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01 Family Medicine > Dermatology COPY > Flashcards

Dermatology COPY Flashcards

1
Q

Define Macule

A

Non palpable

Well circumscribed change in skin color

Less than 1 cm

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2
Q

What is the primary morphology of this lesion?

A

Macule

(labial melanotic macule)

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3
Q

Define Patch

A

nonpalpable

well circumscribed change in skin color

Larger than 1 cm

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4
Q

What is the primary morphology of this lesion?

A

Patch

(Speckled Nevus)

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5
Q

Define Plaque

A

Palpable

Elevated

Solid skin lesion

Greater than 1 cm

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6
Q

Define Papule

A

Palpable

Elevated

Solid skin lesion

Less than 1 cm

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7
Q

What is the primary morphology of this lesion?

A

Papule

(Spitz nevus)

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8
Q

What is the primary morphology of this lesion?

A

Plaque

(Psoriatic plaque)

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9
Q

Define Wheal

A

Transient

Smooth papule or plaque

Seen in urticaria

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10
Q

What is the primary morphology of this lesion?

A

Wheal

(Urticaria)

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11
Q

Define Vesicle

A

Small

Fluid containing blister

Less than 1 cm

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12
Q

What is the primary morphology of this lesion?

A

Vesicles

(Herpes Zoster)

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13
Q

Define Bulla

A

Large

Fluid containing blister

Greater than 1 cm

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14
Q

Define Pustule

A

Vesicle containing pus

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15
Q

Define Nodule

A

Solid

Non-superficial skin mass

1-2 cm

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16
Q

Define Tumor

A

Solid skin mass

Greater than 2 cm

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17
Q

What is the primary morphology of this lesion?

A

Bulla

(Bullous Pemphigoid)

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18
Q

What is the primary morphology of this lesion?

A

Pustules

(Pustular psoriasis)

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19
Q

What is the primary morphology of this lesion?

A

Nodule

(Large pigmented dermatofibroma)

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20
Q

What is the primary morphology of this lesion?

A

Tumor

(Melanoma)

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21
Q

Define Scale

A

Flaking off of the stratum corneum

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22
Q

Define Crust

A

Dried exudate

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23
Q

Define Excoriation

A

Linear skin damage

Due to scratching or scraping

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24
Q

Define Lichenification

A

Skin thickening

Prominent skin lines

Due to repeated rubbing or scratching

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25
Q

Define Erosions

A

Loss of areas of the epidermis

Due to:

  • Manipulatin of the skin
  • Popping blistered areas
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26
Q

Define Ulcers

A

Deep areas of skin loss

Extends at least into the deeper dermis

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27
Q

Define Fissures

A

Cracking of the skin

Somewhat linear pattern

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28
Q

Define Atrophy

A

Thinning of the skin

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29
Q

Define Hypertrophy

A

Thickening of the skin

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30
Q

What is the secondary morphology of this lesion?

A

Scale

(Scalp psoriasis)

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31
Q

What is the secondary morphology of this lesion?

A

Crust

(Superficial infected atopic dermatitis)

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32
Q

What is the secondary morphology of this lesion?

A

Excoriations

(Scratching due to psoriasis)

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33
Q

What is the secondary morphology of this lesion?

A

Lichenification

(Lichen simplex chronicus)

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34
Q

What is the secondary morphology of this lesion?

A

Erosions

(Bullous pemphigoid after blisters have broken)

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35
Q

What is the secondary morphology of this lesion?

A

Ulcer

(Venous stasis ulcer)

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36
Q

What is the secondary morphology of this lesion?

A

Fissures

(Hand dermatitis)

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37
Q

What is the secondary morphology of this lesion?

A

Atrophy

(Discoid lupus)

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38
Q

What is the secondary morphology of this lesion?

A

Hypertrophy

(Hypertrophic lichen planus)

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39
Q

Define Erythematous

A

Pink to red

Increase in blood flow to the area

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40
Q

Define hypopigmented

A

Decrease in pigmentation from the surrounding area

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41
Q

Define Depigmented

A

Complete lack of pigmentation

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42
Q

Define Hyperpigmented

A

Increase in pigmentation from the surrounding skin

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43
Q

What is color/pigmentation of this lesion?

A

Erythematous

(Erythrodermic psoriasis)

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44
Q

What is color/pigmentation of this lesion?

