context week 6 Flashcards

1
Q

why are the terms “buckle fracture” and “greenstick fracture” associated with children #

A

children’s bones tend to buckle/partially fracture/splinter with some degree of continuity of some “fibres” of bone (like breaking a green stick from a tree) rather than break completely.

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2
Q

why do childrens # heal quicker than adults?

A

thicker periosteum which is a rich source of osteoblasts.

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3
Q

what does the periosteum do?

A

serves to increase the width/circumference of growing long bones

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4
Q

what are the differences in periosteum in children and adults

A

in kids is much thicker and tends to remain intact which can help stability and can assist reduction if required.

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5
Q

what do children bones have a greater potential for?

A

remodelling

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6
Q

why do childrens bone have a greater remodelling potential

A

because they grow with bone being formed along the line of stress and children can correct angulation up to 10° per year of growth remaining in that bone.

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7
Q

are kids # treatment in comparison to adults

A

less likely to need surgery with greater degrees of displacement or angulation can be accepted.

If the fracture position is unaccepatable, manipulation and casting may be all that is required accepting a degree of residual angulation or displacement.

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8
Q

when are childs # treated like adults

A

puberty [12-14]

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9
Q

around where has the potential to disturb growth

A

physis (growth plate)

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10
Q

what happens if physis is disturbed by #

A

shortened limb or an angular deformity if one side of the physis is affected by growth arrest.

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11
Q

what classification is used for paediatric physeal #’s?

A

Salter-Harris classification of physeal fractures

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12
Q

what is salter-harris I #

A

pure physeal separation.

best prognosis and is least likely to result in growth arrest.

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13
Q

what is salter-harris 2 #

A

similar to 1 but has a small metaphyseal fragment attached to the physis and epiphysis.

growth disturbance risk is low.

commonest physeal #.

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14
Q

Salter‐Harris III and IV #’s

A

’s reduced and stabilized to ensure a congruent articular surface and minimize growth disturbance.

intra‐articular and with the fracture splitting the physis

greater potential for growth arrest

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15
Q

Salter‐Harris V injury

A

compression injury to the physis with subsequent growth arrest.

cannot be diagnosed on initial x‐rays; only detected once angular deformity has occurred.

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16
Q

Non accidental injury (NAI) risk factors

A

poverty,

children with special needs or disability.

parents who are substance abusers.

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17
Q

what raises concern of child abuse/NAI

A

Multiple fractures of varying ages (with varying amounts of callus or healing)

multiple trips to A&E with different injuries

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18
Q

other features that raise suspicion of NAI

A

Inconsistent / changing history of events

Discrepancy of history between parents / carers

History not consistent with injury

Injuries not consistent with age of child eg non walking child

Multiple bruises of varying ages

Atypical injuries eg cigarette burns, genital injuries, torn frenulum, dental injuries, lower limb
and trunk burns

Rib fractures

Metaphyseal fractures in infants

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19
Q

what should occur in NAI suspected case

A

paediatrics involved ASAP and admitted for saftey.

Full exam and history taking performed

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20
Q

examples of common paediatric fractures

A

distal radius, forearm, supracondylar of elbow, femoral shaft, tibial #’s

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21
Q

what are some examples of paediatric distal radius #’s

A

buckle, greenstick, salter-harris II

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22
Q

Buckle fractures (how stable? treatment?)

A

stable

require only 3‐4 weeks of splintage.

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23
Q

Greenstick fractures (describe variation + treatment)

A

may be angulated

may require manipulation + casting if significant deformity (particularly in the older child)

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24
Q

Salter‐Harris II fractures. (where, problems and treatment)

A

distal radial physis (occurs in older children)Angulation with deformity requires manipulation. Growth problems are highly unlikely (as with most Salter Harris II fractures).

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25
Q

Complete distal radius fractures

A

may displace as well as angulate with dorsal displacement and angulation more common than volar.

The dorsal periosteum usually remains intact which prevents overcorrection of the deformity and aids stability.

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26
Q

treatment of Complete distal radius fractures

A

If the fracture is fairly stable, casting may suffice.

If a complete fracture is very unstable after reduction, wire stabilization or plate fixation may be employed.

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27
Q

forearm #’s

A

Monteggia and Galeazzi fracture‐dislocations

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28
Q

treating forearm #’s in kids

A

These injuries go against the usual principles of children’s fractures, in that anatomic reduction and rigid fixation with plates and screws is typically used to treat these injuries. There is a high rate of re‐dislocation of the radial head or distal radio-ulnar joint (DRUJ) if only manipulation and casting is used.

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29
Q

fractures of both bones of the forearm (types and treatment)

A

angulated fractures → have an intact periosteum and the instability may only be in one plane → controlled with a cast after manipulation.

Displaced fractures → unstable and flexible → intramedullary nail

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30
Q

what are Supracondylar fractures of the elbow common

A

relatively weak point in the growing upper limb

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31
Q

two types of Supracondylar fractures

A

extension - more common and occur due to a heavy fall onto the outstretched hand

flexion - a fall onto the point of the flexed elbow.

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32
Q

treatment of supracondylar #’s

A

Undisplaced fractures → stable → splint.

Angulated, rotated or displaced # → closed reduction and pinning with wires → prevent deformity

also severely displaced / off‐ended fractures (see later card)

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33
Q

severely displace/off-ended supracondylar # problems

A

brachialis muscle may be tethered in the fracture site

With off‐ended extension type fractures the distal fragment displaces posteriorly with stretch and pressure on the brachial artery and median nerve (predominantly its anterior interosseous branch – the patient is unable to make the “OK” sign due to loss of FPL and FDP to the index).

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34
Q

supracondylar Displaced fractures treatment

A

reduced fairly soon to avoid swelling which can make reduction more difficult.

radial pulse is absent or reduced in volume then emergency surgery ASAP [Closed reduction may be performed with wiring and the pulse may return if the artery is no longer under stretch. if hand remains pulseless after reduction, open surgical exploration is required.]

nerve injury then surgery urgently. majority are neurapraxias and occasionally axonotmesis [normally improve]. neuralgic pain or no improvement may indicate entrapment of the nerve. do surgical release

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35
Q

why do Femoral shaft fractures occur?

A

children due to a fall onto a flexed knee or by indirect bending or rotational forces.

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36
Q

what can occur in femoral shaft fracture healing process in kids

A

overgrowth tends to occur after fracture healing and therefore some shortening can be accepted (more with younger children).

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37
Q

femoral shaft # In children less than 2 years old [cause and treatment]

A

more than half of femoral shaft fractures are due to NAI .

Gallows traction and early hip spica cast

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38
Q

femur shaft # in children aged between 2 and 6 treatment

A

Thomas splint or a hip spica cast.

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39
Q

femur shaft # in children between 6 and 12 treatment

A

femur is large enough to accommodate flexible intramedullary nails which obviate the need for traction or cast.

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40
Q

children aged 12 and above femur shaft # treatment

A

adult type intramedullary nail is typically used.

