bad
  • AST: 500-1000U/l = 2.1x risk, over 1000U/l = 6.9x risk
  • CK: 10,000-25,000U/l = 1.5x risk, >25,000 = 4.6x risk
  • lateral recumbency - pressure on lungs, abdominal organs, muscles of back legs, saliva build up that isn’t being expelled
22
Q

When to consider PTS

A
  • incurable conditions e.g. fractures, tendon ruptures
  • lack of response to tx of primary condition within a reasonable time period
  • alert down cow becoming non-alert (indicating further complication0
  • pain & suffering
  • unable to maintain sternal recumbency despite adequate tx
  • not willing to eat or drink despite adequate tx
  • obvious bed sores or swollen joints/limbs
  • insufficient facilities, labour or expertise
23
Q

Salvage procedures

A
  • clay removal *
  • perimortem caesarean
  • tibial neurectomy *
  • teat removal (but have teat cannula that opens it up and allows it to drain, therefore have no need for teat removal)
  • emergency slaughter *
  • ovary removal
  • semen harvesting
  • toe amputation
24
Q

Spastic paresis and tibial neurectomy

A
  • progressive hindlimb condition of unknown origin leading to contraction of gastrocnemius muscle and associated tendons
  • it will be possible to manually flex the hock without pain
  • if seen in older cattle, likely involve spinal lesions
  • can treat with a tenotomy, but neurectomy preferred
  • most surgeons find it too fiddly to dissect out the relevant bits of the tibial nerve, and so cut the whole thing - this is acceptable
  • might help get calf to slaughter/market weight but no bigger, therefore not suitable for breeding, etc
25
Q

Sign of hypophosphataemia

A
  • sat in sternal recumbency, might try to get up but just don’t have the muscle strength to
  • slide on elbows and knees as trying but failing to get up
26
Q

Signs of fog fever

A
  • eyes congested
  • elbows abducted
  • snotty nose
  • seems like bad pneumonia - but not pyrexic
27
Q

Blood tubes

A
  • all haematology from a purple topped tube: EDTA, on most blood machines
  • all biochem rom a green topped tube: lithium heparin, on most blood machines
  • Zinc needs a different tube
  • Copper needs a different bung