A

Hypopigmented

(Postinflammatory hypopigmentation with treatment of psoriasis)

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45
Q

What is color/pigmentation of this lesion?

A

Depigmented

(Vitiligo)

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46
Q

What is color/pigmentation of this lesion?

A

Hyperpigmented

(Postinflammatory hyperpigmentation and Acanthosis nigricans)

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47
Q

Define Ecchymosis

A

Bruising from bleeding under the skin

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48
Q

Define Petechiae

A

Pinpoint bleeding in the skin

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49
Q

Define Purpura

A

Larger areas of bleeding or vascular inflammation

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50
Q

Define Telangiectasis

A

Small dilated vessels

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51
Q

Define Nummular

A

Coin shaped

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52
Q

Define Serpiginous

A

Snakelike 🐍

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53
Q

Define Annular

A

Bordered by a raised ring

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54
Q

Define Reticular

A

Netlike

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55
Q

Define Umbilicated

A

Like an umbilicus

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56
Q

Describe the distribution of Acral

A

Hands and Feet

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57
Q

Describe the distribution of Intertriginous

A

In areas of skin folds

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58
Q

Describe the distribution of Photo-distributed

A

In areas prone to sun exposure

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59
Q

Define Verrucous

A

Wartlike with a rough surface

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60
Q

What vascular term would be used to describe this lesion?

A

Petechiae

(Thrombocytopenia)

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61
Q

What vascular term would be used to describe this lesion?

A

Purpura

(Palpable Purpura in Henoch-Schonlein purpura)

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62
Q

What vascular term would be used to describe this lesion?

A

Telangiectasias

(Rosacea)

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63
Q

Describe the surface characteristics of this lesion

A

Verrucous

(Wart)

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64
Q

Describe the surface characteristics of this lesion

A

Pearly and smooth

(Basal Cell Carcinoma)

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65
Q

Describe the shape of this lesion

A

Nummular

(Nummular Dermatitis)

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66
Q

Describe the shape of this lesion

A

Serpiginous

(Cutaneous larva migrans)

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67
Q

Describe the shape of this lesion

A

Annular

(Granuloma annulare)

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68
Q

Describe the shape of this lesion

A

Reticular

(Livedo Reticularis)

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69
Q

Describe the shape of this lesion

A

Umbilicated

(Molluscum)

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70
Q

How would you describe the location of this condition?

A

Intertriginous

(Inverse psoriasis)

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71
Q

Differential diagnosis for skin lesion with plaques and scales?

A

Psoriasis

Chronic cutaneous discoid lupus

Tinea Corporis

Paget’s Disease

Lichen Planus

Bowen’s Disease

Cutaneous T-cell lymphoma (mycosis fungoides)

Pityriasis rosea

Secondary Syphilis

Ichthyosis

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72
Q

Describe Psoriasis

A

Oval, erythematous plaques

Extensor surfaces or sites of trauma

Pitting fingernails

Associated with asymmetric polyarthritis

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73
Q

What is the treatment of psoriasis if <5% of the body is involved?

A

Topical:

corticosteroids

Calcipotriene (Vit D3 analog)

Retinoids (tazarotene)

Systemic:

Calcineurin Inhibitors- Tacrolimus

Cyclosporine

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74
Q

What is the treatment of psoriasis if >5% of the body is involved?

A

Vitamin D analogs +/- phototherapy

Sysemtic/Biologic agents

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75
Q

What is the treatment for severe, recalcitrant disabling psoriasis?

A

Phototherapy, UVB

Systemic Agents:

Methotrexate

Acitretin

Cyclosporine

Apremilast

Anti-TNF agents
(Ex: Infliximab)

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76
Q

What is the differential for Bullous Lesions?

A

Herpes Simplex

Herpes Zoster

Impetigo

Dermatitis Herpetiformis

Burns

Bullous pemphigoid

Pemphigus Vulgaris

Eyrthema Multiforme

Porphyria Cutanea Tarda

Fixed drug eruption

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77
Q

Describe Herpes Zoster lesions

A

Vesicles of varying size on erythematous base

Typically in a single dermatome

Successive crops over 7 days followed by crusting for 2-3 weeks

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78
Q

How does Herpex zoster present?

A

Pre-eruptive pain, burning, itching (usually in one dermatome)

Fever, headache, malaise

Postherpetic neuralgia (pain after lesions clear up)

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79
Q

What is the treatment of Herpes Zoster?