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41
Q

what to remember to check in femur fractures

A

femur is a common site for benign and malignant bone tumors and the fracture may be pathological with osteolysis and cortical thinning

(also if young then NAI)

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42
Q

undisplayed tibial #’s occur in what age group and are treated with what

A

Undisplaced spiral fractures of the tibial shaft are common in toddler’s (the injury is known as a “toddler’s fracture”)

require a short time in cast.

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43
Q

treating tibial #s

A

mainly cast for majority.

compartment syndrome risk less than adult.

up to 10 degrees angulation allowed or manipulated

serial x-rays to check doesn’t drift into excessive angulation. shortening or malrotation also unaccepted

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44
Q

very unstable or open tibial #s

A

stabilise = flexible intramedullary nails, plates and screws or external fixation.
Adolescents with a closed proximal tibial physis give adult intramedullary nail.

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45
Q

C-spine #

A

high energy injuries (RTA, fall from height) + associated with head injury.

Potentially dangerous unstable fractures may be missed in the unconscious or confused patient which may result in spinal cord injury. therefore C‐spine immobilization with a hard collar and sand bags or blocks on a spinal board

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46
Q

how to clinically clear C-spine injury

A

No history of loss of consciousness

GCS 15 with no alcohol intoxication

No significant distracting injury (such as head injury, chest trauma or other fractures including more distal spinal fractures)

No neurological symptoms in the upper or lower limbs

No midline tenderness on palpation of the c-spine

No pain on gentle active neck movement (ask the patient to gently flexed forward, then rotate to each side)

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47
Q

what to do if c-spine cannot be clinically cleared

A

collar must stay in situ.

Further imaging in the form of X‐Rays (AP & lateral views +/‐ odontoid peg open mouth view) or CT scan of the c‐spine is required so that a c-spine injury can be radologically cleared

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48
Q

what to do in any suspected c-spine injury

A

full trauma assessment (ABCD) and a full neurological examination including:

peripheral motor function
coarse touch sensation
upper & lower limb reflexes
cranial nerve evaluation
rectal examination, and
assessment of bulbocavernous reflex.
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49
Q

what height are c-spine dislocations/’s especially fatal

A

c3 or above (as phrenic nerve suppling diaphragm is C3,4,5.)

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50
Q

treatment of stable c-spine injuries

A

firm cervical collar

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51
Q

treatment of unstable injuries of c-spine

A

immbolise in “halo vest” (external fixation)

surigcal stabilisation with fusion/wiring/internal fixation

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52
Q

c-spine Subluxations and dislocations treament

A

traction for reduction and halo application or operative stabilization

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53
Q

burst fracture with neurological deficits treatment

A

traction to decompress spinal cord

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54
Q

why do thoracolumbar spine fractures occur

A
  • motor vehicle accidents or falls from a height. give burst fractures or chance fracture-dislocation
  • elderly with osteoporosis osteoporotic “wedge” insufficiency fractures ( do not require anything other than symptomatic treatment.)
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55
Q

what does any thoracolumbar injury require?

A

full trauma evaluation and neurological assessment

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56
Q

treatment of thoracolumbar injury

A

stable of throacic spine= brace to limit flexion/kyphosis

stable lumbar spine = plaster jacket for presrve lordosis.

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57
Q

examination of thoracolumbar spinal fractures

A

posterior bony or ligamentous involvement detected by local tenderness, swelling and palpable defect on exam

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58
Q

indication for surgery of thoracolumbar #

A

Presence of neurological deficit (especially if progressive or very unstable injury)

Unstable injury pattern with substantial loss of vertebral height, displacement or involvement of the posterior ligamentous structures.

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59
Q

what might surgery of thoracolumbar # involve

A

Surgery may involve stabilization with pedicle screws and rods, spinal fusion and decompression.

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60
Q

what can cause spinal cord injuries?

A

contusion, compression, stretch or laceration.

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61
Q

what does vascular disruption and oedema cause in spinal cord injuries?

A

further ischaemic damage and hypotension, hypoxia and inflammatory responses may also result in secondary damage.

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62
Q

what is Spinal shock

A

physiologic response to injury with complete loss of sensation and motor function and loss of reflexes below the level of the injury. Spinal shock usually resolves in 24 hours with return of reflexes and the severity of a spinal cord injury may not be determined until after spinal shock has resolved

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63
Q

what reflexes can be asessed in spinal shock?

A

The bulbocavernous reflex

contraction of the anal sphincter with either a squeeze of the glans penis, tapping the mons pubis or pulling on a urethral catheter. The bulbocavernous reflex is absent in spinal shock and its return signals the end of spinal shock.

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64
Q

what is Neurogenic shock

A

occurs secondary to temporary shutdown of sympathetic outflow from the cord from T1 to L2, usually due to injury in the cervical or upper thoracic cord leading to hypotension and bradycardia which usually resolves within 24‐48 hours.

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65
Q

what can occur in neurogenic shock

A

Priapism (prolonged erection), due to unopposed parasympathetic stimulation

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66
Q

treating neurogenic shock

A

Neurogenic shock is treated with IV fluid therapy.

Neurogenic shock must be differentiated from other forms of shock (e.g.: hypovolemic shock also responds to fluid replacement )

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67
Q

two classifications of spinal cord injury

A

complete or incomplete

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68
Q

Complete spinal cord injury

A
  • results in no sensory or voluntary motor function below the level of the injury (reflexes should return).
  • The level of the injury is determined by the most distal spinal level with partial function (after spinal shock has resolved) as determined by the presence of dermatomal sensation and myotomal skeletal muscle voluntary contraction.
  • The prognosis for recovery from complete cord injuries is poor.
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69
Q

incomplete spinal cord injuries,

A
  • some neurologic function (sensory and/or motor) is present distal to the level of injury.
  • In general, the greater the function present, the faster the recovery is and the better the prognosis.
  • Sacral sparing with preservation of perianal sensation, voluntary anal sphincter contraction and big toe flexion (FHL muscle, S1/2) indicates some continuity of the corticospinal (motor) and spinothalamic (course touch, pain, temperature) tracts. The presence of sacral sparing indicates an incomplete cord injury with a better prognosis than a complete injury.
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70
Q

treatment of spinal cord injury

A

Full ATLS primary survey with resuscitation and protection of the cervical and thoracolumbar spine is mandatory.
appropriate immobilisation, traction, surgery, ventilation support, multi-disiplanry approach

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71
Q

name the 4 types of incomplete spinal cord injuries

A

central, anterior, posterior cord syndromes and brown-swquard syndromes

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72
Q

Central cord syndrome signs

A

commoonest, hyperextension injury in C-spine with OA.

often no associated fracture or dislocation. paralysis or arms more than legs. (as central in cord is upper motor neurones mainly) sacral sparing typical .

