Anxiety Disorders Flashcards

1
Q

Who developed the two factor model of anxiety?

A

Mowrer 1947

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2
Q

What is the two factor model of anxiety?

A
  • Classical and operant conditioning
  • Conditioned stimulus (dog) paired with unconditioned stimulus (dog bite, pain) eliciting a conditioned response (fear)
  • Avoidance (safety) behaviour sustains fear (phobia)
  • Extensions of this theory are modelling (Fredrikson, 1997) and verbal instruction (Rachman , 1978)
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3
Q

What are criticisms of the two factor model?

A
  • Not all phobias are reported as a result of conditioning
  • Not all individuals who experience aversive encounters develop phobias
  • Phobias are predominantly specific to life threatening objects and situations (snakes, spiders, heights)
  • Prepared learning
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4
Q

What is prepared learning (Seligman, 1971)?

A

Fear circuit in brain prepared by evolutionary exposure to threats (we learn fear for survival)

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5
Q

Explain study of prepared learning and modelling

A

Cook and Mineka (1989) showed rhesus monkeys videos of another monkey expressing fear of toy objects (snake, croc, rabbit and flower). Monkeys only developed fear of snake and croc. Suggests fear is more easily conditioned when object is a potential threat.

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6
Q

Which anxiety disorders are common to conditioning models?

A
  • Specific phobias
  • Social anxiety
  • Panic disorder
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7
Q

What are criticisms of Seligman’s (1971) prepared learning theory?

A

Biologically prepared fears do not always pose as much threat as other things that are generally not feared. For example:

  • Species of spiders that are not poisonous far outweigh those that are, with only 200 poisonous species reported world wide.
  • Whereas there are over 100 known poisonous mushrooms in the USA alone
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8
Q

What are examples of safety (avoidance) behaviours in social anxiety disorder

A
  • Avoiding situation that causes anxiety
  • Avoiding eye contact
  • Disengaging from conversation
  • Seperating from others
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9
Q

Describe classical conditioning model in relation to panic disorder

A
  • Panic attacks are classically conditioned responses to situations that evoke anxiety or bodily states of arousal (interoceptive conditioning)
  • Interoceptive conditioning refers to the physical signs of anxiety that produce the first panic attack which subsequently becomes a conditioned response to physical change
  • People with panic disorder sustain classically conditioned fears longer (Michael et al., 2007)
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10
Q

What support is there for Rachman’s (1978) vicarious conditioning?

A

Ollendick and King (1991) found that fears in children were mostly attributable to information from parents and vicarious learning as opposed to direct conditioning

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11
Q

What are three cognitive factors relating to anxiety disorders?

A
  • Sustained negative beliefs about the future
  • Perceived control
  • Attention to threat
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12
Q

What types of anxiety can be caused by cognitive factors?

A
  • Panic disorder
  • Social anxiety disorder
  • Agoraphobia
  • Generalised anxiety disorder
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13
Q

What is Clark and Wells (1995) theory of cognitive causes of anxiety?

A
  • Too much focus on negative self-evaluation
  • People have unrealistic negative beliefs about the consequences of their social behaviour
  • Believe they will be rejected (blushing)
  • Constantly think abut how other perceive them
  • Constantly pay too much attention to inner feelings
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14
Q

What models support Clark and Wells (1995) theory?

A
  • Gerlach et al., (2001) looked at blushing and found that participants with social anxiety were more likely to overestimate how much they would blush when made to carry out tasks (singing)
  • Ashbaugh et al., (2005) asked participants to rate their performance after a speech. Socially anxious participants were more self critical than controls
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15
Q

What is Clark’s (1996) cognitive model relating to panic disorders?

A

Body sensations misinterpreted as impending doom (heart attack)

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16
Q

Evidence for Clark’s (1996) model

A
  • Rapee et al (1986) used high levels of carbon dioxide to induce a panic attack
  • Found that those who were informed of sensations were less likely to have an attack than those who were given no explanation
17
Q

What is the fear of fear hypothesis and who proposed it?

A
  • Proposed by Goldstein & Chambless (1978)

- Agoraphobia driven by negative thoughts about consequences of anxiety in a public place

18
Q

What is Borkovec’s (1998) cognitive model of G.A.D?

A
  • Explains why some worry more than others
  • Worry is reinforcing and distracts from more powerful negative emotion (past traumatic events: abuse, death)
  • Worrying decreases psychophysiological states (emotion avoidance, find it hard to understand emotions and regulate them)
19
Q

What evidence is there for neurobiological causes in panic disorder?

A
  • Redmond (1977) found high activity in locus ceruleus when monkeys were exposed to fearful stimuli.
  • In humans some drugs that increase activity in the locus ceruleus can induce panic attacks and vice versa
20
Q

What risk factors are involved in developing an anxiety disorder?

A
  • Cognitive
  • Neurobiological
  • Personality
  • Behavioural
  • Genetic
21
Q

Explain genetic risk factors

A
  • Twin studies show 20-40 % heritability for specific phobias, social anxiety disorders and generalised anxiety disorder
  • 50% heritability for panic disorder
  • Family member with a phobia increases riak of developing anxiety disorder
22
Q

Explain neurobiological risk factors

A
  • People with anxiety disorders show increased amygdala activity
  • Less activity in medial prefrontal cortex
  • Deficient pathways linking amygdala and medial prefrontal cortex interfere with regulation and extinction of anxiety
  • Poor functioning of serotonin and high norepinephrine transmitters