Dissociative Identity Disorder Flashcards
What is Dissociative Identity Disorder (DID)?
- An individual with two or more personalities (alters) that have their own unique behaviours, memories and relationships
What is the DSM V criteria for DID?
- Disruption of identity characterised by two or more personality states (alters)
- At least two alters recurrently control behaviour
- One alter has an inability to recall personal information
- Symptoms are not due to medication or cultural/religious practice
Describe epidemiology for DID
- Prevalence is hugely variable
- Sar et al (‘02, ‘03) found 12-13.8% in psychiatric outpatients
- Sar et al (‘14) found almost half (45.2%) of adolescent patients had a dissociative disorder with 16.4% diagnosed with DID
- More commonly found in women
- Rarely diagnosed until adulthood
What mental disorders are comorbid with DID?
- PTSD
- Major depression
- Substance abuse
- Somatic symptoms (physical)
- BPD
- Phobias
What are the two major theories in DID?
- Sociocognitive model
- Postraumatic model
Describe the sociocognitive model
DID is a form of role play in suggestible individuals brought on by therapist either explicitly or implicitly (Iatrogenesis) (false memories)
Describe the posttraumatic model
DID is a result of severe psychological and/or sexual abuse in childhood
What evidence is there to support the sociocognitive model?
Pope et al (‘06) found:
- no reports of DID before 1800
- major increases since 1970
- Thought to be because of increased awareness due to popular culture (three faces of Eve, Sybil)
Acocelia (‘99):
- Women hospitalised for depression, therapist used suggestive questions and diagnosed DID
- McNally (‘03); patients have sued therapist for implanting false memories
- Californian hillside murderer; used DID as plea
- Spanos et al (‘85); showed DID can be role played study in students (fake hypnotised), we are very suggestible
What does the sociocognitive model (Reiber, ‘06) say about discovery of Sybil?
- Sybil never presented with DID symptoms
- Psychiatrist encouraged multiple personalities which Sybil later revealed
- Psychiatrist gained fame and money
- No documentations of childhood abuse
- Transcripts of therapy show abuse was constructed
Explain sociocognitive views on DID
- Iatrgenesis; multiple personalities occur during treatment
- Diagnosis differs with clinician; not many experts
- Thigpen & Cleckley (‘84) argue out of thousands of cases other than Eve they have only seen one other case
What is the sociocognitive evidence for false memory in DID?
Porter et al (‘99) - implanted false memories in students using guided imagery
Lasko et al (‘04) - Same phisiological arousal between false memories and real memories. Particpants believe false memories (alien abductions)
Huntjen et al (‘03) - People report no sharing of memory between alters in explicit tests but show similar memory between alters compared to controls in implicit tests
Huntjen et al (‘12) - Tested autobiographical memory in DID patients. Interidentity amnesia again not supported, transfer between implicit tests
What are the causes of DID according to the posttraumatic model?
- almost all cases of DID report severe CSA
- dissociation is a means of coping with trauma
What evidence is there for posttraumatic model?
Kisiel & Lyons (‘01) - Children who dissociate are more likely to develop psychological symptoms. Created child dissociative checklist
Taylor & Martin (‘44) - reviewed DID cases before it was widely known about, inconsistent with sociocognitive model
Lewis ('99) :- - 20 year study into 150 murderers - Trances in childhood - Imaginary friends Corroborated by third parties - Distinct handwriting of alters (expert opinion)
What evidence is there for posttraumatic model of memory deficits?
- Putnam (‘95) suggest amnesia for traumatic events are defense mechanism.
- Adreano & Cahill (‘06) found stress hormones can interfere with memory formation
- Neisser & Harsch (‘91) found particpants memory recall of a specific disaster was grossly inaccurate 2 years later compared to 1 day recall
What is the neurobiological evidence for DID?
- MRI study, smaller volume of hippocampus and amygdala, however highly comorbid with PTSD
- Stmulation of hippocampus produced dissociative symptoms
- Ketamine (atagonist of NDMA receptors concentrated in hippocampus produce dissociative symptoms