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73
Q

anterior cord syndrome signs

A

loss of motor function, coarse touch, pain, temp sensation below injury height.

proprioception, vibration sense and light touch preserved

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74
Q

posterior cord syndrome signs

A

loss of dorsal column function is rare (vibration sense, proprioception, light touch)

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75
Q

Brown‐Sequard syndrome signs

A

results from hemisection of the cord usually from penetrating injury eg stab wound. Ipsilateral paralysis and loss of dorsal column sensation occurs with contralateral loss of pain, temperature and coarse touch sensation. This is due to nerve fibres of the spinothalamic tracts crossing to the other side of the cord one or two levels above their entry into the cord whilst the nerve fibres of the other tracts cross higher up in the medulla

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76
Q

pelvic #

A

young=high energy

old= low energy possible due to osteoporosis

if pelvic ring disputed then always > 1 place (polo mint)

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77
Q

what forms the pelvic ring?

A

sacrum, ilium, ischium and pubic bones with strong supporting ligaments.

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78
Q

pelvis # complications

A

Branches of the internal iliac arterial system and the pre‐sacral venous plexus are prone to injury with risk of serious hypovolaemia.

Nerve roots and branches of the lumbo‐sacral plexus are prone to injury.

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79
Q

what are the three main patterns of injury in pelvis?

A

lateral compression #, vertical shear #, anteroposterior compression injury

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80
Q

lateral compression fracture

A

occurs with a side impact (eg RTA) where one half of the pelvis (hemipelvis) is displaced medially. Fractures through the pubic rami or ischium are accompanied by a sacral compression fracture or SI joint disruption.

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81
Q

vertical shear fracture

A

occurs due to axial force on one hemipelvis (eg fall from height, rapid deceleration) where the affected hemipelvis is displaced superiorly. The sacral nerve roots and lumbosacral plexus are at high risk of injury and major haemorrhage may occur. The leg on the affected side will appear shorter.

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82
Q

anteroposterior compression injury

A

may result in wide disruption of the pubic symphysis the pelvis opening up like the pages of a book – the so‐called open book pelvic fracture. Substantial bleeding from torn vessels occurs and as the pelvic volume increases exponentially with the degree of displacement, with widely displaced injuries the pelvis can contain several litres of blood (ie the entire circulating volume) before tamponade and clotting will occur.

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83
Q

high-energy pelvic # treatment

A

associated with other injuries

blood loss treat

with open book pelvic # promptly reduce the displacement and minimise pelvic volume to allow tamponade of bleeding to occur. Application of a tied sheet or a special pelvic binder around the outside of the pelvis will hold the reduction temporarily and allow clotting of the vessels. An external fixator will provide more secure initial stabilization.

Ongoing haemodynamic instability despite these measures may require angiogram and embolization or open packing of the pelvis if laparotomy is required for co‐existing intra‐abdominal injuries.

Bladder and urethral injuries (blood at the urethral meatus) may also occur and urinary catheterization may risk further injury. Urological assessment and intervention may be required. A PR exam is mandatory to assess sacral nerve root function and to look for the presence of blood. The presence of blood indicates a rectal tear rendering the injury an open fracture and carries a higher risk of mortality. General surgical review is mandatory and defunctioning colostomy may be required.

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84
Q

treating low energy pelvic #’s

A

Low energy pubic rami fractures in the elderly tend to be minimally displaced lateral compression injuries (with sacral fracture or SI joint disruption posteriorly) and settle with conservative management over time.

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85
Q

acetabulum #’s

A

Acetabular fractures are usually high energy injuries in the younger patient but can be low energy in the older patient.

Posterior wall fractures may be associated with a hip dislocation. In these cases, the posterior wall is fractured as the head of the femur is pushed out the back of the joint. (RTA)

The pattern of the fracture can be difficult to determine on plain X‐rays (oblique views may help) and CT scans help to determine the pattern of the fracture and are essential for surgical planning.

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86
Q

treatment of acetabulum #’a

A

Undisplaced fractures or small wall fractures may be treated conservatively. As with most intra‐ articular fractures, unstable or displaced fractures require anatomic reduction and rigid fixation in the younger patient to reduce the risk of post traumatic OA. Older patients may be treated with total hip replacement – either early (with an uncemented cup and screws) or delayed.

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87
Q

examples of shoulder trauma

A

humeral neck #, shoulder (gleno-humeral) dislocations, ACJ (Acromioclavicular joint) injuries

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88
Q

humeral neck #’s common cause

A

Proximal humerus fractures are common with the majority being low energy injuries in osteoprotic bone due to a fall onto the outstretched hand or directly onto the shoulder.

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89
Q

humeral neck # pattern of #

A

The most common pattern is a fracture of the surgical neck (rather than the anatomic neck) with medial displacement of the humeral shaft due to pull of the pectoralis major muscle. The greater and lesser tuberosities may also be avulsed with the attachments of Supraspinatus, Infraspinatus and teres minor for the greater tuberosity and subscapularis for the lesser tuberosity. Isolated fractures of the greater tuberosity and head‐splitting intra‐articular fractures can also occur.

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90
Q

treatment of humeral neck #

A

Many minimally displaced proximal humerus fractures are treated conservatively with a sling and gradual return to mobilization

displaced= the position often improves once muscle spasm settles.

permanametly displaced = internal fixation (plate, screws, wires or intramedullary nail) but stiffness, chronic pain and failure of fixation can occur particularly in the older patient.

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91
Q

consequences of humeral head #

A

pain and/or loss of motion. AVN of humeral head causing chronic pain. bone fragments may need cut out or fixed as leads to failure, shoulder replacement can be used. (Head splitting fractures usually require shoulder replacement unless the patient is younger with very good bone quality.)

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92
Q

gleno-humeral dislocation which type is more common?

A

Anterior shoulder dislocation is much more common than posterior dislocation (the latter contributing only 2‐5% of all shoulder dislocations).

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93
Q

Traumatic anterior shoulder dislocation occurs due to what?

A

excessive external rotation force or a fall onto the back of the shoulder. also seizure (watch for bilateral dislocations)

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94
Q

what is the pathophysiology of a Anterior shoulder dislocation

A

often detachment of the anterior glenoid labrum and capsule known as a Bankart lesion whilst the posterior humeral head can impact on the anterior glenoid producing an impaction fracture of the posterior head (Hill‐Sachs lesion)

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95
Q

what can be compromised in Anterior shoulder dislocation

A

auxillary nerve can be stretched as it passed through quadrilateral space witle axially artery and other bracial plexus nerves can be affected.

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96
Q

finding on clinical assessment of anterior shoulder dislocation

A

Loss of symmetry is seen with loss of roundness of the shoulder and the arm held in an adducted position supported by the patients other arm. The principle sign of axillary nerve injury is loss of sensation in the regimental badge area. It may be difficult to determine deltoid contraction in the acute phase. Full distal neurovascular assessment should be carried out. In older patients, tears of the rotator cuff are very common but again these can be difficult to assess in the acute setting.

