Dental Anomolies Flashcards
missing teeth major differential
also called?
Ectodermal dysplasia
hypodontia, oligodontia, anodontia
macrodontia disease mechanism
cause unknown - but vascualr abnormalities
see macrodontia on one side?
bilaterally – more likely congenital / unknonw cause
if one area - could be significant
has transposition been reported in primary teeth?
no
fusion vs gemination difference
fusion – one less tooth
number of teeth is decreased by one with this
concrescence
fusion of roots together by the cementum
gemination
looks like fusion but when count number of teeth – will remain normal
NORMAL NUMBER OF TEETH
main way to recognoze dilaceration
bullseye – ROOT apex curves and then can see either buccal or lingually the opening
*dens invaginatis
ingrwoing of enamel into pulp space
common location is lateral incisor
more severe where deforamties in crown of tooth
- dens in dente
spectrum of dens invaginatus
dens invaginiatis < in dente < dialted odontoma
dialted odontoma
associated with third molar
most common appearance of AI
hypoplastic type
Dentinogenesis and OI appears? *
bulbous crown
narrow roots
cervical constriction!!
root canals absent or thin
Dentin Dysplasia * type I
radicualr
type II of dentin dysplasia *
coronal
bulcous crowns
Dentinogen Imperfecta
narrow roots
DI
flame shaped pulp
dysplasia type II
- coronal
which has normal root?
type II outline
W shaped molar root?
Type I dentin dysplasia
ghost teeth
regional odontodysplasia
enamel pearl * location
APICAL TO CEJ on the roots
enamel pearl * location
APICAL TO CEJ on the roots
turners hypoplasia location?
localized
- not bilateral
- usually infection or trauma
perm tooth with local enamle hypoplastic defect in crown caused by infection from primary tooth or trauama
turners hypoplasia location?
localized
- not bilateral
- usually infection or trauma
perm tooth with local enamle hypoplastic defect in crown caused by infection from primary tooth or trauama
mulberry molars associated with?
congenital syphilis cases
attrition *
PHYSIOLOGICAL WEARING
DD with atterition
AI – this will be affecting all the teeth and congenital
abrasion *
NON-PHYSIOLOGIC FRICTION INDUCED WEARING
EROSION by?
CHEMICAL ACTION
-
erosion DD?
abrasion
but in erosion – esdges of erosion lesionos are usually more roundede than abrasion
*external resorption
PDL space is in tact
can have overlap of pulp space
also if on the side more apparent for external
*internal resorption
widening the canal space
pulp stones implication
root canal treatment – more difficult
*hemifacial microsomia DD
condylar hypoplasis
- but this one EFFECTS THE EARS TOO
*Treacher collins syndrome
underdevelopment of zygomatic bones
- mandible will be down-turned
downward inclination of palpebral fissure
underdevelopemetn
see a lot of supernumerary teeth?
Cleidocranial Dysaplsia
Cleidocranial Dysaplsia * clinical and imaging features
hypoplastic maxilla and aplasia or HYPOPLASIA OF CLAVICLES BUT NORMAL SIZED MANDIBLE WITH MULTIPLE SUPERNUMERARY TETEH
*lingual salivary gland depression location?
more often than not BELOW THE CANAL
salivary gland depression appearance
WD corticated round , ovoid
*focal osteoporotic bone marrow
variation of normal anatomy
lamina dura in tact *
surrounding bone is normal trabeculation
if missing and conicical shaped think?
ectodermal dysplasia
hypercementosis
be able to recognize this — LD and PDLspace encompasses the enlargement