A

Analgesics

Antiviral therapy- Acyclovir, Famciclovir, or Valacyclovir for 7 days

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80
Q

What is the role of corticosteroids in Herpes Zoster treatment?

A

May decrease pain in acute phase

No reduction in risk of postherpetic neuralgia

May increase risk of secondary infection

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81
Q

What is the treatment for postherpetic neuralgia?

A

Pregabalin or Gabapentin

TCAs

Opioids

Sympathetic nerve block - Bupivacaine

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82
Q

When is Shingrix (HZV) vaccine given?

A

50 years and older

Regardless of hx of varicella or other zoster vaccines

2 doses - 2 to 6 months apart

90% effective

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83
Q

Which vaccine is prefered for HZV?

A

Shingrix

Higher effectivity than Zostavax

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84
Q

What is the differential for pruritic erythematous papules?

A

Miliaria Rubra

Atopic dermatitis

Urticaria

Insect bites

Scabies

Pruritic papular Eruption

Pruritic urticarial papules and plaques of pregnancy (PUPPP)

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85
Q

What skin condition is commonly on flexor surfaces v. extensor surfaces?

A

Flexor- Atopic dermatitis

Extensor- Psoriasis

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86
Q

Describe scabies lesions

A

Curved or linear burrows

Vesicles or papules

Pustules indicated secondary infection

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87
Q

What is scabies?

A

Hypersensitivity reaction to Sarcoptes scabiei

May present with nocturnal pruritus

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88
Q

Where are scabies lesions commonly seen?

A

Axilla

Breasts

Elbows, wrists

Finger Webs

Waist

Buttocks, Genitals

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89
Q

How is scabies diagnosed?

A

Mark burrow with felt tip pen

Examine scraping under microscope with KOH or mineral oil

Mites will appear black

May also see eggs and feces

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90
Q

What is Norwegian Crusted Scabies?

A

Overwhelming infestation

Crusted lesions

Not as pruritic as regular scabies

Typically seen in immunocompromised patients

91
Q

What is the treatment for scabies?

A

Wash all clothes and bedding used in past 48 hours in hot water or seal in plastic for 3 days

Treat all members of household and close contacts

5% Permethrin cream

Ivermectin

92
Q

What is the best medical treatment for Norwegian Scabies?

A

Ivermectin 200 mcg/kg PO

Repeat in 1-2 weeks

93
Q

What is Rhus Dermatitis?

A

Contact with urushiol

  • Poison Ivy, Poison Oak
  • Poison sumac
  • Cashew, Mango
  • Ginkgo, Japanese lacquer tree
94
Q

Describe Rhus Dermatitis lesions

A

Linear

Vesicles, erythema

May appear 8 hours to 1 week after exposure

95
Q

What is the treatment for Rhus Dermatitis?

A

Decontaminate within 10-15 minutes of exposure

Wet compress, calamine lotion

High potency topical steroids

Systemic steroids

96
Q

Describe Folliculitis v. Furuncle v. Carbuncle

A

Folliculitis- superficial bacterial infection of hair follicles, purulent material in epidermis

Furuncle- deep infection of hair follicle, purulent material through dermis and subcutaneous tissue

Carbuncle- coalescence of several furuncles into single mass, purulent drainage from multiple follicles

97
Q

What is sycosis barbae?

A

Folliculitis in the beard area

98
Q

What organisms can cause foliculitis?

A

Staph Aureus

Pseudomonas (Hot tubs)

Candida

Non TB mycobacteria

99
Q

What is the treatment for folliculitis?

A

May resolve spontaneously with or without drainage

Warm compresses TID

Avoid shaving

Antibiotics if not resolving in 2-3 weeks

100
Q

What is the treatment for hot tub folliculitis?

A

Most cases self-limited

Avoid exposure to contaminated water source

Acetic acid compresses 2-4 times per day

If severe, use ciprofloxacin

101
Q

Antibiotic treatment for folliculitis?

A

Usually self resolves, but if lasting 2-3 weeks or extensive involvement:

Mild Staph: Topical mupirocin

Not MRSA: cephalexin, dicloxacillin

MRSA: Bactrim, clindamycin, doxycycline

Fungal: Topical azoles

102
Q

What is Impetigo?

A

Contagious superficial bacterial infection

Common in kids 2-5 years old

103
Q

What is primary v. secondary Impetigo?