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97
Q

investigations of anterior shoulder dislocation

A

x-rays to confirm.

also surgical neck and greater tuberoity # can occur with shoulder dislocation

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98
Q

Management of ant shoulder dislocation

A

Closed reduction (under sedation or anaesthetic) is the mainstay of treatment with neurovascular assessment before and after reduction. Radiographs are repeated to confirm reduction. The patient is placed in a sling for 2‐3 weeks to allow the detached capsule to heal then rehabilitation with physiotherapy is commenced. Delayed presentation dislocations (eg alcoholics) may be difficult to reduce by closed means and may require open reduction..

associated fracture of the greater tuberosity, this usually reduces to an acceptable position with reduction of the shoulder however ORIF is usually required if it remains displaced. Fracture‐dislocations involving the surgical neck usually require surgery.

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99
Q

risk of shoulder redislocation

A

The risk of recurrent dislocation is predicted by the age of the patient at the time of initial dislocation.

Patients less than 20 have an 80% chance of re‐dislocation and many surgeons advocate stabilization surgery after first time dislocation in this age group. Patients over 30 have only a 20% risk of further dislocation and the re‐dislocation rate reduced further with increasing age. Recurrent dislocations can be stabilized by a Bankart repair with reattachment of the torn labrum and capsule by arthroscopic or open means.

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100
Q

Some shoulder dislocations occur in patients with marked ligamentous laxity (who and treatment)

A

EDS/marfan’s causes this. atraumatic multi-directional dislocation (which may be painful), open tightening gf shoulder capsule possible + physic to strengthen RC is treatment

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101
Q

causes of Posterior shoulder dislocations

A

posterior force on the adducted and internally rotated arm

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102
Q

investigating Posterior shoulder dislocations

A

The humeral head may be palpated posteriorly. They are often missed as the radiographic findings are much less obvious than anterior dislocation. The main Xray finding is the “light bulb” sign where the excessively internally rotated humeral head looks symmetrical like a light bulb on an AP view. Special lateral xray views assist in the diagnosis.

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103
Q

treatment of Posterior shoulder dislocations

A

Closed reduction and a period of immobilization followed by physiotherapy are again the mainstay of treatment.

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104
Q

why do ACJ injuries occur?

A

Injuries of the acromioclavicular joint usually occur after a fall onto the point of the shoulder. They are a fairly common sporting injury.

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105
Q

classification of ACJ injuries

A

sprained, subluxed (the acromioclavicular ligaments are ruptured) or dislocated. ( the coracoclavicular ligaments (conoid and trapezoid ligaments) are also disrupted )

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106
Q

treatment of ACJ injuries

A

Most injuries are treated with conservative management wearing a sling for a few weeks followed by physiotherapy.

surgery (reconstruction of the coracoclavicular ligaments) is reserved for those with chronic pain (although some surgeons advocate early reconstruction for younger athletes with dislocation – controversial).

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107
Q

what causes a humeral shaft #

A

direct rauama (RTA) results in transverse or comminuted #.

fall +/- twisting injury resulting in oblique or spiral #.

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108
Q

recovery from humeral shaft #

A

Union rates are high (90%) and due to the mobility of the ball and socket shoulder joint proximally and the elbow joint distally, up to 30° of angulation can be accepted.

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109
Q

complications of humeral shaft #

A

The radial nerve in the spiral groove is susceptible to injury which present with a wrist drop and loss of sensation in the first dorsal web space.

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110
Q

treatment of humeral shaft #

A

Most cases are treated non-operatively with a functional humeral brace which compresses the fragments into acceptable alignment and provides some stability.

Internal fixation with an intramedullary nail or plate and screws may afford a quicker recovery and intramedullary nailing may be used in polytrauma. Non unions require plating and bone grafting.

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111
Q

common elbow injuries

A

supracondylar # (paediartic )

intra-articular distal humerus #

olecranon #

radial head and neck #

elbow dislocation and fracture dislocation

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112
Q

Intra‐articular distal humerus fractures treatment

A

As with most intra‐articular fractures, these require open reduction, internal fixation (ORIF) with anatomic reduction and rigid fixation to minimize loss of function. Special plates are available to follow the complex contours of the distal humerus. Elbow replacement can be considered in highly comminuted fractures in the elderly.

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113
Q

Olecranon fracture

A

common, due to fall onto pint of elbow with contraction of tricep. mostly ORIF to restore tricep function and articular surface.

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114
Q

Olecranon fracture treatment

A

A simple transverse avulsion fracture can be fixed with tension band wiring which compresses the tension side of the fractures. Comminuted fractures don’t have a fulcrum for the tension band and require ORIF with a plate and screws.

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115
Q

Radial head & neck fractures due to what?

A

Radial head and neck fractures usually occur due to a fall onto the outstretched arm.

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116
Q

investigating radial head and neck #s

A

Some undisplaced fractures may not show up on x‐ray other than a fat pad sign on the lateral x‐ray (a triangle like a sail anterior to the distal humerus) with lateral elbow pain on supination / pronation.

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117
Q

treating Radial head & neck fractures

A

Undisplaced or minimally displaced fractures are treated conservatively with a sling for comfort followed by early elbow exercises to minimize stiffness. Patients often lose 10‐15° of terminal extension. Displaced intra‐articular radial head fractures may require surgery if the displaced fragment causes a mechanical block to full extension. Aspitation of the haemarthrosis and injection of local anaesthetic may help to exclude those with restricted ROM due to pain. ORIF is performed if the fragment is large enough or excision if not amenable to fixation.

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118
Q

elbow dislocation

A

Most elbow dislocations occur in the posterior direction after a fall onto the outstretched hand. They may be associated with neurovascular injury. Uncomplicated dislocations require closed reduction under sedation assessing neurovascular status pre‐ and post‐reduction. A short period in sling (1‐3 weeks) followed by elbow exercises is typically required.

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119
Q

In elbow fracture-dislocation, what are the common # that occur with elbow dislocation

A

Associated fractures of the radial head, humeral epicondyles or coronoid process of the ulna.

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120
Q

treating elbow fracture dislocation

A

Surgery may be required if entrapped bony fragments prevent reduction or block motion. Radial neck or head fractures may require ORIF or excision and replacement with a prosthetic radial head to maintain stability.

Epicondyle fractures are fixed with a screw. Large coronoid fractures may need ORIF with small screws to prevent recurrent dislocation. Some cases with recurrent instability may be helped with lateral elbow ligament reconstruction.

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121
Q

forearm # principles

A

forearm consists of radius nd ulna bone connected proximally and distally by strong ligaments around the respective radio-ulnar joints (where supination/pronation occurs).

because of strong ligaments forearm acts as a ring (so ifone bone #/dislocated then other is likely to be as well)

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122
Q

Fracture of ulnar shaft is know as?

A

nightstick #

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123
Q

what can cause an ulnar shaft #

A

direct blow (nightstick because used to b after hit by truncheon/nightstick)

check there is no associated Monteggia injury.

many

124
Q

treatment of ulnar shaft #/nightstick #

A

conservative management

ORIF if need earlier return of function/reduced risk of non-union

125
Q

treatment for # of both bones of forearm

A

diaphysial fracture of both bone in adults needs ORID with plates and screws as is highly unstable and needed to prevent dislocation and maximise function.

children can do cast if minimally angulated # as angulation corrects with age

substantially angulated # or displace # with intact periostem are only unstable in one direction so plaster (+/-MUA)

if fracture is very unstable after reduction then flexible intramedullary screws used

126
Q

MUA means?

