Disorders of esophagus stomach small intestine Flashcards

1
Q

What is dysphagia, and what’s its risk factor?
ESOPHAGUS

A

is the difficulty swallowing, muscular neuropathy dysfunction
RF: aspiration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is esophagitis?

A

inflammation of the lining of the esophagus by gastric acid
stomach acid(vomiting)
medication (NSAIDS)
fungal infection (Candida albicans)

patient may complain of burning sensation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is GERD gastroesophageal reflux disorder and what is it’s s/s?
ESOPHAGUS

A

allows regurgitation of stomach acids
metaplasia (replacement of 1 cell to another)
Barrett’s esophagus can occur (stomach acid coming out and pancreas change to cells)
s/s: heart burn (acid indigestion, dysphagia, epigastric pain)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What is Upper GI bleed and what is its s/s and causes?

A

any part of upper GI (stomach, duodenum, esophagus) can be acute or chronic
Causes: Mallory-Weiss, PUD, esophageal varices, cancer
s/s: hematemesis (vomiting blood)
melena (blood-causing black tarry stools)
vital changes (decreased BP, increased HR)

Chronic = blood loss

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What is esophageal varices?

A

engorged vein in the lower end of the esophagus due to portal vein hypertension (liver cirrhosis). engorged veins may rupture, leading to liver cirrhosis (degenerative disease of liver, scarring, and liver failure)
s/s: jaundice

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What are the 2 types of Esophageal cancer and what are the causes and s/s?

A
  1. Squamous cell carcinoma: cancer invades the lining of esophagus due to chronic alcohol and tobacco use, also HPV
  2. Adenocarcinoma: cancer in distal esophagus and gastroesophageal junction (> 70% of new cases in US)
    Cause: tobacco use, GERD, Barretts esophagus
    s/s: weight loss, changes in eating patterns, dysphagia (difficulty swallowing)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What is a hiatal hernia and it’s s/s?
STOMACH

A

Part of the stomach pushes through opening in diaphragm by increased intra-abdominal pressure. asymptomatic

SLIDING hiatal hernia are more common and may become EMERGENT

s/s: GERD, dysphagia, epigastric discomfort

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What is pyloric stenosis, and what are the s/s?
STOMACH

A

hardening of sphincter so it’s not dilating stomach content constriction of pyloric sphincter so it’s not dilating stomach content constriction of pyloric sphincter gastric propulsion of contents is compromised and can be congenital.
s/s: gastroparesis (distended stomach),
firm abdomen over pylorus (lower stomach), nausea, projectile vomiting, SURGICAL REPAIR NEEDED

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is gastritis, and what are the causes and s/s?
STOMACH

A

inflammation of the mucosa, acute or chronic
Acute: due to injury to the mucosal layer from drugs or chemicals (NSAIDS, alcohol, aspirin)
Chronic: thinning and degeneration of stomach wall, more common in elderly
Cause: medications (NSAIDS, aspirin, corticosteroids), infection, acute stress, bile reflux, alcohol abuse.
Chronic s/s: burning, gnawing epigastric pain, hematemesis, weight loss, risk of stomach cancer

PRECURSOR OF STOMACH CANCER (H. pylori, chronic)
H. pylori causes irritation and erosion of the mucosa, atrophy of the stomach lining (smaller), and atrophic gastritis.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is PUD peptic ulcer disease, its causes, and s/s?
STOMACH

A

inflammatory erosion of stomach, duodenum, lower esophagus, hypersecretion of hydrochloric acid HCI, ineffective GI mucus production, poor cellular repair

Cause: H. pylori, NSAIDS (take away/ decreased gastric mucus production), alcohol abuse, stress, genetics, smoking, excessive caffeine

s/s: epigastric abdominal pain (occurs between meals/ 2-3 hrs after eating), intense pain, burning/gnawing sensation

Complications: bleeding peptic ulcers, perforation (hole) in stomach and intestine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is dumping syndrome and it’s 2 phases?

A

emptying of stomach before acids does, goes from stomach to esophagus, rapid gastric emptying, common after bariatric surgery (600ib life/ 1000ibs siters), hypertonic fluid enters intestines causing fluid shift

2 phases:
Early: 30 mins after eating
Late: 2-3 hrs after eating
dietary management is key

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What is a hernia?
small intestine

A

protrusion of the intestine through the abdominal wall (based on the location), most common hernia: inguinal hernia (above the crotch), symptoms and severity depend on location and extent
1. Reducible: returned to normal position w/ manual pressure
2. Incarceration: loop of intestine trapped
3. Strangulation: ischemia of bowel tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is gastroenteritis and its s/s?

A

irritation of stomach, small or large intestine by pathogen or toxin (virus, bacteria, parasite, chemical toxin) transmitted person to person, waterborne or foodborne infectious microorganisms, and people in close contact increase risk
s/s: electrolyte imbalance condition lasting 48–72 hours, diarrhea, abdominal cramping, nausea, vomiting, IV FLUID REPLACEMENT may be needed

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What is celiac disease?

A

sprune, gluten sensitivity enteropathy, hypersensitivity to gluten (allergic to gluten), unknown cause, autoimmune
s/s:
gluten ingestion = abdominal pain, bloating, gas, difficulty digesting carbohydrates, proteins, and fats; malnutrition and vitamin deficiencies are concerns.
Steatorrhea may develop (loss of fat in stools)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is short-bowel syndrome?

A

malabsorption/less nutriton intake, andy process that leaces less than 200cm of small intestine (normal length is 600cm), remaining intesetine adapts and increased absorptive capabilities

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What are the 4 types of small bowel obstructions and the s/s?

A

Acute: adhesions (post-surgical, major cause), herniation
Chronic: inflammatory, tumors
Partial: decreased flow of intestial contents
Complete: prevents passage of all type of content and fluids
s/s: hyperactive bowel sounds =diarrhea (may be present in partial), pain, nausea, vomiting, s/s depend on severity of obstruction

17
Q

What is peritonitis and the classic triad?

A

inflammation of the peritoneum, bacterial infection or leakage of intestial contents into peritoneal cavity
blood and fluid shift toward abdomen (ascites)
Classic triad AAR:
Abdominal pain (inflamed tissues)
Abdominal rigidty
Rebound tenderness