Dermatology Flashcards
how do you measure the extent of a burn
wallaces rule of 9’s
- head & neck - each arm - each anterior leg - each posterior leg - anterior chest - posterior chest - anterior abo - posterior abdo
what is the most accurate method of measuring the extent of a burn
lund and browden chart
red and painful burn which is dry with no blisters
superficial epidermal (1st)
pale pink burn which is painful, blistered and has a slow capillary refill
superficial dermal (2nd)
white burn with patches of non-blanching erythema, reduced sensation and pain on deep pressure
deep dermal (2nd)
white waxy, brown leathery or black burn with no blisters and no pain
full thickness (3rd)
give some initial management for burns
first aid
ANALGESIA
early intubation if burns to face/inhalation injuries
urinary catheter
IV fluids
management of superficial epidermal
analgesia and emollients
management of superficial dermal
no emollients, non-adherent dressing to keep the blister intact
management of severe full thicknesss burns
escharotomies
where are deep dermal, full thickness, superficial dermal more than 3% in adults or 2% in paeds, inhalational injuries, electrical or chemical burns managed
secondary care
burns more than 10% in adult or 5% in child or complex burn
burns unit
what causes oedema weeks after a burn
loss of plasma proteins
what is the formula for IV fluid calculation in burns patients
PARKLAND FORMULA
% SA of the burn x weight x 4
give half in first 8 hours and half in next 16
skin cancer presenting as a painless bleeding ulcer on sun exposed skin with RF of sunlight, smoking, leg ulcers, genetics, bowens disease, actinic keratosis
squamous cell carcinoma
management of skin cancers
surgery with wide excision
what factors contribute towards a poor prognosis in skin cancer
deep/large diameter or patient immunosuppressed
precancerous dermatosis
slow growing red scaly patches in sun exposed areas
managed with topical flurouracil
bowen’s disease (-> SCC)
premalignant condition with small crusty, scaly and itchy lesions in the sun
managed with topical flurouracil, diclofenac or surgery
actinic keratosis
benign tumour similar to an SCC
dome crater filled with keratin
keratocanthoma
skin cancer with a pearly white edge
BCC
most common BCC raised translucent papule on face with local destruction
nodular BCC
what are the 4 other BCC types
superficial: trunk in 50’s
morpheaform: flat irregular plaque
cystic: clear blue-grey
basosquamous: very invasive
most common type of malignant melanoma presenting as a slow growing mole in a young person
superficial spreading malignant melonoma
second most common malignant melanoma presenting as a red/black bleeding lump in sun exposed areas of middle aged which metastasises early
nodular malignant melanoma
least common malignant melanoma presenting as a growing mole in elderly
lentigo maligna
rare form of malignant melanoma in the nails, palms or soles of darker skinned individuals which exhibits hutchinsons sign
acral lentiginous
what are the surgical margins of malignant melanoma based on
breslow thickness
premalignant condition in smokers presenting with white hard spots on the mucous membranes of the mouth
leucoplakia
what are the two types of dermatitis
irritant: non allergic on hands from cleaning/cement
allergic: T4 hypersensitivity with weeping eczema
management of allergic dermatitis
patch test and steroid
20-45 year old woman presenting with clusters n the perioral region which worsen with steroids and are treated with ABx
perioral dermatitis
dermatitis around the anus
zinc deficiency
eczematous rash on the face associated with otitis externa and blepharitis treated with ketoconazole/steroids
seborrheic dermatitis
SE of ketoconazole
gynaecomastia
autoimmune condition causing IgA deposits in coeliac disease presenting with itchy vesicular lesions on extensors treated by removing gluten or dapsone
dermatitis herpetiformis
antibodies against desmosomes causing mucosal ulcers and skin blisters
managed with steroids and immunosuppressants
pemphigus vulgaris
blistering skin in the elderly treated with steroids
bullous pemphigold
pathophysiology of vitiligo
reduced melanocytes causes well demarcated depigmented skin
precipitated by trauma
management of vitiligo
suncream, make up, tacrolimus, steroids, phototherapy
what must you test for in patients who have been diagnosed with vitiligo or alopecta acreta
other autoimmune diseases
which organism causes impetigo
staph aureus/pyrogens
presentation of impetigo
golden crusted lesions