A

manipluation under anathesisa

127
Q

what is a Monteggia Fracture dislocation

A

fracture of the ulna occurs with dislocation of the radial head at the elbow

128
Q

Monteggia Fracture dislocation investigation

A

forearms X-rays may not be easy to see

therefore do elbow X-rays too

129
Q

Monteggia Fracture dislocation treatment

A

ORIF of ulna fracture (even in kids). which should result in reduction of RCJ [manipulation alone risks re-dislocation due to unstable nature or injury]

130
Q

what is Galeazzi fracture dislocation

A

fracture of the radius with dislocation of the ulna at the distal radioulnar joint.

131
Q

Galeazzi fracture dislocation investigation

A

forearms X-rays may not be easy to see

therefore lateral X-ray of wrist is mandatory

132
Q

Galeazzi fracture dislocation treatment

A

ORIF of radius which should allow the DRUJ to reduced

133
Q

what are some common Distal Radial #s

A

colles#, Smith’s#, Barton’s#, comminuted IA distal #

134
Q

distal radial # common cause?

A

due to FOOSH (falling into an outstretched hand)

135
Q

what is a Colles #

A

extra‐articular fracture of the distal radius within an inch of the articular surface and with dorsal displacement or angulation.

(there is often associated # of ulnar styloid)

136
Q

why does Colles # occur

A

FOOSH with wrist extended.

137
Q

treatment of Colles #

A

minimal angulation/shortening = splintage alone

any angulation past neutral = manipulation.

may be held with plaster cast alone or if has dorsal comminution or is felt unstable after reduction then percutaneous wires or ORIF with plate + screws.

138
Q

what accompanies a Colles #

A

assocaited # of ulnar styloid.

median nerve compression from nerve stretch or a bleed into carpal tunnel.

[Reduction may relive the pressure on the nerve and the fracture is usually stabilized with fixation. The carpal tunnel may need surgically decompressed.]

139
Q

what is a specific late local complication of a Colles #

A

Extensor Polligus Longus tendon rupture (require tendon transfer)

140
Q

what is Smith’s #

A

volarly displaced or angulated extra‐articular fracture of the distal radius which usually occurs after falling onto the back of a flexed wrist.

141
Q

treatment for Smith’s #

A

ORIF using plate and screws as are highly unstable.

142
Q

examining Smith’s #

A

grip strength and wrist extension are greatly reduced if there is a malunion with excessive volar angulation.

143
Q

what is Barton’s #

A

intra‐articular fractures of the distal radius involving the dorsal or volar rim, where the carpal bones of the wrist joint sublux with the displaced rim fragment.

144
Q

how can Barton’s # be classified

A

They can be classified as volar Barton’s fractures (an intra-articular Smith’s fracture) or a dorsal Barton’s fracture (an intra-articular Colles’ fracture)

145
Q

Barton’s # treatment

A

require ORIF (as with most IA injuries)

146
Q

why does a Comminuted intra‐articular distal radius fracture occur?

A

when the distal radius fractures are so comminuted (due to high energy or poor bone quality) that stable fixation of the joint fragments is not possible.

147
Q

what is treatment for Comminuted intra‐articular distal radius fracture?

A

external fixture across wrist joint (restore the shortening and hold the wrist in reasonable alignment and therefore limit functional deficit). supplementary wires can be used to pin larger fragments

148
Q

scaphoid # PC

A

after FOOSH.

tenderness in anatomical snuff box and pain on compressing thumb metatarsal

149
Q

investigating scaphoid #

A

AP, lateral and 2 oblique views taken due to kindness shaped bone makes it difficult to detect #. Some aren’t visible initially but can bee seen 2 weeks later after reabsorption of fracture ends.

150
Q

what to do if scaphoid # is suspected but X-rays fail to show #

A

wrist is splinted and further assessment in 2 weeks time.

called “clinical scaphoid #”

151
Q

treatment for scaphoid #

A

Undisplaced # are usually treated with plaster cast for 6‐12 weeks.

Displaced # = compression screw to prevent non-union.

152
Q

complications of scaphoid #

A

non-union problems due to SF inhibitng # healing. CT helpful to see if union has occurred.

AVN of proximal pole.

153
Q

how are non-union scaphoid # treated

A

CT scan shows non-union

screw fixation and bone grafting.

154
Q

how is AVN of scaphoid # treated

A

difficult, once has established usually symptomatic partial or total wrist fusion

155
Q

what is a peri-lunate dislocation

A

dislocation of one of the carpal bones around the lunate (another carpal bone)

uncommon, classic “missed diagnosis” with up to 25% of cases being missed on initial presentation.

156
Q

peri-lunate dislocation PC

A

severe high energy wrist injury resulting in hyperdorsiflexion

157
Q

investigation of peri-lunate dislocation

A

x-ray: not obvious. demonstrate lose of alignment of the capitate and lunate with the concave lunate fossa being empty

exam: median nerve injury/acute carpal tunnel syndrome may be present

158
Q

treatment of peri-lunate dislocation

A

emergency - closed reduction + percutaneous pinning/open reduction. carpal tunnel decompression may be required.

159
Q

what is a lunate dislocation

A

high energy injury where the lunate dislocates (usually volarly) whilst the remainder of the carpal bones remain enlocated.

160
Q

investigating lunate dislocation

A

X-raysplit cup sign.

exam: acute carpal tunnel syndrome common.

161
Q

treatment of lunate dislocation

A

emergency closed/open reduction and pinning

162
Q

why does a Scapho‐lunate dissociation occur?

A

occurs when the scapho‐lunate ligaments ruptures.

163
Q

what is seen on Xray in Scapho‐lunate dissociation

A

increased gap between the scaphoid and lunate on AP X-ray.

164
Q

what happens if Scapho‐lunate dissociation is left untreated

A

abnormal forces on wrist and carpus causing early OA

165
Q

treatment of Scapho‐lunate dissociation

A

surgical with closed reduciotn and K-wiring +/-scapholunate ligament repair.

166
Q

how are chronic cases of Scapho‐lunate dissociation without OA treated?

A

soft tissue tethering to the distal pole of the scaphoid to prevent hyperflexion (dorsal capsulodesis)

167
Q

how are chronic cases of Scapho‐lunate dissociation with OA treated?

A

partial or total wrist fusion (to improve symptoms)

168
Q

name some common hand injuries

A

penetrating injury, extensor tendon injuries, mallet finger, flexor tendon injuries, metacarpal #, phalangeal #

169
Q

what do volar injuries risk damage (palm side)

A

flexor tendons, digital nerves, digital arteries

170
Q

what do dorsal injuries risk damage (back of hand side)

A

extensor tendons

171
Q

investigating hand penetrating injury

A

assessment of neurovascular and tendon function.

