Cortex Localization Flashcards

1
Q

how did brodmann define his original areas?

A

slight differences in the cellular composition and laminar structure w/in the cortex

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2
Q

executive v perceptual corticies

A

frontal- executive

caudal- perceptual

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3
Q

association cortex

A

input from multiple sensory modalities

associated w/ higher processes such as memory, planning, language, math

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4
Q

parietal association cortex

A

includes brodmann area 7

involved w/ spatial localization , attention, agency

important for working memory w/ frontal

deficits can result in neglect- contralateral neglect occurs when damage occurs in non dominant hemisphere

deficits also cause optic apraxia, optic ataxia, and tactile agnosia

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5
Q

contralateral neglect syndrome

A

caused by damage to non dominant parietal cortex

non dominant parietal lobe attends to both sides of world

spatial attention in dominant hemisphere is only contralateral, thus loss of non dominant parietal cortex leads to contralateral neglect

deficits may be severe or very mild (extinction)

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6
Q

optic apraxia/ataxia

A

apraxia- deficits in visual scanning of the environments

ataxia- deficit in hand eye coordination

both caused by parietal damage

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7
Q

balints syndrome

A

co occurrences of optic apraxia, ataxia, and simultanagnosia (inability to perceive multiple objects in visual field

occurs following bilateral parietal damage

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8
Q

tactile agnosia

A

inability to identify objects by touch despite normal sensory ability

requires parietal cortex

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9
Q

temporal association cortex

A

brodmanns areas 21 and 22

process auditory and visual info, important for object recognition and language comprehension

deficits: visual agnosia, prosoagnosia, auditory agnosia, receptive aphasia, acalculia

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10
Q

visual agnosia

A

inability to recognize objects by sight

temporal association

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11
Q

prosopagnosia

A

inability to recognize faces

occurs after lesions to non dominant inferior temporal lobe

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12
Q

auditory agnosia

A

inability to recognize or make sense of complex sounds (words)

temporal association

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13
Q

receptive aphasia

A

wernickes aphasia

damage to wernickes area leads to deficits in language comprehension

temporal association

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14
Q

acalculia

A

impairs ability to do math

occurs w/ damage to dominant temporal lobe and angular gyrus

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15
Q

dorsolateral cortex

A

areas 9 10 46

executive functions- working memory, judgement, planning, sequencing of activity, dividing attention, abstract reasoning

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16
Q

orbitomedial prefrontal cortex

A

areas 11 and 12

impulse control, personality, reactivity to surroundings, mood

17
Q

anterior cingulate gyrus

A

part of orbitomedial prefrontal cortex

associated w/ mood

18
Q

frontal cortex and motor behaviors

A

frontal cortex inhibits motor behaviors, but this can be impaired if the frontal cortex is damaged

ex. utilization behaviors- individuals grasp nearby objects in meaningful ways but at inappropriate times

perseveration- repetition of a normal motor pattern after it should stop

19
Q

vountary movement

A

associated w/ a sense of agency that stems from parietal cortex

however, the decisions themselves are associated w/ increased activity in supplementary motor cortex before their is conscious awareness of the decision- readiness potential

20
Q

abulia

A

inability to initate movements

21
Q

alien hand syndrome

A

damage to the connections between frontal and parietal association cortex

there is no sense of agency,
and the hand seems to move as it it were independently controlled

22
Q

dominant v non dominant hemispheres

A

applies to which hemisphere shows language specialization

most people- left is dominant and has language

23
Q

Wada test

A

anesthetic injected into internal carotid only on one side while they are doing a task that requires verbal skills- identifies langauge dominant hemisphere

24
Q

language cortex

A

wenickes (22) and brocas (44 and 45- inferior frontal gyrus) areas

wernickes- receptive aphasia- problems decoding the meaning of words

brocas- motor aphasia- difficulty generating spoken or written words

both are also used for abstract symbolism- both used in deaf individuals for sign language

25
Q

transcortical aphasias

A

wernickes and brocas are intact, but areas around them are damaged

repeat very well, but cannot understand complex phrases or generate spontaneous language

echolalia- parrot back whatever is said to them

26
Q

non dominant wernickes and brocas areas

A

used to understand or generate inflections that put language into context

27
Q

self identification v identification of others

A

self ident- occurs in dominant hemi

others- occurs in non dom- including facial recognition

28
Q

prefrontal laterlization

A

damage to dominant produces depression

damage to non dom produces mania