A

Primary- direct invasion of normal skin

Secondary- Infection at sites with previous minor trauma (ex: abrasions, eczema)

104
Q

Describe the lesions of nonbullous impetigo

A

Most common

Papules surrounded by erythema that progress to pustules

Pustules form thick crusts (golden)

105
Q

What organism is the most common cause of Impetigo?

A

Staph Aureus

(Beta hemolytic strep causes a minority of cases)

106
Q

Describe the lesions of Bullous Impetigo

A

Vesicles enlarge and form flaccid bullae (clear yellow fluid)

Then become dark and turbid

Ruptured bullae leave brown crust

107
Q

What is a rare complication of streptococcal Impetigo?

A

Poststreptococcal glomerulonephritis

108
Q

What is the treatment of Impetigo?

A

Topical- Mupirocin, Retapmulin

PO- Dicloxacillin, cephalexin, clindamycin

-MRSA of PCN allergic- Clindamycin, Bactrim, Linezolid

109
Q

What is the pathogenesis of Acne Vulgaris?

A

Androgen-mediated disorder of pilosebaceous units

  • Androgens stimulate sebum production and keratinocyte proliferation
  • Keratin plugs obstruct follicle os
  • Cutibacterium acnes (P. acnes) proliferates and produces inflammation
110
Q

What is the name of whiteheads v. blackheads?

A

Whiteheads- closed comedones

Blackheads- open comedones

111
Q

Why are the face, chest, back and arms the most common areas for acne vulgaris?

A

Areas with highest concentration of sebaceous glands

112
Q

What is the treatment for comedonal acne?

A

Topical retinoids

If uneffective add benzoyl peroxide

Maintenance therapy: Topical retinoid

113
Q

What is the treatment for mild inflammatory acne with papules and pustules?

A

Topical retinoid plus benzoyl peroxide

If uneffective, add topical or oral antibiotic

Maintenance therapy: Topical retinoid

114
Q

What is the treatment for moderate inflammatory acne with papules and pustules?

A

Topical retinoid and benzoyl peroxide and topical antibiotic

If uneffective, add PO antibiotic

Maintenance therapy: Topical retinoid and benzoyl peroxide

115
Q

What is the treatment for moderate inflammatory acne with nodules?

A

Topical retinoid and benzoyl peroxide plus PO antibiotics

If uneffective, add oral isotretinoin

Maintenance therapy: Topical retinoid and benzoyl peroxide

116
Q

What is the treatment for severe inflammatory acne with papules and pustules and nodules?

A

Oral isotretinoin

Maintenance therapy: Topical retinoid and benzoyl peroxide and topical or PO antibiotic

117
Q

Name some nonantibiotic topical treatments for acne vulgaris

A

Azelaic acid

Benzoyl peroxide

Dapsone

118
Q

Name some topical antibiotics used to treat acne vulgaris

A

Clindamycin

Erythromycin

119
Q

Name some topical retinoids use to treat acne vulgaris

A

Adapalene

Tazarotene

Tretinoin

120
Q

Name some systemic antibiotics use to treat acne vulgaris

A

Doxycycline

Erythromycin

Minocycline

Tetracycline

Bactrim

121
Q

Can topical antibiotics be used as monotherapy for acne vulgaris?

A

No

Always use with benzoyl peroixde to prevent antibiotic resistance

122
Q

What is the maximum length of PO antibiotic therapy for treating acne vulgaris?

A

12 weeks

Can stop if inflammatory lesions resolve or switch to topical without taper

Do not use topical or PO antibiotics together

123
Q

What are two medications to consider for females with acne vulgaris?

A

Oral contraceptives

Spironolactone

124
Q

What are the side effects of Isotretinoin?

A

Dry, red peeling skin

Chelitis (Chapped lips)

Hyperlipidemia

Increased intracranial pressure

Teratogen (iPledge program)

125
Q

Name some alternative treatment options for acne vulgaris

A

UVA/UVB

Comedone extraction

Steroid injection into large cysts

Topical tea tree oil

126
Q

Describe Rosacea

A

Erythema, transient symmetric flushing

Papules, pustules

Telangiectasia

Forehead, cheeks, nose, ocular area

127
Q

What is the prevalence of rosacea and who is most commonly affected?

A

>15 million in US

“Curse of the Celts” (Irish descent)

Women > Men

More common after 30

128
Q

What is the treatment for Rosacea?