(be prepared for surgery as tendon can seem functionally normal but have little continuity/attachment meaning rupture is common)

172
Q

treatment for Complete or significant partial tendon injuries

A

surgery

173
Q

surgery for Complete or significant partial tendon injuries involves

A

if both digital arteries injured then microsurgical repair

if pulsated bleeding/arteries injury to digit then higher chance of congruent injury to adjacent digital nerve

Digital nerve injuries proximal to the DIPJ warrant repair with sensation to the thumb, index and little fingers particularly important.

174
Q

Extensor tendon injuries treatment

A

Extensor tendon divisions of 50% or more usually require surgical repair with splintage in extension for 6 weeks as any flexion within this period may cause failure of the repair.

175
Q

what is mallet finger

A

Mallet finger is an avulsion of the extensor tendon from its insertion into the terminal phalanx and is caused by forced flexion of the extended DIPJ, often from a ball at sport.

176
Q

PC of mallet finger

A

due to sport usually, The patient presents with pain, a drooped DIPJ of the affected finger and inability to extend at the DIPJ. The injury may be a purely tendinous avulsion or may have a bony fragment.

177
Q

treatment of mallet finger

A

mallet splint holding the DIPJ extended which should be worn continuously for a minimum of 4 weeks.

178
Q

what problems do flexor tendon injuries cause?

A

tendons cannot run smoothly within tendon sheath and under the pulley.

digital nerves and arteries that run adjacent to the tendons can be injured too

injuries to FDS, FDP or both may occur

concomitant injury to the interdigital nerves or arterial system.

179
Q

how to tell difference between FDS and FDP?

A

FDP has more distal insertion than FDS

180
Q

what are FDS and FDP

A

FDS = flexor digitorium superficalis

FDP = deep (extends to distal)

181
Q

what is flexor in thumb?

A

FPB - flexor pollicus brevis

182
Q

how to treat palmar injuries

A

all need explored

smooth over partial divisions or repair complete divisions. (special suture technique)

183
Q

why does the tendon sheath requires careful repair with preservation of the pulleys?

A

to avoid bow-stringing of tendon

184
Q

what about rehab post-surgery of flexor tendon injury

A

splinted in flexed position.

often with elastic traction to allow early active gentle extension and passive flexion

(to prevent stiffness and adhesion in tendon sheath)

185
Q

Penetrating injuries in the volar forearm risk injury to what?

A

wrist flexors (FCU and FCR)

long flexors to the fingers and thumb

injuries to median and ulnar nerves

injuries to ulnar and radial arteries

186
Q

how are # of 3, 4, 5th metacarpals treated and why?

A

conservatively (usually)

The 3rd + 4th metacarpals have strong intermetacarpal ligaments proximally and distally. gives stability to # and minimal displacement.

187
Q

why do Fractures of the 5th metacarpal often occur?

A

punching injury

Esp 5th matacapral neck # (aka: boxer’s #).

188
Q

what is tolerate for boxer’s #

A

up to 45 degrees angulation - as don’t affect hand function

189
Q

treatement for boxer’s #

A

neighbour strapping

early motion to maintain function.

check rotational alignment/grip problems/overlapping fingers when making a fist

190
Q

how would you fix rotational alignment/grip problems/overlapping fingers when making a fist

A

mainulaltion + neighbour stapping/k-wire stabilisation

191
Q

what is a “fight bite”?

A

laceration sustained to the puncher’s hand from the punchee’s tooth!

192
Q

what complications can “fight bite “ cause?

A

MCPj disruption

extensor tendon rupture

intra-oral organsis cause aggressive infection cause septic arthritis.

193
Q

treatment of fight bite

A

explored and thoroughly washed out in theatre and certainly not sutured closed in A&E.(due to infection risk)

194
Q

how to treat phalangeal #’s

A

neighbour strapping/spintage.

if significantly displaced → MUA.

unstable # or IA #→ Kwiring or fixation with small screens.

195
Q

what two things are hip # related to?

A

falls + osteoporosis

196
Q

outcomes and mortality from hip #

A

variable some fine, some need walking aids, some wheelchair

Mortality from hip fracture is around 10% at one month, 20% at four months and 30% at one year.

because of significant co-morbidities (MI, ARF, RespF, Chest infections, surgical complication, CVS failure, reduced physiological reserves….)

197
Q

what increase a patient risk of falling?

A

age, co-morbiditiies (cerebrovascular insufficiency, cardiac arrhythmias, postural hypotension etc.)

198
Q

treatment for hip #

A

surgery despite risks. (as non-op just as high)

199
Q

non-op management of hip #

A

prolonged bed rest for several weeks → very sore for toileting and bathing → problems with recumbency (pressure sores, chest infections) + # may not heal. also muscular atrophy making rehab difficult

200
Q

post hip # surgery what is done in 24hrs?

A

mobilisation (reduces surgical risks and physio time)

201
Q

when would surgery not be indicated for hip #

A

ery high risk patients who are expected to die very soon after the injury.

202
Q

what are the two types of hip #?

A

intra and extra capsular

can also be described as sub capital, intertrochanteric and subtrochanteric [intra, extra, extra]

203
Q

why is it relevant if # is intra/extracapsular?

A

likelihood of blood supply being interrupted in greater in intra (femoral head AVN and poor healing/non-union)

204
Q

what is the arterial supply of the femoral head?

A

come from a ring anastomoses of the circumflex femoral artier (at insertion of hip papule at base of femoral neck).

medial and lateral circumflex arteries travel up femoral neck and into the femoral head.

205
Q

medial and lateral circumflex arteries are branches of which artery?

A

profunda femoral artery

206
Q

intracapsular # problems

A

femoral head AVN and poor healing/non-union

207
Q

treatment of intracapsular # of hip

A

replacement femoral head which can either be a hemi‐arthroplasty or THR

208
Q

what is the difference between THR + hemi‐arthroplasty?

A

hemi‐arthroplasty - replacing the femoral head alone.

THR - replacing the acetabulum as well as the femoral head

209
Q

why choose THR? why choose hemi‐arthroplasty?

A

THR - better function (given to higher functioning patients)

hemi‐arthroplasty - give to resirtited mobility and cognitively impaired patient. (as THR dislocation risk is high in cognitively impaired)

210
Q

why is internal fixation used for extra-capsular hip #?

A

bc want to keep natural joint as AVN and non-union rate low bc have good blood supply both sides

211
Q

treatment for extra-capsular hip #?

A

internal fixation EG; compression or dynamic hip screw (screw slides promoting # healing)

212
Q

why do Subtrochanteric # occur?

A

elderly patient with osteoporosis with fall onto their side

213
Q

healing of subtrochanteric #

A
  • relatively poor blood supply
  • area of bone under considerable bending stress.

-therefore inc time healing and non‐union occurs fairly frequently.

214
Q

treating Subtrochanteric #

A

Strong indirect fixation (without further disruption to the blood supply) = intramedullary nail

Thomas split may help relieve pre-op pain and stabilise #

215
Q

why do femoral shaft # occur? (give 5 reasons)

A

high energy injuries (high risk of concomitant# elsewhere)

[also osteoporotic bone, metatstatic disease, patients with Paget’s disease and paradoxically with long term bisphosphonate use for osteoporosis.]

216
Q

what is main problem with displaced femoral shaft fractures?

A
  • substantial blood loss of up to 1.5L can occur.

- Fat from the medullary canal → enter venous system → fat embolism → confusion, hypoxia and risk of ARDS.

217
Q

management of femoral shaft #

A

initially = initial resuscitation (optimizing analgesia [morphine IV/femoral nerve block] + application of a Thomas splint)

Definitive = usually closed reduction and stabilization with an intramedullary nail
(minimally invasive plate fixation with minimal disruption to the fracture site blood supply possible)

218
Q

why is thomas splint used in immediate management of femoral shaft #?

A

stabilises the fracture minimizing further blood loss and fat embolism.)

219
Q

knee range of movement

A

mainly flexion and extension

small degree of rotation

220
Q

what is the knee the articulation of?

A

distal end of the femur and proximal tibia

221
Q

what force of the MCL resist

A

valgus force

222
Q

what force of the LCL resist

A

varus force

223
Q

what does the ACL + PCL do?

A

stabilise the tibia in the sagittal and rotational planes for stability.

224
Q

what do distal femoral # occur?

A

osteoporotic bone with a fall onto the flexed knee.

can be extra (supracondylar) or intra articular (intercondylar)

225
Q

treatment of distal femoral #

A

-surgery with plate and screws (as the fracture position is difficult to maintain in a cast.)

226
Q

when do true knee dislocations occur?

A

High energy injuries

severe hyperextension and/or rotational forces with a sporting injury.

227
Q

what should you think when you see a knee dislocation

A

surgical emergency

228
Q

why is knee dislocation a surgical emergency?

A

high incidence of vascular injury (intimal tears, vascular occlusion, complete transection), nerve injury and compartment syndrome

229
Q

treatment of knee dislocation

A

reduced urgently → neurovascular assessment

230
Q

what to do if knee dislocation causes abnormal neurovascular assessment

A

vascular surgery referral if any doubt with further investigation (Doppler, duplex scan or angiogram) and revascularization (endovascular procedures or bypass) as required.

231
Q

what can be done in very unstable knee (post-dislocation)

A

external fixator

232
Q

what must occur for knee to dislocate?

A

Multi‐ligament tears/rupture → needs reconstruction

233
Q

why can true knee dislocations be difficult to tell apart from part from partial and how do you do it?

A

can spontaneuosly reduce back

check neurovasuclar status carefully

234
Q

why do patellar dislocation occur?

A
  • direct blow (clash of knees at sport)

- contraction of quads with rotation force with patella not engaged in trochlea

235
Q

patellar dislocation treatment (initially)

A

Manipulation for reduction but more commonly reduce when knee is straightened

236
Q

facts about patellar dislocations (epidimeilology and risk factors)

A

common, mainly occur in adolecscent females.

lax ligaments, valgus alignment of the knee,
rotational malalignment (including femoral neck anteversion), and
a shallow trochlear groove. = inc risk

Subluxations can also occur without frank dislocation.

237
Q

Examination of patellar dislocation shows what?

A
  • tenderness over the medial retinaculum (where the medial patellofemoral ligament is torn)
  • haemarthrosis [from impaction of the medial patellar facet on the outer aspect of the lateral femoral condyle]
  • osteochondral # can occur (fix or remove fragments)
238
Q

how many people who have first time patellar dislocation will have another one and how many will have multiple?

A

10% have another one

50% of those will be recurrent

239
Q

treatment of patellar dislocation

A

reduction

temporary splinter then physio

occasionally surgical stabilisation (with either bony procedure for malalignment or soft tissue reconstruction is required)

240
Q

what does the physio try to strengthen post-patellar dislocation?

A

vastus medialis - prevents further dislocations (many adolescent patient stabilise with age).

241
Q

why do Proximal tibia (plateau) #s occur?

A

high energy in young

osteoporotic bone in old

242
Q

Proximal tibia (plateau) # characterisitics

A

IA # with either split in bone or depression of the articular surface or both.

243
Q

how are Proximal tibia (plateau) #s classified

A

Schatzker system (beyond undergrad level)

244
Q

what may Proximal tibia (plateau) # have associated in high energy injuries?

A

neurovascular injury or compartment syndrome.

245
Q

treatment of Proximal tibia (plateau) #

A

(as with most IA #s) surgery to reduce articular surface and riding fixation with early motion (combat stiffness and post-trauma OA).

246
Q

what investigations are useful in Proximal tibia (plateau) # surgical planning

A

CT

247
Q

what can valgus stress to the knee cause?

A

lateral plateau fracture with failure of the MCL and possibly ACL with increasing force

248
Q

what can a direct blow (car bumper) to the knee cause?

A

proximal fibular fracture and injury to the common peroneal nerve with foot-drop (due to loss of power to tibialis anterior)

249
Q

what can a varus injury to the knee cause?

A

medial plateau fracture (less common) with potential for LCL rupture and stretch injury to the common peroneal nerve.

250
Q

what is the treatment for lateral plateau, medical plateau and proximal fibular #?

A

Plates and screws for fixation.

once depressed # has ben elevated then void in bone filled by bone graft (morsellised packed cancellous autograft from iliac crest) to provide support.

251
Q

what often occurs with high energy #s and what is done about it?

A

substantial soft tissue swelling

temporary external fixator spanning the joint → initial stability → allows the swelling to resolve → do ORIF (some surgeons use external fixture as definitive management not ORIF)

252
Q

what is prognosis of surgery

A

Despite efforts to restore the articular surface → results often disappointing → need TKR

253
Q

why do tibial #s occur?

A

indirect force and either (transverse #) bending or rotational energy (spiral #), compressive force from decelleration (oblique #), or a combination (comminuted #)

254
Q

what is common to occur in tibial shaft #?

A

open # (as shaft is subcutaneous) and compartment syndrome risk very high (particularly anterior compartment of the leg)

255
Q

treatment of tibial shaft # non-op/conservative

A

non-op: up to 50% displacement and 5 degrees angulation accepted. above knee cast

internal rotation of the distal fragment is poorly tolerated.

position difficult to control in a cast → frequent cast changes and check X-rays required:

fibula not # → tibia often drifts into varus

fibula is also fractured → valgus alignment common.

256
Q

treatment of tibial shaft # operative

A

internal fixation controls position and removes need for cast (early mobility and # quicker rehab)

257
Q

how long does tibial shaft # take to heal?

A

average time to union of around 16 weeks and can take up to a year to heal.

258
Q

what to do in comminuted tibial shaft #?

A

v unstable needs surgery to stabilise. (open # need plastic surgeon).

compartment syndrome needs fasciotomy.

ORIF with plate/screws give rigid stability. Intramedullary nailing is the commonest method of surgical stabilization - can give pain kneeling

external fixation used sometimes but infection and loosening = problems

259
Q

IA distal tibial # are known as what?

A

pilon #s

260
Q

how is extra-articular # of distal tibia is it treated?