A

Topical antibiotics- metonidazole, erythromycin

  • Azelaic acid or benzoyl peroxide
  • PO antibiotics
  • Isotretinoin if severe or resistant
  • Brimonidine (alpha 2 agonist) for erythema
  • Carvedilol PO
129
Q

Who is most likley to get seborrheic dermatitis?

A

Infants 2 weeks - 12 months

Adolescents and adults

Increased incidence in patient with HIV or Parkinsons

130
Q

What is the treatment of seborrheic dermatitis?

A

Low potency topical steroids

Topical antifungals and/or shampoos

131
Q

What is the treatment for perioral dermatitis?

A

Topical pimecrolimus or metronidazole

PO Doxycyline, tetracycline or Minocycline

132
Q

Describe the most common rash seen in Lupus Erythematosus?

A

Erythema over the cheeks and nose

Spares nasolabial folds

Worse with sun exposure

(Butterfly rash)

133
Q

What is the treatment of Discoid Lupus Erythematous?

A

Photoprotection, avoid aggravating drugs, smoking cessation

Topical steroids

Topical calcineurin inhibitors

PO Antimalarials

134
Q

Differential for acquired hypopigmented lesions

A

Pityriasis alba

Vitiligo

Tinea Versicolor

Postinflammatory Hypopigmentation

Leprosy

Halo Nevus

Phytophotodermatitis

135
Q

What causes Tinea Versicolor?

A

Pityrosporum (Malassezia) species

Worse with heat, humidity, pregnancy, steroids, immunosupression

136
Q

Describe tinea versicolor lesions

A

Circular macules

Tan, dark brown or hypopigmented

Powdery scale

Upper trunk, neck, abdomen

pale yellow on woods lamp

137
Q

What is the treatment for tinea versicolor?

A

Topical antifungals- cream, shampoo

PO for extensive or resistant disease

138
Q

What causes Erythrasma?

A

Corynebacterium minutissium

Less inflammatory than tinea

Coral pink on woods lamp

139
Q

What is the treatment for erythrasma?

A

Topical clindamycin, erythromycin

Topical clotrimazole

PO clindamycin, erythromycin if severe

140
Q

What is the treatment for tinea capitis?

A

PO only:

  • Griseofulvin 6-12 weeks
  • Terbinafine 2-4 weeks
  • Itraconazole 4-6 weeks
  • Fluconazole 3-6 weeks
141
Q

What causes Alopecia areata?

A
142
Q

What is seen with alopecia areata?

A

Exclamation point hairs (proximal end is narrower than distal end)

  • on the edges of patches
  • with short broken hairs
  • extracted with minimal traction
143
Q

Alopecia totalis v. universalis

A

Totalis- complete loss of scalp hair

Universalis- loss of all scalp and body hair

144
Q

What is the treatment of alopecia areata?

A

Corticosteroids- topical, systemic, or intralesional

Topical minoxidil 5%

Topical immunotherapy

145
Q

What is the prognosis of alopecia areata?

A

50% recover within a year

May persist for several years

10% do not regrow hair

146
Q

What is the treatment of pediculosis capitis?

A

(Head Lice)

Pyrethroids (Permethrin)

Malathion lotion

Benzyl alcohol

Topical or oral ivermectin

Topical spinosad

147
Q

How long do children with pediculosis capitis (lice) need to be out of school?

A

Once treatment has started, no need to be excluded from school

Examine and treat household members

148
Q

What causes warts?

A

Infection with HPV

Type 1- plantar warts

Types 6 & 11- genital warts

149
Q

What is the treatment of genital warts?

A

Patient administered (for about 16 weeks):

-Podofilox 5%, Imiquimod 5% or Sinecatechins 15%

Provider administered:

-Cyrotherapy, BCA/TCA, surgical or laser removal

150
Q

How can you differentiate warts v. callus?

A

Callus have skin lines and warts have no skin lines

151
Q

What causes molluscum contagiosum?

A

Double stranded DNA Pox Virus

Spread by skin to skin contact

152
Q

Describe molluscum contagiosum lesions

A

Umbilicated, firm, flesh colored, dome shaped papules

Kids- anywhere except palms and soles

Adults- usually genital area

153
Q

What is the treatment for molluscum contagiosum?

A

May self resolve in 6 months to 4 years

Cryotherapy, Curettage, Laser

Imiquimod, Podofilox, KOH

Topical retinoids, salicylic acid

154
Q

Differential for nodular lesions?