A

if in acceptable position either op/non-op with early motion. (intramedullary nail or distal locking screw).

261
Q

how is an (IA) pilon # treated

A

emergency - temporary external fixation (as soft tissue swelling) then definitive ORIF to ensure congruent articular surface

(impacted articular fracture fragment need to be removed and any voids filled by bone graft).

(ankle arthrodesis may be required for post-trauma OA)

262
Q

why do pilon # occur?

A

high energy caused by fall from height or rapid deceleration.

263
Q

what is the pathology of a pilon #?

A

talus driven into the distal tibial article surface → substantial disruption, comminution or impaction of articular cartilage.

soft tissue swelling/ bruising/blistering.

other injuries common

264
Q

how is pilon # investigated?

A

CT scan.

265
Q

ankle sprain due to what?

A

very common, with inversion or rotational injury force on planted foot

266
Q

what are soft tissue sprain in ankle PC

A

sprains of lateral ankle ligaments are commonplace:

pain, bruising and mild/moderate tenderness in involved ligaments. (higher force causes #s)

267
Q

what criteria is used for ankle injuries to determine if need Xray? what merits Xray?

A

Ottowa.

Any severe localized tenderness (known as bony tenderness) of the distal tibia or fibula or inability to weight bear for four steps merits an xray

268
Q

what is difference between stable and unstable ankle # and why is this important?

A

Isolated distal fibular fractures with no medial fracture or rupture of the deltoid ligament are stable and are common and stable.

Distal fibular fractures with rupture of the deltoid ligament (suspected by bruising and tenderness medially) are unstable.

important as treatment differs

269
Q

what is treatment for stable ankle #?

A

a walking cast or splint for around 6 weeks.

270
Q

what is treatment for unstable ankle #?

A

ORIF (with plate/screws).

271
Q

what must have occurred if there is talar shift/talar tilt?

A

definition the deltoid ligament must be ruptured if there is no medial malleolar fracture

272
Q

what is difference between talar shift + talar tilt?

A

talar shift - asymmetric increased space around the talus within the ankle mortise (on AP X-ray view)

talar tilt - talus and tibial plafond being non parallel

273
Q

why is talar shift important to spot?

A

Ankle joint contact pressures greatly increase with even 1mm of talar shift with subsequent risk of post traumatic OA

274
Q

treating talar shift

A

anatomic reduction and rigid internal fixation is required to minimize this risk with any talar shift.

275
Q

what is treatment for bimalleolar #s?(# both medial and lateral malleoli)

A

unstable so undergo ORIF

276
Q

what are ankle # associated with (soft tissue wise). why is this relevent?

A

substantial soft tissue swelling and fracture blisters

ORIF may be delayed by 1‐2 weeks to allow the soft tissues to settle and reduced the risk of wound healing problems and infection.

277
Q

when to check for Talar shift? (do AP X-ray for it)

A

if ottawa criteria suggests Xray (bony tenderness or inability to bear weight)

278
Q

how is the foot anatomically arrangement

A

forefoot - matatarsal + phlanx

midfoot - the rest (navicular, cuboid, lateral, median and middle cuneiforms)

rearfoot - talus and calcaneus

279
Q

calcaneal # MOI

A

due to fall from height onto the heel

280
Q

calcaneal # pathology

A

can bc EA or involve subtalar joint (IA).

substantial soft tissue swelling + compartment syndrome

heel tends to drift into valgus with a widened heel causing impingement of the lateral ankle tendons

281
Q

what is calcaneal # prognosis dependant on?

A

extent of involvement of the subtalar joint and the degree of comminution.

282
Q

treatment of calcaneal #

A

controversial. ORIF to restore artilar surface in young.

however, risk of wound healing and breakdown high → difficult to achieve skin coverage without footwear problems. Smoking, vascular disease, inc age = risk factors for healing problems/infection. Also no evidence ORIF improves pain/function long term.

283
Q

what is a complication of cancaneal # involving subtler joint and how is treated?

A

post-trauam OA →chronic pain → subtalar arthrodesis

284
Q

why do talar # occur?

A

of talar neck occur with forced dorsiflexion of foot from rapid deceleration (RTA). high energy

285
Q

talar # types

A

undisplaced or displaced with subluxation of of subtalar joint.

286
Q

why is talus at risk of AVN?

A

blood supply from distally with anastomoses around and vessels traversing the talar neck.

287
Q

treatment of talar #s?

A

displaced = closed or open reduction and screw fixation

AVN of the talus is not always symptomatic but secondary symptomatic OA may require ankle fusion.

288
Q

midfoot #/dislocation know as

A

lisfranc injury

289
Q

what is a lisfranc injury?

A

base # of the 2nd metatarsal in associated with dislocation of the base of the 2nd metatarsal +/- dislocation of the other metatarsals at the tarso‐metatarsal joints.

290
Q

investigating Lisfranc injury

A

X-ray (can look normal as # can be small flake # and easy missed) → CT if doubt

291
Q

PC of lisfranc #

A

swollen, bruised foot, can’t bear weight.

292
Q

what happens if lisfranc injury is untreated?

A

high risk of pain + disability

293
Q

treatment for lisfranc/midfoot #

A

closed/open reduction with fixation using screws

294
Q

metatarsal injuries: how likely are 5th metatarsal # and why do they occur?

A

of base of 5th metatarsal are common.

inversion injury with an avulsion fracture at the insertion of the peroneus brevis tendon

295
Q

treatment for 5th metatarsal #

A

These heal predictably and require a walking cast, supportive bandage or wearing of a stout boot for 4‐6 weeks. Even with those which fail to achieve bony union, many have a stable fibrous non‐union which is usually asymptomatic.

296
Q

what is a jones #?

A

where the bone fractures in the region of the proximal diaphysis

297
Q

why is Jones # important?

A

area has poorer blood supply and risk of non-union higher (25%) even in undisplaced #s

298
Q

treatment for jones #?

A

Fixation if displaced/inactive patient.(usually with single screw).

non-union = bone graft and fixation

299
Q

how common is 1st metatarsal # and what is treatment

A

uncommon (due to thickness and strength). normally fixed as important

300
Q

how common are the lesser metatarsal #s and what is treatment

A

commonly fractured, often with multiple fractures.

minimal displacement= conservatively with a cast.

Multiple displaced fractures = K‐wires to reduce the risk of chronic pain.

301
Q

what is a common metatarsal site for a stress #?

A

2nd metatarsal.

occur spontaneously/after period of inc activity.

302
Q

investigating 2nd metatarsal #

A

x-ray may not show until callus formation has started.

Bone scan may aid diagnosis

303
Q

treatment for 2nd metatarsal #

A

cast until pain stops

304
Q

toe # treatment

A

rarely require anything other than protection in a stout boot.

305
Q

treatment of IA # at base of proximal phalanx of hallus

A

reduction and fixation if the fragment(s) are sizeable

306
Q

what is treatment of open toes #?

A

Open fractures require debridement +/- wire stabilising

307
Q

how are toe dislocations treated?

A

closed reduction + neighbour stepping or wiring.