A

Basal cell carcinoma

Squamous cell carcinoma

Keratocanthoma

Sebaceous hyperplasia

Melanoma

Neurofibroma

Hemangioma

Prurigo nodularis

155
Q

What is the epidemiology of basal cell carcinoma?

A

Most common skin cancer

Male > female

Mostly age 40 and older

85% on head or neck

156
Q

What is the treatment of basal cell carcinoma?

A

Excisional biopsy

Cryotherapy or electrodesiccation/curettage (<6mm)

Mohs surgery- large, recurrent, nose/eyelid, sclerosing

Radiation or chemotherapy if advanced

157
Q

What is the epidemiology of actinic keratosis?

A

Risk increases with age, fair skin, sun exposure

Males > females

On sun exposed areas

158
Q

Describe actinic keratosis lesions

A

Rough, scaly patches

Skin tone to red/brown

Well circumscribed

1mm to 25 mm

Often multiple present

159
Q

What is the prognosis of actinic keratosis?

A

Potential to progress to squamous cell carcinoma (most do not)

*60% of SCC arise from AKs

26% regress spontaneously

160
Q

What is the treatment of actinic keratosis?

A

Cryotherapy

Curettage

Aminolevulinic acid

Topical fluorouracil, imiquimod, diclofenac or ingenol mebutate

161
Q

Name some precursors to squamous cell carcinoma

A

Actinic Keratosis

Keratocanthoma

Cutaneous horn

Bowen’s disease

Erythroplasia of Queyrat

162
Q

What are some risk factors for squamous cell carcinoma?

A

Chronic injury or disease of the skin

Exposure to UV radiation

Immunosuppression

163
Q

How is squamous cell carcinoma diagnosed?

A

Shave biopsy- if raised

Punch biopsy

164
Q

What is the treatment for squamous cell carcinoma?

A

Surigical excision/Mohs surgery

Electrodesiccation/curettage if small/low risk

Radiotherapy if high risk, unable to surgically remove

165
Q

Differential for pigmented lesions?

A

Intradermal nevus

Melanoma

Seborrheic keratosis

Kaposi’s Sarcoma

Cherry angioma

Pigmented basal cell carcinoma

166
Q

Epidemiology of malignant melanoma?

A

Non-hispanic caucasians at highest risk

Median age 57

Men > women

167
Q

Risk factors for malignant melanoma?

A

Large number of atypical nevi

Hx of other skin cancers

Hx of congenital giant nevus

Family hx of melanoma

Immunosuppression

UV radiation exposure

168
Q

What are the ABCDEs of melanoma?

A

Asymmetry

Border irregularity

Color variegation

Diameter > 6mm

Evolving

Ugly duckling sign (looks different than other nevi)

169
Q

What feature of melanoma determines its prognosis?

A

Thickness

AJCC staging used

170
Q

How is melanoma diagnosed?

A

Full thickness biopsy

  • Excision with at least 2 mm borders
  • (Very wide excision not necessary)
171
Q

What is the treatment of seborrheic keratosis?

A

Cryosurgery

Curettage +/- electrodessication or shave excision

Observation

172
Q

What is the sign of Leser-trelat?

A

Sudden appearance of numerous seborrheic keratoses

Sudden growth of existing SKs

Associated with malignancy (gastric adenocarcinoma)

173
Q

What do Epidermal Cysts contain?

A

Keratin

174
Q

What is the treatment for epidermal cysts?

A

If no infection/inflammation can resolve without treatment

Injection of triamcinolone

Excision (4-6 weeks after inflammation resolves)

I&D if infection or severe inflammation

PO abx if not responding after I&D

175
Q

What is the treatment of Keloid?

A

Intralesional steroids

70% improve

50% recurrence at 5 years

*may cause atrophy, hypopigmentation

176
Q

What is the most likely diagnosis of this skin finding?

A

Plaque Psoriasis

177
Q

What is the most likely diagnosis of this skin finding in the axilla?

A

Tinea corporis

(Annular pururitic lesion with concentric rings)

178
Q

What is the most likely diagnosis of this skin finding?

A

Pityriasis rosea

(Arrow points to herald patch)

(Can have “Christmas tree” distribution)

179
Q

What is the most likely diagnosis of this skin finding?

A

Bowen disease

(Squamous cell carcinoma in situ)

*lesion on upper edge of photo is an actinic keratosis

180
Q

What is the most likely diagnosis of this skin finding?

A

Pityriasis rosea

(Can have “Christmas tree” distribution)

181
Q

What is the most likely diagnosis of this skin finding?

A

Ichthyosis vulgaris

182
Q

What is the most likely diagnosis of this skin finding on flexor surface of the wrist?

A

Lichen planus

183
Q

What is the most likely diagnosis of this skin finding?

A

Herpes Zoster

184
Q

What is the most likely diagnosis of this skin finding?

A

Bullous pemphigoid

185
Q

What is the most likely diagnosis of this skin finding?

A

Erythema multiforme

186
Q

What is the most likely diagnosis of this skin finding on the palms?

A

Scabies

(Notice the burrows)

187
Q

What is the most likely diagnosis of this skin finding?

A

Atopic dermatitis

188
Q

What is the most likely diagnosis of this skin finding?

A

Allergic contact dermatitis from poison ivy

189
Q

What is the most likely diagnosis of this skin finding?

A

Contact dermatitis

(Likely from pants button or belt)

190
Q

What is the most likely diagnosis of this skin finding?

A

“Hot tub” folliculitis

(Distribution around swim suit or water line)

191
Q

What is the most likely diagnosis of this skin finding?

A

Pseudofolliculitis barbae

(Shaving worsens condition)

192
Q

What is the most likely diagnosis of this skin finding?

A

Impetigo

(Honey crusted plaque)

193
Q

What is the most likely diagnosis of this skin finding?

A

Severe inflammatory acne

194
Q

What is the most likely diagnosis of this skin finding?

A

Comedonal acne

195
Q

What is the most likely diagnosis of this skin finding?

A

Inflammatory acne with pustules and nodules

196
Q

What is the most likely diagnosis of this skin finding?

A

Rosacea

(Erythema and telangiectasias)

197
Q

What is the most likely diagnosis of this skin finding?

A

Papulopustular rosacea

198
Q

What is the most likely diagnosis of this skin finding?

A

Rhinophymatous rosacea

199
Q

What is the most likely diagnosis of this skin finding?

A

Seborrheic dermatitis

200
Q

What is the most likely diagnosis of this skin finding?

A

Perioral dermatitis

(tiny papules, scaling, erythema around mouth and nose

201
Q

What is the most likely diagnosis of this skin finding?

A

Tinea versicolor

202
Q

What is the most likely diagnosis of this skin finding?

A

Pityriasis alba

203
Q

What is the most likely diagnosis of this skin finding?

A

Erythrasma

(Coral red flourescence with Wood’s lamp)

204
Q

What is the most likely diagnosis of this skin finding on scalp?

A

Tinea capitis

205
Q

What is the most likely diagnosis of this skin finding?

A

Allopecia areata

206
Q

What finding do these arrows indicate in alopecia areata?

A

Exclamation point sign

(Hair is narrow at the base, wide at the end)

207
Q

What is the most likely diagnosis of this skin finding?

A

Traction alopecia

208
Q

What is the most likely diagnosis of this skin finding?

A

Melasma

209
Q

What is the most likely diagnosis of this skin finding?

A

Plantar Wart

(Notice that skin lines are disrupted)

210
Q

What is the most likely diagnosis of this skin finding?

A

Molluscum contagiosum

211
Q

What is the most likely diagnosis of this skin finding?

A

Superficial basal cell carcinoma

(Notice the pearly border and areas of pigmentation)

212
Q

What is the most likely diagnosis of this skin finding?

A

Squamous cell carcinoma

213
Q

What is the most likely diagnosis of this skin finding?

A

Melanoma

214
Q

What is the most likely diagnosis of this skin finding?

A

Actinic keratosis

215
Q

What is the most likely diagnosis of this skin finding?

A

Keratoacanthoma

(Notice telangiectasias ans central keratin core)

216
Q

What is the most likely diagnosis of this skin finding?

A

Keratoacanthoma

(Central keratin core)

217
Q

What is the most likely diagnosis of this skin finding?

A

Seborrheic keratosis

(Notice visible horn cysts)

218
Q

What is the most likely diagnosis of this skin finding?

A

Pyogenic granuloma

219
Q

What is the most likely diagnosis of this skin finding?

A

Compound dysplastic nevus

220
Q
A
221
Q
A
222
Q
A
223
Q
A

01 Family Medicine (16 decks)

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